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Nature, ISSN 0028-0836, 03/2010, Volume 464, Issue 7285, pp. 104 - 107
Injury causes a systemic inflammatory response syndrome (SIRS) that is clinically much like sepsis. Microbial pathogen-associated molecular patterns (PAMPs)... 
SHOCK | SIRS | OPERATED CALCIUM INFLUX | HUMAN NEUTROPHILS | PEPTIDES | MODELS | MOBILIZATION | CHEMOTAXIS | MULTIDISCIPLINARY SCIENCES | RECEPTORS | TRAUMA | Liver - pathology | Phosphorylation | Humans | Male | Mitochondria - immunology | Liver - injuries | N-Formylmethionine Leucyl-Phenylalanine - metabolism | Liver - immunology | Systemic Inflammatory Response Syndrome - blood | Muscle, Skeletal - immunology | Sepsis - metabolism | Receptors, Formyl Peptide - metabolism | Systemic Inflammatory Response Syndrome - immunology | Neutrophils - metabolism | Calcium Signaling | Femur - injuries | Sepsis - immunology | CpG Islands - immunology | Neutrophils - enzymology | Wounds and Injuries - pathology | Fractures, Bone - immunology | Wounds and Injuries - blood | Cells, Cultured | Neutrophils - immunology | Wounds and Injuries - complications | Rats | Wounds and Injuries - immunology | Acute Lung Injury - pathology | DNA, Mitochondrial - blood | Rats, Sprague-Dawley | DNA, Mitochondrial - immunology | Immunity, Innate - immunology | Systemic Inflammatory Response Syndrome - pathology | Animals | Mitochondria - secretion | Acute Lung Injury - immunology | Fractures, Bone - pathology | Systemic Inflammatory Response Syndrome - complications | Muscle, Skeletal - pathology | Toll-Like Receptor 9 - metabolism | Sepsis - microbiology | Mitogen-Activated Protein Kinases - metabolism | N-Formylmethionine Leucyl-Phenylalanine - immunology | Infection | Peptides | Physiological aspects | Mitochondrial DNA | Inflammation | Research | Risk factors | Fractures | Bacterial proteins | Rheumatoid arthritis | Trauma | Deoxyribonucleic acid--DNA
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 02/2017, Volume 114, Issue 8, pp. E1490 - E1499
DNA methylation and specifically the DNA methyltransferase enzyme DNMT3A are involved in the pathogenesis of a variety of hematological diseases and in... 
DNA methylation | Epigenetics | Inflammation | Mast cells | TRANSCRIPTION FACTORS | DNA METHYLTRANSFERASE 3A | MULTIDISCIPLINARY SCIENCES | ACUTE MYELOID-LEUKEMIA | mast cells | DE-NOVO METHYLATION | epigenetics | HUMAN GENOME | inflammation | HEMATOPOIETIC STEM-CELLS | IN-VIVO | DIFFERENTIATION | T-CELLS | GENE-TRANSCRIPTION | Biological Sciences | PNAS Plus
Journal Article
Injury, ISSN 0020-1383, 2017, Volume 48, pp. S10 - S14
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e46551
Aims: Accumulating evidence suggest that numerous microRNAs (miRNAs) play important roles in cell proliferation, apoptosis, and differentiation, as well as... 
IMMUNE-RESPONSE | HEART-DISEASE | SIGNALING PATHWAY | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | ACUTE MYOCARDIAL-INFARCTION | LIPID UPTAKE | CYTOKINE | INDUCTION | EXPRESSION | CORONARY-ARTERY-DISEASE | Leukocytes, Mononuclear - metabolism | Plaque, Atherosclerotic - genetics | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Humans | Male | MicroRNAs - metabolism | MAP Kinase Signaling System | Inflammation - metabolism | Inflammation Mediators - metabolism | Lipoproteins, LDL - metabolism | Plaque, Atherosclerotic - pathology | Biomarkers - metabolism | Atherosclerosis - pathology | Gene Expression | Plaque, Atherosclerotic - metabolism | MAP Kinase Kinase Kinases - genetics | Coronary Artery Disease - metabolism | Cytokines - secretion | Gene Expression Regulation | MAP Kinase Kinase Kinases - metabolism | Atherosclerosis - metabolism | Mice, Knockout | Macrophages - metabolism | Animals | Apolipoproteins E - genetics | Coronary Artery Disease - genetics | Inflammation - genetics | Mice | MicroRNAs - blood | MicroRNAs - genetics | Medical research | MicroRNA | Atherosclerosis | Medicine, Experimental | Inflammation | B cells | Apolipoproteins | Mitogens | Protein kinases | Coronary heart disease | Apoptosis | Cell proliferation | Post-transcription | Atherogenesis | Heart attacks | Inflammatory response | Cardiovascular disease | Feedback loops | Kinases | Liver cancer | Negative feedback | Apolipoprotein E | Aorta | Cardiology | Heart diseases | Immune system | Cytokines | Dendritic cells | Coronary artery | MiRNA | MAP kinase | Thorax | Gene expression | Ribonucleic acid--RNA | Coronary artery disease | Patients | Polymerase chain reaction | Medicine | Studies | Protein kinase | MicroRNAs | Arteriosclerosis | Coronary vessels | Biomarkers | Control theory | Cardiovascular diseases | Ribonucleic acids
Journal Article
Journal Article