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Journal Article
Nature Medicine, ISSN 1078-8956, 05/2016, Volume 22, Issue 5, pp. 547 - 556
Doxorubicin is an anthracycline chemotherapy agent effective in treating a wide range of malignancies, but it causes a dose-related cardiotoxicity that can... 
MEDICINE, RESEARCH & EXPERIMENTAL | OXIDATIVE STRESS | RISK-FACTORS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DEXRAZOXANE | INDUCED CARDIOMYOPATHY | MATURATION | MUSCLE GENE-EXPRESSION | CELL BIOLOGY | CHILDHOOD-CANCER | INHIBITION | ANTHRACYCLINE-INDUCED CARDIOTOXICITY | CONGESTIVE-HEART-FAILURE | Mitochondria, Heart - metabolism | Reactive Oxygen Species - metabolism | Antibiotics, Antineoplastic - pharmacology | Apoptosis - drug effects | Calcium - metabolism | Humans | Middle Aged | Transcriptome | Antibiotics, Antineoplastic - adverse effects | Mitochondria, Heart - drug effects | Membrane Potential, Mitochondrial - drug effects | Flow Cytometry | Adult | Female | Real-Time Polymerase Chain Reaction | Cardiotoxicity - genetics | DNA Damage - drug effects | Cell Survival - drug effects | Disease Susceptibility | Heart Failure - genetics | Breast Neoplasms - drug therapy | Myocytes, Cardiac - drug effects | Fluorescent Antibody Technique | Myocytes, Cardiac - metabolism | Aged | Polymorphism, Single Nucleotide | Oxidative Stress - drug effects | Induced Pluripotent Stem Cells | Doxorubicin - adverse effects | Doxorubicin - pharmacology | Heart Failure - chemically induced | Complications and side effects | Patient outcomes | Stem cells | Development and progression | Breast cancer | Transplantation | Cardiovascular diseases | Drug therapy | Doxorubicin | Methods | Heart failure | Chemotherapy | Toxicity | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 2012, Volume 485, Issue 7400, pp. 593 - 598
The reprogramming of adult cells into pluripotent cells or directly into alternative adult cell types holds great promise for regenerative medicine. We... 
HEART | DEFINED FACTORS | STEM-CELLS | REPAIR | MULTIDISCIPLINARY SCIENCES | DIRECT CONVERSION | INJURY | GENE-TRANSFER | DIFFERENTIATION | EXPRESSION | ADULT | Heart - physiology | Male | Regenerative Medicine - methods | Cellular Reprogramming | Myocardial Infarction - therapy | Myocardial Infarction - pathology | Cell Transdifferentiation | Female | Myocardial Infarction - physiopathology | Cicatrix - therapy | MEF2 Transcription Factors | Fibroblasts - metabolism | Heart - physiopathology | GATA4 Transcription Factor - metabolism | Myocytes, Cardiac - cytology | Myogenic Regulatory Factors - genetics | Thymosin - pharmacology | Biomarkers - analysis | Gene Expression Regulation | Myogenic Regulatory Factors - metabolism | GATA4 Transcription Factor - genetics | Myocardium - pathology | Fibroblasts - pathology | T-Box Domain Proteins - genetics | Genetic Vectors - genetics | T-Box Domain Proteins - metabolism | Myocardium - cytology | Cell Lineage | Animals | Myocardial Infarction - drug therapy | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - physiology | Fibroblasts - drug effects | Myocytes, Cardiac - metabolism | Cicatrix - pathology | Fibroblasts - cytology | Mice | Thymosin - therapeutic use | Medical research | Regenerative medicine | Heart cells | Physiological aspects | Fibroblasts | Medicine, Experimental | Research | Proteins | Musculoskeletal system | Cardiomyocytes | Heart attacks | Rodents | Stem cells | Index Medicus | Cellular reprogramming | Cardiac fibroblast | Thymosin β4 | Cardiomyocyte
Journal Article
Circulation Research, ISSN 0009-7330, 02/2017, Volume 120, Issue 4, pp. 627 - 629
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2014, Volume 111, Issue 24, pp. 8850 - 8855
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2016, Volume 68, Issue 19, pp. 2086 - 2096
Abstract Background Brugada syndrome (BrS), a disorder associated with characteristic electrocardiogram precordial ST-segment elevation, predisposes afflicted... 
Cardiovascular | Internal Medicine | genome editing | Ca2+ transient | SCN5A | action potential | arrhythmia | gene expression | transient | Ca | RECAPITULATE | CARDIAC & CARDIOVASCULAR SYSTEMS | SEGMENT ELEVATION | CARDIOTOXICITY | MECHANISMS | LONG-QT SYNDROME | DILATED CARDIOMYOPATHY | MODELS | MUTATION | SUDDEN CARDIAC DEATH | ASSOCIATION | Brugada Syndrome - genetics | Humans | Middle Aged | Male | RNA - genetics | Young Adult | Brugada Syndrome - metabolism | Brugada Syndrome - pathology | Polymerase Chain Reaction | Electrocardiography | Adult | Cell Differentiation | NAV1.5 Voltage-Gated Sodium Channel - genetics | Induced Pluripotent Stem Cells - cytology | NAV1.5 Voltage-Gated Sodium Channel - biosynthesis | Induced Pluripotent Stem Cells - metabolism | Heart Conduction System - pathology | Myocytes, Cardiac - cytology | Gene Expression Regulation | Genotype | Phenotype | Heart Conduction System - physiopathology | Pedigree | Adolescent | Myocytes, Cardiac - metabolism | Heart | Medical colleges | Analysis | Electrocardiogram | Genomics | Stem cells | Genetic aspects | Cardiology | Cardiovascular agents | Cardiomyopathy | Editing | Cardiomyocytes | Genomes | Family medical history | Gene expression | Patients | Cell adhesion & migration | Studies | Genotype & phenotype | Hypotheses | Sodium | Fibroblasts | Genetic engineering | Mutation | Methods | Index Medicus | Abridged Index Medicus | cardiomyocytes | Induced pluripotent stem cells | Brugada Syndrome
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 03/2012, Volume 16, Issue 3, pp. 468 - 482
Journal Article
Nature, ISSN 0028-0836, 07/2017, Volume 547, Issue 7662, pp. 227 - 231
The regenerative capacity of the adult mammalian heart is limited, because of the reduced ability of cardiomyocytes to progress through mitosis(1). Endogenous... 
PRESSURE-OVERLOAD | GENE | ADULT HEART REGENERATION | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MOUSE MODEL | MUSCLE | DUCHENNE MUSCULAR-DYSTROPHY | MICE | Protein-Serine-Threonine Kinases - deficiency | Phosphorylation | Cell Proliferation | Glycoproteins - metabolism | Dystroglycans - metabolism | Male | Phosphoproteins - metabolism | Heart Failure - prevention & control | Multiprotein Complexes - metabolism | Dystrophin - deficiency | Mice, Inbred mdx | Cardiomyopathies | Glycoproteins - deficiency | Multiprotein Complexes - deficiency | Dystrophin - metabolism | Protein-Serine-Threonine Kinases - metabolism | Myocytes, Cardiac - cytology | Mice, Inbred C57BL | Organ Size | Heart Failure - genetics | Pressure | Multiprotein Complexes - chemistry | Animals | Dystrophin - genetics | Myocytes, Cardiac - metabolism | Protein Binding | Mice | Muscular Dystrophy, Duchenne - metabolism | Muscular Dystrophy, Duchenne - genetics | Adaptor Proteins, Signal Transducing - metabolism | Heart | Genes | Homeostasis | Genomes | Kinases | Muscular dystrophy | Cell growth | Actin | Duchenne's muscular dystrophy | Cell cycle | Extracellular matrix | Heart diseases | Dystrophin | Deoxyribonucleic acid--DNA | Heart failure | Dystroglycan | Glycoprotein | Cardiomyocytes | Studies | Yes-associated protein | Regeneration | Point mutation | Cytoskeleton | Dystrophy | Mutation | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 9, pp. 1 - 13
Background: It has long been thought that mammalian cardiomyocytes are terminally-differentiated and unable to proliferate. However, myocytes in more primitive... 
PROGENITORS | ABNORMAL QT PROLONGATION | MULTIPOTENT | FIBROBLAST | MULTIDISCIPLINARY SCIENCES | MOUSE | CARDIAC STEM-CELLS | ZEBRAFISH HEART REGENERATION | EXPRESSION | ADULT | ATRIAL-FIBRILLATION | Cell Proliferation | GATA4 Transcription Factor - metabolism | Myocytes, Cardiac - cytology | Homeodomain Proteins - metabolism | Cells, Cultured | Rats | Rats, Inbred WKY | GATA4 Transcription Factor - genetics | Male | Mice, Transgenic | Transcription Factors - genetics | Homeodomain Proteins - genetics | Transcription Factors - metabolism | Homeobox Protein Nkx-2.5 | Animals | Myocytes, Cardiac - metabolism | Cell Differentiation | Mice | Fluorescence | Genetic engineering | Stem cells | Heart cells | Heart | Cell proliferation | Cell culture | Cardiac arrhythmia | Transcription factors | Physicians | Centrifugation | Myocytes | Kinases | Genotype & phenotype | Cell fate | Transgenic animals | Rodents | Cell cycle | Plasticity | Fibroblasts | Extracellular matrix | Cardiology | Heart diseases | Nkx2.5 protein | Explants | Antigens | Cardiac muscle | Transgenic mice | MiRNA | Zebrafish | Cardiomyocytes | Mammals | Tamoxifen | Gene expression | c-Kit protein | Endothelial cells | Medicine | Studies | Polymerase chain reaction | Microscopy | Striations | Green fluorescent protein | Plastic properties | Diabetes | Gene mapping | Pluripotency | Plastic foam | Index Medicus
Journal Article