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Journal of Cellular Biochemistry, ISSN 0730-2312, 08/2017, Volume 118, Issue 8, pp. 2271 - 2284
ABSTRACT A typical hallmark of diabetic kidney disease (DKD) is an excessive deposition of extracellular matrix (ECM) in the glomerulus and renal... 
DIABETIC MICE | EXTRACELLULAR MATRIX | SRC HOMOLOGY 2 DOMAIN‐CONTAINING INOSITOL 5’‐PHOSPHATASE | PHOSPHATIDYLINOSITOL 3‐KINASE/PROTEIN KINASE B | TRANSFORMING GROWTH FACTOR‐β1 | RENAL TUBULAR CELLS | TRANSFORMING GROWTH FACTOR-β1 | SRC HOMOLOGY 2 DOMAIN-CONTAINING INOSITOL 5’-PHOSPHATASE | PHOSPHATIDYLINOSITOL 3-KINASE/PROTEIN KINASE B | FIBROSIS | ACTIVATION | PROTEIN KINASE B | PHOSPHATIDYLINOSITOL 3-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSFORMING GROWTH FACTOR-1 | SRC HOMOLOGY 2 DOMAIN-CONTAINING INOSITOL 5'-PHOSPHATASE | DIABETIC-NEPHROPATHY | CELL BIOLOGY | TO-MESENCHYMAL TRANSITION | DISEASE | SHIP-1 | STRESS | EXPRESSION | PROGRESSION | TGF-BETA-1 | Epithelial Cells - metabolism | Cadherins - metabolism | Epithelial Cells - drug effects | Humans | Transforming Growth Factor beta1 - metabolism | Extracellular Matrix - metabolism | Inositol Polyphosphate 5-Phosphatases - genetics | Dioxoles - pharmacology | Benzamides - pharmacology | Cadherins - genetics | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Phosphatidylinositol 3-Kinase - metabolism | Phosphatidylinositol-3,4,5-Trisphosphate 5-Phosphatases - metabolism | Cell Line | Phosphatidylinositol-3,4,5-Trisphosphate 5-Phosphatases - genetics | Extracellular Matrix - drug effects | Inositol Polyphosphate 5-Phosphatases - metabolism | Morpholines - pharmacology | Glucose - pharmacology | Signal Transduction - genetics | Transforming Growth Factor beta1 - genetics | Kidney Tubules - cytology | Phosphatidylinositol 3-Kinase - genetics | Signal Transduction - drug effects | Proto-Oncogene Proteins c-akt - drug effects | Inositol | Phosphatases | Collagen | Bone morphogenetic proteins | Phospholipids | Kidney diseases | Glucose | Muscle proteins | Transforming growth factors | Dextrose | Surgical implants | Epithelial cells | Electrochemical machining | Smooth muscle | Homology | AKT protein | Activation | Biochemistry | Kinases | Phosphatase | E-cadherin | Signal transduction | Biomedical materials | Actin | Ships | Extracellular matrix | Attenuation | Inhibition | Deposition | Diabetes mellitus | Src protein | Inositol 5'-phosphatase | 1-Phosphatidylinositol 3-kinase | Inhibitors | Protein kinase | Fibrosis | Glomerulus | Mice | Diabetes | In vitro methods and tests | Clear cell-type renal cell carcinoma | Kidney transplantation
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 09/2018, Volume 146, Issue 6, pp. 686 - 702
Astrocytes are glial cells that help maintain brain homeostasis and become reactive in neurodegenerative processes releasing both harmful and beneficial... 
3‐nitropropionic acid | neuro protection | antioxidant action | astrocytes | BDNF | TrkB‐T1 signaling | TrkB-T1 signaling | 3-nitropropionic acid | INDUCED TOXICITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | RAT ASTROCYTES | MITOCHONDRIAL TOXIN | FACTOR EXPRESSION | NEUROSCIENCES | SIGNAL-TRANSDUCTION | NEUROPATHIC PAIN | SPINAL-CORD-INJURY | POSTNATAL-DEVELOPMENT | GROWTH-FACTOR | Reactive Oxygen Species - metabolism | Membrane Glycoproteins - metabolism | Rats, Wistar | Apoptosis - drug effects | Protein-Tyrosine Kinases - metabolism | Apoptosis - genetics | Receptor, trkB - genetics | PC12 Cells | Brain-Derived Neurotrophic Factor - pharmacology | Neuroprotective Agents - pharmacology | Benzamides - pharmacology | Astrocytes - drug effects | Gene Expression Regulation - genetics | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Membrane Glycoproteins - genetics | Azepines - pharmacology | Gene Expression Regulation - drug effects | Animals | Signal Transduction - drug effects | Indole Alkaloids - pharmacology | Carbazoles - pharmacology | Cell Cycle - drug effects | Receptor, trkB - metabolism | Culture Media, Serum-Free - toxicity | Astrocytes - metabolism | Prevention | Antioxidants | Tyrosine | Neurons | Analysis | Tumor proteins | Muscle proteins | Apoptosis | Neuroprotection | Brain | Reactive oxygen species | Calcium | p53 Protein | Homeostasis | AKT protein | Glial cells | Caspase-3 | Neuronal-glial interactions | Protein-tyrosine kinase receptors | Conditioning | Protein-tyrosine kinase | Glutathione | Deprivation | Tropomyosin | Astrocytes | Mortality | Extracellular signal-regulated kinase | Caspase | Brain-derived neurotrophic factor | Cell death | Pheochromocytoma cells | TrkB receptors | Viability
Journal Article
IUBMB Life, ISSN 1521-6543, 06/2018, Volume 70, Issue 6, pp. 519 - 528
Benign prostate hypertrophy (BPH) is among the most common diseases with a huge impact on the quality of life of elderly men. There is a current need for the... 
rats | IGF‐1R | Akt | testosterone | CAPE | benign prostate hypertrophy | IGF-1R | OXIDATIVE STRESS | CANCER CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROLIFERATION | BETA-CATENIN | HYPERPLASIA | CELL BIOLOGY | NF-KAPPA-B | EXPRESSION | PROGRESSION | GROWTH-FACTOR-I
Journal Article
Journal Article
Neuroscience, ISSN 0306-4522, 2011, Volume 183, pp. 212 - 220
Journal Article
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 11/2019, Volume 234, Issue 11, pp. 19911 - 19920
Journal Article
PLOS ONE, ISSN 1932-6203, 04/2019, Volume 14, Issue 4, p. e0215871
Tubule-interstitial injury (TII) is a critical step in the progression of renal disease. It has been proposed that changes in proximal tubule (PT) albumin... 
SURVIVAL | ALBUMIN ENDOCYTOSIS | SIGNALING PATHWAYS | INFLAMMATION | PKB | MULTIDISCIPLINARY SCIENCES | DEPHOSPHORYLATION | CUBILIN | MEGALIN | PROXIMAL TUBULE | RENAL INJURY | Epithelial Cells - metabolism | Mechanistic Target of Rapamycin Complex 2 - agonists | Apoptosis - drug effects | Epithelial Cells - drug effects | Humans | Male | Proto-Oncogene Proteins c-akt - genetics | Protective Agents - pharmacology | Low Density Lipoprotein Receptor-Related Protein-2 - genetics | Mechanistic Target of Rapamycin Complex 2 - genetics | Proto-Oncogene Proteins c-akt - metabolism | Cell Line | Macrophages - pathology | Albumins - metabolism | Endocytosis - drug effects | Gene Expression Regulation | Proteinuria - metabolism | Epithelial Cells - pathology | Kidney Tubules, Proximal - injuries | Low Density Lipoprotein Receptor-Related Protein-2 - metabolism | Proteinuria - physiopathology | Cell Movement - drug effects | Mechanistic Target of Rapamycin Complex 2 - metabolism | Macrophages - metabolism | Animals | Proteinuria - drug therapy | Signal Transduction - drug effects | Lithium Carbonate - pharmacology | Proto-Oncogene Proteins c-akt - agonists | Kidney Tubules, Proximal - metabolism | Macrophages - drug effects | Mice | Mice, Inbred BALB C | Kidney Tubules, Proximal - drug effects | Usage | Care and treatment | Chemical properties | Depression, Mental | Lithium compounds | Albumin | Kidney diseases | Protein kinases | Analysis | Collagen | Phosphorylation | Animal models | Lithium | AKT protein | Activation | Fluorides | Kinases | Macrophages | Proteins | Endocytosis | Chloride | Sodium | Rodents | Phenols | Infiltration | Injuries | Proteinuria | Apoptosis
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2018, Volume 13, Issue 2, p. e0193184
Journal Article
Journal Article