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Molecular and Cellular Biology, ISSN 0270-7306, 10/2006, Volume 26, Issue 19, pp. 7046 - 7055
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Receptors, Tumor Necrosis Factor - metabolism | Apoptosis Regulatory Proteins - pharmacology | Apoptosis - drug effects | Jurkat Cells | Humans | Membrane Glycoproteins - pharmacology | Cells, Cultured | GPI-Linked Proteins | Enzyme Activation - drug effects | Receptors, TNF-Related Apoptosis-Inducing Ligand | Death Domain Receptor Signaling Adaptor Proteins | Receptors, Tumor Necrosis Factor, Member 10c | Tumor Necrosis Factor Receptor-Associated Peptides and Proteins - metabolism | Tumor Necrosis Factor Decoy Receptors | Tumor Necrosis Factor-alpha - pharmacology | Caspases - metabolism | Membrane Glycoproteins - antagonists & inhibitors | TNF-Related Apoptosis-Inducing Ligand | Membrane Microdomains - drug effects | Protein Binding - drug effects | Apoptosis Regulatory Proteins - antagonists & inhibitors | HeLa Cells | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Index Medicus | Membrane Microdomains | Tumor Necrosis Factor Receptor-Associated Peptides and Proteins | Biochemistry, Molecular Biology | Tumor Necrosis Factor-alpha | Life Sciences | Apoptosis Regulatory Proteins | Receptors, Tumor Necrosis Factor | Protein Binding | Caspases | Enzyme Activation | Membrane Glycoproteins | Hela Cells | Apoptosis
Journal Article
Journal Article
Cytokine & growth factor reviews, ISSN 1359-6101, 12/2015, Volume 26, Issue 6, pp. 673 - 685
.... The disease is initiated by the activation of the endothelium by various risk factors leading to chemokine-mediated recruitment of immune cells... 
Advanced Basic Science | Therapeutic avenues | Inflammation | Cytokines | Chemokines | Atherosclerosis | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Atherosclerosis - immunology | Cytokines - metabolism | Atherosclerosis - physiopathology | Humans | Endothelium, Vascular - physiopathology | Extracellular Matrix - metabolism | Inflammation - immunology | Lipoproteins - blood | Molecular Targeted Therapy | Extracellular Matrix - chemistry | Macrophages - enzymology | Animals | Atherosclerosis - therapy | Muscle, Smooth, Vascular - chemistry | Myocardial Infarction - physiopathology | Chemokines - metabolism | Mice | Matrix Metalloproteinases - metabolism | Macrophages - immunology | Chemokines - immunology | Cytokines - immunology | Endothelium, Vascular - immunology | Inflammation - physiopathology | Development and progression | Blood lipids | Health aspects | GM-CSF, granulocyte macrophage colony-stimulating factor | ECM, extracellular matrix | RCT, reverse cholesterol transport | ICAM-1, intercellular adhesion molecule-1 | LDLr, low-density lipoprotein receptor | PECAM-1, platelet endothelial cell adhesion molecule-1 | ABC, ATP-binding cassette | IL-1RA, IL-1 receptor antagonist | ADRP, adipocyte differentiation related protein | SREBP, sterol response element binding protein | wt, wild type | G-CSF, granulocyte colony-stimulating factor | PAI, plasminogen activator inhibitor | CSF, colony-stimulating factor | PRR, pattern recognition receptor | TIMP, tissue inhibitor of metalloproteinases | NPC, Niemann-Pick disease, type C | TNF, tumor necrosis factor | TL1A, TNF-like protein 1A | NLR, NOD-like receptor | SMCs, smooth muscle cells | CR, chemokine receptor | Tregs, regulatory T cells | Survey | GDF-15, growth differentiation factor-15 | NK, natural killer | TNFSF12, TNF superfamily member 12 | IL, interleukin | NLRP3, NLR family, pyrin domain containing 3 | mmLDL, minimally modified LDL | LIGHT, homologous to lymphotoxin, exhibits inducible expression, and competes with HSV glycoprotein D for herpes virus entry mediator, a receptor expressed by T lymphocytes | TF, tissue factor | Th, T-helper | MMP, matrix metalloproteinase | OxLDL, oxidized LDL | CVD, cardiovascular disease | TLR, Toll-like receptor | DCs, dendritic cells | IL-18BP, IL-18 binding protein | miRNA, micro RNA | CANTOS, Canakinumab Anti-inflammatory Thrombosis Outcomes Study | TRAIL, TNF-related apoptosis-inducing ligand | ACAT-1, acyl-CoA acyl transferase-1 | ECs, endothelial cells | TWEAK, TNF-related weak inducer of apoptosis | TGF, transforming growth factor | LFA1, lymphocyte function-associated antigen 1 | ROS, reactive oxygen species | CIRT, Cardiovascular Inflammation Reduction Trial | LDL, low-density lipoprotein | IFN, interferon | MIF, macrophage migration inhibitory factor | SR, scavenger receptor | eNOS, endothelial nitric oxide synthase | STAT1, signal transducer and activator of transcription-1 | Fn14, fibroblast growth factor-inducible 14 | VCAM-1, vascular cell adhesion molecule-1 | LXR, liver X receptors | MHC, major histocompatibility complex | CCR2, CC-chemokine receptor-2 | NOD, nucleotide-binding oligomerization domain | VLA4, very late antigen 4 | BMT, bone marrow transplantation | SR-PSOX, SR that binds phosphatidyl serine and oxidized lipoprotein | ERK, extracellular signal-regulated kinase | SOCS, suppressor of cytokine signaling | Apo, apolipoprotein | M-CSF, macrophage-colony stimulating factor | ER, endoplasmic reticulum
Journal Article
Cancer science, ISSN 1347-9032, 05/2014, Volume 105, Issue 5, pp. 520 - 527
Although tumor necrosis factor‐related apoptosis‐inducing ligand (TRAIL) has shown efficacy in a phase 2 clinical trial, development of resistance to TRAIL by tumor cells is a major roadblock... 
quercetin | CCAAT enhancer‐binding protein homologous protein | tumor necrosis factor‐related apoptosis‐inducing ligand | death receptor 5 | reactive oxygen species | Death receptor 5 | Reactive oxygen species | Tumor necrosis factor-related apoptosis-inducing ligand | CCAAT enhancer-binding protein homologous protein | Quercetin | Life Sciences & Biomedicine | Oncology | Science & Technology | Transcription Factor CHOP - genetics | Up-Regulation | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | Caspase 3 - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Receptors, TNF-Related Apoptosis-Inducing Ligand - metabolism | Receptors, TNF-Related Apoptosis-Inducing Ligand - biosynthesis | DNA-Binding Proteins - metabolism | TNF-Related Apoptosis-Inducing Ligand - pharmacology | RNA Interference | Membrane Potential, Mitochondrial | Female | Ovarian Neoplasms - drug therapy | Promoter Regions, Genetic | Endoplasmic Reticulum Stress - drug effects | Quercetin - therapeutic use | Antioxidants - pharmacology | TNF-Related Apoptosis-Inducing Ligand - metabolism | Xenograft Model Antitumor Assays | Antioxidants - therapeutic use | Animals | Quercetin - pharmacology | Mice, Nude | Cell Line, Tumor | Mice | RNA, Small Interfering | Enzyme Activation | Transcription Factor CHOP - metabolism | Transcription factors | Oxygen | Cell survival | Tumor cells | Caspase | Kinases | Cancer therapies | Ovarian cancer | Proteins | Studies | Signal transduction | Flavonoids | Tumor necrosis factor | Cell death | Natural products | Xenografts | Tumor necrosis factor-TNF | Ligands | TRAIL protein | Endoplasmic reticulum | Apoptosis | Index Medicus | tumor necrosis factor-related apoptosis-inducing ligand | Original
Journal Article