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Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 09/2015, Volume 136, Issue 3, pp. 769 - 780
.... Methods Mice were exposed to ozone, and lung inflammation, airway hyperresponsiveness (AHR), and mitochondrial function were determined... 
Allergy and Immunology | airway hyperresponsiveness | antioxidant | proliferation | MitoQ | inflammation | mitochondria | airway smooth muscle | Ozone | oxidative stress | chronic obstructive pulmonary disease | Allergy | Immunology | Life Sciences & Biomedicine | Science & Technology | Respiratory System - pathology | Reactive Oxygen Species - metabolism | Humans | Middle Aged | Male | Pulmonary Disease, Chronic Obstructive - pathology | Bronchial Hyperreactivity - genetics | Ubiquinone - pharmacology | Muscle, Smooth - drug effects | Smoking - physiopathology | Organophosphorus Compounds - pharmacology | Myocytes, Smooth Muscle - drug effects | Pneumonia - genetics | Pulmonary Disease, Chronic Obstructive - genetics | Bronchial Hyperreactivity - chemically induced | Signal Transduction | Mitochondria - pathology | Smoking - metabolism | Bronchial Hyperreactivity - pathology | Reactive Oxygen Species - antagonists & inhibitors | Electron Transport Chain Complex Proteins - metabolism | Pulmonary Disease, Chronic Obstructive - chemically induced | Respiratory System - drug effects | Airway Remodeling - genetics | Mice | Oxidative Stress - drug effects | Bronchial Hyperreactivity - drug therapy | Myocytes, Smooth Muscle - pathology | Pneumonia - pathology | Membrane Potential, Mitochondrial - drug effects | Pneumonia - chemically induced | Adult | Female | Pulmonary Disease, Chronic Obstructive - metabolism | Myocytes, Smooth Muscle - metabolism | Ubiquinone - analogs & derivatives | Gene Expression Regulation | Hydrogen Peroxide - pharmacology | Electron Transport Chain Complex Proteins - genetics | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - drug effects | Muscle, Smooth - metabolism | Animals | Pneumonia - drug therapy | Respiratory System - metabolism | Aged | Muscle, Smooth - pathology | Oxidative stress | Care and treatment | Lung diseases, Obstructive | Inflammation | Pharmaceutical industry | Biomedical research | Disease | Laboratories | Mortality | Colleges & universities | Smooth muscle | Mitochondrial DNA | Metabolism | Defects | Antioxidants | Mitochondria | Hospitals | Biopsy | Chronic obstructive pulmonary disease | Apoptosis | Smoking | Index Medicus | Abridged Index Medicus | NO, Nitric oxide | ΔΨm, Mitochondrial membrane potential | ASM, Airway smooth muscle | NAC, N-acetylcysteine | RL, Lung resistance | BAL, Bronchoalveolar lavage | OCR, Oxygen consumption rate | dTPP, Decyltriphenylphosphonium bromide | AHR, Airway hyperresponsiveness | logPC100, Concentration of acetylcholine that increased lung resistance by 100 | GOLD, Global Initiative for Chronic Obstructive Lung Disease | ATP, Adenosine triphosphate | KC, Keratinocyte-derived cytokine | JC-1, 5,5′,6,6′-Tetrachloro-1,1′,3,3′-tetraethylbenzimidazolylcarbocyanine iodide | COPD, Chronic obstructive pulmonary disease | ROS, Reactive oxygen species | Mechanisms of Allergy and Clinical Immunology
Journal Article
International journal of chronic obstructive pulmonary disease, ISSN 1178-2005, 04/2018, Volume 13, pp. 1135 - 1144
Background: Airway hyperresponsiveness (AHR) is associated with airway inflammation and a rapid decline in lung function and is a predictor of future risk of COPD among smokers... 
Matrix metalloproteinase-9 | P38 mitogen-activated protein kinase | Alveolar macrophage | Extracellular signal-regulated kinase | Tissue inhibitor of metalloproteinase-1 | Airway hyperresponsiveness | Smoking | Life Sciences & Biomedicine | Respiratory System | Science & Technology | Smoking - adverse effects | Spirometry | Humans | Middle Aged | Bronchial Provocation Tests | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Male | Pulmonary Disease, Chronic Obstructive - physiopathology | Extracellular Signal-Regulated MAP Kinases - metabolism | Pulmonary Disease, Chronic Obstructive - etiology | Lung - enzymology | Forced Expiratory Volume | Young Adult | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Matrix Metalloproteinase 9 - metabolism | Time Factors | Smoking - physiopathology | Respiratory Hypersensitivity - physiopathology | Adult | Female | Respiratory Hypersensitivity - enzymology | p38 Mitogen-Activated Protein Kinases - metabolism | Biomarkers - metabolism | Smokers | Smoking - blood | Respiratory Hypersensitivity - etiology | Cells, Cultured | Lung - physiopathology | Disease Progression | Macrophages, Alveolar - enzymology | Pulmonary Disease, Chronic Obstructive - enzymology | Vital Capacity | Macrophages, Alveolar - drug effects | Lung - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Aged | Protein Kinase Inhibitors - pharmacology | Lung diseases, Obstructive | Mitogens | Protein kinases | Enzyme-linked immunosorbent assay | Index Medicus | matrix metalloproteinase-9 (MMP)-9 | airway hyperresponsiveness (AHR) | p38 mitogen-activated protein kinase (MAPK) | tissue inhibitor of metalloproteinase-1 (TIMP-1) | extracellular signal-regulated kinase (ERK) | alveolar macrophage (AM)
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 04/2006, Volume 34, Issue 4, pp. 426 - 433
Journal Article
Journal Article