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Journal of Clinical Investigation, ISSN 0021-9738, 05/2009, Volume 119, Issue 5, pp. 1109 - 1123
Imatinib mesylate (IM), a potent inhibitor of the BCR/ABL tyrosine kinase, has become standard first-line therapy for patients with chronic myeloid leukemia... 
CHRONIC MYELOGENOUS LEUKEMIA | MEDICINE, RESEARCH & EXPERIMENTAL | MALIGNANT GLIOMA-CELLS | BLAST CRISIS | CLINICAL RESISTANCE | BCR-ABL MUTATIONS | ENDOPLASMIC-RETICULUM | CYTOCHROME-C RELEASE | CASPASE ACTIVATION | IMATINIB RESISTANCE | CHRONIC MYELOID-LEUKEMIA | Transcription Factor CHOP - genetics | Neoplastic Stem Cells - cytology | Gene Expression - drug effects | Calcium - metabolism | Gene Expression - genetics | Microtubule-Associated Proteins - metabolism | Neoplastic Stem Cells - drug effects | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Endoplasmic Reticulum - metabolism | Antineoplastic Agents - therapeutic use | Autophagy - physiology | Thiazoles - therapeutic use | Autophagy - drug effects | Chloroquine - pharmacology | Neoplastic Stem Cells - metabolism | RNA Interference | Endoplasmic Reticulum - drug effects | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Macrolides - pharmacology | Antineoplastic Agents - pharmacology | Cell Death - drug effects | Dasatinib | Chloroquine - therapeutic use | Piperazines - therapeutic use | Pyrimidines - pharmacology | Imatinib Mesylate | Piperazines - pharmacology | Mice, Inbred C3H | Xenograft Model Antitumor Assays | Fusion Proteins, bcr-abl - genetics | Animals | Cell Death - physiology | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Fusion Proteins, bcr-abl - antagonists & inhibitors | Cell Line, Tumor | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Mice | Protein Kinase Inhibitors - pharmacology | Thiazoles - pharmacology | Benzamides | Macrolides - therapeutic use | Protein-Tyrosine Kinases - antagonists & inhibitors | Causes of | Physiological aspects | Genetic aspects | Chronic myeloid leukemia | Research | Drug therapy | Phagocytosis
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2014, Volume 211, Issue 6, pp. 1153 - 1166
Journal Article
Human Reproduction, ISSN 0268-1161, 04/2016, Volume 31, Issue 4, pp. 700 - 711
Abstract STUDY QUESTION Are the immune regulatory molecules programmed cell death-1 (PD-1) and T-cell immunoglobulin mucin-3 (Tim-3) involved in regulating... 
CD4+T | miscarriage | Th2 bias | pregnancy | trophoblast | PD-1 | Tim-3 | ALLOGRAFT-REJECTION | IMMUNITY | PROTECTION | THYMIC STROMAL LYMPHOPOIETIN | OBSTETRICS & GYNECOLOGY | REPRODUCTIVE BIOLOGY | COSTIMULATORY PATHWAYS | CD4(+)T | DIFFERENTIATION | PDL1 | EXHAUSTION | FETOMATERNAL TOLERANCE | Abortion, Habitual - pathology | Fetal Resorption - prevention & control | Coculture Techniques | Humans | Hepatitis A Virus Cellular Receptor 2 - metabolism | Abortion, Habitual - immunology | Fetal Resorption - metabolism | Programmed Cell Death 1 Receptor - antagonists & inhibitors | Th2 Cells - immunology | CD4-Positive T-Lymphocytes - pathology | Abortion, Habitual - metabolism | Pregnancy Trimester, First | Programmed Cell Death 1 Receptor - blood | Th2 Cells - drug effects | Abortion, Induced | CD4-Positive T-Lymphocytes - immunology | Hepatitis A Virus Cellular Receptor 2 - antagonists & inhibitors | Abortion, Habitual - blood | Antibodies, Neutralizing - therapeutic use | Female | Fetal Resorption - pathology | Maternal-Fetal Exchange - drug effects | Th2 Cells - pathology | Immune Tolerance - drug effects | Decidua - immunology | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Hepatitis A Virus Cellular Receptor 2 - blood | Programmed Cell Death 1 Receptor - metabolism | Antibodies, Neutralizing - pharmacology | Decidua - pathology | Fetal Resorption - immunology | Th2 Cells - metabolism | Decidua - metabolism | Pregnancy | Tocolytic Agents - pharmacology | Tocolytic Agents - therapeutic use | Animals | Cell Proliferation - drug effects | Mice | Decidua - drug effects | CD4-Positive T-Lymphocytes - drug effects | Crosses, Genetic
Journal Article
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 139, Issue 5, pp. 1468 - 1477.e2
Journal Article
Immunity, ISSN 1074-7613, 02/2012, Volume 36, Issue 2, pp. 262 - 275
The emerging notion of environment-induced reprogramming of Foxp3 regulatory T (Treg) cells into helper T (Th) cells remains controversial. By genetic fate... 
TGF-BETA | DNA METHYLATION | TRANSCRIPTION FACTOR FOXP3 | IN-VIVO | GENE-EXPRESSION | IMMUNOLOGICAL SELF-TOLERANCE | MICE | INDUCTION | IMMUNOLOGY | LINEAGE | ANTIGEN | T-Lymphocyte Subsets - immunology | Inflammation - pathology | T-Lymphocytes, Regulatory - metabolism | T-Lymphocyte Subsets - cytology | Epigenesis, Genetic | Humans | CD2 Antigens - genetics | Lymphopenia - pathology | T-Lymphocytes, Regulatory - immunology | CD4-Positive T-Lymphocytes - immunology | DNA Methylation | Inflammation - metabolism | Forkhead Transcription Factors - metabolism | Lymphopenia - metabolism | T-Lymphocytes, Regulatory - cytology | CD2 Antigens - metabolism | Cell Differentiation | Cell Lineage - genetics | Gene Expression | Lymphocyte Activation | CD4-Positive T-Lymphocytes - cytology | CD4-Positive T-Lymphocytes - metabolism | Mice, Transgenic | Inflammation - immunology | Forkhead Transcription Factors - genetics | Mice, Knockout | Animals | Interleukin-2 Receptor alpha Subunit - metabolism | T-Lymphocyte Subsets - metabolism | Immunologic Memory | Lymphopenia - immunology | Mice | In Vitro Techniques | Cell Lineage - immunology | Transcription factors | Cytokines | Science | Cell division | Homeostasis | Artificial chromosomes | Proteins | Genotype & phenotype | Lymphocytes | Rodents | DNA methylation | Software | Deoxyribonucleic acid--DNA
Journal Article