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Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 2015, Volume 308, Issue 8, pp. F878 - F887
Dipeptidyl peptidase (DPP)-4 is an enzyme that cleaves and inactivates incretin hormones capable of stimulating insulin secretion from pancreatic beta-cells.... 
Nondiabetic kidney disease | Glucagon-like peptide-1 | Dipeptidyl peptidase-4 | GLOMERULONEPHRITIS | ATTENUATES KIDNEY | PHYSIOLOGY | glucagon-like peptide-1 | BETA-CELL FUNCTION | ISCHEMIA-REPERFUSION INJURY | nondiabetic kidney disease | INDUCED DIABETIC-RATS | dipeptidyl peptidase-4 | KIDNEY INJURY | UROLOGY & NEPHROLOGY | AMELIORATES PROTEINURIA | RENAL INJURY | Dipeptidyl Peptidase 4 - metabolism | Glomerulonephritis - prevention & control | Male | Dipeptidyl-Peptidase IV Inhibitors - pharmacology | Antigens, CD - metabolism | Proteinuria - prevention & control | Uracil - pharmacology | Piperidines - pharmacology | Glomerulonephritis - enzymology | Macrophages, Peritoneal - drug effects | Cytoprotection | Disease Models, Animal | Kidney Glomerulus - enzymology | Receptors, Glucagon - agonists | Cell Line | Proteinuria - immunology | Anti-Inflammatory Agents - pharmacology | Kidney Glomerulus - drug effects | Mice, Inbred C57BL | Macrophages, Peritoneal - immunology | Receptors, Glucagon - metabolism | Glomerulonephritis - immunology | Chemotaxis - drug effects | Kidney Glomerulus - pathology | Pyrimidines - pharmacology | Rats, Sprague-Dawley | Peptides - pharmacology | Proteinuria - enzymology | Animals | Chemokine CCL2 - pharmacology | Glucagon-Like Peptide-1 Receptor | Signal Transduction - drug effects | Antigens, Differentiation, Myelomonocytic - metabolism | Glomerulonephritis - pathology | Antilymphocyte Serum | Venoms - pharmacology | Kidney Glomerulus - immunology | Uracil - analogs & derivatives | Care and treatment | Kidney glomerulus | Proteases | Health aspects | Diabetic nephropathies | Models | Glucagon | Anti-inflammatory drugs | Blood sugar | Analysis | Proteins | Enzymes | Signal transduction | Rodents | Physiology | Diabetes | Kidney diseases | Index Medicus
Journal Article
The Journal of Pathology, ISSN 0022-3417, 04/2013, Volume 229, Issue 5, pp. 672 - 684
Mesangioproliferative glomerulonephritis is the most common nephritis worldwide. We examined the effects of low‐ and high‐dose telmisartan, an angiotensin II... 
IgA | chemokine | platelet‐derived growth factor receptor | telmisartan | nephropathy | fibrosis | mesangioproliferative glomerulonephritis | renin–angiotensin system | chemokine receptor | IgA nephropathy | platelet-derived growth factor receptor | renin-angiotensin system | reninangiotensin system | TUBULOINTERSTITIAL INJURY | PDGF-D | ANTAGONISM | PATHOLOGY | MESENCHYMAL TRANSITION | ONCOLOGY | NEPHRITIS | AMELIORATES PROTEINURIA | GROWTH-FACTOR | EXPRESSION | RENAL INTERSTITIAL FIBROSIS | Antihypertensive Agents - pharmacology | Humans | Hypertension - drug therapy | Male | Gene Expression Profiling | RNA, Messenger - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Inflammation - metabolism | Benzimidazoles - administration & dosage | Nephrectomy | RNA Interference | Time Factors | Glomerular Mesangium - metabolism | Chemokine CCL20 - genetics | Glomerular Mesangium - drug effects | Glomerulonephritis, Membranoproliferative - metabolism | Angiotensin II Type 1 Receptor Blockers - administration & dosage | Cytoprotection | Disease Models, Animal | Glomerulonephritis, Membranoproliferative - pathology | Macrophages - pathology | Rats | Receptors, Platelet-Derived Growth Factor - genetics | Podocytes - pathology | Hypertension - metabolism | Proteinuria - physiopathology | Glomerulonephritis, Membranoproliferative - genetics | Macrophages - metabolism | Receptors, Platelet-Derived Growth Factor - metabolism | Fibrosis | Receptors, CCR6 - genetics | Benzoates - pharmacology | Benzimidazoles - pharmacology | Glomerular Mesangium - physiopathology | Mice | Rats, Wistar | Hydralazine - pharmacology | Antihypertensive Agents - administration & dosage | Isoantibodies | Glomerulonephritis, Membranoproliferative - drug therapy | Transfection | Hydrochlorothiazide - pharmacology | Inflammation - drug therapy | Receptors, CCR6 - metabolism | Blood Pressure - drug effects | Atenolol - pharmacology | Cell Line | Podocytes - metabolism | Cell Dedifferentiation - drug effects | Gene Expression Regulation | Proteinuria - metabolism | Hypertension - physiopathology | Benzoates - administration & dosage | Glomerular Mesangium - pathology | Animals | Proteinuria - drug therapy | Glomerulonephritis, Membranoproliferative - physiopathology | Podocytes - drug effects | Macrophages - drug effects | Receptors, Platelet-Derived Growth Factor - drug effects | Glomerulonephritis, Membranoproliferative - etiology | Inflammation - physiopathology | Index Medicus | Blood Pressure | Benzimidazoles | Genomics | Podocytes | Cell Dedifferentiation | Chemokine CCL20 | Macrophages | Life Sciences | Angiotensin II Type 1 Receptor Blockers | Receptors, Platelet-Derived Growth Factor | Glomerulonephritis, Membranoproliferative | Antihypertensive Agents | Hypertension | Benzoates | Biochemistry, Molecular Biology | Glomerular Mesangium | Inflammation | Hydrochlorothiazide | Atenolol | Receptors, CCR6 | Hydralazine | RNA, Messenger | Proteinuria
Journal Article
European Journal of Nutrition, ISSN 1436-6207, 12/2012, Volume 51, Issue 8, pp. 961 - 973
Diabetic glomerulosclerosis is the hardening of the renal glomeruli that can lead to kidney failure. In the early stage of glomerulosclerosis occur renal... 
Glomerulosclerosis | Purple corn anthocyanin | Chemistry | Nutrition | db/db mice | Renal fibrosis | Mesangial cells | High glucose | DB/DB mice | FIBROSIS | ACTIVATION | TGF-BETA | NEPHROPATHY | NUTRITION & DIETETICS | TISSUE GROWTH-FACTOR | AMELIORATES HYPERGLYCEMIA | ZEA-MAYS L | ACCUMULATION | BLOCKADE | PROGRESSION | Zea mays - chemistry | Fibrosis - drug therapy | Plant Extracts - pharmacology | Humans | STAT Transcription Factors - metabolism | TYK2 Kinase - metabolism | Diabetic Nephropathies - drug therapy | Male | Intracellular Signaling Peptides and Proteins - metabolism | Proteinuria - pathology | Platelet-Derived Growth Factor - genetics | Mesangial Cells - drug effects | Membrane Proteins - metabolism | Interleukin-8 - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Albuminuria - pathology | Collagen Type IV - metabolism | Anthocyanins - pharmacology | Diabetic Nephropathies - pathology | Blood Glucose - analysis | Signal Transduction | Membrane Proteins - genetics | Biomarkers - blood | Mesangial Cells - metabolism | Diabetic Nephropathies - complications | Platelet-Derived Growth Factor - metabolism | STAT Transcription Factors - genetics | Animals | Proteinuria - drug therapy | Transforming Growth Factor beta - genetics | Albuminuria - drug therapy | Collagen Type IV - genetics | Cell Proliferation - drug effects | Connective Tissue Growth Factor - genetics | Mice | Fibrosis - pathology | TYK2 Kinase - genetics | Proteinuria - complications | Transforming Growth Factor beta - metabolism | Connective Tissue Growth Factor - metabolism | Index Medicus
Journal Article
Kidney International, ISSN 0085-2538, 09/2012, Volume 82, Issue 5, pp. 537 - 547
β-Catenin is a unique intracellular protein functioning as an integral component of the cell–cell adherens complex and a principal signaling protein mediating... 
β-catenin | apoptosis | acute kidney injury | Wnt | beta-catenin | INTEGRIN-LINKED KINASE | PROAPOPTOTIC BAX | GAMMA-CATENIN | MECHANISMS | PODOCYTE INJURY | EPITHELIAL-CELLS | UROLOGY & NEPHROLOGY | AMELIORATES PROTEINURIA | UP-REGULATION | RENAL INTERSTITIAL FIBROSIS | Reperfusion Injury - etiology | Wnt1 Protein - genetics | Phosphorylation | Epithelial Cells - metabolism | Cadherins - metabolism | Inhibitor of Apoptosis Proteins - genetics | Humans | Acute Kidney Injury - genetics | Wnt1 Protein - metabolism | Adherens Junctions - metabolism | Transfection | Time Factors | Acute Kidney Injury - chemically induced | Inhibitor of Apoptosis Proteins - metabolism | Kidney Tubules - pathology | Kidney Tubules - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | Repressor Proteins - metabolism | Disease Models, Animal | Folic Acid | Cell Line | Reperfusion Injury - pathology | Acute Kidney Injury - pathology | Mice, Inbred C57BL | beta Catenin - deficiency | Tumor Suppressor Protein p53 - metabolism | bcl-2-Associated X Protein - metabolism | gamma Catenin - metabolism | Repressor Proteins - genetics | Genotype | Survivin | beta Catenin - metabolism | beta Catenin - genetics | Mice, Knockout | Phenotype | Animals | Creatinine - blood | Mice | Acute Kidney Injury - metabolism | Apoptosis | Index Medicus
Journal Article
by Zhang, J and Li, Y and Shan, K and Wang, LL and Qiu, W and Lu, YL and Zhao, D and Zhu, GQ and He, FX and Wang, YW
FASEB JOURNAL, ISSN 0892-6638, 03/2014, Volume 28, Issue 3, pp. 1511 - 1525
Journal Article
Molecular Medicine Reports, ISSN 1791-2997, 08/2016, Volume 14, Issue 2, pp. 1771 - 1784
Journal Article
Journal Article
Cellular Physiology and Biochemistry, ISSN 1015-8987, 10/2017, Volume 42, Issue 6, pp. 2441 - 2452
Journal Article
Journal Article