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Journal Article
Journal Article
Nature Immunology, ISSN 1529-2908, 02/2013, Volume 14, Issue 2, pp. 162 - 171
Signaling through the G protein-coupled receptors for the complement fragments C3a and C5a (C3aR and C5aR, respectively) by dendritic cells and CD4(+) cells... 
Forkhead Transcription Factors - immunology | Cell Communication - immunology | T-Lymphocytes, Regulatory - metabolism | TOR Serine-Threonine Kinases - metabolism | Transforming Growth Factor beta1 - metabolism | Complement C5a - metabolism | Receptors, Complement - metabolism | T-Lymphocytes, Regulatory - immunology | Signal Transduction - immunology | Transforming Growth Factor beta1 - immunology | Forkhead Transcription Factors - metabolism | Class Ib Phosphatidylinositol 3-Kinase - metabolism | T-Lymphocytes, Regulatory - cytology | Interleukin-10 - metabolism | Receptor, Anaphylatoxin C5a - immunology | Cell Differentiation | Proto-Oncogene Proteins c-akt - metabolism | Complement C3a - metabolism | Interleukin-6 - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | Receptors, Chemokine - metabolism | Gene Expression Regulation | Mice, Transgenic | Receptors, Chemokine - immunology | Receptor Cross-Talk - immunology | TOR Serine-Threonine Kinases - immunology | Animals | Proto-Oncogene Proteins c-akt - immunology | Receptors, Complement - immunology | Cyclic AMP-Dependent Protein Kinases - immunology | Interleukin-6 - immunology | Mice | Complement C3a - immunology | Complement C5a - immunology | Class Ib Phosphatidylinositol 3-Kinase - immunology | Interleukin-10 - immunology | Receptor, Anaphylatoxin C5a - metabolism
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2015, Volume 10, Issue 5, p. e0126765
4-1BB (CD137), a member of the tumor necrosis factor receptor superfamily (TNFRSF), is primarily expressed on activated T cells and is known to enhance... 
RESPONSES IN-VIVO | INTERLEUKIN-2 | ACTIVATION | IMMUNE-RESPONSES | MULTIDISCIPLINARY SCIENCES | PROLIFERATION | IL-2 | INFECTION | COSTIMULATION | INDUCTION | PROGRESSION | Interleukin-2 - metabolism | Autocrine Communication | Gene Expression | Lymphocyte Activation | CD4-Positive T-Lymphocytes - metabolism | Receptors, Interleukin-2 - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Tumor Necrosis Factor Receptor Superfamily, Member 9 - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Mice, Knockout | CD4-Positive T-Lymphocytes - immunology | Receptors, Interleukin-2 - genetics | Animals | Signal Transduction - drug effects | CD8-Positive T-Lymphocytes - metabolism | Mice | Protein Kinase Inhibitors - pharmacology | CD8-Positive T-Lymphocytes - immunology | Proto-Oncogene Proteins c-akt - metabolism | CD27 antigen | Cell proliferation | CD8 antigen | Lymphocytes T | Biology | Kinases | T-cell receptor | Cell activation | Cell growth | Immunology | CD30 antigen | Interleukin 2 | Lymphocytes | Cell cycle | CD25 antigen | Tumor necrosis factor-TNF | Autocrine signalling | Expansion | Interleukin 2 receptors | Immunological memory | Memory cells | Population growth | CD137 antigen | Cytokines | CD4 antigen | Tumor necrosis factor | Cell death | Monoclonal antibodies | Laboratory animals | Viral infections | Cancer
Journal Article