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PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e34892
Background: New therapeutic targets for cardiac hypertrophy, an independent risk factor for heart failure and death, are essential. HNO is a novel redox... 
CARDIAC SARCOPLASMIC-RETICULUM | NATRIURETIC-PEPTIDE | ANTIHYPERTROPHIC ACTIONS | OXIDATIVE STRESS | ADULT-RAT CARDIOMYOCYTES | CYCLIC-GMP | MULTIDISCIPLINARY SCIENCES | II-INDUCED HYPERTROPHY | IN-VIVO | NITRIC-OXIDE | ANTIOXIDANT ACTIONS | Reactive Oxygen Species - metabolism | Natriuretic Peptide, Brain - metabolism | NADPH Oxidases - metabolism | Cardiomegaly - pathology | Phosphoproteins - metabolism | Superoxides - metabolism | Microfilament Proteins - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Angiotensin II - adverse effects | Phosphorylation - drug effects | Endothelin-1 - metabolism | Cardiomegaly - drug therapy | Rats | Guanylate Cyclase - metabolism | Soluble Guanylyl Cyclase | Cell Adhesion Molecules - metabolism | Nitrites - pharmacology | Myocytes, Cardiac - pathology | Reactive Oxygen Species - antagonists & inhibitors | Animals | Myocytes, Cardiac - drug effects | Cyclic GMP - metabolism | Signal Transduction - drug effects | Nitrogen Oxides - pharmacology | Cardiomegaly - chemically induced | Myocytes, Cardiac - metabolism | Cardiomegaly - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | Oxidases | Heart failure | Heart | Heart enlargement | Vasodilators | Protein biosynthesis | Superoxide | Muscle proteins | Angiotensin | Myosin | Health aspects | Protein kinases | Mitogens | Natriuretic peptides | Neonates | Phosphorylation | Salts | Peptides | Medical innovations | Protein kinase G | Oxidase | Guanylate cyclase | NAD(P)H oxidase | Vasodilation | Risk factors | Rodents | Cyclic GMP | CYBB protein | Angiotensin II | Heart diseases | Hypertension | Brain natriuretic peptide | Therapeutic applications | MAP kinase | Cardiomyocytes | Pharmacology | Vasodilator-stimulated phosphoprotein | Signaling | Sodium | Protein synthesis | Protein kinase | Diabetes | Laboratory animals | Hypertrophy | Apoptosis
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e44481
Background: Although evidence now suggests cGMP is a negative regulator of cardiac hypertrophy, the direct consequences of the soluble guanylyl cyclase (sGC)... 
ANTIHYPERTROPHIC ACTIONS | VENTRICULAR HYPERTROPHY | ATRIAL-NATRIURETIC-PEPTIDE | ADULT-RAT CARDIOMYOCYTES | CYCLIC-GMP | PULMONARY-HYPERTENSION | MULTIDISCIPLINARY SCIENCES | II-INDUCED HYPERTROPHY | CARDIAC-HYPERTROPHY | THERAPEUTIC TARGET | NITRIC-OXIDE | Benzoates - therapeutic use | Reactive Oxygen Species - metabolism | Cardiomegaly - pathology | Phosphoproteins - metabolism | Microfilament Proteins - metabolism | Fibroblasts - metabolism | Endothelin-1 - metabolism | Cardiomegaly - drug therapy | Cells, Cultured | Rats | Guanylate Cyclase - metabolism | Soluble Guanylyl Cyclase | Receptors, Cytoplasmic and Nuclear - agonists | Enzyme Activation - drug effects | Fibroblasts - pathology | Cell Adhesion Molecules - metabolism | Rats, Sprague-Dawley | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Cyclic GMP - metabolism | Signal Transduction - drug effects | Fibroblasts - drug effects | Benzoates - pharmacology | Myocytes, Cardiac - metabolism | Cardiomegaly - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | Heart | Superoxide | Comparative analysis | Heart enlargement | Vasodilators | Neonates | Phosphorylation | Heart attacks | Endothelin | Smooth muscle | Kinases | Guanylate cyclase | Eutrophication | Rodents | Cyclic GMP | Cell cycle | Inhibition | Heart diseases | Heart failure | Hypertension | Protein biosynthesis | Cardiomyocytes | Pharmacology | Endothelin 1 | Vasodilator-stimulated phosphoprotein | Signaling | Protein synthesis | Nitric oxide | Fibrosis | Ligands | Diabetes | Hemodynamics | Hypertrophy
Journal Article
Hypertension, ISSN 0194-911X, 03/2005, Volume 45, Issue 3, pp. 341 - 346
Journal Article
Journal Article
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