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The New England Journal of Medicine, ISSN 0028-4793, 03/2016, Volume 374, Issue 12, pp. 1123 - 1133
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2017, Volume 69, Issue 16, pp. 2054 - 2063
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2017, Volume 292, Issue 39, pp. 16122 - 16134
Angptl4 (Angiopoietin-like 4) is a circulating protein secreted by white and brown adipose tissues and the liver. Structurally, Angptl4 contains an N-terminal... 
ADIPOCYTES | GENE | BEIGE FAT DEVELOPMENT | INSULIN-RESISTANCE | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | IMPROVES GLUCOSE-TOLERANCE | MICE | ANGPTL4 | LIPOPROTEIN-LIPASE | BROWN FAT | Up-Regulation | Liver - enzymology | Adipose Tissue, Beige - pathology | Male | Muscle, Skeletal - metabolism | Obesity - blood | Recombinant Fusion Proteins - metabolism | Angiopoietin-like 4 Protein | Adiposity | Peptide Fragments - blood | Protein Interaction Domains and Motifs | Peptide Fragments - genetics | Angiopoietins - metabolism | Muscle, Skeletal - enzymology | Peptide Fragments - metabolism | Abdominal Fat - pathology | Adipose Tissue, Beige - cytology | Liver - metabolism | Mice, Inbred C57BL | Oligopeptides - genetics | Cells, Cultured | Mice, Transgenic | Oligopeptides - metabolism | Recombinant Fusion Proteins - chemistry | Angiopoietins - blood | Lipolysis | Recombinant Fusion Proteins - blood | Obesity - metabolism | Obesity - pathology | Triglycerides - metabolism | Abdominal Fat - metabolism | Peptide Fragments - chemistry | Abdominal Fat - cytology | Animals | Angiopoietins - chemistry | Energy Metabolism | Models, Biological | Obesity - prevention & control | Triglycerides - blood | Mutation | Angiopoietins - genetics | Adipose Tissue, Beige - metabolism | Index Medicus | type 2 diabetes | angiopoietin-like | adipocyte | Energy expenditure | Metabolism | metabolic disease | obesity
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 07/2017, Volume 377, Issue 3, pp. 211 - 221
Loss-of-function variants in ANGPTL3 were associated with lower plasma levels of lipids and lower rates of coronary disease. A monoclonal antibody against... 
MEDICINE, GENERAL & INTERNAL | HEART-DISEASE | REMNANT CHOLESTEROL | PLASMA-LIPIDS | IMMUNOGLOBULIN GENES | ATHEROSCLEROSIS | RISK | CORONARY-ARTERY-DISEASE | FAMILIAL COMBINED HYPOLIPIDEMIA | LIPOPROTEIN-LIPASE GENE | TRANSGENIC MICE | Cardiovascular Diseases - prevention & control | Humans | Middle Aged | Antibodies, Monoclonal - adverse effects | Male | Angiopoietins - antagonists & inhibitors | Dose-Response Relationship, Drug | Dyslipidemias - blood | Angiopoietin-like Proteins | Lipids - blood | Female | Disease Models, Animal | Dyslipidemias - drug therapy | Atherosclerosis - drug therapy | Double-Blind Method | Antibodies, Monoclonal - pharmacology | Coronary Artery Disease - metabolism | Mice, Inbred Strains | Atherosclerosis - metabolism | Animals | Antibodies, Monoclonal - administration & dosage | Coronary Artery Disease - genetics | Lipid Metabolism - drug effects | Aged | Mice | Mutation | Angiopoietins - genetics | Cholesterol, LDL | Research | Antibiosis | Atherosclerosis | Lipoproteins (low density) | Medical research | Angiopoietin | Genomics | Genes | Coronary artery | Triglycerides | Biochemistry | Gene expression | Cholesterol | Serum levels | Proteins | Antagonism | Arteriosclerosis | Plasma levels | Cardiovascular diseases | Lipoproteins (high density) | Heart diseases | Drug dosages | Pharmaceuticals
Journal Article
by Stitziel, Nathan O and Stirrups, Kathleen E and Masca, Nicholas G. D and Erdmann, Jeanette and Ferrario, Paola G and König, Inke R and Weeke, Peter E and Webb, Thomas R and Auer, Paul L and Schick, Ursula M and Lu, Yingchang and Zhang, He and Dube, Marie-Pierre and Goel, Anuj and Farrall, Martin and Peloso, Gina M and Won, Hong-Hee and Do, Ron and van Iperen, Erik and Kanoni, Stavroula and Kruppa, Jochen and Mahajan, Anubha and Scott, Robert A and Willenborg, Christina and Braund, Peter S and van Capelleveen, Julian C and Doney, Alex S. F and Donnelly, Louise A and Asselta, Rosanna and Merlini, Piera A and Duga, Stefano and Marziliano, Nicola and Denny, Josh C and Shaffer, Christian M and El-Mokhtari, Nour Eddine and Franke, Ane and Gottesman, Omri and Heilmann, Stefanie and Hengstenberg, Christian and Hoffmann, Per and Holmen, Oddgeir L and Hveem, Kristian and Jansson, Jan-Håkan and Jöckel, Karl-Heinz and Kessler, Thorsten and Kriebel, Jennifer and Laugwitz, Karl L and Marouli, Eirini and Martinelli, Nicola and McCarthy, Mark I and van Zuydam, Natalie R and Meisinger, Christa and Esko, Tõnu and Mihailov, Evelin and Escher, Stefan A and Alver, Maris and Moebus, Susanne and Morris, Anew D and Müller-Nurasyid, Martina and Nikpay, Majid and Olivieri, Oliviero and Perreault, Louis-Philippe Lemieux and AlQarawi, Alaa and Robertson, Neil R and Akinsanya, Karen O and Reilly, Dermot F and Vogt, Thomas F and Yin, Wu and Asselbergs, Folkert W and Kooperberg, Charles and Jackson, Rebecca D and Stahl, Eli and Strauch, Konstantin and Varga, Tibor V and Waldenberger, Melanie and Zeng, Lingyao and Kraja, Aldi T and Liu, Chunyu and Ehret, Georg B and Newton-Cheh, Christopher and Chasman, Daniel I and Chowdhury, Rajiv and Ferrario, Marco and Ford, Ian and Jukema, J. Wouter and Kee, Frank and Kuulasmaa, Kari and Nordestgaard, Børge G and Perola, Markus and Saleheen, Danish and Sattar, Naveed and Surenan, Praveen and Tregouet, David and Young, Robin and Howson, Joanna M. M and Butterworth, Adam S and Danesh, John and Ardissino, Diego and Bottinger, Erwin P and Erbel, Raimund and ... and Myocardial Infarction Genetics and CARDIoGRAM Exome Consortia Investigators and Medicinska fakulteten and Institutionen för folkhälsa och klinisk medicin and Umeå universitet and Medicin
The New England Journal of Medicine, ISSN 0028-4793, 03/2016, Volume 374, Issue 12, pp. 1134 - 1144
Journal Article
Journal Article