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PLoS Genetics, ISSN 1553-7390, 2013, Volume 9, Issue 12, pp. e1003977 - e1003977
Cilia are microtubule-based cell appendages, serving motility, chemo-/mechano-/photo- sensation, and developmental signaling functions. Cilia are comprised of... 
COMPLEX | CAENORHABDITIS-ELEGANS | TRANSITION ZONE | ARL13B | SIGNALING PROTEINS | GENETICS & HEREDITY | TRAFFICKING | C-ELEGANS | CILIOGENESIS | SENSORY CILIA | INTRAFLAGELLAR TRANSPORT | Polycystic Kidney Diseases - genetics | Cytoskeletal Proteins - genetics | Humans | Biological Transport, Active - genetics | Cerebellum - abnormalities | Membranes - metabolism | Polycystic Kidney Diseases - pathology | Retinitis Pigmentosa | Cerebellar Diseases - pathology | Bardet-Biedl Syndrome - pathology | Kidney Diseases, Cystic - genetics | Bardet-Biedl Syndrome - genetics | Cytoskeletal Proteins - metabolism | ADP-Ribosylation Factors - metabolism | ADP-Ribosylation Factors - genetics | Encephalocele - genetics | Caenorhabditis elegans - metabolism | Caenorhabditis elegans - genetics | Abnormalities, Multiple | Transcription Factors - genetics | Eye Abnormalities - genetics | Cilia - metabolism | Cilia - genetics | Kidney Diseases, Cystic - pathology | Transcription Factors - metabolism | Animals | Cerebellar Diseases - genetics | Ciliary Motility Disorders - pathology | Encephalocele - pathology | Eye Abnormalities - pathology | Retina - abnormalities | Retina - pathology | Ciliary Motility Disorders - genetics | Cilia and ciliary motion | Biological transport, Active | Joubert syndrome | Physiological aspects | Genetic research | Genetic aspects | Research | Health aspects | Index Medicus | Proteins | Studies | Genotype & phenotype | Membranes | Motility | Peptides | Proteomics | Diffusion | Experiments
Journal Article
Journal Article
Molecular Cell, ISSN 1097-2765, 10/2013, Volume 52, Issue 2, pp. 272 - 285
Poly(ADP-ribos)ylation (PARylation) is a reversible posttranslational modification found in higher eukaryotes. However, little is known about PARylation... 
DNA-DAMAGING AGENTS | REPAIR | INHIBITION | CHROMATIN | RNA | PARP1 | BIOCHEMISTRY & MOLECULAR BIOLOGY | ADP-RIBOSYLATION | SITES | BINDING PROTEIN | MAMMALIAN-CELLS | CELL BIOLOGY | TATA-Binding Protein Associated Factors - metabolism | Oxidants - pharmacology | Proteome - genetics | Humans | Antineoplastic Agents, Alkylating - pharmacology | Green Fluorescent Proteins - genetics | DNA-Binding Proteins - metabolism | Tandem Mass Spectrometry | Cell Nucleus - metabolism | Protein Processing, Post-Translational - drug effects | RNA Interference | Chromatography, Liquid | Proteomics - methods | Signal Transduction - radiation effects | Nuclear Proteins - genetics | Radiation, Ionizing | Green Fluorescent Proteins - metabolism | Electrophoresis, Polyacrylamide Gel | Hydrogen Peroxide - pharmacology | Protein Processing, Post-Translational - radiation effects | Nuclear Proteins - metabolism | Signal Transduction - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | TATA-Binding Protein Associated Factors - genetics | Transcription Factors - metabolism | Microscopy, Confocal | Poly Adenosine Diphosphate Ribose - metabolism | Cell Nucleus - genetics | Signal Transduction - drug effects | Cell Line, Tumor | DNA Damage | HeLa Cells | Methyl Methanesulfonate - pharmacology | Proteome - metabolism | Index Medicus
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 11/2016, Volume 17, Issue 11, pp. 1869 - 1869
Tumor necrosis factor receptor-associated factor 2 (TRAF2) is a critical mediator of tumor necrosis factor-alpha (TNF-alpha) signaling. However, the regulatory... 
TRAF2 | JNK | Tumor necrosis factor-α (TNF-α) | BIG1 | Apoptosis | APOPTOSIS | LOCALIZATION | ACTIVATION | PROTEIN | NECROPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | tumor necrosis factor-alpha (TNF-alpha); apoptosis | CHEMISTRY, MULTIDISCIPLINARY | CELL-DEATH | ADP-RIBOSYLATION FACTORS | RECYCLING ENDOSOMES | ARF-FAMILY | NF-KAPPA-B | RNA, Small Interfering - genetics | Luciferases - metabolism | Apoptosis - drug effects | Humans | TNF Receptor-Associated Factor 2 - genetics | JNK Mitogen-Activated Protein Kinases - metabolism | NF-kappa B - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Luciferases - genetics | Proteolysis - drug effects | Ubiquitination - drug effects | Guanine Nucleotide Exchange Factors - metabolism | JNK Mitogen-Activated Protein Kinases - genetics | Genes, Reporter | Guanine Nucleotide Exchange Factors - genetics | NF-kappa B - antagonists & inhibitors | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Gene Expression Regulation | TNF Receptor-Associated Factor 2 - metabolism | Signal Transduction - genetics | Receptors, Tumor Necrosis Factor, Type I - antagonists & inhibitors | Receptors, Tumor Necrosis Factor, Type I - genetics | Protein Transport | TNF Receptor-Associated Factor 2 - antagonists & inhibitors | Tumor Necrosis Factor-alpha - pharmacology | Guanine Nucleotide Exchange Factors - antagonists & inhibitors | NF-kappa B - genetics | Cell Line, Tumor | Protein Binding | HeLa Cells | RNA, Small Interfering - metabolism | Signal transduction | Tumor necrosis factor-TNF | Adenosine triphosphatase | Index Medicus | apoptosis | tumor necrosis factor-α (TNF-α)
Journal Article
Nature Cell Biology, ISSN 1465-7392, 01/2012, Volume 14, Issue 1, pp. 80 - 86
Journal Article
Genes and Development, ISSN 0890-9369, 11/2004, Volume 18, Issue 21, pp. 2614 - 2626
Alveolar rhabdomyosarcoma is an aggressive childhood muscle cancer for which outcomes are poor when the disease is advanced. Although well-developed mouse... 
FoxO1A | Pax3:Fkhr | Pax3 | Satellite cell | Alveolar rhabdomyosarcoma | Fkhr | Chromosome translocation | CHILDHOOD RHABDOMYOSARCOMA | STEM-CELLS | Pax3 : Fkhr | chromosome translocation | SATELLITE CELLS | DEVELOPMENTAL BIOLOGY | SKELETAL-MUSCLE DEVELOPMENT | MUTANT MICE | satellite cell | alveolar rhabdomyosarcoma | GENETICS & HEREDITY | GENE-EXPRESSION | FUSION PROTEIN | PAX7 EXPRESSION | MUTATIONS | HUMAN CANCER | Humans | Muscle, Skeletal - metabolism | Tumor Suppressor Protein p53 - genetics | DNA-Binding Proteins - metabolism | Cell Differentiation - genetics | Gene Expression Regulation, Developmental | Mice, Mutant Strains | Rhabdomyosarcoma, Alveolar - pathology | ADP-Ribosylation Factors - metabolism | ADP-Ribosylation Factors - genetics | Disease Models, Animal | Genetic Engineering - methods | PAX3 Transcription Factor | Myogenic Regulatory Factors - genetics | Rhabdomyosarcoma, Alveolar - genetics | Paired Box Transcription Factors | Tumor Suppressor Protein p53 - metabolism | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Myogenic Regulatory Factors - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Transcription Factors - metabolism | Animals | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Forkhead Box Protein O1 | Mice | Muscle, Skeletal - pathology | Mutation | Forkhead Transcription Factors | Muscles | Genetic research | Research | Observations | Rhabdomyosarcoma | Index Medicus | Research Papers
Journal Article
Genes and Development, ISSN 0890-9369, 03/2011, Volume 25, Issue 5, pp. 485 - 498
Journal Article
Molecular Biology and Evolution, ISSN 0737-4038, 01/2013, Volume 30, Issue 1, pp. 45 - 56
Journal Article
The American Journal of Human Genetics, ISSN 0002-9297, 10/2018, Volume 103, Issue 4, pp. 612 - 620
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2016, Volume 11, Issue 1, pp. e0148097 - e0148097
High reactive oxygen species (ROS) levels and enhanced vascular smooth muscle cells (VSMC) proliferation are observed in numerous cardiovascular diseases. The... 
MIGRATION | PATHWAYS | COMPLEX | PROTEIN | TYROSINE KINASE | MULTIDISCIPLINARY SCIENCES | GROWTH | CARDIOVASCULAR-DISEASE | RECEPTOR | RAC1 | DOWNSTREAM ACTIVATION | RNA, Small Interfering - genetics | Receptor, Epidermal Growth Factor - genetics | Reactive Oxygen Species - metabolism | Muscle, Smooth, Vascular - metabolism | Receptor, Angiotensin, Type 1 - genetics | Receptor, Epidermal Growth Factor - metabolism | MAP Kinase Kinase 4 - metabolism | ADP-Ribosylation Factors - antagonists & inhibitors | Mitogen-Activated Protein Kinase 1 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | ADP-Ribosylation Factors - metabolism | Myocytes, Smooth Muscle - drug effects | NADH, NADPH Oxidoreductases - metabolism | ADP-Ribosylation Factors - genetics | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Muscle, Smooth, Vascular - drug effects | Vasoconstrictor Agents - pharmacology | Cell Line | NADH, NADPH Oxidoreductases - genetics | Angiotensin II - pharmacology | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Gene Expression Regulation | Rats | p38 Mitogen-Activated Protein Kinases - genetics | Muscle, Smooth, Vascular - cytology | NADPH Oxidase 1 | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Receptor, Angiotensin, Type 1 - metabolism | MAP Kinase Kinase 4 - genetics | Cell Proliferation - drug effects | rac1 GTP-Binding Protein - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | RNA, Small Interfering - metabolism | rac1 GTP-Binding Protein - genetics | Cell proliferation | Angiotensin | Physiological aspects | Smooth muscle | Genetic aspects | Research | Guanosine triphosphatase | ADP-ribosylation factor | Molecular machines | GTP-binding protein | Oxidase | Intracellular signalling | Activation | Hormones | Kinases | ADP | ADP-ribosylation factor 6 | NAD(P)H oxidase | Cell adhesion & migration | Proteins | Signal transduction | Cell growth | Epidermal growth factor | Rodents | Aorta | Ribosylation | Angiotensin II | Heart diseases | ADP-ribosylation | Enzymes | Immunoglobulins | Epidermal growth factor receptors | RNA-mediated interference | Muscles | Extracellular signal-regulated kinase | MAP kinase | Rac1 protein | JNK protein | Superoxide | Pharmacology | Gene expression | Ribonucleic acid--RNA | Medicine | Cardiovascular diseases | Guanosinetriphosphatase | Index Medicus | RNA | Ribonucleic acid
Journal Article