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Neurobiology of Disease, ISSN 0969-9961, 2013, Volume 58, pp. 102 - 114
Abstract Toll-like receptor 4 (TLR4) activation in neuron and astrocytes by High Mobility Group Box 1 (HMGB1) protein is a key mechanism of seizure generation.... 
Neurology | Cell loss | Alarmin | Inflammation | Damage associated molecular pattern | Glia | Neurogenesis | Toll-like receptor | DAMP | Seizures | GRANULE CELL DISPERSION | RAGE | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | NEURITE OUTGROWTH | MOUSE MODEL | MOBILITY GROUP BOX-1 | DENTATE GYRUS | END-PRODUCTS | HIPPOCAMPAL NEUROGENESIS | Electric Stimulation - adverse effects | Microtubule-Associated Proteins - metabolism | Humans | Male | Seizures - metabolism | Electroencephalography | Hippocampus - drug effects | Seizures - chemically induced | Cell Death - genetics | Toll-Like Receptor 4 - deficiency | HMGB1 Protein - metabolism | Up-Regulation - physiology | Cell Death - drug effects | Kainic Acid - toxicity | Epilepsy, Temporal Lobe - pathology | Receptor for Advanced Glycation End Products | Disease Models, Animal | HMGB1 Protein - administration & dosage | Epilepsy, Temporal Lobe - chemically induced | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Receptors, Immunologic - deficiency | Mice, Transgenic | Neuropeptides - metabolism | Up-Regulation - genetics | Epilepsy, Temporal Lobe - etiology | Epilepsy, Temporal Lobe - metabolism | Gene Expression Regulation - drug effects | Animals | Excitatory Amino Acid Agonists - toxicity | Seizures - etiology | Mice | Hippocampus - physiology | Receptors, Immunologic - metabolism | Temporal lobe epilepsy | Chromosomal proteins | Seizures (Medicine) | Glutamate | Analysis | Index Medicus | Advanced glycosylation end products
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2012, Volume 109, Issue 39, pp. 15888 - 15893
The epidemics of insulin resistance (IR) and type 2 diabetes (T2D) affect the first world as well as less-developed countries, and now affect children as well.... 
Receptors | 3T3 L1 cells | Myasthenia gravis | Diet | Insulin resistance | Mice | Adipocytes | Type 2 diabetes mellitus | Diabetes | Insulin | Aging | Glycotoxins | Obesity | LIFE-SPAN | OXIDATIVE STRESS | METABOLIC SYNDROME | OXIDANT STRESS | MULTIDISCIPLINARY SCIENCES | DOWN-REGULATION | RISK-FACTOR | glycotoxins | MESANGIAL CELLS | aging | INFLAMMATORY RESPONSE | END-PRODUCTS | obesity | METHYLGLYOXAL | Sirtuin 1 - metabolism | Deoxyglucose - metabolism | Diabetes Mellitus, Type 2 - genetics | Humans | Diabetes Mellitus, Experimental - genetics | Metabolic Syndrome - metabolism | Diabetes Mellitus, Type 2 - metabolism | Insulin Receptor Substrate Proteins - metabolism | Metabolic Syndrome - chemically induced | Sirtuin 1 - genetics | Inflammation - drug therapy | Glycation End Products, Advanced - adverse effects | Diabetes Mellitus, Experimental - metabolism | Insulin Receptor Substrate Proteins - genetics | Receptor for Advanced Glycation End Products | Macrophages - pathology | Administration, Oral | Oxidative Stress - genetics | 3T3-L1 Cells | Adipocytes - pathology | Proto-Oncogene Proteins c-akt | Macrophages - metabolism | Animals | Glycation End Products, Advanced - pharmacology | Metabolic Syndrome - genetics | Adipocytes - metabolism | Deoxyglucose - genetics | Diabetes Mellitus, Experimental - pathology | Inflammation - genetics | Diabetes Mellitus, Type 2 - pathology | Metabolic Syndrome - pathology | Oxidative Stress - drug effects | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | Type 2 diabetes | Antioxidants | Cell receptors | Physiological aspects | Glycosylation | Properties | Proteins | Oxidative stress | Genotype & phenotype | Epidemics | Index Medicus | Biological Sciences
Journal Article
Journal Article
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 2019, Volume 10, pp. 855 - 855
Multiple sclerosis (MS) is a demyelinating autoimmune disease in which innate and adaptive immune cells infiltrate the central nervous system (CNS) and damage... 
Neuroinflammation | Advanced glycation endproducts | Multiple sclerosis | α-dicarbonyl | Astrocytes | OXIDATION | BIOMARKER | multiple sclerosis | RECEPTOR | MECHANISMS | IMMUNOLOGY | advanced glycation endproducts | CEREBROSPINAL-FLUID | INDIVIDUALS | neuroinflammation | DISEASE | GENE-EXPRESSION | END-PRODUCTS AGES | CHOROID-PLEXUS | alpha-dicarbonyl | astrocytes
Journal Article
Journal Article