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Neuroscience, ISSN 0306-4522, 2017, Volume 357, pp. 84 - 98
Highlights • M1 receptor expression is increased in the dorsomedial striatum of dtsz hamsters. • M2/M4 receptor binding is altered in limbic structures of dtsz... 
Neurology | muscarinic | basal ganglia | acetylcholine | movement disorders | striatum | PRIMARY PAROXYSMAL DYSTONIA | PARVALBUMIN-POSITIVE INTERNEURONS | PROJECTION NEURONS | SUBTYPE-SPECIFIC ANTIBODIES | NEUROSCIENCES | MUTANT HAMSTERS | disorders | AGE-RELATED-CHANGES | GABAERGIC INTERNEURONS | STRIATAL CHOLINERGIC INTERNEURONS | ANIMAL-MODEL | movement | RODENT MODEL | Limbic System - pathology | Dystonia - pathology | Male | Receptor, Muscarinic M1 - antagonists & inhibitors | Dystonia - metabolism | Limbic System - metabolism | Thiophenes - administration & dosage | Corpus Striatum - metabolism | Dose-Response Relationship, Drug | Mesocricetus | Female | Anti-Dyskinesia Agents - administration & dosage | Drug Therapy, Combination | Corpus Striatum - pathology | Disease Models, Animal | Limbic System - drug effects | Severity of Illness Index | Pyridines - administration & dosage | Receptor, Muscarinic M4 - metabolism | Dystonia - drug therapy | Disease Progression | Muscarinic Antagonists - administration & dosage | Animals | Corpus Striatum - drug effects | Tropicamide - administration & dosage | Mutation | Receptor, Muscarinic M4 - antagonists & inhibitors | Trihexyphenidyl - administration & dosage | Receptor, Muscarinic M1 - metabolism | Hamsters | Analysis | Parasympatholytic agents | Movement disorders | Index Medicus
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2005, Volume 102, Issue 26, pp. 9194 - 9199
Loss of immune function and an increased incidence of myeloid leukemia are two of the most clinically significant consequences of aging of the hematopoietic... 
T lymphocytes | Up regulation | Biological Sciences | B lymphocytes | Genes | Stem cells | Mice | Transplantation | Progenitor cells | Hematopoietic stem cells | Cells | Lineage potential | Ontogeny | Leukemia microarray | leukemia | CAPACITY | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | IN-VIVO PROLIFERATION | IDENTIFICATION | ontogeny | LINEAGE COMMITMENT | microarray | AGE-RELATED-CHANGES | CALORIC RESTRICTION | lineage potential | MICE | GENE-EXPRESSION PROFILE | TRANSCRIPTIONAL PROFILE | Up-Regulation | Oligonucleotide Array Sequence Analysis | Cell Separation | Down-Regulation | Mice, Inbred C57BL | Gene Expression Regulation | Cell Transplantation | Leukemia - metabolism | Stem Cells - cytology | Reverse Transcriptase Polymerase Chain Reaction | Stem Cells - metabolism | Cell Lineage | Animals | Flow Cytometry | Cell Transformation, Neoplastic | Cellular Senescence | Hematopoiesis | Hematopoietic Stem Cells - cytology | Software | Genome | Myelocytic leukemia | Causes of | Aging | Nonlymphoid leukemia | Research | Leukemia | Immune system | Index Medicus | Neurologi | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Immunologi inom det medicinska området | Klinisk medicin | Clinical Medicine | Neurology | Medicinska och farmaceutiska grundvetenskaper | Immunology in the medical area
Journal Article
Neurobiology of Aging, ISSN 0197-4580, 2014, Volume 35, Issue 6, pp. 1325 - 1333
Journal Article
Behavioural Brain Research, ISSN 0166-4328, 07/2016, Volume 308, pp. 187 - 195
Senescence-accelerated mouse (SAM) is inbred lines of mice originally developed from AKR/J mice. Among the six SAM prone (SAMP) substrains, 8- to 12-month-old... 
Learning and memory | Senescence-accelerated mouse (SAM) | Animal model of dementia | OXIDATIVE STRESS | LONG-TERM-MEMORY | CIRCADIAN-RHYTHM | ALZHEIMERS-DISEASE | NEUROSCIENCES | OBJECT EXPLORATION | SUPRACHIASMATIC NUCLEUS | AGE-RELATED-CHANGES | WHEEL-RUNNING ACTIVITY | MITOCHONDRIAL DYSFUNCTION | BEHAVIORAL SCIENCES | CEREBRAL-BLOOD-FLOW | Memory | Analysis | Batteries
Journal Article
JOURNALS OF GERONTOLOGY SERIES A-BIOLOGICAL SCIENCES AND MEDICAL SCIENCES, ISSN 1079-5006, 06/2019, Volume 74, Issue 6, pp. 860 - 871
Journal Article
Journal Article
Journal Article
Neurobiology of Aging, ISSN 0197-4580, 2015, Volume 36, Issue 12, pp. 3321 - 3333
Journal Article