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Cardiovascular Diabetology, ISSN 1475-2840, 05/2017, Volume 16, Issue 1, pp. 71 - 11
Mechanisms underlying the pathogenesis of ischemia/reperfusion injury are particularly complex, multifactorial and highly interconnected. A complex and... 
Antiplatelet therapy | Type 2 diabetes | Aspirin | Cardioprotection | CARDIAC & CARDIOVASCULAR SYSTEMS | RISK-FACTORS | CARDIOVASCULAR EVENTS | ISCHEMIA-REPERFUSION INJURY | CHEMERIN LEVELS | LOW-DOSE ASPIRIN | ENDOCRINOLOGY & METABOLISM | CORONARY-ARTERY-DISEASE | PRIMARY PREVENTION | ACTIVATING-FACTOR | INSULIN THERAPY | Ischemic Preconditioning, Myocardial - adverse effects | Myocardial Reperfusion Injury - etiology | Diabetes Mellitus, Type 2 - genetics | Humans | Risk Factors | MicroRNAs - metabolism | Myocardium - pathology | Treatment Outcome | Diabetes Mellitus, Type 2 - blood | Myocardial Reperfusion Injury - pathology | Animals | Signal Transduction - drug effects | Aspirin - adverse effects | Blood Platelets - metabolism | Myocardium - metabolism | Aspirin - therapeutic use | MicroRNAs - genetics | Blood Platelets - drug effects | Platelet Aggregation Inhibitors - therapeutic use | Diabetes Mellitus, Type 2 - complications | Drug Resistance | Myocardial Reperfusion Injury - blood | Myocardial Reperfusion Injury - prevention & control | Platelet Aggregation Inhibitors - adverse effects | Drugs | Therapy | Heart attacks | Laboratories | Pathogenesis | Property damage | Risk | Myocardial ischemia | Cardiovascular disease | Reperfusion | Ischemia | Blood platelets | Drug dosages | Injuries | Adenosine | Complications | Diabetes mellitus | MiRNA | Health risks | Patients | Thrombosis | Diseases | Disease prevention | MicroRNAs | Coronary vessels | Diabetes | Acute coronary syndromes | Cardiovascular diseases | Platelets
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2011, Volume 58, Issue 7, pp. 740 - 748
Journal Article
Cardiovascular Diabetology, ISSN 1475-2840, 06/2013, Volume 12, Issue 1, pp. 91 - 91
Background: Diabetic patients, through incompletely understood mechanisms, endure exacerbated ischemic heart injury compared to non-diabetic patients.... 
Ischemia-reperfusion | Inflammatory | Oxidative stress | Diabetes | Intermedin | Apoptosis | NADPH OXIDASE | ISCHEMIA/REPERFUSION INJURY | METABOLIC SYNDROME | CARDIAC & CARDIOVASCULAR SYSTEMS | NITRIC-OXIDE SYNTHASE | CARDIAC DYSFUNCTION | ACUTE CORONARY SYNDROME | INFARCT SIZE | SIGNALING PATHWAY | ENDOCRINOLOGY & METABOLISM | CARDIOVASCULAR-DISEASE | Diabetes Mellitus, Experimental - drug therapy | Diabetic Cardiomyopathies - metabolism | Apoptosis - drug effects | Streptozocin | Male | Adrenomedullin - pharmacology | Diabetic Cardiomyopathies - physiopathology | Myocardial Reperfusion Injury - pathology | Time Factors | Myocardial Infarction - pathology | Neuropeptides - pharmacology | Inflammation Mediators - metabolism | Diabetic Cardiomyopathies - prevention & control | Myocardial Infarction - physiopathology | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Diabetes Mellitus, Experimental - physiopathology | Cytokines - metabolism | Ventricular Function, Left - drug effects | Neuropeptides - metabolism | Adrenomedullin - metabolism | Cardiotonic Agents - pharmacology | Myocardial Infarction - metabolism | Rats, Sprague-Dawley | Myocardial Reperfusion Injury - physiopathology | Myocardial Reperfusion Injury - metabolism | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Diabetes Mellitus, Experimental - pathology | Myocytes, Cardiac - metabolism | Diabetic Cardiomyopathies - pathology | Myocardial Infarction - prevention & control | Oxidative Stress - drug effects | Myocardial Reperfusion Injury - prevention & control | Proteins | Hyperglycemia | Heart attacks | Mortality | Nitric oxide | Rodents | Cardiovascular disease
Journal Article
Journal Article
Journal Article
by Zhang, M and Zhu, JY and Qin, X and Zhou, M and Zhang, XL and Gao, Y and Zhang, TX and Xiao, DX and Cui, WT and Cai, XX
ACS APPLIED MATERIALS & INTERFACES, ISSN 1944-8244, 08/2019, Volume 11, Issue 34, pp. 30631 - 30639
Journal Article
Circulation Research, ISSN 0009-7330, 09/2017, Volume 121, Issue 8, pp. 930 - 940
RATIONALE:Clinical benefits of reperfusion after myocardial infarction are offset by maladaptive innate immune cell function, and therapeutic interventions are... 
Efferocytosis | Inflammation | Ischemia reperfusion injury | Phagocytosis | Macrophage | phagocytosis | CLEARANCE | CARDIAC & CARDIOVASCULAR SYSTEMS | efferocytosis | macrophage | ischemia reperfusion injury | MECHANISMS | TAM RECEPTORS | ACUTE-INFLAMMATION | INFARCTION | inflammation | APOPTOTIC CELLS | DISEASE | PERIPHERAL VASCULAR DISEASE | GROWTH-FACTOR | HEMATOLOGY | RECEPTOR TYROSINE KINASE | Receptors, CCR2 - genetics | Receptors, CCR2 - immunology | Humans | Middle Aged | Receptor Protein-Tyrosine Kinases - immunology | Male | Monocytes - immunology | Myocardial Reperfusion Injury - pathology | Time Factors | Proteolysis | Disease Models, Animal | Myocardial Reperfusion Injury - immunology | Genetic Predisposition to Disease | Macrophages - pathology | Signal Transduction | Proto-Oncogene Proteins - deficiency | Receptor Protein-Tyrosine Kinases - metabolism | Myocardial Reperfusion Injury - physiopathology | Macrophages - enzymology | Mice, Knockout | Monocytes - enzymology | Phenotype | Receptors, CCR2 - metabolism | Histocompatibility Antigens Class II - immunology | ST Elevation Myocardial Infarction - enzymology | Mice | Myocardium - immunology | Myocardial Reperfusion Injury - enzymology | c-Mer Tyrosine Kinase | Proto-Oncogene Proteins - immunology | Female | Histocompatibility Antigens Class II - metabolism | Macrophages - immunology | Cytokines - immunology | Proto-Oncogene Proteins - metabolism | ST Elevation Myocardial Infarction - immunology | Receptor Protein-Tyrosine Kinases - deficiency | Cytokines - metabolism | Mice, Inbred C57BL | Myocardium - pathology | Proto-Oncogene Proteins - genetics | Immunity, Innate | ST Elevation Myocardial Infarction - physiopathology | Myocardium - enzymology | Animals | Receptor Protein-Tyrosine Kinases - genetics | ST Elevation Myocardial Infarction - pathology | Apoptosis | Heart | Myocardial infarction | Tyrosine | CC chemokine receptors | Heart attacks | Cytokines | Therapeutic applications | Transforming growth factor | Immune clearance | Wounds | Myocardial ischemia | Macrophages | Phagocytes | Reperfusion | Major histocompatibility complex | Ischemia | CCR2 protein | Rodents | Histocompatibility | Heart diseases | Protein-tyrosine kinase | Monocyte chemoattractant protein 1
Journal Article
Journal Article