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Journal Article
Plant and Cell Physiology, ISSN 0032-0781, 2014, Volume 55, Issue 4, pp. 737 - 749
Journal Article
Plant Physiology, ISSN 0032-0889, 2015, Volume 167, Issue 2, pp. 394 - 410
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2014, Volume 111, Issue 5, pp. 2005 - 2010
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2016, Volume 11, Issue 7, pp. e0159341 - e0159341
Poly(ADP-ribose) polymerases (PARPs) are the first proteins involved in cellular DNA repair pathways to be targeted by specific inhibitors for clinical... 
BLOOMS-SYNDROME GENE | POLY(ADP-RIBOSE) POLYMERASE | RECOMBINATION | DNA-DAMAGE RESPONSE | MULTIDISCIPLINARY SCIENCES | HOMOLOGY-DIRECTED REPAIR | MICE | CHROMOSOME STABILITY | MAMMALIAN-CELLS | CANCER | SISTER-CHROMATID EXCHANGES | DNA Damage - drug effects | DNA Repair - drug effects | Gene Expression | Humans | Homologous Recombination | Poly(ADP-ribose) Polymerase Inhibitors - adverse effects | Cisplatin - pharmacology | Piperazines - adverse effects | Dose-Response Relationship, Drug | Drug Synergism | Poly(ADP-ribose) Polymerases - metabolism | Sister Chromatid Exchange - drug effects | Poly(ADP-ribose) Polymerases - genetics | Chromosome Aberrations - drug effects | Karyotyping | Genomic Instability - drug effects | Female | Benzimidazoles - adverse effects | Phthalazines - adverse effects | Care and treatment | DNA damage | Analysis | Risk factors | Ovarian cancer | Cancer | Toxicity | Epithelial cells | Sister chromatid exchange | Homologous recombination | Genotoxicity | Poly(ADP-ribose) | Clinical trials | Cytotoxicity | Homology | Kinases | ADP | DNA repair | Genomic instability | Proteins | Ribose | Rodents | Ribosylation | Repair | Deoxyribonucleic acid--DNA | ADP-ribosylation | BRCA2 protein | Medical research | Lymphoid cells | Stability | BRCA1 protein | Hypersensitivity | Poly(ADP-ribose) polymerase | Breast cancer | Double-strand break repair | Cisplatin | Inhibitors | Cell lines | Mutation | Aberration | Prostate cancer | Prostate | Tumors | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Nature Cell Biology, ISSN 1465-7392, 05/2007, Volume 9, Issue 5, pp. 493 - 505
Activating Ras mutations can induce either proliferation or senescence depending on the cellular context. To determine whether Ras activation has... 
ACTIVATION | PREMATURE SENESCENCE | K-RAS ONCOGENE | CONDITIONAL EXPRESSION | CELL-CYCLE ARREST | PROLIFERATION | MICE | GLAND | TUMOR INITIATION | P53 | CELL BIOLOGY | Up-Regulation | Receptors, Estrogen - metabolism | Cell Proliferation | Epithelial Cells - metabolism | Epithelial Cells - drug effects | Hyperplasia | Gene Expression Regulation, Neoplastic | Cellular Senescence - drug effects | Precancerous Conditions - metabolism | Mammary Neoplasms, Experimental - metabolism | Promoter Regions, Genetic - drug effects | Mammary Neoplasms, Experimental - genetics | Oncogene Protein p21(ras) - metabolism | Receptors, Progesterone - metabolism | Dose-Response Relationship, Drug | Time Factors | Cell Transformation, Neoplastic - genetics | Mammary Neoplasms, Experimental - pathology | Female | Precancerous Conditions - pathology | ADP-Ribosylation Factors - metabolism | Cellular Senescence - genetics | Signal Transduction | Mammary Glands, Animal - pathology | Cell Cycle Proteins - metabolism | Tumor Suppressor Protein p53 - metabolism | Epithelial Cells - pathology | Mice, Transgenic | Mammary Glands, Animal - drug effects | Cell Transformation, Neoplastic - metabolism | Precancerous Conditions - genetics | Protein Transport | Animals | Mammary Glands, Animal - metabolism | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Doxycycline - pharmacology | Mice | Cell Transformation, Neoplastic - drug effects | Mutation | Cell Transformation, Neoplastic - pathology | Oncogene Protein p21(ras) - genetics | Gene mutations | Doxycycline | Causes of | Breast cancer | Genetic aspects | Research | Gene expression | Health aspects | Index Medicus
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Journal Article
Developmental Cell, ISSN 1534-5807, 01/2012, Volume 22, Issue 1, pp. 211 - 222
Journal Article
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