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Journal of Immunology, ISSN 0022-1767, 06/2018, Volume 200, Issue 11, pp. 3729 - 3738
Nonsteroidal anti-inflammatory drugs (NSAIDs) inhibit cyclooxygenase (COX) activity and are commonly used for pain relief and fever reduction. NSAIDs are used... 
INFLAMMATORY LIPID MEDIATORS | DENDRITIC CELLS | GENE-EXPRESSION | RECEPTOR | ARACHIDONIC-ACID | INDUCTION | IMMUNOLOGY | PROSTAGLANDIN E-2 | MICE LACKING | T-CELLS | I INTERFERON | Cyclooxygenase 2 Inhibitors - pharmacology | Immunity, Innate - drug effects | Mice, Inbred C57BL | Immunity - immunology | Anti-Inflammatory Agents, Non-Steroidal - immunology | Male | Membrane Proteins - immunology | Listeria monocytogenes - immunology | Dinoprostone - immunology | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Immunity, Innate - immunology | Animals | Acetaminophen - pharmacology | CD8-Positive T-Lymphocytes - drug effects | Listeriosis - immunology | Female | Mice | CD8-Positive T-Lymphocytes - immunology | Cyclooxygenase 2 - immunology | Cyclooxygenase 1 - immunology | CD8 antigen | Prostaglandin E2 | Lymphocytes T | Immunity | Immunity (cell-mediated) | Immunosuppressive agents | Prostaglandin endoperoxide synthase | Pain | Listeria | Analgesics | Immunotherapy | Inhibition | Children | Nonsteroidal anti-inflammatory drugs | Phenotypes | Antiinflammatory agents | Immune response | Listeria monocytogenes | Pharmacology | Inflammation | T cell receptors | Celecoxib | Fever | Nonsteroidal antiinflammatory drugs | Immune systems | Inhibitors | Cyclooxygenase-1 | Acetaminophen | Antitumor activity | Indomethacin | Cyclooxygenase-2 | Cancer | Index Medicus | Abridged Index Medicus
Journal Article
International Review of Cell and Molecular Biology, ISSN 1937-6448, 01/2019, Volume 343, pp. 111 - 127
Glycosylation and its by-product, the glycan, play a crucial role in many cellular processes. Aberrant glycan structures and mutations of the glycosylation... 
O-linked glycosylation | Antitumor immunity | Adaptive immunity | Glycans | Glycosylation | Immune evasion | N-linked glycosylation
Conference Proceeding
Nature, ISSN 0028-0836, 11/2015, Volume 527, Issue 7577, pp. 249 - 253
Epigenetic silencing including histone modifications and DNA methylation is an important tumorigenic mechanism. However, its role in cancer immunopathology and... 
Polycomb Repressive Complex 2 - antagonists & inhibitors | CD8-Positive T-Lymphocytes - cytology | Immunotherapy - methods | Prognosis | Histones - chemistry | Chemokine CXCL9 - biosynthesis | Humans | Ovarian Neoplasms - pathology | DNA (Cytosine-5-)-Methyltransferases - antagonists & inhibitors | Th1 Cells - immunology | DNA (Cytosine-5-)-Methyltransferases - metabolism | Th1 Cells - metabolism | Chemokine CXCL10 - biosynthesis | Chemokine CXCL10 - immunology | Female | Epigenesis, Genetic - drug effects | Lysine - metabolism | Tumor Cells, Cultured | Chemokines - immunology | Tumor Escape - immunology | Chemokines - biosynthesis | DNA (Cytosine-5-)-Methyltransferase 1 | Gene Silencing | Chemokine CXCL9 - immunology | Chemokine CXCL9 - genetics | Chemokines - genetics | Enhancer of Zeste Homolog 2 Protein | Ovarian Neoplasms - enzymology | Xenograft Model Antitumor Assays | B7-H1 Antigen - metabolism | Animals | Chemokine CXCL10 - genetics | Ovarian Neoplasms - therapy | Mice | Histones - metabolism | CD8-Positive T-Lymphocytes - immunology | Polycomb Repressive Complex 2 - metabolism | DNA Methylation - drug effects | Lymphocytes, Tumor-Infiltrating - immunology | Ovarian Neoplasms - immunology | Index Medicus | checkpoint | CXCL9 | histone modification | chemotherapy | epigenetics | Chemokine | CXCL10 | DNMT | T cell therapy | DNA methylation | PD-L1 | B7-H1 | cancer | trafficking | PD-1 | EZH2
Journal Article
Nature, ISSN 0028-0836, 08/2014, Volume 512, Issue 7514, pp. 324 - 327
Journal Article
Journal Article
Immunity, ISSN 1074-7613, 08/2015, Volume 43, Issue 2, pp. 240 - 250
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 08/2012, Volume 18, Issue 8, pp. 1248 - 1253
Interleukin-9 (IL-9) is a T cell cytokine that acts through a gamma C-family receptor on target cells and is associated with inflammation and allergy. We... 
MEDICINE, RESEARCH & EXPERIMENTAL | TGF-BETA | ANTITUMOR RESPONSES | IFN-GAMMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | LARGE ESTABLISHED MELANOMA | CELL BIOLOGY | T(H)17 CELLS | IL-9 PRODUCTION | IN-VIVO | ROR-GAMMA | GROWTH | DIFFERENTIATION | T-Lymphocyte Subsets - immunology | Humans | Neoplasm Proteins - physiology | Receptors, Interleukin - deficiency | Vaccination | Carcinoma, Lewis Lung - immunology | Recombinant Fusion Proteins - physiology | Skin Neoplasms - chemistry | Gene Expression Profiling | Interleukin-9 - physiology | Interleukin-9 - biosynthesis | Melanoma, Experimental - immunology | Cancer Vaccines | Nuclear Receptor Subfamily 1, Group F, Member 3 - deficiency | Neoplasm Proteins - genetics | Melanoma - chemistry | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Interleukin-9 - analysis | Skin - immunology | Skin Neoplasms - pathology | Mast Cells - immunology | Immunotherapy, Adoptive | Skin Neoplasms - immunology | Neoplasm Proteins - biosynthesis | Melanoma, Experimental - therapy | Mice, Inbred C57BL | Melanoma, Experimental - pathology | Lymphatic Metastasis | Radiation Chimera | Melanoma - pathology | Tumor Burden | Mast Cells - drug effects | Receptors, Interleukin - genetics | Disease Progression | Homeodomain Proteins - genetics | Melanoma - secondary | Mice, Knockout | Animals | Interleukin-9 - genetics | Melanoma - immunology | T-Lymphocyte Subsets - metabolism | Carcinoma, Lewis Lung - pathology | Neoplasm Proteins - analysis | Mice | Lymphocytes, Tumor-Infiltrating - immunology | Care and treatment | Melanoma | Interleukin-9 | Genetic aspects | Cellular immunity | Research | Diagnosis | T cells | Health aspects | Metastasis | Cytokines | Gene expression | Lymphocytes | Skin cancer | Immune system | Index Medicus
Journal Article
CANCER RESEARCH, ISSN 0008-5472, 11/2016, Volume 76, Issue 21, pp. 6183 - 6192
Journal Article
OncoImmunology, ISSN 2162-4011, 03/2016, Volume 5, Issue 3, pp. e1085146 - e1085146
Programmed death one (PD-1) is a well-established co-inhibitory regulator that suppresses proliferation and cytokine production of T cells. Despite remarkable... 
Antitumor immunity | Programmed death one (PD-1) | liver cancer | dendritic cell | hepatocellular carcinoma | cancer immunotherapy | ACTIVATION | HEPATOCELLULAR-CARCINOMA PATIENTS | RECEPTOR | DEATH | IMMUNOLOGY | CANCER | IMMUNORECEPTOR | ONCOLOGY | MICE | INFECTION | BLOCKADE | TUMOR-IMMUNOTHERAPY
Journal Article