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index medicus (80) 80
humans (77) 77
annexins (48) 48
animals (43) 43
female (39) 39
cancer (35) 35
male (35) 35
annexin a10 (34) 34
expression (33) 33
proteins (33) 33
oncology (29) 29
annexins - metabolism (28) 28
cell biology (27) 27
gene expression (26) 26
immunohistochemistry (25) 25
annexins - genetics (24) 24
middle aged (24) 24
analysis (22) 22
biochemistry & molecular biology (21) 21
metastasis (20) 20
mice (20) 20
pathology (20) 20
aged (19) 19
gene (19) 19
apoptosis (18) 18
research (18) 18
cell line, tumor (17) 17
prognosis (16) 16
adult (15) 15
calcium (15) 15
down-regulation (15) 15
identification (14) 14
annexin (13) 13
biomarkers, tumor - analysis (13) 13
cell proliferation (13) 13
protein (13) 13
blotting, western (12) 12
carcinoma (12) 12
cells (12) 12
colorectal cancer (12) 12
development and progression (12) 12
gene expression profiling (12) 12
hepatocellular-carcinoma (12) 12
progression (12) 12
protein binding (12) 12
signal transduction (12) 12
tumors (12) 12
annexin a2 - metabolism (11) 11
dna methylation (11) 11
life sciences (11) 11
mutation (11) 11
genetic aspects (10) 10
genetics & heredity (10) 10
growth (10) 10
microsatellite instability (10) 10
reverse transcriptase polymerase chain reaction (10) 10
rna, messenger - metabolism (10) 10
binding proteins (9) 9
binding sites (9) 9
kinases (9) 9
medicine (9) 9
neurosciences (9) 9
poor-prognosis (9) 9
research article (9) 9
aged, 80 and over (8) 8
annexin a2 (8) 8
biochemistry, general (8) 8
biomarkers, tumor - genetics (8) 8
biomarkers, tumor - metabolism (8) 8
braf mutation (8) 8
gene-expression (8) 8
immunology (8) 8
invasion (8) 8
medical prognosis (8) 8
polyps (8) 8
rna (8) 8
adenocarcinoma (7) 7
annexins - analysis (7) 7
anxa10 (7) 7
apoptosis - drug effects (7) 7
binding (7) 7
biology (7) 7
calcium - metabolism (7) 7
care and treatment (7) 7
case-control studies (7) 7
cell growth (7) 7
colorectal neoplasms - pathology (7) 7
diagnosis, differential (7) 7
differentiation (7) 7
gene expression regulation, neoplastic (7) 7
genes (7) 7
genetic research (7) 7
inflammation (7) 7
multidisciplinary sciences (7) 7
neoplasm invasiveness (7) 7
neoplasm staging (7) 7
oligonucleotide array sequence analysis (7) 7
oncology, experimental (7) 7
physiological aspects (7) 7
protein transport (7) 7
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1. Full Text Gene expression profiling of serrated polyps identifies annexin A10 as a marker of a sessile serrated adenoma/polyp
by Gonzalo, David Hernandez and Lai, Keith K and Shadrach, Bonnie and Goldblum, John R and Bennett, Ana E and Downs‐Kelly, Erinn and Liu, Xiuli and Henricks, Walter and Patil, Deepa T and Carver, Paula and Na, Jie and Gopalan, Banu and Rybicki, Lisa and Pai, Rish K
The Journal of Pathology, ISSN 0022-3417, 08/2013, Volume 230, Issue 4, pp. 420 - 429
Sessile serrated adenomas/polyps (SSA/Ps) are precursors of colon cancer, particularly those that exhibit microsatellite instability. Distinguishing SSA/Ps... 
sessile serrated polyp/adenoma | annexin A10 | hyperplastic polyp | colon cancer | HYPERPLASTIC POLYPS | EMERGING ROLES | sessile serrated polyp | MICROSATELLITE INSTABILITY | PREVALENCE | PATHOLOGY | TUMORIGENICITY | ISLAND METHYLATOR PHENOTYPE | adenoma | ONCOLOGY | BRAF MUTATION | PATHWAY | COLORECTAL-CANCER | CARCINOMA | Immunohistochemistry | Predictive Value of Tests | Colonic Neoplasms - genetics | Oligonucleotide Array Sequence Analysis | Adenoma - genetics | Colonic Polyps - pathology | Humans | Middle Aged | Case-Control Studies | Colonic Polyps - genetics | Colonic Neoplasms - chemistry | Trefoil Factor-2 | Real-Time Polymerase Chain Reaction | Diagnosis, Differential | Biomarkers, Tumor - analysis | Gene Expression Profiling - methods | Adenoma - chemistry | Colonic Polyps - chemistry | Annexins - genetics | Annexins - analysis | Biopsy | Colonic Neoplasms - pathology | Adenoma - pathology | Aged | Biomarkers, Tumor - genetics | Cluster Analysis | Polyps (Pathology) | Colon cancer | DNA microarrays | Analysis | Genes | Genetic research | Gene expression | Medical research | Colon | Colorectal cancer | Adenoma
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2. Full Text Annexin A10 Expression in Microsatellite-unstable Colorectal Cancers: Is it Specific to Sporadic Tumors?
by Kim, Jung Ho and Kang, Gyeong Hoon
The American Journal of Surgical Pathology, ISSN 0147-5185, 11/2014, Volume 38, Issue 11, pp. 1577 - 1579
SURGERY | LYNCH SYNDROME | PATHOLOGY | IMMUNOHISTOCHEMISTRY | Biomarkers, Tumor - analysis | Colorectal Neoplasms, Hereditary Nonpolyposis - diagnosis | Humans | Female | Male | Annexins - biosynthesis | Colorectal Neoplasms - diagnosis
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3. Full Text Simvastatin Up‐Regulates Annexin A10 That Can Inhibit the Proliferation, Migration, and Invasion in Androgen‐Independent Human Prostate Cancer Cells
by Miyazawa, Yoshiyuki and Sekine, Yoshitaka and Kato, Haruo and Furuya, Yosuke and Koike, Hidekazu and Suzuki, Kazuhiro
The Prostate, ISSN 0270-4137, 03/2017, Volume 77, Issue 4, pp. 337 - 349
BACKGROUND Statins have recently been studied for their proapoptotic and antimetastatic effects. However, the exact mechanisms of their anticancer actions... 
S100 calcium binding protein A4 | prostate cancer | annexin A10 | statin | CONTROLLED-TRIAL | DIFFERENTIAL EXPRESSION | PLUS PREDNISONE | DOWN-REGULATION | CASTRATION | ENDOCRINOLOGY & METABOLISM | UROLOGY & NEPHROLOGY | S100A4 | RECURRENCE | CARCINOMA | PROGRESSION | ENZALUTAMIDE | Simvastatin - therapeutic use | Humans | Cell Proliferation - physiology | Male | Prostatic Neoplasms, Castration-Resistant - drug therapy | Simvastatin - pharmacology | Prostatic Neoplasms, Castration-Resistant - metabolism | Cell Movement - physiology | Prostatic Neoplasms, Castration-Resistant - pathology | Up-Regulation - drug effects | Cell Movement - drug effects | Animals | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Mice, Nude | Neoplasm Invasiveness - pathology | Hydroxymethylglutaryl-CoA Reductase Inhibitors - therapeutic use | Up-Regulation - physiology | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Mice, Inbred BALB C | Annexins - biosynthesis
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4. Full Text Immunohistochemistry for Annexin A10 Can Distinguish Sporadic From Lynch Syndrome–associated Microsatellite-unstable Colorectal Carcinoma
by Pai, Reetesh K and Shadrach, Bonnie L and Carver, Paula and Heald, Brandie and Moline, Jessica and Church, James and Kalady, Matthew F and Burke, Carol A and Plesec, Thomas P and Lai, Keith K and Gonzalo, David H and Pai, Rish K
The American Journal of Surgical Pathology, ISSN 0147-5185, 04/2014, Volume 38, Issue 4, pp. 518 - 525
Differentiating sporadic microsatellite-unstable colorectal carcinoma due to MLH1 promoter hypermethylation from Lynch syndrome (LS)-associated tumors due to... 
BRAF | MLH1 | Annexin A10 | Sessile serrated polyp | Lynch syndrome | SURGERY | PROMOTER HYPERMETHYLATION | INSTABILITY | DNA METHYLATION | sessile serrated polyp | PATHOLOGY | POLYPS | ISLAND METHYLATOR PHENOTYPE | REAL-TIME PCR | COLON-CANCER | annexin A10 | BRAF MUTATION | SESSILE SERRATED ADENOMAS | MLH1 METHYLATION | Immunohistochemistry | MutL Protein Homolog 1 | Diagnosis, Differential | Microsatellite Instability | Biomarkers, Tumor - analysis | Colorectal Neoplasms - genetics | Colorectal Neoplasms, Hereditary Nonpolyposis - genetics | Humans | Male | Reverse Transcriptase Polymerase Chain Reaction | Colorectal Neoplasms - diagnosis | Annexins - analysis | Colorectal Neoplasms, Hereditary Nonpolyposis - metabolism | Colorectal Neoplasms, Hereditary Nonpolyposis - diagnosis | Adaptor Proteins, Signal Transducing - genetics | Female | Aged | Annexins - biosynthesis | Nuclear Proteins - genetics | Colorectal Neoplasms - metabolism
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5. Full Text Annexin A10 is a candidate marker associated with the progression of pancreatic precursor lesions to adenocarcinoma
by Zhu, Jianhui and Wu, Jing and Pei, Xiucong and Tan, Zhijing and Shi, Jiaqi and Lubman, David M
PLoS ONE, ISSN 1932-6203, 03/2017, Volume 12, Issue 4, pp. e0175039 - e0175039
Annexins are a multigene family of calcium and phospholipid-binding proteins that play important roles in calcium signaling, cell motility, differentiation and... 
GENETICS | NEOPLASIA | MULTIDISCIPLINARY SCIENCES | MEMBRANE-BINDING PROTEINS | BIOLOGY | CONSENSUS | CLASSIFICATION | SERRATED PATHWAY | COLORECTAL-CARCINOMA | EXPRESSION | CANCER | Multigene Family - genetics | Pancreatic Neoplasms - blood | Adenocarcinoma - pathology | Annexins - metabolism | Adenocarcinoma - blood | CD24 Antigen - metabolism | Humans | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - genetics | Pancreas - metabolism | Pancreatitis, Chronic - genetics | Carcinoma, Pancreatic Ductal - pathology | Annexins - genetics | Disease Progression | Pancreatic Ducts - metabolism | Biomarkers, Tumor - blood | Carcinoma, Pancreatic Ductal - blood | Pancreatitis, Chronic - pathology | Pancreatitis, Chronic - blood | CD24 Antigen - blood | Biomarkers, Tumor - metabolism | Adenocarcinoma - genetics | Annexins - blood | Adenocarcinoma | Development and progression | Annexins | Health aspects | Risk factors | Index Medicus | Calcium | Epithelial cells | Differentiation (biology) | Phospholipids | Tissue analysis | Tissues | Calcium signalling | Proteins | Precursors | Quality | Rodents | Surgery | Classification | Lesions | Pancreas | Statistical analysis | Tumor cells | Ducts | Pancreatitis | Mass spectroscopy | Statistics | Pathology | Pancreatic cancer | Proteomics | Mass spectrometry | Binding sites | Cancer | Tumors
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6. Full Text Annexin A10 expression in colorectal cancers with emphasis on the serrated neoplasia pathway
by Jeong Mo Bae Jung Ho Kim Ye-Young Rhee Nam-Yun Cho Tae-You Kim Gyeong Hoon Kang
世界胃肠病学杂志：英文版, ISSN 1007-9327, 2015, Volume 21, Issue 33, pp. 9749 - 9757
AIM: To validate the utility of Annexin A10 as a surrogate marker of the serrated neoplasia pathway in invasive colorectal cancers(CRCs).METHODS: A total of... 
A10;Serrated | pathway;CpG | island | Annexin | neoplasia | CpG island methylator phenotype | Serrated neoplasia pathway | Annexin A10 | Colorectal cancer | BRAF mutation | MARKER | MICROSATELLITE INSTABILITY | ADENOCARCINOMA | ANTIBODY | COLON | LYNCH SYNDROME | ISLAND METHYLATOR PHENOTYPE | GASTROENTEROLOGY & HEPATOLOGY | CARCINOMA | IMMUNOHISTOCHEMISTRY | Immunohistochemistry | Predictive Value of Tests | Microsatellite Instability | Proto-Oncogene Proteins p21(ras) - genetics | Tissue Array Analysis | Colorectal Neoplasms - genetics | Humans | Middle Aged | Colorectal Neoplasms - chemistry | Male | Colorectal Neoplasms - therapy | DNA Methylation | DNA Mutational Analysis | Female | Retrospective Studies | Republic of Korea | Colorectal Neoplasms - mortality | Reproducibility of Results | Biomarkers, Tumor - analysis | Neoplasm Invasiveness | Risk Factors | Hospitals, University | Kaplan-Meier Estimate | Proportional Hazards Models | Treatment Outcome | Annexins - analysis | Disease-Free Survival | Proto-Oncogene Proteins B-raf - genetics | CpG Islands | Aged | Biomarkers, Tumor - genetics | Mutation | Colorectal Neoplasms - pathology | Neoplasm Staging | Retrospective Study
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7. Full Text Prognostic Significance of Increased Expression of Annexin A10 (ANXA10) in Serous Epithelial Ovarian Cancer
by Wang, J and Zhao, SS and Wang, FF and Zhang, YH
MEDICAL SCIENCE MONITOR, ISSN 1643-3750, 07/2019, Volume 25, pp. 5666 - 5673
Background: Epithelial ovarian cancer (EOC) is a gynecological malignancy that is associated with high mortality. Annexin A10 (ANXA10) is variably expressed in... 
MEDICINE, RESEARCH & EXPERIMENTAL | Prognosis | Neoplasm Invasiveness | STATISTICS | DYSREGULATION | Chemotherapy, Adjuvant | Ovarian Neoplasms | Annexins
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8. Full Text Annexin A10 is a marker for the serrated pathway of colorectal carcinoma
by Sajanti, Sara A and Sajanti, Sara A and Väyrynen, Juha P and Väyrynen, Juha P and Sirniö, Päivi and Sirniö, Päivi and Klintrup, Kai and Klintrup, Kai and Mäkelä, Jyrki and Mäkelä, Jyrki and Tuomisto, Anne and Tuomisto, Anne and Mäkinen, Markus J and Mäkinen, Markus J
Virchows Archiv, ISSN 0945-6317, 1/2015, Volume 466, Issue 1, pp. 5 - 12
Serrated adenocarcinoma (SAC), representing at least 10 % of colorectal carcinomas (CRC), differs from conventional carcinomas not only by its histology, but... 
Immunohistochemistry | Pathology | Medicine & Public Health | Colorectal cancer | Serrated adenocarcinoma | BRAF | Annexin A10 | METHYLATION | MICROSATELLITE INSTABILITY | PREVALENCE | PATHOLOGY | CANCER | POLYPS | MUTATIONS | EXPRESSION | PROGRESSION | ras Proteins - genetics | Adenoma - diagnosis | Diagnosis, Differential | Proto-Oncogene Proteins p21(ras) | Adenocarcinoma - pathology | Annexins - metabolism | Humans | Middle Aged | Male | Proto-Oncogene Proteins - genetics | Mutation - genetics | Case-Control Studies | Colorectal Neoplasms - diagnosis | Adenoma - metabolism | Adenocarcinoma - diagnosis | Adenocarcinoma - metabolism | Proto-Oncogene Proteins B-raf - genetics | Sensitivity and Specificity | Adenoma - pathology | Biomarkers, Tumor - metabolism | Female | Aged | Colorectal Neoplasms - pathology | Colorectal Neoplasms - metabolism | Gene mutations
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9. Full Text Annexin A10 is involved in the development and maintenance of neuropathic pain in mice
by Lu, Ying and Ni, Sujie and He, Li-Na and Gao, Yong-Jing and Jiang, Bao-Chun
Neuroscience Letters, ISSN 0304-3940, 09/2016, Volume 631, pp. 1 - 6
ANXA10 (annexin A10) is a member of the annexin family, and its biological effects are mediated primarily through the calcium-dependent phospholipid-binding... 
Spinal nerve ligation | Spinal cord | Mice | Anxa10 | Neuropathic pain | TARGET | CENTRAL SENSITIZATION | SPINAL-CORD | CANCER | NEUROSCIENCES | MEMBRANE DYNAMICS | EXPRESSION | NOCICEPTION | Up-Regulation | Neuralgia - genetics | Spinal Cord - metabolism | Annexins - metabolism | Hyperalgesia - metabolism | Humans | Neuralgia - metabolism | Male | RNA, Messenger - metabolism | Annexins - genetics | Mice, Inbred ICR | Animals | Ligation | Hyperalgesia - genetics | HEK293 Cells | Neurons - metabolism | Neuralgia - physiopathology | Annexins - physiology | Spinal Nerves - injuries | Astrocytes - metabolism | Gene expression | Safety and security measures | Food | Phosphates | Care and treatment | Pain | DNA microarrays | RNA | Genes | Annexins
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10. Full Text The tumor suppressor annexin A10 is a novel component of nuclear paraspeckles
by Quiskamp, Nina and Quiskamp, Nina and Poeter, Michaela and Poeter, Michaela and Raabe, Carsten Alexander and Raabe, Carsten Alexander and Hohenester, Ulli Martin and Hohenester, Ulli Martin and König, Simone and König, Simone and Gerke, Volker and Gerke, Volker and Rescher, Ursula and Rescher, Ursula
Cellular and Molecular Life Sciences, ISSN 1420-682X, 1/2014, Volume 71, Issue 2, pp. 311 - 329
Annexin A10 is the latest identified member of the annexin family of Ca2+- and phospholipid-binding proteins. In previous studies, downregulation of annexin... 
Life Sciences | Biochemistry, general | Nucleolar caps | Life Sciences, general | Paraspeckles | Annexin A10 | Biomedicine general | Cell Biology | Apoptosis | F-ACTIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | ACUTE MYELOID-LEUKEMIA | I-HETEROTETRAMER | CELL BIOLOGY | MYC MESSENGER-RNA | HEPATOCELLULAR-CARCINOMA | GASTRIC-CARCINOMA | CA2+-BINDING SITES | INVERTED REPEATS | BINDING PROTEINS | Antibiotics, Antineoplastic - toxicity | RNA Polymerase II - antagonists & inhibitors | Annexins - metabolism | Apoptosis - drug effects | Calcium - metabolism | Humans | Nuclear Proteins - metabolism | RNA Polymerase II - metabolism | Cell Nucleus - ultrastructure | RNA, Messenger - metabolism | Annexins - genetics | Doxorubicin - toxicity | PTB-Associated Splicing Factor | Annexins - analysis | Animals | Cell Nucleus - metabolism | Protein Isoforms - metabolism | Dogs | Madin Darby Canine Kidney Cells | HeLa Cells | RNA-Binding Proteins - metabolism | Development and progression | Genetic transcription | RNA | Binding proteins | Tumors | Protein binding | Proteins | Signal transduction | Biochemistry | Gene expression
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11. Full Text Annexin A10 in Human Oral Cancer: Biomarker for Tumoral Growth via G1/S Transition by Targeting MAPK Signaling Pathways
by Shimizu, Toshihiro and Kasamatsu, Atsushi and Yamamoto, Ayumi and Koike, Kazuyuki and Ishige, Shunsaku and Takatori, Hiroaki and Sakamoto, Yosuke and Ogawara, Katsunori and Shiiba, Masashi and Tanzawa, Hideki and Uzawa, Katsuhiro
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, p. e45510
Background: Annexins are calcium and phospholipid binding proteins that form an evolutionary conserved multigene family. Considerable evidence indicates that... 
HEPATOCELLULAR-CARCINOMA | PROTEIN | CYCLIN D1 EXPRESSION | POOR-PROGNOSIS | MULTIDISCIPLINARY SCIENCES | PANCREATIC-CANCER | SQUAMOUS-CELL CARCINOMA | DOWN-REGULATION | PROLIFERATION | KINASE ACTIVATION | ACUTE PROMYELOCYTIC LEUKEMIA | Cell Cycle - genetics | Annexins - metabolism | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Mouth Neoplasms - metabolism | Annexins - genetics | Gene Knockdown Techniques | MAP Kinase Signaling System | RNA Interference | Aged, 80 and over | Cell Line, Tumor | Mouth Neoplasms - pathology | Female | Aged | Biomarkers, Tumor - genetics | Neoplasm Staging | Mouth Neoplasms - genetics | Physiological aspects | Development and progression | Genetic aspects | Research | Annexins | Mouth cancer | Mitogen-activated protein kinases | Cell proliferation | Immunohistochemistry | RNA-directed DNA polymerase | Calcium | Oral cancer | Immunoblotting | Phospholipids | Kinases | Inactivation | Carcinogenesis | Small intestine | Maxillofacial surgery | Proteins | Cyclin-dependent kinase | Carcinogens | Evolutionary conservation | Rodents | Surgery | Bioindicators | Cyclin-dependent kinase inhibitors | G1 phase | Squamous cell carcinoma | Deactivation | Cloning | Cyclin-dependent kinases | Extracellular signal-regulated kinase | MAP kinase | Oral squamous cell carcinoma | Dentistry | Gene expression | Polymerase chain reaction | Medicine | Signaling | Biomarkers | Protein expression | Molecular biology
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12. Full Text Annexin A10 contributes to chronic constrictive injury-induced pain through activating ERK1/2 signalling in rats
by Li, Feng and Xue, Zhou-Ya and Liu, Xiang and Bai, Gang and Wang, Yuan-Lin
International Journal of Neuroscience, ISSN 0020-7454, 02/2018, Volume 128, Issue 2, pp. 125 - 132
Purpose: The current study aims at investigating the downstream targets of spinal Annexin A10 in modulating neuropathic pain. Materials and methods: Paw... 
ERK1/2 | pain | TNF-α | IL-1β | Annexin A10 | TNF-alpha | RECEPTOR | IL-1 beta | NEUROSCIENCES | NEUROPATHIC PAIN | MAINTENANCE | INHIBITION | PATHWAY | MICE | HYPERALGESIA | NOCICEPTION | Peripheral Nerve Injuries - complications | Physical Stimulation | Tumor Necrosis Factor-alpha - metabolism | MAP Kinase Signaling System - physiology | Phosphorylation | Spinal Cord - metabolism | Annexins - metabolism | Hyperalgesia - metabolism | Neuralgia - metabolism | Rats | Male | Rats, Sprague-Dawley | Annexins - genetics | Gene Knockdown Techniques | Animals | Interleukin-1beta - metabolism | Peripheral Nerve Injuries - metabolism | Neuralgia - etiology | Hyperalgesia - etiology | RNA, Small Interfering | Disease Models, Animal
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13. Full Text Annexin A10 is an immunohistochemical marker for adenocarcinoma of the upper gastrointestinal tract and pancreatobiliary system
by Lu, Su‐Hsi and Yuan, Ray‐Hwang and Chen, Yu‐Ling and Hsu, Hey‐Chi and Jeng, Yung‐Ming
Histopathology, ISSN 0309-0167, 11/2013, Volume 63, Issue 5, pp. 640 - 648
Aims Annexin A10 (ANXA10) is a calcium‐ and phospholipid‐binding protein expressed normally in the gastric mucosa. In this study, we evaluated the potential... 
cancer of an unknown primary site | annexin A10 | immunohistochemistry | Immunohistochemistry | Annexin A10 | Cancer of an unknown primary site | PATHOLOGY | UNKNOWN PRIMARY | CANCER | TUMORS | CELL BIOLOGY | HEPATOCELLULAR-CARCINOMA | GASTRIC-CARCINOMA | POOR-PROGNOSIS | EXPRESSION | PROGRESSION | Diagnosis, Differential | Pancreatic Neoplasms - metabolism | Stomach Neoplasms - diagnosis | Annexins - metabolism | Pancreatic Neoplasms - diagnosis | Humans | Stomach Neoplasms - metabolism | Stomach - metabolism | Gastric Mucosa - metabolism | Biliary Tract Neoplasms - diagnosis | Adenocarcinoma - diagnosis | Adenocarcinoma - metabolism | Biliary Tract Neoplasms - metabolism | Biomarkers, Tumor - metabolism | Adenocarcinoma | Liver cancer | Gastrointestinal system | Protein binding | Mucous membrane
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14. Full Text Aberrant expression of annexin A10 is closely related to gastric phenotype in serrated pathway to colorectal carcinoma
by Tsai, Jia-Huei and Lin, Yu-Lin and Cheng, Yi-Chen and Chen, Chien-Chuan and Lin, Liang-In and Tseng, Li-Hui and Cheng, Mei-Ling and Liau, Jau-Yu and Jeng, Yung-Ming
Modern Pathology, ISSN 0893-3952, 02/2015, Volume 28, Issue 2, pp. 268 - 278
Annexin A10 (ANXA10) is a member of the ANX family that is normally expressed in gastric mucosa. ANXA10 was recently observed to be upregulated in sessile... 
ISLAND METHYLATOR PHENOTYPE | MOLECULAR-FEATURES | DNA METHYLATION | BRAF MUTATION | MARKER | DIETARY FIBER | ADENOMA | PATHOLOGY | CANCER | MUC6 | POLYPS | Immunohistochemistry | Microsatellite Instability | Adenocarcinoma - pathology | Biomarkers, Tumor - analysis | Colorectal Neoplasms - genetics | Humans | Precancerous Conditions - metabolism | Stomach - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Cell Transformation, Neoplastic - metabolism | Precancerous Conditions - genetics | DNA Methylation | Phenotype | Adenocarcinoma - metabolism | Cell Transformation, Neoplastic - genetics | DNA Mutational Analysis | CpG Islands | Precancerous Conditions - pathology | Adenocarcinoma - genetics | Annexins - biosynthesis | Cell Transformation, Neoplastic - pathology | Colorectal Neoplasms - pathology | Colorectal Neoplasms - metabolism
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15. Full Text Annexin A10 expression correlates with serrated pathway features in colorectal carcinoma with microsatellite instability
by Kim, Jung Ho and Rhee, Ye‐Young and Kim, Kyung‐Ju and Cho, Nam‐Yun and Lee, Hye Seung and Kang, Gyeong Hoon
APMIS, ISSN 0903-4641, 12/2014, Volume 122, Issue 12, pp. 1187 - 1195
Annexin A10 (ANXA10) has recently been identified as a marker of sessile serrated adenomas/polyps of the colorectum. Although the serrated neoplasia pathway is... 
microsatellite instability | colorectal cancer | Annexin A10 | serrated pathway | Serrated pathway | Microsatellite instability | Colorectal cancer | MOLECULAR-FEATURES | HYPERPLASTIC POLYPS | ADENOCARCINOMA | MICROBIOLOGY | COLON | IMMUNOLOGY | PATHOLOGY | CANCER | ISLAND METHYLATOR PHENOTYPE | BRAF MUTATION | GASTRIC-CARCINOMA | KRAS | ADENOMAS | Immunohistochemistry | MutL Protein Homolog 1 | ras Proteins - genetics | Microsatellite Instability | Proto-Oncogene Proteins p21(ras) | Colorectal Neoplasms - genetics | Humans | Middle Aged | ras Proteins - metabolism | Male | DNA Methylation | DNA Mismatch Repair | Biomarkers, Tumor - metabolism | Female | Nuclear Proteins - genetics | Microsatellite Repeats | Proto-Oncogene Proteins B-raf - metabolism | Proto-Oncogene Proteins - metabolism | Promoter Regions, Genetic | Annexins - metabolism | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Annexins - genetics | Proto-Oncogene Proteins B-raf - genetics | Adaptor Proteins, Signal Transducing - genetics | CpG Islands - genetics | Biomarkers, Tumor - genetics | Mutation | Adaptor Proteins, Signal Transducing - metabolism | Genetic aspects | Methylation | Analysis | Cancer
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