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1999, Current topics in microbiology and immunology, ISBN 3540647317, Volume 237., 189
Book
Scientific Reports, ISSN 2045-2322, 02/2016, Volume 6, Issue 1, pp. 19857 - 19857
Through both gain-and loss-of-TTF-1 expression strategies, we show that TTF-1 positively regulates vascular endothelial growth factor (VEGF) and that the VEGF... 
SURVIVAL | MORPHOGENESIS | EPITHELIAL-CELLS | GM-CSF | VEGF | MULTIDISCIPLINARY SCIENCES | LUNG ADENOCARCINOMA | GENE-EXPRESSION | NKX2-1 | CANCER | ENDOTHELIAL GROWTH-FACTOR | Exosomes - metabolism | Angiogenic Proteins - genetics | Neovascularization, Physiologic - drug effects | Thyroid Nuclear Factor 1 | TOR Serine-Threonine Kinases - metabolism | Humans | Lung Neoplasms - metabolism | Culture Media, Conditioned - pharmacology | Lung Neoplasms - pathology | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor Receptor-2 - immunology | Vascular Endothelial Growth Factor A - genetics | Cell Hypoxia | Transcription Factors - secretion | Nuclear Proteins - secretion | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | RNA Interference | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Cytokines - genetics | Real-Time Polymerase Chain Reaction | Lung Neoplasms - blood supply | A549 Cells | Promoter Regions, Genetic | Cytokines - metabolism | Enzyme-Linked Immunosorbent Assay | Cell Survival | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Indoles - toxicity | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Vascular Endothelial Growth Factor A - analysis | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Blotting, Western | Antibodies - pharmacology | Transcription Factors - metabolism | Up-Regulation - drug effects | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Pyrroles - toxicity | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Angiogenic Proteins - metabolism | RNA, Small Interfering - metabolism | TOR protein | Statistical analysis | Granulocyte-macrophage colony-stimulating factor | Lung | Lung cancer | Media | Rapamycin | Secretome | Exosomes | Macrophages | Angiogenesis | Hypoxia | Colony-stimulating factor | Vascular endothelial growth factor | Thyroid transcription factor 1 | Thyroid | Index Medicus
Journal Article
Journal Article
Journal Article
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 07/2010, Volume 56, Issue 5, pp. 414 - 422
Journal Article
Cancer Research, ISSN 0008-5472, 01/2008, Volume 68, Issue 2, pp. 521 - 529
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2014, Volume 34, Issue 3, pp. 635 - 643
OBJECTIVE—Vascular lineage differentiation of stem/progenitor cells can contribute to both tissue repair and exacerbation of vascular diseases such as in vein... 
Tumor necrosis factor-alpha | Vascular smooth muscle | Macrophages | Endothelial cells | Stem cells | PROGENITOR CELLS | stem cells | endothelial cells | vascular smooth muscle | ANGIOGENESIS | ATHEROSCLEROSIS | tumor necrosis factor-alpha | PATHOGENESIS | macrophages | EMBRYONIC STEM-CELLS | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | VEIN BYPASS GRAFTS | MICE | TNF-ALPHA | DIFFERENTIATION | HEMATOLOGY | Endothelium, Vascular - cytology | Neovascularization, Physiologic - drug effects | Antigens, CD - biosynthesis | Tumor Necrosis Factor-alpha - genetics | Veins - transplantation | Culture Media, Conditioned - pharmacology | NF-kappa B - metabolism | Tumor Necrosis Factor-alpha - deficiency | Antigens, CD - genetics | Cadherins - biosynthesis | Receptors, Tumor Necrosis Factor, Type I - drug effects | Thrombophilia - etiology | Tissue Scaffolds | RNA Interference | Nitric Oxide Synthase Type III - physiology | Cadherins - genetics | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - cytology | Angiogenic Proteins - pharmacology | Adult Stem Cells - drug effects | Gene Expression Regulation, Developmental - physiology | Cell Line | Promoter Regions, Genetic | Adult Stem Cells - cytology | Signal Transduction | Thrombophilia - physiopathology | Macrophages, Peritoneal - physiology | Mice, Inbred C57BL | RNA, Small Interfering - pharmacology | Gene Expression Regulation, Developmental - drug effects | Radiation Chimera | Recombinant Proteins - pharmacology | Macrophages, Peritoneal - secretion | Muscle, Smooth, Vascular - cytology | Mice, Knockout | Cell Lineage | Tumor Necrosis Factor-alpha - pharmacology | Animals | Interleukin-6 - pharmacology | Transcription Factor RelA - metabolism | Endothelial Cells - cytology | Receptors, Tumor Necrosis Factor, Type I - physiology | Neointima - pathology | Tumor Necrosis Factor-alpha - physiology | Mice | Apoptosis | Endothelial Cells - drug effects | Index Medicus
Journal Article
International Journal of Cancer, ISSN 0020-7136, 01/2013, Volume 132, Issue 1, pp. 29 - 41
Increased levels of hypoxia and hypoxia‐inducible factor 1α (HIF‐1α) in human sarcomas correlate with tumor progression and radiation resistance. Prolonged... 
hypoxia | HIF‐1α | sarcomas | VEGF‐A | HIF-1α | VEGF-A | ACTIVATION | HIF-1 alpha | METASTASIS | RANDOMIZED-TRIAL | TUMOR ANGIOGENESIS | CANCER-THERAPY | ONCOLOGY | MOUSE MODEL | ELECTRON-BEAM RADIOTHERAPY | ANTI-ANGIOGENIC THERAPY | UP-REGULATION | SOFT-TISSUE SARCOMA | Genetic Therapy | Cell Hypoxia - physiology | Apoptosis - drug effects | Carbonic Anhydrases - genetics | Humans | Apoptosis - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor A - genetics | Sarcoma - therapy | Bevacizumab | Carbonic Anhydrases - metabolism | DNA-Binding Proteins - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Antibodies, Monoclonal, Humanized - pharmacology | Antigens, Neoplasm - metabolism | Sarcoma - genetics | Carbonic Anhydrase IX | Antigens, Neoplasm - genetics | Cell Line | DNA-Binding Proteins - antagonists & inhibitors | Endothelial Cells - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Antibodies, Monoclonal - pharmacology | DNA-Binding Proteins - genetics | Sarcoma - pathology | Sarcoma - metabolism | Animals | Cell Line, Tumor | Mice | Endothelial Cells - pathology | Doxorubicin - pharmacology | Endothelial Cells - drug effects | Index Medicus | Cancer Cell Biology
Journal Article
Circulation Research: Journal of the American Heart Association, ISSN 0009-7330, 11/2003, Volume 93, Issue 11, pp. 1074 - 1081
ABSTRACT—Understanding molecular mechanisms regulating angiogenesis may lead to novel therapies for ischemic disorders. Hypoxia-inducible factor 1 (HIF-1)... 
Gene therapy | Angiogenesis | Hypoxia | FACTOR VEGF | hypoxia | HIF-1-ALPHA/VP16 HYBRID | CARDIAC & CARDIOVASCULAR SYSTEMS | angiogenesis | HYDROXYLATION | gene therapy | MODEL | RETINAL NEOVASCULARIZATION | FACTOR 1-ALPHA | IN-VIVO | HINDLIMB ISCHEMIA | PERIPHERAL VASCULAR DISEASE | MICE | HEMATOLOGY | HIF-ALPHA | Angiogenic Proteins - genetics | Neovascularization, Physiologic - drug effects | Cell Hypoxia - physiology | Eye - blood supply | Angiopoietin-2 - genetics | Humans | Pregnancy Proteins - genetics | Male | Pregnancy Proteins - metabolism | Stem Cells - cytology | Vascular Endothelial Growth Factor A - metabolism | Angiopoietin-1 - metabolism | RNA, Messenger - metabolism | Stem Cells - metabolism | Vascular Endothelial Growth Factor A - genetics | Angiopoietin-2 - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit | Genes, Dominant | Transcription Factors - pharmacology | Adenoviridae - genetics | Eye - drug effects | Fibroblasts - metabolism | Transcription Factors - physiology | Proto-Oncogene Proteins c-sis - genetics | Myocytes, Cardiac - cytology | Proto-Oncogene Proteins c-sis - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation - physiology | Rats | Transcription Factors - genetics | Gene Expression Regulation - drug effects | Angiopoietin-1 - genetics | Neovascularization, Physiologic - physiology | Animals | Myocytes, Cardiac - drug effects | Fibroblasts - drug effects | Myocytes, Cardiac - metabolism | Placenta Growth Factor | Fibroblasts - cytology | Mice | Angiogenic Proteins - metabolism | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, pp. e0121568 - e0121568
Osteosarcoma (OS) is a hyperproliferative malignant tumor that requires a high vascular density to maintain its large volume. Vascular Endothelial Growth... 
BREAST-CANCER | SUPPRESSES TUMOR-GROWTH | IN-VITRO | OSTEOBLAST DIFFERENTIATION | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | FACTOR EXPRESSION | CELL-CYCLE ARREST | INDUCIBLE FACTOR-1 | ANGIOGENIC FACTORS | PROTEIN-KINASE-II | Transcription Factor AP-1 - genetics | Humans | Transcriptional Activation - drug effects | Male | Peptides, Cyclic - pharmacology | Transplantation, Heterologous | Autocrine Communication - drug effects | Endothelial Growth Factors - pharmacology | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Bone Neoplasms - pathology | Transcription Factor AP-1 - metabolism | Bone Neoplasms - metabolism | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | RNA Interference | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Transcription, Genetic | Vascular Endothelial Growth Factor A - pharmacology | Osteosarcoma - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Neoplasm Invasiveness | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Sulfonamides - pharmacology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - antagonists & inhibitors | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - genetics | Animals | Signal Transduction - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Benzylamines - pharmacology | Osteosarcoma - pathology | RNA, Small Interfering - metabolism | Osteosarcoma | Physiological aspects | Development and progression | Genetic aspects | Research | Vascular endothelial growth factor | Protein kinases | Therapy | Chromatin | Transcription factors | Immunoprecipitation | Transcription | Calcium | Paracrine signalling | Kinases | Proteins | Angiogenesis | Biomedical materials | Rodents | Calcium-binding protein | Biocompatibility | Inhibition | Autocrine signalling | Growth factors | Calmodulin | Calcium (intracellular) | Aggressive behavior | Secretion | Tumor cells | Activator protein 1 | Pharmacology | Tumorigenicity | Gene expression | Vascular endothelial growth factor receptors | Inhibitors | Human behavior | Calcium ions | Ca2+/calmodulin-dependent protein kinase II | Tumors | Index Medicus
Journal Article
15.