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alpha-tropomyosin (503) 503
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PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, p. e61449
Rationale: Vasoactive Intestinal Peptide (VIP), a pulmonary vasodilator and inhibitor of vascular smooth muscle proliferation, is absent in pulmonary arteries... 
LACKING | FAMILIAL HYPERTROPHIC CARDIOMYOPATHY | PULMONARY ARTERIAL-HYPERTENSION | CARDIAC TROPONIN-T | MULTIDISCIPLINARY SCIENCES | MUTATION | SMOOTH-MUSCLE-CELLS | PROLIFERATION | DILATED CARDIOMYOPATHY | INTESTINAL-PEPTIDE GENE | ALPHA-TROPOMYOSIN | Up-Regulation | Oligonucleotide Array Sequence Analysis | Mice, Inbred C57BL | Heart Failure - physiopathology | Transcriptome | Vasoactive Intestinal Peptide - deficiency | Heart Failure - genetics | Male | Heart Failure - metabolism | Stroke Volume - genetics | Mice, Knockout | Cardiomyopathies - genetics | Cardiomyopathies - physiopathology | Phenotype | Animals | Cardiomyopathies - metabolism | Gene Deletion | Female | Mice | Vasoactive Intestinal Peptide - genetics | Heart failure | DNA microarrays | Cardiomyopathy | Vasodilators | Genes | Genomics | Genetic aspects | Heart diseases | Pulmonary hypertension | Nuclear magnetic resonance--NMR | Laboratories | Homeostasis | Smooth muscle | Genomes | Gene deletion | Kinases | Arteries | Proteins | Clonal deletion | Intestine | Vasoactive agents | Rodents | Electrocardiography | Deletion | Stretching | Deoxyribonucleic acid--DNA | Enlargement | Hypertension | Pulmonary arteries | Body temperature | Muscles | Wall thickness | Pulmonary artery | Heart rate | Magnetic resonance imaging | Lungs | Vasoactive intestinal peptide | Ventricle | Mutation | RADIATION CHEMISTRY, RADIOCHEMISTRY, AND NUCLEAR CHEMISTRY | WT (wild type) mice | MRI (magnetic resonance imaging) | VIP Gene Deletion in Mice Causes | Deoxyribonucleic acid | Nuclear magnetic resonance | NMR | DNA
Journal Article
Circulation Research, ISSN 0009-7330, 2013, Volume 112, Issue 11, pp. 1491 - 1505
Journal Article
Mayo Clinic Proceedings, ISSN 0025-6196, 2008, Volume 83, Issue 6, pp. 630 - 638
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2014, Volume 64, Issue 24, pp. 2589 - 2600
Abstract Background Mild hypertrophy but increased arrhythmic risk characterizes the stereotypic phenotype proposed for hypertrophic cardiomyopathy (HCM)... 
Cardiovascular | Internal Medicine | end-stage | diastolic function | troponin | triphasic filling | genotype to phenotype correlation | correlation triphasic filling troponin | genotype to phenotype | diastolic function end-stage | CARDIAC & CARDIOVASCULAR SYSTEMS | CARDIOVASCULAR MAGNETIC-RESONANCE | HEARTS BEARING | PREVALENCE | LEFT-VENTRICULAR HYPERTROPHY | TASK-FORCE | CARDIAC TROPONIN-T | DISEASE | DYSFUNCTION | ALPHA-TROPOMYOSIN | DOPPLER-ECHOCARDIOGRAPHY | Follow-Up Studies | Humans | Middle Aged | Male | Cardiomyopathy, Hypertrophic - complications | Death, Sudden, Cardiac - etiology | Ventricular Fibrillation - etiology | Ventricular Outflow Obstruction - etiology | Actins - genetics | Ventricular Dysfunction, Left - genetics | Ventricular Fibrillation - genetics | Adult | Female | Heart Function Tests | Actin Cytoskeleton - genetics | Severity of Illness Index | Troponin T - genetics | Ventricular Outflow Obstruction - genetics | Cardiomyopathy, Hypertrophic - genetics | Genetic Predisposition to Disease | MAP Kinase Kinase Kinases - genetics | Ventricular Dysfunction, Left - etiology | Patient Outcome Assessment | Cardiomyopathy, Hypertrophic - diagnosis | Disease Progression | Cardiomyopathy, Hypertrophic - physiopathology | Italy | Mutation | Sects | Genetic aspects | Cardiomyopathy, Hypertrophic | Gene mutations | Heart | Medical research | Tachycardia | Actin | Genes | Myosin | Medicine, Experimental | Protein binding | Heart attacks | Cardiology | Drug therapy | Clinical outcomes | MYH7, myosin heavy chain | TNNT2, cardiac troponin T gene | HR, hazard ratio | SCD, sudden cardiac death | ICD, implantable cardioverter-defibrillator | TPM1, cardiac α-tropomyosin gene | LVH, left ventricular hypertrophy | MYBPC3, myosin binding protein C | LV, left ventricular | ECG, electrocardiography | LGE, late gadolinium enhancement | AF, atrial fibrillation | NSVT, nonsustained ventricular tachycardia | ACTC, cardiac α-actin gene | Original Investigation | CMR, cardiac magnetic resonance | HCM, hypertrophic cardiomyopathy | TNNI3, cardiac troponin I gene | NYHA, New York Heart Association
Journal Article
Pediatric Research, ISSN 0031-3998, 2018, Volume 84, Issue 5, pp. 733 - 742
BACKGROUND: Left ventricular noncompaction (LVNC) is a primary cardiomyopathy with heterogeneous genetic origins. The aim of this study was to elucidate the... 
MYOCARDIUM | E180G MUTATION | HYPERTROPHIC CARDIOMYOPATHY | GENOMICS | PROTEIN GENES | CARDIAC TROPONIN-C | HEART-FAILURE | PEDIATRICS | PREVALENCE | ASSOCIATION | ALPHA-TROPOMYOSIN | Genes | Mutation
Journal Article
Journal Article
CIRCULATION JOURNAL, ISSN 1346-9843, 09/2019, Volume 83, Issue 9, pp. 1908 - 1908
Background: Hypertrophic cardiomyopathy (HCM) is associated primarily with pathogenic mutations in sarcomeric genes. The aim of this study was to identify the... 
BINDING-PROTEIN-C | CARDIAC & CARDIOVASCULAR SYSTEMS | Genetic mutations | GUIDELINES | PREVALENCE | IDENTIFICATION | Next-generation sequencing | MOLECULAR DIAGNOSIS | CARDIAC TROPONIN-T | DISEASE | Hypertrophic cardiomyopathy | SPECTRUM | JAPANESE PATIENTS | ALPHA-TROPOMYOSIN
Journal Article
AMERICAN JOURNAL OF MEDICAL GENETICS PART A, ISSN 1552-4825, 08/2016, Volume 170, Issue 8, pp. 2186 - 2190
Ebstein anomaly of the tricuspid valve (EA) can be associated with left ventricular non-compaction (LVNC), a rare congenital cardiomyopathy. We report a 2... 
MYOCARDIUM | heart failure | Ebstein anomaly | ACTIN | TPM1 | ADULTS | NONCOMPACTION CARDIOMYOPATHY | PREVALENCE | left ventricular noncompaction | CHILDREN | mutation | tropomyosin | genetics | genetic testing | DISEASE | GENETICS & HEREDITY | cardiomyopathy | BINDING | congenital heart defects | ALPHA-TROPOMYOSIN | MYH7 MUTATION | Heart failure | Heart | Genetic aspects | Gene mutations | Muscle proteins | Genes
Journal Article
BBA - Molecular Basis of Disease, ISSN 0925-4439, 08/2017, Volume 1863, Issue 8, pp. 2056 - 2063
Journal Article
Circulation Journal, ISSN 1346-9843, 2012, Volume 76, Issue 2, pp. 453 - 461
Journal Article