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International Journal of Molecular Sciences, ISSN 1661-6596, 09/2014, Volume 15, Issue 11, pp. 20486 - 20499
Journal Article
BBA - Molecular and Cell Biology of Lipids, ISSN 1388-1981, 10/2019, Volume 1864, Issue 10, pp. 1458 - 1471
Monocyte chemoattractant protein-1-induced protein-1 (MCPIP1) acts as an endonuclease that degrades selected mRNAs, viral RNAs and pre-miRNAs. MCPIP1 inhibits... 
Peroxisome proliferator-activated receptor gamma | Obesity | TXNIP | MCPIP1 | Fatty liver | Physiological aspects | Liver diseases | Ribonuclease | RNA | Liver | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2012, Volume 109, Issue 8, pp. 3143 - 3148
The endocrine hormone fibroblast growth factor 21 (FGF21) is a powerful modulator of glucose and lipid metabolism and a promising drug for type 2 diabetes.... 
Bone resorption | Bone formation | Bone marrow | Bones | Osteoclasts | Fibroblast growth factors | Gene expression regulation | Adipocytes | Type 2 diabetes mellitus | Osteoblasts | Osteoblast | Adipocyte | Nuclear receptor | Bone metabolism | Thiazolidinediones | thiazolidinediones | FIBROBLAST-GROWTH-FACTOR-21 | MULTIDISCIPLINARY SCIENCES | ALPHA | osteoblast | ADIPOGENESIS | INSULIN SENSITIVITY | OSTEOCLAST | bone metabolism | METABOLISM | adipocyte | nuclear receptor | PPAR-GAMMA | MICE | ROSIGLITAZONE | FGF21 | Bone and Bones - pathology | RANK Ligand - metabolism | Adipogenesis - drug effects | Humans | PPAR gamma - metabolism | Bone and Bones - drug effects | Fibroblast Growth Factors - metabolism | Fibroblast Growth Factors - administration & dosage | Bone and Bones - metabolism | Bone Resorption - metabolism | Thiazolidinediones - pharmacology | Bone Marrow - drug effects | Osteoblasts - drug effects | Osteogenesis - drug effects | Drug Resistance - drug effects | Fibroblast Growth Factors - pharmacology | Mice, Knockout | Osteoblasts - pathology | Organ Size - drug effects | Animals | Bone Marrow - pathology | Bone Resorption - pathology | Mice | Osteoprotegerin - metabolism | Osteoblasts - metabolism | Drugs | Fibroblast growth factor | Energy metabolism | Mesenchyme | Diabetes mellitus | Homeostasis | Bone turnover | Hormones | Glucose metabolism | Side effects | Bone mass | Osteoblastogenesis | adipogenesis | Stem cells | rosiglitazone | Lipid metabolism | Peroxisome proliferator-activated receptors | Bone loss | Index Medicus | Biological Sciences
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2010, Volume 107, Issue 19, pp. 8772 - 8777
Crohn's disease (CD), a major form of human inflammatory bowel disease, is characterized by primary immunodeficiencies. The nuclear receptor peroxisome... 
Crohn disease | Inflammatory bowel diseases | Receptors | Antimicrobials | Mucosa | Antibodies | Biopsies | Immunity | Caco 2 cells | Ulcerative colitis | Microbiota | Crohn's disease | Nutrition | β-defensin 1 | PPAR-γ | microbiota | beta-defensin 1 | CROHNS-DISEASE | MULTIDISCIPLINARY SCIENCES | PPAR-gamma | BETA | nutrition | CANDIDA-ALBICANS | SIGNALING PATHWAY | HUMAN BETA-DEFENSIN-1 | INFLAMMATORY BOWEL DISEASES | ASSOCIATION | EXPRESSION | SINGLE-NUCLEOTIDE POLYMORPHISMS | Crohn Disease - genetics | Gene Frequency - genetics | Humans | Colon - immunology | Ileum - immunology | PPAR gamma - metabolism | Promoter Regions, Genetic - genetics | Fungi - immunology | Bacteria - immunology | PPAR gamma - deficiency | beta-Defensins - metabolism | Cell Line | beta-Defensins - genetics | Colon - pathology | Mice, Inbred C57BL | Gene Expression Regulation | Genotype | Immunity, Innate - immunology | Models, Immunological | Animals | Crohn Disease - pathology | Protein Binding | Colon - microbiology | Mice | Ileum - microbiology | Physiological aspects | Research | Microbiota (Symbiotic organisms) | Homeostasis | Bacteria | Colon | Gastroenterology | Crohns disease | Immune system | Index Medicus | Immunodeficiency | Antimicrobial agents | Nuclear receptors | Promoters | Defensins | Intestine | Colitis | Intestinal microflora | Peroxisome proliferator-activated receptors | Biological Sciences
Journal Article
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR AND CELL BIOLOGY OF LIPIDS, ISSN 1388-1981, 10/2019, Issue 10, pp. 1458 - 1471
Monocyte chemoattractant protein-l-induced protein-1 (MCPIP1) acts as an endonuclease that degrades selected mRNAs, viral RNAs and pre-miRNAs. MCPIP1 inhibits... 
Obesity | CELLS | PGC-1-ALPHA | BIOCHEMISTRY & MOLECULAR BIOLOGY | MCPIP1 | FATTY-ACIDS | CELL BIOLOGY | Fatty liver | BIOPHYSICS | Peroxisome proliferator-activated receptor | INFLAMMATION | LIPOGENESIS | IMPAIRS ADIPOGENESIS | TXNIP | PPAR-GAMMA | PROTEIN-INDUCED PROTEIN-1 | DIFFERENTIATION | EXPRESSION | gamma
Journal Article
Journal Article
Journal of Animal Science, ISSN 0021-8812, 2017, Volume 95, Issue 7, pp. 2898 - 2906
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 03/2010, Volume 285, Issue 10, pp. 7153 - 7164
The recent insight that brown adipocytes and muscle cells share a common origin and in this respect are distinct from white adipocytes has spurred questions... 
SKELETAL-MUSCLE | NONSHIVERING THERMOGENESIS | FAT-CELLS | MITOCHONDRIAL BIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTIONAL CONTROL | UNCOUPLING PROTEIN-1 | GENE-EXPRESSION | ADULT HUMANS | COLD-ACCLIMATION | ADIPOSE-TISSUE | Adipocytes, White - cytology | Coculture Techniques | Ion Channels - genetics | Male | Epididymis - cytology | Mitochondrial Proteins - genetics | PPAR gamma - metabolism | Oxygen Consumption - physiology | Thermogenesis - physiology | Adipocytes, White - drug effects | Mitochondrial Proteins - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Trans-Activators - genetics | Thiazolidinediones - pharmacology | Cell Differentiation - physiology | PPAR gamma - genetics | Biomarkers - metabolism | Adipocytes, Brown - drug effects | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Hypoglycemic Agents - pharmacology | Mice, Knockout | Adipocytes, White - physiology | Voltage-Dependent Anion Channels - genetics | Adipocytes, Brown - physiology | Animals | Ion Channels - metabolism | PPAR gamma - agonists | Trans-Activators - metabolism | Mice | Transcription Factors | Uncoupling Protein 1 | Adipocytes, Brown - cytology | Voltage-Dependent Anion Channels - metabolism | Index Medicus | Adipocyte | Subcellular Organelles | UCP1 | Brown Adipocytes | Microscopic Imaging | PPARγ | Cell Biology | Receptors | Mitochondria | Nuclear | Mouse | Development Differentiation | Rosiglitazone | White Adipocytes
Journal Article
Journal Article
Kidney International, ISSN 0085-2538, 10/2012, Volume 82, Issue 7, pp. 771 - 789
Glomerular podocytes are highly specialized epithelial cells whose injury in glomerular diseases causes proteinuria. Since mitochondrial dysfunction is an... 
mitochondrial dysfunction | podocytes | SIRT1 | aldosterone | PGC-1a | OXIDATIVE STRESS | PGC-1 alpha | INDUCED APOPTOSIS | PGC-1-ALPHA | TRANSCRIPTIONAL COACTIVATOR | CELL APOPTOSIS | METABOLIC REGULATOR | UROLOGY & NEPHROLOGY | RAT PODOCYTES | FOCAL SEGMENTAL GLOMERULOSCLEROSIS | CALORIE RESTRICTION | Sirtuin 1 - metabolism | Apoptosis - drug effects | Receptors, Mineralocorticoid - metabolism | Podocytes - virology | Respiratory Syncytial Virus Infections - metabolism | Stilbenes - pharmacology | Aldosterone | Sirtuin 1 - genetics | Kidney Diseases - genetics | Receptors, Mineralocorticoid - drug effects | Transfection | RNA Interference | Time Factors | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Kidney Diseases - chemically induced | Mineralocorticoid Receptor Antagonists - pharmacology | Trans-Activators - genetics | Respiratory Syncytial Virus Infections - virology | Genes, Reporter | Cytoprotection | Disease Models, Animal | Kidney Diseases - metabolism | Cell Line | Promoter Regions, Genetic | Podocytes - metabolism | Kidney Diseases - pathology | Kidney Diseases - prevention & control | Mice, Inbred C57BL | Gene Expression Regulation | Respiratory Syncytial Virus Infections - genetics | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Podocytes - pathology | Animals | Podocytes - drug effects | Mitochondria - virology | Trans-Activators - metabolism | Mice | Transcription Factors | Enzyme Activation | Respiratory Syncytial Viruses - pathogenicity | Index Medicus
Journal Article
Journal of Cancer Research and Therapeutics, ISSN 0973-1482, 2018, Volume 14, Issue 9, pp. S306 - S310
Journal Article