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Genes and Development, ISSN 0890-9369, 05/2013, Volume 27, Issue 10, pp. 1101 - 1114
Tumorigenesis results from dysregulation of oncogenes and tumor suppressors that influence cellular proliferation, differentiation, apoptosis, and/or... 
Huwe1 | c-Myc | Mule | Ras | p21 | Miz1 | DNA-DAMAGE | KERATINOCYTE GROWTH | DEVELOPMENTAL BIOLOGY | CELL BIOLOGY | BASE EXCISION-REPAIR | HUWE1 UBIQUITIN LIGASE | NEGATIVE REGULATION | GENETICS & HEREDITY | ARF TUMOR-SUPPRESSOR | DIFFERENTIATION | MIZ-1 | HUMAN CANCER | Protein Inhibitors of Activated STAT - deficiency | Tetradecanoylphorbol Acetate - pharmacology | 9,10-Dimethyl-1,2-benzanthracene - pharmacology | Male | Protein Inhibitors of Activated STAT - metabolism | Cyclin-Dependent Kinase Inhibitor p15 - biosynthesis | Oncogene Protein p21(ras) - metabolism | Cyclin-Dependent Kinase Inhibitor p16 | Oncogene Protein p21(ras) - antagonists & inhibitors | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Cell Transformation, Neoplastic - genetics | Cyclin-Dependent Kinase Inhibitor p15 - genetics | Nuclear Proteins - deficiency | Protein Inhibitors of Activated STAT - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Female | Nuclear Proteins - genetics | Protein Inhibitors of Activated STAT - antagonists & inhibitors | Skin Neoplasms - pathology | Cyclin-Dependent Kinase Inhibitor p21 - biosynthesis | Signal Transduction | Down-Regulation | Cells, Cultured | Ubiquitin-Protein Ligases - metabolism | Nuclear Proteins - metabolism | Skin Neoplasms - chemically induced | Proto-Oncogene Proteins c-myc - metabolism | Mice, Knockout | Skin Neoplasms - metabolism | Keratinocytes - pathology | Animals | Tumor Suppressor Protein p53 | Keratinocytes - drug effects | Keratinocytes - metabolism | Nuclear Proteins - antagonists & inhibitors | Proto-Oncogene Proteins c-myc - deficiency | Skin Neoplasms - genetics | Proto-Oncogene Proteins c-myc - antagonists & inhibitors | Ubiquitin-Protein Ligases - deficiency | Mice | Proto-Oncogene Proteins c-myc - genetics | Cyclin-Dependent Kinase Inhibitor p15 - metabolism | Genes, ras | Ubiquitin-Protein Ligases - genetics | Oncogene Protein p21(ras) - genetics | Carcinogenesis | Ras genes | Analysis | Index Medicus | Research Paper
Journal Article
Genes and Development, ISSN 0890-9369, 01/2007, Volume 21, Issue 1, pp. 43 - 48
Here we report that RNA interference against ATM inhibited p53 accumulation in cells expressing oncogenic STAT5 and cooperated with Rb inactivation to suppress... 
Senescence | DNA damage | E2F1 | STAT5 | ATM | RasV12 | p53 | ACTIVATION | P19(ARF) | RAS | PHOSPHORYLATION | DEVELOPMENTAL BIOLOGY | CELL BIOLOGY | senescence | PREMATURE SENESCENCE | IN-VIVO | GENETICS & HEREDITY | TUMOR-SUPPRESSOR | HUMAN FIBROBLASTS | CHECKPOINT | Protein Kinases - metabolism | Phosphorylation | Tumor Suppressor Proteins - antagonists & inhibitors | Humans | Oncogenes - physiology | Cell Cycle Proteins - antagonists & inhibitors | DNA-Binding Proteins - metabolism | Genes, ras - physiology | STAT5 Transcription Factor - metabolism | Tumor Suppressor Proteins - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Cell Cycle Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Fibroblasts - metabolism | Tumor Suppressor Proteins - metabolism | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction | RNA, Small Interfering - pharmacology | Retinoblastoma Protein - metabolism | Cell Cycle Proteins - metabolism | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Tumor Suppressor Protein p53 - metabolism | Cellular Senescence - physiology | Ataxia Telangiectasia Mutated Proteins | E2F1 Transcription Factor - metabolism | DNA-Binding Proteins - genetics | Cell Transformation, Neoplastic | Fluorescent Antibody Technique | Retinoblastoma Protein - antagonists & inhibitors | Checkpoint Kinase 2 | Checkpoint Kinase 1 | Fibroblasts - cytology | DNA Damage | Mutation | Aging | Genetic research | Research | Index Medicus | Research Communication
Journal Article
The Plant Cell, ISSN 1040-4651, 3/2009, Volume 21, Issue 3, pp. 749 - 766
Plasma membrane proteins internalized by endocytosis and targeted for degradation are sorted into lumenal vesicles of multivesicular bodies (MVBs) by the... 
Proteins | Cotyledons | Auxins | Cell membranes | Plants | Embryos | Freight | Endosomes | Plant cells | Seedlings | CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MIT DOMAIN | SACCHAROMYCES-CEREVISIAE | PLANT SCIENCES | CELL BIOLOGY | AAA-ATPASE VPS4 | STRUCTURAL BASIS | ENDOSOMAL TRANSPORT | EFFLUX CARRIER | III COMPLEX | MEMBRANE ASSOCIATION | GNOM ARF-GEF | Immunohistochemistry | Arabidopsis - physiology | Vesicular Transport Proteins - metabolism | Humans | Molecular Sequence Data | Arabidopsis - embryology | Phylogeny | Genetic Complementation Test | Recombinant Fusion Proteins - metabolism | Endosomes - metabolism | Arabidopsis Proteins - metabolism | Membrane Transport Proteins - genetics | Membrane Transport Proteins - metabolism | Membrane Proteins - metabolism | Carrier Proteins - classification | Amino Acid Sequence | Arabidopsis Proteins - genetics | Indoleacetic Acids - metabolism | Endosomal Sorting Complexes Required for Transport | Membrane Proteins - genetics | Seedlings - cytology | Arabidopsis - cytology | Vesicular Transport Proteins - genetics | Saccharomyces cerevisiae Proteins - genetics | Endocytosis - physiology | Protein Transport | Arabidopsis - genetics | Carrier Proteins - genetics | Phenotype | Sequence Alignment | Animals | Carrier Proteins - metabolism | Seedlings - physiology | Saccharomyces cerevisiae Proteins - metabolism | Recombinant Fusion Proteins - genetics | Vacuoles - metabolism | Auxin | Arabidopsis | Plant genetics | Physiological aspects | Genetic aspects | Research | Membrane proteins | Index Medicus | s
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 09/2017, Volume 292, Issue 39, pp. 16174 - 16187
Digestion in the stomach depends on acidification of the lumen. Histamine-elicited acid secretion is triggered by activation of the PKA cascade, which... 
ADP ribosylation factor (ARF) | MST2 (mammalian sterile 20-like kinase 2) | exocytosis | ezrin | protein phosphorylation | MIGRATION | LOCALIZATION | ACTIVATION | ERM PROTEINS | PARIETAL-CELLS | EZRIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | MICROTUBULE PLUS-END | IDENTIFICATION | POLARIZATION | AURORA | Cell Polarity | Phosphorylation | Cytoskeletal Proteins - genetics | Protein Multimerization | Parietal Cells, Gastric - metabolism | GTPase-Activating Proteins - metabolism | Recombinant Fusion Proteins - metabolism | Databases, Protein | Cytoskeletal Proteins - metabolism | Cell Membrane - metabolism | Protein-Serine-Threonine Kinases - metabolism | Microscopy, Electron, Transmission | Rabbits | H(+)-K(+)-Exchanging ATPase - metabolism | Cells, Cultured | Computational Biology | Protein-Serine-Threonine Kinases - genetics | Cell Membrane - ultrastructure | Cytoskeletal Proteins - chemistry | GTPase-Activating Proteins - chemistry | Recombinant Fusion Proteins - chemistry | Parietal Cells, Gastric - cytology | Protein Transport | Cell Membrane - enzymology | Animals | Models, Biological | Parietal Cells, Gastric - ultrastructure | Protein Conformation | Protein Processing, Post-Translational | Protein-Serine-Threonine Kinases - chemistry | GTPase-Activating Proteins - genetics | Mutation | Parietal Cells, Gastric - secretion | Amino Acid Substitution | Index Medicus | Cell Biology
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2009, Volume 106, Issue 52, pp. 22540 - 22545
Auxin regulates most aspects of plant growth and development. The hormone is perceived by the TIR1/AFB family of F-box proteins acting in concert with the... 
Receptors | Phenotypes | RNA | Genes | Alleles | Auxins | Gene expression regulation | Plants | Seedlings | Plant cells | Plant hormone | Plant development | plant hormone | plant development | ARABIDOPSIS-THALIANA | DOMAIN | MEMBERS | ARF | MULTIDISCIPLINARY SCIENCES | FUNCTIONAL GENOMIC ANALYSIS | BOX PROTEIN TIR1 | TRANSCRIPTIONAL REPRESSION | DEGRADATION | AUX/IAA PROTEINS | Arabidopsis Proteins - genetics | Plant Roots - metabolism | Indoleacetic Acids - metabolism | Arabidopsis - growth & development | F-Box Proteins - metabolism | Receptors, Cell Surface - metabolism | RNA, Plant - genetics | Recombinant Fusion Proteins - metabolism | Arabidopsis - metabolism | Arabidopsis - genetics | Arabidopsis Proteins - metabolism | Plant Proteins - genetics | Indoleacetic Acids - pharmacology | Plants, Genetically Modified | Gene Expression Regulation, Plant | Recombinant Fusion Proteins - genetics | MicroRNAs - genetics | Plant Proteins - metabolism | Mutation | F-Box Proteins - genetics | Genes, Plant - drug effects | Plant Roots - growth & development | Receptors, Cell Surface - genetics | Plant proteins | Auxin | Research | Arabidopsis | Growth | Proteins | Genotype & phenotype | Genetics | Plant growth | Index Medicus | RNA, Plant | Genes, Plant | Receptors, Cell Surface | Recombinant Fusion Proteins | Life Sciences | F-Box Proteins | Arabidopsis Proteins | Plant Roots | MicroRNAs | Plant Proteins | Vegetal Biology | Indoleacetic Acids | Biological Sciences
Journal Article
Oncogene, ISSN 0950-9232, 02/2012, Volume 31, Issue 6, pp. 752 - 763
MYCN amplification is a major biomarker of poor prognosis, occurring in 25-30% of neuroblastomas. MYCN has contradictory roles in promoting cell growth and... 
neuroblastoma | apoptosis | MYCN | growth inhibition | MDM2-p53 antagonists | P53-DEPENDENT APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | P53/MDM2/P14(ARF) PATHWAY | DIRECT TRANSCRIPTIONAL TARGET | CELL-DEATH | CELL BIOLOGY | G ARREST | AMPLIFICATION | ONCOLOGY | P53 GENE-MUTATIONS | IN-VIVO | GENETICS & HEREDITY | N-MYC | Caspase 7 - metabolism | Oncogene Proteins - genetics | Proto-Oncogene Proteins c-mdm2 - genetics | Tumor Suppressor Protein p53 - antagonists & inhibitors | Apoptosis - drug effects | Humans | Caspase 3 - metabolism | Drug Resistance, Neoplasm | Tumor Suppressor Protein p53 - genetics | Dose-Response Relationship, Drug | Flow Cytometry | RNA Interference | Indoles - pharmacology | N-Myc Proto-Oncogene Protein | Nuclear Proteins - genetics | Proto-Oncogene Proteins c-mdm2 - antagonists & inhibitors | Proto-Oncogene Proteins c-mdm2 - metabolism | Tetracycline - pharmacology | Oncogene Proteins - metabolism | Tumor Suppressor Protein p53 - metabolism | Nuclear Proteins - metabolism | Imidazoles - pharmacology | Piperazines - pharmacology | Blotting, Western | Signal Transduction - drug effects | Models, Biological | Cell Line, Tumor | Cell Proliferation - drug effects | Mutation | Tumor Suppressor Protein p14ARF - genetics | Cell Cycle - drug effects | Tumor Suppressor Protein p14ARF - metabolism | Spiro Compounds - pharmacology | Gene mutations | Physiological aspects | Development and progression | Research | Neuroblastoma | Tumor proteins | Apoptosis | Proteins | Nervous system | Cell growth | Gene expression | Cancer | MDM2 protein | Prognosis | Risk groups | Transcription | biomarkers | Antagonists | Caspase-3 | Neuroblastoma cells | p53 protein | p14ARF protein | Index Medicus
Journal Article
The EMBO Journal, ISSN 0261-4189, 10/2012, Volume 31, Issue 20, pp. 4085 - 4094
Journal Article
Oncogene, ISSN 0950-9232, 08/2012, Volume 31, Issue 32, pp. 3721 - 3732
MDMX is a heterodimeric partner of MDM2 and a critical regulator of p53. The MDMX level is generally elevated in tumors with wild-type p53 and contributes to... 
MDM2 | nutlin | ARF | ubiquitination | MDMX | p53 | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | CELL BIOLOGY | ACIDIC DOMAIN | MDM2-DEFICIENT MICE | AUTOREGULATORY FEEDBACK LOOP | ONCOLOGY | SMALL-MOLECULE INHIBITORS | P53 ACTIVATION | IN-VIVO | GENETICS & HEREDITY | EMBRYONIC LETHALITY | RING DOMAIN | Tumor Suppressor Protein p14ARF - deficiency | Immunoprecipitation | Humans | Protein Multimerization | Proto-Oncogene Proteins - chemistry | Proto-Oncogene Proteins c-mdm2 - chemistry | Ubiquitination | Proteolysis | Indoles - pharmacology | Protein Interaction Domains and Motifs | Protein Stability | Proto-Oncogene Proteins c-mdm2 - antagonists & inhibitors | Proto-Oncogene Proteins c-mdm2 - metabolism | Proto-Oncogene Proteins - metabolism | Gene Expression | Tumor Suppressor Protein p53 - metabolism | Nuclear Proteins - metabolism | Imidazoles - pharmacology | Nuclear Proteins - chemistry | Piperazines - pharmacology | Cell Cycle Checkpoints | Cell Line, Tumor | Protein Binding | Protein Processing, Post-Translational | Proteasome Endopeptidase Complex - metabolism | Tumor Suppressor Protein p14ARF - genetics | Proteasome Inhibitors | Spiro Compounds - pharmacology | Research | Tumor proteins | Health aspects | Decomposition (Chemistry) | Tumors | Proteins | Cellular biology | Gene expression | Binding sites | Index Medicus | MDM2 protein | Language | Tumor cells | Stress | p53 protein | Nutlin
Journal Article
Experimental Cell Research, ISSN 0014-4827, 2012, Volume 318, Issue 3, pp. 228 - 237
Epithelial cells are largely immotile under normal circumstances, but become motile during development, repair of tissue damage and during cancer metastasis.... 
Arf | CNK3 | IPCEF1 | HGF | Cytohesin | PHOSPHATIDYLINOSITOL 3-KINASE | HEPATOCYTE GROWTH-FACTOR | PLASMA-MEMBRANE | CELL BIOLOGY | ONCOLOGY | LIVER-REGENERATION | EPITHELIAL-CELL MIGRATION | ACTIN CYTOSKELETON | INTEGRIN BETA-1 | SCATTER-FACTOR | DOWNSTREAM ACTIVATION | EXCHANGE FACTORS | Humans | Recombinant Fusion Proteins - physiology | Molecular Sequence Data | Hepatocyte Growth Factor - pharmacology | Recombinant Fusion Proteins - metabolism | Cell Movement - genetics | Cell Movement - physiology | Protein Isoforms - metabolism | Membrane Proteins - physiology | Membrane Proteins - metabolism | ADP-Ribosylation Factors - metabolism | ADP-Ribosylation Factors - genetics | Caco-2 Cells | Carrier Proteins - physiology | Membrane Proteins - genetics | RNA, Small Interfering - pharmacology | Carrier Proteins - antagonists & inhibitors | Cells, Cultured | Protein Structure, Tertiary - genetics | Recombinant Fusion Proteins - chemistry | Protein Isoforms - physiology | Gene Expression Regulation - drug effects | Carrier Proteins - genetics | Cell Movement - drug effects | Animals | Carrier Proteins - metabolism | Membrane Proteins - antagonists & inhibitors | Dogs | Recombinant Fusion Proteins - genetics | Protein Isoforms - antagonists & inhibitors | Protein Isoforms - genetics | Protein Structure, Tertiary - physiology | Cells | G proteins | Proteins | Genes | Cell adhesion & migration | Cancer | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, pp. e53631 - e53631
Journal Article
eLife, ISSN 2050-084X, 01/2013, Volume 2013, Issue 2, pp. e00160 - e00160
Planar cell polarity (PCP) requires the asymmetric sorting of distinct signaling receptors to distal and proximal surfaces of polarized epithelial cells. We... 
COPII VESICLE | CLATHRIN ADAPTER | TISSUE POLARITY | LOOP-TAIL | BIOLOGY | PLANAR CELL POLARITY | ARF FAMILY | MEMBRANE TRAFFICKING | PLASMA-MEMBRANE | SORTING SIGNALS | RIGHT DIRECTION | Cell Polarity | Phosphorylation | Adaptor Protein Complex gamma Subunits - metabolism | Adaptor Protein Complex mu Subunits - metabolism | Epithelial Cells - metabolism | Cadherins - metabolism | Humans | Cercopithecus aethiops | Molecular Sequence Data | Intracellular Signaling Peptides and Proteins - metabolism | Adaptor Protein Complex 1 - metabolism | trans-Golgi Network - metabolism | Transfection | RNA Interference | Protein Kinase C - metabolism | Membrane Proteins - metabolism | Protein Interaction Domains and Motifs | ADP-Ribosylation Factors - metabolism | ADP-Ribosylation Factors - genetics | Binding Sites | Intracellular Signaling Peptides and Proteins - genetics | Amino Acid Sequence | Membrane Proteins - genetics | Frizzled Receptors - metabolism | Receptor Protein-Tyrosine Kinases - metabolism | Cell Adhesion Molecules - metabolism | Protein Transport | Animals | Protein Binding | HeLa Cells | Mutation | COS Cells | Enzymes | GTP-binding protein | Clathrin | Transcription factors | Frizzled protein | Epithelial cells | Lipids | siRNA | Cholesterol | Defects | Mammalian cells | Proteins | Mutagenesis | Insects | Polarity | Protein transport | Localization | Golgi cells | Index Medicus
Journal Article
Nature genetics, ISSN 1061-4036, 2000, Volume 26, Issue 3, pp. 291 - 299
To identify new immortalizing genes with potential roles in tumorigenesis, we performed a genetic screen aimed to bypass the rapid and tight senescence arrest... 
LYMPHOMAGENESIS | APOPTOSIS | STABILIZES P53 | AMPLIFICATION | INK4A LOCUS | GENETICS & HEREDITY | C-MYC | MDM2 | GENE FAMILY | CELL-CYCLE ARREST | ARF TUMOR-SUPPRESSOR | T-Box Domain Proteins - physiology | Protein Biosynthesis | Humans | E2F Transcription Factors | Cyclin-Dependent Kinase Inhibitor p16 | T-Box Domain Proteins - isolation & purification | Adenocarcinoma - metabolism | Cell Transformation, Neoplastic - genetics | Gene Deletion | Repressor Proteins - isolation & purification | Neoplastic Syndromes, Hereditary - genetics | Genes, BRCA1 | Neoplasm Proteins - genetics | Tumor Suppressor Protein p14ARF | Oncogenes | DNA-Binding Proteins | Fibroblasts - metabolism | Cell Cycle Proteins - isolation & purification | Repressor Proteins - genetics | Transcription Factor DP1 | Breast Neoplasms - genetics | Polycomb Repressive Complex 1 | Neoplasm Proteins - isolation & purification | Proto-Oncogene Proteins p21(ras) - antagonists & inhibitors | Proto-Oncogene Proteins c-myc - antagonists & inhibitors | Fibroblasts - cytology | Mice | COS Cells | Cell Cycle Proteins - physiology | Neoplasm Proteins - physiology | Gene Expression Regulation, Neoplastic | Chromosomes, Human, Pair 17 - genetics | Breast Neoplasms - metabolism | Repressor Proteins - physiology | Transfection | Cell Cycle Proteins - genetics | Female | Adenocarcinoma - genetics | Tumor Cells, Cultured | Nuclear Proteins - genetics | Cellular Senescence - genetics | Promoter Regions, Genetic | E2F1 Transcription Factor | Neoplasm Proteins - biosynthesis | Cells, Cultured | Proto-Oncogene Proteins - genetics | Transcription Factors - antagonists & inhibitors | T-Box Domain Proteins - genetics | Proteins - genetics | Carrier Proteins - genetics | Animals | Gene Amplification | Retinoblastoma-Binding Protein 1 | Fibroblasts | Physiological aspects | Development and progression | Breast cancer | Genetic aspects | Research | Risk factors | Bmi1 gene | p19ARF gene | Cdkn2a gene | TBX2 gene | HRAS protein | Myc protein | E2F1 protein | Index Medicus
Journal Article