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JAMA, ISSN 0098-7484, 12/2003, Volume 290, Issue 21, pp. 2805 - 2816
Journal Article
Circulation, ISSN 0009-7322, 03/2016, Volume 133, Issue 9, pp. 849 - 858
Journal Article
Seminars in Dialysis, ISSN 0894-0959, 09/2008, Volume 21, Issue 5, pp. 411 - 414
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 11/2017, Volume 232, Issue 11, pp. 3088 - 3102
The Pyk2 inhibitor PF‐4594755 potently inhibits Pyk2 phosphorylation of vascular smooth muscle cells stimulated by FBS or PDGF‐BB. This is accompanied by... 
phenotype | calcium | SERCA | vascular remodeling | PF‐459475 | PF-459475 | ACTIVATION | PHYSIOLOGY | PHENOTYPIC MODULATION | HEART-FAILURE | TRANSCRIPTION | TYROSINE KINASE PYK2 | CELL BIOLOGY | REGULATES SERCA2 | CA2 | GROWTH | GENE-EXPRESSION | ION-CHANNEL | Large-Conductance Calcium-Activated Potassium Channel beta Subunits - metabolism | RNA, Messenger - metabolism | Focal Adhesion Kinase 2 - antagonists & inhibitors | Muscle, Smooth, Vascular - physiopathology | Dose-Response Relationship, Drug | Focal Adhesion Kinase 2 - genetics | Transfection | Focal Adhesion Kinase 2 - metabolism | RNA Interference | Time Factors | Kruppel-Like Transcription Factors - metabolism | Carotid Artery Injuries - genetics | Myocytes, Smooth Muscle - drug effects | Proto-Oncogene Proteins c-sis - pharmacology | Organ Culture Techniques | Large-Conductance Calcium-Activated Potassium Channel beta Subunits - genetics | Muscle, Smooth, Vascular - drug effects | ORAI1 Protein - metabolism | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Carotid Artery, Common - physiopathology | ORAI1 Protein - genetics | Myocytes, Smooth Muscle - enzymology | RNA, Messenger - genetics | Gene Expression Regulation | Becaplermin | Rats, Sprague-Dawley | Vasoconstriction - drug effects | Cell Movement - drug effects | Phenotype | Animals | Calcium Channels, L-Type - genetics | Carotid Artery Injuries - enzymology | Calcium Signaling - drug effects | Carotid Artery Injuries - physiopathology | Carotid Artery, Common - enzymology | Cell Differentiation - drug effects | Carotid Artery, Common - drug effects | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Calcium Channels, L-Type - metabolism | Sarcoplasmic Reticulum Calcium-Transporting ATPases - genetics | Kruppel-Like Transcription Factors - genetics | Muscle, Smooth, Vascular - enzymology | Smooth muscle | Calcium channels | Platelet-derived growth factor | RNA | Protein kinases | Cell proliferation | Cell culture | Electric potential | Calcium | Disorders | Stimulation | Segments | mRNA | Kinases | Carotid arteries | Calcium influx | Arteries | Potassium channels (calcium-gated) | Ca2+-transporting ATPase | Calcium signalling | Genotype & phenotype | KLF4 protein | Rodents | Modulation | Inhibition | Carotid artery | Calcium (intracellular) | Calcium channels (voltage-gated) | Markers | Contractility | Gene expression | Orai1 protein | Muscle contraction | Platelet-derived growth factor BB | Resistance | Inhibitors | Protein kinase | Calcium conductance | Calcium channels (L-type) | Organ culture | Conductance | Intracellular | Focal adhesion kinase | Differentiation | Potassium | Veins & arteries | Integrity | Index Medicus | Kardiologi | Clinical Medicine | Cardiac and Cardiovascular Systems | Medical and Health Sciences | Medicin och hälsovetenskap | Klinisk medicin
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2014, Volume 9, Issue 10, pp. e109432 - e109432
Background and Aims: Endothelial small- and intermediate-conductance K-Ca channels, SK3 and IK1, are key mediators in the endothelium-derived hyperpolarization... 
K(CA)3.1 CHANNELS | BRADYKININ-INDUCED RELAXATION | HYPERPOLARIZING FACTOR | NITRIC-OXIDE RELEASE | MULTIDISCIPLINARY SCIENCES | CA2+-ACTIVATED K+ CHANNELS | TRPV4 CHANNELS | FUNCTIONAL-ROLE | EDHF RESPONSES | ACTIVATED POTASSIUM CHANNELS | SMALL-CONDUCTANCE | Myography | Up-Regulation | Coronary Vessels - drug effects | Small-Conductance Calcium-Activated Potassium Channels - agonists | Small-Conductance Calcium-Activated Potassium Channels - genetics | Intermediate-Conductance Calcium-Activated Potassium Channels - genetics | Calcium Signaling - physiology | Rats | Endothelium, Vascular - drug effects | Male | Intermediate-Conductance Calcium-Activated Potassium Channels - metabolism | Obesity - genetics | Small-Conductance Calcium-Activated Potassium Channels - metabolism | Coronary Vessels - metabolism | Intermediate-Conductance Calcium-Activated Potassium Channels - agonists | Obesity - metabolism | Rats, Zucker | Animals | Calcium Signaling - drug effects | Endothelium, Vascular - metabolism | Oximes - pharmacology | Indoles - pharmacology | Vasodilation - drug effects | Vasodilation - physiology | Immunohistochemistry | Obesity | Acetylcholine | Nitric oxide | Endothelium | Calcium oxide | Calcium | Fluorescence | Smooth muscle | Kinases | Channels | Arteries | Potassium channels (calcium-gated) | Calcium signalling | Rodents | Calcium channels | Calcium (intracellular) | Muscles | Coronary circulation | Muscle contraction | Resistance | Conductance | Microvasculature | Hyperpolarization | Calcium ions | Veins & arteries | Index Medicus
Journal Article
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 03/2006, Volume 47, Issue 6, pp. 1196 - 1206
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 10/2013, Volume 369, Issue 16, pp. 1537 - 1543
Journal Article