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Journal of Biological Chemistry, ISSN 0021-9258, 07/2011, Volume 286, Issue 28, pp. 24638 - 24648
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2015, Volume 290, Issue 22, pp. 13875 - 13887
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, p. e81733
Heart failure is a major clinical problem worldwide. Previous studies have demonstrated an important role or G protein coupled receptors, including... 
ISCHEMIA-REPERFUSION INJURY | HYPERTROPHY | ISCHEMIA/REPERFUSION INJURY | PAR-1 | MULTIDISCIPLINARY SCIENCES | INNATE IMMUNE-RESPONSE | FACTOR DEFICIENCY | CARDIAC FIBROSIS | TISSUE FACTOR EXPRESSION | MAST-CELL TRYPTASE | FIBROBLASTS | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Receptor, PAR-2 - metabolism | Heart Failure - physiopathology | Male | Receptor, PAR-2 - deficiency | Hypertrophy - metabolism | Protease Inhibitors - pharmacology | Myocardial Infarction - pathology | Myocardial Infarction - physiopathology | Myocardial Infarction - etiology | Receptor, PAR-2 - genetics | Animals, Newborn | Heart - physiopathology | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Mice, Transgenic | Heart Failure - metabolism | Heart Failure - pathology | Myocardial Infarction - metabolism | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Fibrosis | Heart - drug effects | Myocytes, Cardiac - metabolism | Coronary Vessels - surgery | Mice | Ligation - adverse effects | Ventricular Myosins - genetics | Heart failure | G proteins | Heart enlargement | Proteases | Heart attack | Myocardial infarction | Neonates | Heart attacks | G protein-coupled receptors | Cardiomyopathy | Pathogenesis | Proteinase | Cardiovascular disease | Activation | Heart function | Remodeling | Kinases | Eutrophication | Proteins | Receptors | Reperfusion | Ischemia | Protease | Rodents | Fibroblasts | Extracellular matrix | Heart diseases | Cytokines | Coronary artery | Cardiomyocytes | Inflammation | Gene expression | Medicine | Signaling | Coronary vessels | Growth hormones | Ligands | Infarction | Alzheimers disease | Monocyte chemoattractant protein 1 | Hypertrophy | Failure
Journal Article
Journal Article
International journal of molecular sciences, ISSN 1422-0067, 11/2018, Volume 19, Issue 11, p. 3530
Cathepsin S (CTSS) activity is increased in tears of Sjögren's syndrome (SS) patients. This elevated CTSS may contribute to ocular surface inflammation. Human... 
ocular surface inflammation | human corneal epithelial cells | matrix metalloproteinase 9 | pro-inflammatory cytokines | proteases | inflammatory dry eye | Sjögren’s syndrome | Cathepsin S | protease-activated receptor-2
Journal Article
Role of protease-activated receptor-2 in inflammation, and its possible implications as a putative mediator of periodontitis, 12/2005
Proteinase-activated receptor-2 (PAR2) belongs to a novel subfamily of G-protein-coupled receptors with seven-transmembrane domains. This receptor is widely... 
protease-activated receptor-2 - alveolar bone loss - inflammation - host response - Porphyromonas gingivalis - periodontitis
Journal
JOURNAL OF BIOLOGICAL CHEMISTRY, ISSN 0021-9258, 06/2019, Volume 294, Issue 26, pp. 10266 - 10277
The collagenase subfamily of matrix metalloproteinases (MMPs) have important roles in the remodeling of collagenous matrices. The proteinase-activated receptor... 
MATRIPTASE | HUMAN FIBROBLAST | BIOCHEMISTRY & MOLECULAR BIOLOGY | RELEASE | CARTILAGE | collagenase | chondrocyte | arthritis | inflammation | ONCOSTATIN-M | matrix metalloproteinase (MMP) | cell signaling | proteinase-activated receptor-2 (PAR2) | TETHERED LIGAND | DEGRADATION | EXPRESSION | proteolysis | extracellular matrix | PROTEASE
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 08/2018, Volume 832, pp. 25 - 32
We synthesized and investigated the effects of a novel peptide B, RRFSLLRY as a novel PAR-2 antagonist. Peptide B decreased calcium levels in HaCat cells... 
Protease activated receptor-2 | Peptide | Inflammation | PAR-2 | CELLS | HUMAN SKIN | TRYPSIN | KERATINOCYTES | BREAST-CANCER | ATOPIC-DERMATITIS | IN-VIVO | PHARMACOLOGY & PHARMACY | MICE | EXPRESSION | Allergy | Keratin | Peptides | Interleukins | Proteases | Skin | Testing
Journal Article
Cellular and Molecular Life Sciences, ISSN 1420-682X, 7/2014, Volume 71, Issue 13, pp. 2517 - 2533
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 06/2011, Volume 183, Issue 12, pp. 1703 - 1714
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2013, Volume 62, Issue 19, pp. 1737 - 1745
Journal Article