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Circulation Research, ISSN 0009-7330, 04/2015, Volume 116, Issue 8, pp. 1312 - 1323
Rationale: Matrix vesicles (MVs), secreted by vascular smooth muscle cells (VSMCs), form the first nidus for mineralization and fetuin-A, a potent circulating... 
exosomes | extracellular matrix | vascular calcification | CALCIPROTEIN PARTICLES | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTEIN | RETICULOCYTE MATURATION | EXTRACELLULAR CALCIUM | MATRIX VESICLES | HUMAN ATHEROSCLEROTIC PLAQUES | MULTIVESICULAR ENDOSOMES | ARTERIAL CALCIFICATION | PERIPHERAL VASCULAR DISEASE | CHRONIC KIDNEY-DISEASE | HEMATOLOGY | FETUIN-A | Exosomes - metabolism | Sphingomyelin Phosphodiesterase - genetics | Muscle, Smooth, Vascular - metabolism | Calcium - metabolism | Humans | Middle Aged | Vascular Calcification - genetics | Myocytes, Smooth Muscle - pathology | Secretory Vesicles - metabolism | Exosomes - pathology | Male | Exocytosis | Case-Control Studies | Young Adult | Intercellular Signaling Peptides and Proteins - metabolism | Vascular Calcification - metabolism | Transfection | RNA Interference | Time Factors | Sphingomyelin Phosphodiesterase - metabolism | Vascular Calcification - physiopathology | Adult | Female | Proteomics - methods | Tetraspanins - metabolism | Myocytes, Smooth Muscle - metabolism | Exosomes - secretion | Cytokines - metabolism | Cells, Cultured | Vascular Calcification - pathology | Myocytes, Smooth Muscle - secretion | Protein Transport | Secretory Vesicles - secretion | Muscle, Smooth, Vascular - pathology | Adolescent | alpha-2-HS-Glycoprotein - metabolism | Secretory Vesicles - pathology | Muscle, Smooth, Vascular - secretion
Journal Article
Circulation Research, ISSN 0009-7330, 10/2015, Volume 117, Issue 10, pp. 870 - 883
RATIONALE:The pathogenesis of pulmonary arterial hypertension (PAH) remains unclear. The 4 microRNAs representing the miR-143 and miR-145 stem loops are... 
microRNAs | hypertension, pulmonary | endothelium | myocytes, smooth muscle | cell movement | exosomes | MIGRATION | CARDIAC & CARDIOVASCULAR SYSTEMS | MECHANISM | smooth muscle | PHENOTYPE | pulmonary | CANCER | myocytes | MIR-145 | GROWTH | DISEASE | PERIPHERAL VASCULAR DISEASE | CLUSTER | HEMATOLOGY | hypertension | EXPRESSION | Exosomes - metabolism | Vascular Remodeling | Muscle, Smooth, Vascular - metabolism | Humans | Myocytes, Smooth Muscle - pathology | Hypertension, Pulmonary - physiopathology | Male | MicroRNAs - metabolism | Muscle, Smooth, Vascular - physiopathology | Case-Control Studies | Hypertension, Pulmonary - prevention & control | Pulmonary Artery - metabolism | Arterial Pressure | Transfection | Time Factors | Cattle | Female | Binding Sites | Myocytes, Smooth Muscle - metabolism | Promoter Regions, Genetic | Signal Transduction | Endothelial Cells - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Cell Communication | Hypertension, Pulmonary - genetics | Hypertension, Pulmonary - metabolism | Pulmonary Artery - physiopathology | Mice, Knockout | Transcription Factors - metabolism | Muscle, Smooth, Vascular - pathology | Animals | MicroRNAs - genetics | Ventricular Pressure | HeLa Cells | Endothelial Cells - pathology | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Ventricular Function, Right | Cell Movement
Journal Article
Cardiovascular Research, ISSN 0008-6363, 03/2018, Volume 114, Issue 4, pp. 513 - 528
Journal Article