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Environmental Health Perspectives, ISSN 0091-6765, 12/2008, Volume 116, Issue 12, pp. 1694 - 1699
Background: Polybrominated diphenyl ether (PBDE) flame retardants have been implicated as disruptors of the hypothalamic-pituitary-thyroid axis. Animals... 
Minnows | Brain | Receptors | Messenger RNA | Halogenated diphenyl ethers | Genes | Liver | Thyroid hormones | Research | Dosage | Neurogenesis | Polybrominated diphenyl ethers | Thyroid-stimulating hormone | Basic transcription element-binding protein | Endocrine disruption | Thyroid hormone | Thyrotropin | PBDE-47 | Thyroid hormone receptor | thyroid hormone | MINNOW PIMEPHALES-PROMELAS | polybrominated diphenyl ethers | basic transcription element-binding protein | ELEMENT-BINDING PROTEIN | brain | endocrine disruption | thyroid-stimulating hormone | ADULT-RAT BRAIN | XENOPUS-LAEVIS | ENVIRONMENTAL SCIENCES | PUBLIC, ENVIRONMENTAL & OCCUPATIONAL HEALTH | thyroid hormone receptor | thyrotropin | BROMINATED FLAME RETARDANTS | RECEPTOR-BETA | CENTRAL-NERVOUS-SYSTEM | FATHEAD MINNOW | POLYCHLORINATED-BIPHENYLS | Pituitary Gland - metabolism | Liver - metabolism | RNA, Messenger - genetics | Male | Brain - drug effects | Brain - metabolism | Halogenated Diphenyl Ethers - toxicity | Tissue Distribution | Halogenated Diphenyl Ethers - administration & dosage | Thyroid Gland - physiology | Transcription, Genetic - physiology | Animals | Receptors, Thyroid Hormone - genetics | Diet | Radioimmunoassay | Halogenated Diphenyl Ethers - pharmacokinetics | Female | Polybrominated Biphenyls | Cyprinidae | Pituitary Gland - drug effects | Thyroid Gland - drug effects | Thyroid Hormones - physiology | Polybrominated biphenyls | Environmental aspects | Physiological aspects | Thyroid gland | Genetic aspects | Gene expression | Health aspects | Index Medicus
Journal Article
Journal of Neurotrauma, ISSN 0897-7151, 12/2009, Volume 26, Issue 12, pp. 2257 - 2267
An important but poorly understood feature of traumatic brain injury (TBI) is the clinically serious problem of spatiotemporal progression ("blossoming'') of a... 
Contusion | Sulfonylurea receptor 1 | Traumatic brain injury | Glibenclamide | Hemorrhage | CELLS | NONSELECTIVE CATION CHANNEL | sulfonylurea receptor 1 | contusion | traumatic brain injury | HEAD TRAUMA | VASCULAR DISRUPTION | NEUROSCIENCES | CLINICAL NEUROLOGY | ADULT-RAT BRAIN | REACTIVE ASTROCYTES | SPINAL-CORD-INJURY | glibenclamide | ISCHEMIC-STROKE | LATERAL FLUID PERCUSSION | hemorrhage | CRITICAL CARE MEDICINE | Oligodeoxyribonucleotides, Antisense - pharmacology | Calcium Channels - metabolism | Receptors, Drug - genetics | Rats, Long-Evans | Blood-Brain Barrier - physiopathology | Male | Brain Injuries - metabolism | Sulfonylurea Receptors | Brain Injuries - physiopathology | Brain Hemorrhage, Traumatic - pathology | Brain - metabolism | Brain Hemorrhage, Traumatic - physiopathology | Neuroprotective Agents - pharmacology | ATP-Binding Cassette Transporters - genetics | Glyburide - pharmacology | Up-Regulation - physiology | ATP-Binding Cassette Transporters - metabolism | Potassium Channels, Inwardly Rectifying - antagonists & inhibitors | Disease Models, Animal | Brain Hemorrhage, Traumatic - metabolism | Endothelial Cells - metabolism | Brain - physiopathology | Rats | Potassium Channels, Inwardly Rectifying - genetics | Down-Regulation - genetics | Receptors, Drug - metabolism | Blood-Brain Barrier - metabolism | Hypoglycemic Agents - pharmacology | Down-Regulation - physiology | Blood-Brain Barrier - pathology | Animals | Brain - pathology | Brain Injuries - pathology | Potassium Channels, Inwardly Rectifying - metabolism | Receptors, Drug - antagonists & inhibitors | ATP-Binding Cassette Transporters - antagonists & inhibitors | Brain | Antisense nucleic acids | Usage | Care and treatment | Development and progression | Research | Health aspects | Injuries | Risk factors | Neurons | Brain damage | Index Medicus | Original
Journal Article
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 09/2005, Volume 25, Issue 9, pp. 1111 - 1118
Delayed administration of vascular endothelial growth factor (VEGF) promotes functional recovery after focal cerebral ischemia. However, early intravenous... 
Focal cerebral ischemia | Neuroprotection | Neuronal apoptosis | VEGF (vascular endothelial growth factor) | REDUCES INFARCT SIZE | ANGIOGENESIS | NITRIC-OXIDE SYNTHASE | NEUROTROPHIC FACTOR | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | neuronal apoptosis | ADULT-RAT BRAIN | neuroprotection | CEREBRAL-ARTERY OCCLUSION | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | TEMPORAL PROFILE | focal cerebral ischemia | HEMATOLOGY | ENDOTHELIAL GROWTH-FACTOR | Immunohistochemistry | Neuroprotective Agents | Cerebrovascular Circulation - physiology | Injections, Intraventricular | Oncogene Protein v-akt | Middle Cerebral Artery - physiology | Vascular Endothelial Growth Factor A - administration & dosage | Vascular Endothelial Growth Factor A - pharmacology | Cerebral Infarction - pathology | Nervous System Diseases - pathology | In Situ Nick-End Labeling | Reperfusion Injury - pathology | Cerebrovascular Circulation - drug effects | Brain Ischemia - physiopathology | Brain Ischemia - prevention & control | Blood-Brain Barrier - drug effects | Blotting, Western | Nervous System Diseases - prevention & control | Animals | Reperfusion Injury - prevention & control | Signal Transduction - drug effects | Brain Ischemia - pathology | Nervous System Diseases - etiology | Hemodynamics - drug effects | Mice | Retroviridae Proteins, Oncogenic - physiology | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2009, Volume 29, Issue 10, pp. 3019 - 3025
Journal Article
Journal of Neurotrauma, ISSN 0897-7151, 10/2011, Volume 28, Issue 10, pp. 2171 - 2183
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2016, Volume 11, Issue 12, pp. e0167359 - e0167359
Therapeutic hypercapnia has the potential for neuroprotection after global cerebral ischemia. Here we further investigated the effects of different degrees of... 
FOCAL CEREBRAL ISCHEMIA/REPERFUSION | THERAPEUTIC HYPERCAPNIA | HEAD-INJURY | ADULT RATS | BARRIER PERMEABILITY | EDEMA | MULTIDISCIPLINARY SCIENCES | PROTEIN EXPRESSION | BLOOD-FLOW | PERINATAL-PERIOD | REOXYGENATION | Hypercapnia - physiopathology | Carbon Dioxide - metabolism | Carotid Artery, Common - physiopathology | Cerebrovascular Circulation - drug effects | Brain Ischemia - therapy | Humans | Blood Gas Analysis | Blood-Brain Barrier - physiopathology | Carbon Dioxide - therapeutic use | Rats | Brain Ischemia - physiopathology | Brain Injuries - therapy | Brain Injuries - physiopathology | Blood-Brain Barrier - drug effects | Animals | Brain Edema - chemically induced | Ligation | Brain Edema - physiopathology | Carotid Artery, Common - drug effects | Aquaporin 4 - genetics | Hypoxia - physiopathology | Brain Edema - blood | Disease Models, Animal | Hypercapnia | Hypoxia | Brain damage | Analysis | Risk factors | Aquaporin 4 | Brain | Neuroprotection | Flow cytometry | Traumatic brain injury | Carbon dioxide | Kinases | Blood | Western blotting | Proteins | Hypoxemia | Ischemia | Education | Cerebral blood flow | Rodents | Blood pressure | Carotid artery | Edema | Damage assessment | Neurons | Aquaporins | Cortex | Biotin | Permeability | Metabolism | Blood flow | DNA nucleotidylexotransferase | Cytometry | Ostomy | Brain research | Ventilation | Anesthesiology | Laboratory animals | Immunofluorescence | Brain injury | Hippocampus | Apoptosis | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, pp. e61988 - e61988
The neurodegenerative processes that underlie Alzheimer's disease are mediated, in part, by soluble oligomeric amyloid beta, a neurotoxic protein that inhibits... 
SPHINGOSINE 1-PHOSPHATE RECEPTORS | MULTIPLE-SCLEROSIS | MESSENGER-RNA | ADULT-RAT | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | CENTRAL-NERVOUS-SYSTEM | SPINAL-CORD-INJURY | AXONAL-TRANSPORT | FULL-LENGTH | SYNAPTIC PLASTICITY | Neurons - pathology | Amyloid beta-Peptides - toxicity | Humans | Neurotoxins - toxicity | Extracellular Signal-Regulated MAP Kinases - metabolism | Organophosphates - pharmacology | Sphingosine - pharmacology | Sphingosine - analogs & derivatives | Animals | MAP Kinase Signaling System - drug effects | Neuroprotective Agents - pharmacology | Neurons - enzymology | Cytoprotection - drug effects | Brain-Derived Neurotrophic Factor - biosynthesis | Brain-Derived Neurotrophic Factor - metabolism | Mice | Neurons - metabolism | Neurons - drug effects | Receptors, Lysosphingolipid - metabolism | Receptor, trkB - metabolism | Protein Multimerization - drug effects | Potentiation | Phosphates | Brain | Neuroprotection | Reactive oxygen species | Multiple sclerosis | Memory | Nervous system | Kinases | Sclerosis | Sphingosine 1-phosphate | Proteins | Neurotoxicity | Lymphocytes | Rodents | Localization | Alzheimer's disease | Medical research | Neurodegenerative diseases | Neurons | Long-term potentiation | Cortex | Environmental health | Extracellular signal-regulated kinase | Pharmacology | Plasticity (hippocampal) | Gene expression | Chemical compounds | Neurological diseases | Medicine | Signaling | Brain research | Brain-derived neurotrophic factor | Plasticity (synaptic) | Inhibitors | FTY720 | TrkB receptors | Alzheimers disease | Hippocampus | Index Medicus
Journal Article
Journal Article
STEM CELLS, ISSN 1066-5099, 06/2012, Volume 30, Issue 6, pp. 1120 - 1133
Journal Article