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Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 10/2018, Volume 142, Issue 4, pp. 1243 - 1256.e17
Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 08/2014, Volume 134, Issue 2, pp. 440 - 450.e3
...) cooperate with TLR4 to mediate HDM sensitization. Objectives To determine the requirement for HMGB1 and RAGE, and their relationship with TLR4, in airway sensitization. Methods TLR4−/− , RAGE−/− , and RAGE-TLR4... 
Allergy and Immunology | TSLP | allergic sensitization | epithelium | IL-25 | RAGE | HMGB1 | TLR4 | IL-1α | IL-33 | Asthma | Allergy | Immunology | Life Sciences & Biomedicine | Science & Technology | Inflammation - pathology | Interleukin-1alpha - immunology | Dermatophagoides pteronyssinus - immunology | Dendritic Cells - immunology | Dendritic Cells - pathology | Male | HMGB1 Protein - immunology | Adoptive Transfer | Respiratory Hypersensitivity - pathology | HMGB1 Protein - genetics | Allergens - administration & dosage | Interleukins - genetics | Allergens - immunology | Toll-Like Receptor 4 - deficiency | Interleukins - immunology | Interleukin-1alpha - genetics | Receptors, Immunologic - immunology | HMGB1 Protein - antagonists & inhibitors | Receptor for Advanced Glycation End Products | Lung - pathology | Signal Transduction | Interleukin-33 | Gene Expression Regulation | Receptors, Immunologic - deficiency | Toll-Like Receptor 4 - genetics | Antibodies, Neutralizing - pharmacology | Inflammation - immunology | Toll-Like Receptor 4 - immunology | Respiratory Hypersensitivity - immunology | Administration, Intranasal | Mice, Knockout | Respiratory Hypersensitivity - genetics | Animals | Lung - drug effects | Blattellidae - immunology | Inflammation - genetics | Mice | Receptors, Immunologic - genetics | Dendritic Cells - transplantation | Lung - immunology | Drugstores | Neurosciences | Pharmacy | Studies | Statistical analysis | Ligands | Allergies | Chemokines | Methods | Immune system | Index Medicus | Abridged Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, pp. e92710 - e92710
To evaluate the effect of airborne particulate matter 2.5 (PM2.5) in winter on airway inflammation, water-soluble supernatant (Sup... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Inflammation - chemically induced | Particulate Matter - toxicity | Inflammation - pathology | NLR Family, Pyrin Domain-Containing 3 Protein | Cell Count | Metals - analysis | Caspase 1 - metabolism | Male | Hypersensitivity - complications | RNA, Messenger - metabolism | Organic Chemicals - analysis | Inflammation - complications | Bronchoalveolar Lavage Fluid - cytology | Interleukin-1beta - secretion | Lung - pathology | Enzyme-Linked Immunosorbent Assay | Acetylcholine - pharmacology | RNA, Messenger - genetics | Administration, Intranasal | Bronchial Hyperreactivity - complications | Chemokines - genetics | Bronchial Hyperreactivity - pathology | Animals | Carrier Proteins - metabolism | Hypersensitivity - pathology | Lung - drug effects | Chemokines - metabolism | Mice | Particulate Matter - administration & dosage | Allergy | Allergens | Cytokines | Air pollution | Inflammation | Allergic reaction | Aluminum oxide | Aluminum | Lymphocytes T | Leucine | Macrophages | Respiratory system | Eotaxin | Caspase-1 | Proteins | Respiratory tract | Inoculation | Histopathology | Respiratory tract diseases | Pharmaceutical sciences | Public health | Particulate emissions | University graduates | Pathogens | Granulation | Airborne particulates | Particulates | Caspase | Interleukin 13 | Dentistry | Gene expression | Eotaxin 1 | Allergies | Asthma | Medicine | Sensitivity | Lungs | Particulate matter | Synergistic effect | Laboratory animals | Chemokines | Index Medicus
Journal Article
American journal of physiology. Lung cellular and molecular physiology, ISSN 1040-0605, 08/2014, Volume 307, Issue 3, pp. L240 - L251
Exposure to cigarette smoke (CS) is the main risk factor for developing chronic obstructive pulmonary disease and can induce airway epithelial cell damage, innate immune responses, and airway inflammation... 
APOPTOSIS | cell survival | BAD | GATEKEEPER | IMMUNOLOGY | mice | OBSTRUCTIVE PULMONARY-DISEASE | NECROSIS | INHIBITION | damage-associated molecular patterns | innate immune response | ALLERGIC SENSITIZATION | LUNG INFLAMMATION | chronic obstructive pulmonary disease | Chronic obstructive pulmonary disease | Mice | Cell survival | Damage-associated molecular patterns | Innate immune response | Respiratory System | Physiology | Life Sciences & Biomedicine | Science & Technology | Bronchoalveolar Lavage Fluid | Smoking - adverse effects | Inflammation - pathology | Lung - pathology | Epithelial Cells - metabolism | Cells, Cultured | Proto-Oncogene Proteins c-pim-1 - metabolism | Epithelial Cells - pathology | Pulmonary Disease, Chronic Obstructive - pathology | HSP70 Heat-Shock Proteins - metabolism | Smoking - metabolism | Inflammation - metabolism | Animals | Cell Death - physiology | Smoking - pathology | Membrane Potential, Mitochondrial - physiology | Female | Chemokines - metabolism | Lung - metabolism | Mice, Inbred BALB C | Pulmonary Disease, Chronic Obstructive - metabolism | Neutrophils - metabolism | Neutrophils - pathology | Lung diseases, Obstructive | Immune response | Epithelial cells | Physiological aspects | Physiological research | Inflammation | Research | Health aspects | Phosphotransferases | Risk factors | Smoking | Airway management | Cytokines | Rodents | Apoptosis | Index Medicus
Journal Article
Journal Article