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Nature Neuroscience, ISSN 1097-6256, 05/2009, Volume 12, Issue 5, pp. 627 - 636
The mechanisms underlying disease manifestations in neurodegeneration remain unclear, but their understanding is critical to devising effective therapies. We... 
NEURODEGENERATIVE DISEASE | UNFOLDED PROTEIN RESPONSE | MOTOR-NEURON DEGENERATION | WILD-TYPE SOD1 | ALS-LINKED SOD1 | MOUSE MODEL | AMYOTROPHIC-LATERAL-SCLEROSIS | ENDOPLASMIC-RETICULUM STRESS | MUTANT SUPEROXIDE-DISMUTASE | NEUROSCIENCES | TRANSGENIC MICE | Microglia - metabolism | Central Nervous System - metabolism | Amyotrophic Lateral Sclerosis - physiopathology | Ubiquitin - metabolism | Central Nervous System - pathology | Endoplasmic Reticulum - metabolism | Gliosis - physiopathology | Thiourea - pharmacology | Mice, Neurologic Mutants | Motor Neurons - classification | Motor Neurons - pathology | Ubiquitination | Neuroprotective Agents - pharmacology | Disease Models, Animal | Wallerian Degeneration - genetics | Biomarkers - metabolism | Genetic Predisposition to Disease - genetics | Gliosis - genetics | Endoplasmic Reticulum - genetics | Wallerian Degeneration - metabolism | Amyotrophic Lateral Sclerosis - genetics | Gliosis - metabolism | Wallerian Degeneration - physiopathology | Biomarkers - analysis | Oxidative Stress - genetics | Cinnamates - pharmacology | Disease Progression | Protein Folding | Motor Neurons - metabolism | Phenotype | Animals | Amyotrophic Lateral Sclerosis - metabolism | Mice | Central Nervous System - physiopathology | Thiourea - analogs & derivatives | Motor neurons | Physiological aspects | Amyotrophic lateral sclerosis | Nervous system | Degeneration | Research | Endoplasmic reticulum
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