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Journal Article
Oncogene, ISSN 1476-5594, 05/2014, Volume 34, Issue 14, pp. 1831 - 1842
Extracellular ATP has been shown to either inhibit or promote cancer growth and migration; however, the mechanism underlying this discrepancy remained elusive.... 
Biochemistry & Molecular Biology | Oncology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Neoplasm Transplantation | Cell Proliferation | Humans | Adenosine Triphosphate - secretion | Bone Neoplasms - secondary | Transplantation, Heterologous | Receptors, Purinergic P2X - genetics | Adenosine Triphosphate - pharmacology | RNA Interference | Apyrase - pharmacology | Adenosine Triphosphate - metabolism | Receptors, Purinergic P2X - metabolism | Female | Receptor, Adenosine A2A - genetics | Osteocytes - metabolism | Signal Transduction | Mice, Inbred C57BL | Receptor, Adenosine A2A - metabolism | Adenosine Triphosphate - analogs & derivatives | Bone Density Conservation Agents - pharmacology | Cell Movement - drug effects | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Mice, Nude | Adenosine - secretion | Alendronate - pharmacology | Adenosine - metabolism | Cell Line, Tumor | Mice | RNA, Small Interfering | Complications and side effects | Bone cancer | Development and progression | Breast cancer | Genetic aspects | Metastasis | Health aspects | Adenosine triphosphate | Bones | Adenosine triphosphatase | Adenosine | Carcinoma | Leukocyte migration | Osteocytes | siRNA | Nucleotides | Bisphosphonates | Purine P2X receptors | Cell adhesion & migration | Metastases | Alendronic acid | Bone growth | Xenografts | Apyrase | Mammary gland | Cell migration | Index Medicus | bone metastasis | adenosine nucleotides | breast cancer
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 09/2011, Volume 301, Issue 3, pp. H1183 - H1189
All four adenosine receptor subtypes have been shown to play a role in cardioprotection, and there is evidence that all four subtypes may be expressed in... 
Knockout mice | Adenosine receptor | Myocardial Infarction - genetics | Receptor, Adenosine A1 - drug effects | Adenosine A1 Receptor Antagonists - pharmacology | Male | Receptor, Adenosine A2B - genetics | Myocardial Reperfusion Injury - pathology | Adenosine A2 Receptor Agonists - pharmacology | Time Factors | Adenosine A1 Receptor Agonists - pharmacology | Myocardial Infarction - pathology | Myocardium - metabolism | Myocardial Infarction - physiopathology | Receptor, Adenosine A2A - genetics | Myocardial Reperfusion Injury - genetics | Disease Models, Animal | Receptor Cross-Talk | Receptor, Adenosine A2B - deficiency | Mice, Inbred C57BL | Ventricular Function, Left - drug effects | Ventricular Pressure - drug effects | Myocardium - pathology | Adenosine - pharmacology | Myocardial Infarction - metabolism | Receptor, Adenosine A2A - metabolism | Receptor, Adenosine A1 - metabolism | Myocardial Reperfusion Injury - physiopathology | Receptor, Adenosine A2B - metabolism | Mice, Knockout | Myocardial Reperfusion Injury - metabolism | Animals | Analysis of Variance | Perfusion | Adenosine - analogs & derivatives | Mice | Myocardial Infarction - prevention & control | Receptor, Adenosine A2A - deficiency | Myocardial Reperfusion Injury - prevention & control | Index Medicus | A2a adenosine receptor | A2b adenosine receptor | knockout mice | Integrative Cardiovascular Physiology and Pathophysiology
Journal Article
PLoS biology, ISSN 1545-7885, 03/2019, Volume 17, Issue 3, pp. e3000161 - e3000161
Adenosine is a constituent of many molecules of life; increased free extracellular adenosine indicates cell damage or metabolic stress. The importance of... 
Biochemistry & Molecular Biology | Biology | Life Sciences & Biomedicine - Other Topics | Life Sciences & Biomedicine | Science & Technology | Blood Pressure - genetics | Male | Receptor, Adenosine A2B - genetics | Hypothermia - chemically induced | Body Temperature - physiology | Hypothermia - metabolism | Receptor, Adenosine A3 - metabolism | Caffeine - pharmacology | Heart Rate - physiology | Female | Receptor, Adenosine A2A - genetics | Blood Pressure - physiology | Uridine - toxicity | Receptor, Adenosine A1 - genetics | Inosine - pharmacology | Heart Rate - genetics | Genotype | Receptor, Adenosine A2A - metabolism | Receptor, Adenosine A1 - metabolism | Receptor, Adenosine A2B - metabolism | Mice, Knockout | Phenotype | Animals | Body Temperature - genetics | Lipopolysaccharides - pharmacology | Receptor, Adenosine A3 - genetics | Mice | Hypothermia - genetics | Physiological aspects | Nucleosides | Adenosine | Temperature requirements | Temperature | Thermoregulation | Kinases | Receptors | Torpor | Temperature effects | Dependence | Physiology | Inhibition | Damage | Supervision | Phenotypes | Adenosine receptors | AMP | Breeding | Body temperature | Hazards | Metabolism | Gene expression | Diurnal | Hypotension | Histamine | Bradycardia | Adenosine kinase | Uridine | Diabetes | Kidney diseases | Endocrinology | Hypothermia | Index Medicus
Journal Article
Journal of cellular physiology, ISSN 0021-9541, 03/2018, Volume 233, Issue 3, pp. 2032 - 2057
Journal Article