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Annals of Dermatology, ISSN 1013-9087, 11/2013, Volume 25, Issue 4, pp. 423 - 427
Journal Article
by Cho, MK
ANNALS OF DERMATOLOGY, ISSN 1013-9087, 11/2013, Volume 25, Issue 4, pp. 423 - 427
Background: Diabetes is characterized by chronic hyperglycemia, and hyperglycemia can increase reactive oxygen species (ROS) production from the mitochondrial... 
Diabetic skin | OXIDATIVE STRESS | AKR1C3 | DAMAGE | DERMATOLOGY | Type 5 17 beta-hydroxysteroid dehydrogenase | KETO REDUCTASE SUPERFAMILY | AMINO-ACIDS | BIOCHEMISTRY | GLUTAMINE-SYNTHETASE | CONSEQUENCES | COMPLICATIONS | RADICALS | RESIDUES | Type 5 17 β-hydroxysteroid dehydrogenase | Original
Journal Article
Annals of Dermatology, ISSN 1013-9087, 2013, Volume 25, Issue 4, p. 423
Background: Diabetes is characterized by chronic hyperglycemia, and hyperglycemia can increase reactive oxygen species (ROS) production from the mitochondrial... 
Diabetic skin | AKR1C3 | Type 5 17 β-hydroxysteroid dehydrogenase
Journal Article
Annals of Dermatology, ISSN 1013-9087, 11/2013, Volume 25, Issue 4, pp. 423 - 427
Diabetes is characterized by chronic hyperglycemia, and hyperglycemia can increase reactive oxygen species (ROS) production from the mitochondrial electron... 
Diabetic skin | Type 5 17 β-hydroxysteroid dehydrogenase | AKR1C3
Journal Article
프로그램북(구 초록집), 2013, Volume 65, Issue 2, p. 305
Background: Diabetes is characterized by chronic hyperglycemia, and hyperglycemia could Increase Reactive Oxygen Species (ROS) production from the... 
AKR1C3 | Aldo-keto reductases | Diabetic Skin
Journal Article
Chemico-Biological Interactions, ISSN 0009-2797, 06/2016, Volume 253, pp. 60 - 65
Journal Article
Biomedicine & Pharmacotherapy, ISSN 0753-3322, 2015, Volume 69, pp. 317 - 325
Abstract Aldo-keto reductase 1C3 (AKR1C3), one member of the aldo-keto reductase superfamily, is involved in a variety of cancers. Recently, AKR1C3 has been... 
Internal Medicine | Medical Education | AKR1C3 | PTEN/Akt | Human breast cancer | DOX resistance | PTEN loss | MEDICINE, RESEARCH & EXPERIMENTAL | STEROID-HORMONE | 5 17-BETA-HYDROXYSTEROID-DEHYDROGENASE AKR1C3 | PROSTATE-CANCER | REVEALS ROLES | PROGNOSTIC MARKER | PHARMACOLOGY & PHARMACY | HUMAN LIVER | MOLECULAR-CLONING | ANTICANCER DRUGS | ALPHA-HYDROXYSTEROID DEHYDROGENASE | IN-SITU | Doxorubicin - therapeutic use | Apoptosis - drug effects | Humans | RNA, Messenger - metabolism | Hydroxyprostaglandin Dehydrogenases - metabolism | Breast Neoplasms - enzymology | Transfection | MCF-7 Cells | 3-Hydroxysteroid Dehydrogenases - metabolism | Female | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Cell Survival - drug effects | Green Fluorescent Proteins - metabolism | RNA, Messenger - genetics | Morpholines - pharmacology | PTEN Phosphohydrolase - metabolism | Enzyme Activation - drug effects | Breast Neoplasms - drug therapy | Blotting, Western | Hydroxyprostaglandin Dehydrogenases - genetics | Breast Neoplasms - genetics | Signal Transduction - drug effects | Aldo-Keto Reductase Family 1 Member C3 | Breast Neoplasms - pathology | Protein Kinase Inhibitors - pharmacology | 3-Hydroxysteroid Dehydrogenases - genetics | Doxorubicin - pharmacology | Drug Resistance, Neoplasm - drug effects | Anthracyclines | Breast cancer | Index Medicus
Journal Article
The Prostate, ISSN 0270-4137, 05/2017, Volume 77, Issue 6, pp. 625 - 638
Journal Article
Journal Article
Journal Article
Oncology Letters, ISSN 1792-1074, 03/2018, Volume 15, Issue 3, pp. 3167 - 3172
Statins have become of interest in research due to their anticancer effects. However, the exact mechanism of their anticancer properties remains unclear. The... 
AKR1C3 | Meclofenamic acid | Prostate cancer | Statins | ANDROGEN RECEPTOR | prostate cancer | LNCAP | ONCOLOGY | statins | ENZYMES | EXPRESSION | meclofenamic acid | LINES | ATORVASTATIN
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 08/2014, Volume 278, Issue 3, pp. 238 - 248
Pharmacokinetic drug resistance is a serious obstacle that emerges during cancer chemotherapy. In this study, we investigated the possible role of aldo-keto... 
Anthracyclines | Aldo-keto reductase 1C3 | Drug resistance | Metabolism | Enzyme induction | DOXORUBICIN | TUMOR-CELLS | INDUCTION | STEROID-HORMONE | DAUNORUBICIN | ENDOMETRIAL CANCER | BREAST-CANCER | 5 17-BETA-HYDROXYSTEROID-DEHYDROGENASE AKR1C3 | PROSTATE-CANCER | PHARMACOLOGY & PHARMACY | TOXICOLOGY | MCF-7 CELLS | Antibiotics, Antineoplastic - pharmacology | Humans | Daunorubicin - agonists | Daunorubicin - pharmacology | Idarubicin - pharmacology | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - metabolism | Anthracyclines - agonists | Hydroxyprostaglandin Dehydrogenases - metabolism | 3-Hydroxysteroid Dehydrogenases - antagonists & inhibitors | Anthracyclines - pharmacology | Biotransformation | Anthracyclines - metabolism | Enzyme Induction - drug effects | 3-Hydroxysteroid Dehydrogenases - metabolism | Doxorubicin - metabolism | Daunorubicin - metabolism | Neoplasm Proteins - genetics | Antibiotics, Antineoplastic - agonists | Carcinoma - drug therapy | Idarubicin - metabolism | Recombinant Proteins - metabolism | Cell Survival - drug effects | Oxidation-Reduction | Flavanones - pharmacology | Enzyme Inhibitors - pharmacology | Recombinant Proteins - chemistry | Hydroxyprostaglandin Dehydrogenases - genetics | Drug Synergism | Hydroxyprostaglandin Dehydrogenases - antagonists & inhibitors | Aldo-Keto Reductase Family 1 Member C3 | Cell Line, Tumor | Kinetics | 3-Hydroxysteroid Dehydrogenases - genetics | Antibiotics, Antineoplastic - metabolism | Idarubicin - agonists | Doxorubicin - pharmacology | Drug Resistance, Neoplasm - drug effects | Physiological aspects | Chemotherapy | Cancer cells | Cancer | Index Medicus | CARBONYLS | METABOLISM | NEOPLASMS | ENZYMES | DRUGS | TOXICITY | INCUBATION | 60 APPLIED LIFE SCIENCES | ENZYME INDUCTION | CHEMOTHERAPY
Journal Article
Chemico-Biological Interactions, ISSN 0009-2797, 10/2017, Volume 276, pp. 194 - 202
Journal Article