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2012, 1. Aufl., ISBN 0470601825, xiii, 533
Book
Molecular bioSystems, ISSN 1742-206X, 05/2017, Volume 13, Issue 5, pp. 981 - 990
Elucidation of the dimerization process of the aryl hydrocarbon receptor (AhR) with the AhR nuclear translocator (ARNT) is crucial for understanding the... 
Index Medicus
Journal Article
European Journal of Immunology, ISSN 0014-2980, 05/2014, Volume 44, Issue 5, pp. 1330 - 1340
The aryl hydrocarbon receptor (AhR) is a ligand‐dependent transcription factor that mediates immunosuppression caused by a variety of environmental... 
Human T lymphocytes | Aryl hydrocarbon receptor | T‐cell activation | T-cell activation | DNA-BINDING | HUMAN MACROPHAGES | AH RECEPTOR | IMMUNOLOGY | ENVIRONMENTAL CONTAMINANT | DEPENDENT INDUCTION | DIFFERENTIATION | LIGAND | EXPRESSION | MODULATION | LYMPHOCYTES | Cell Nucleus - immunology | Receptors, Aryl Hydrocarbon - biosynthesis | Cytochrome P-450 CYP1A1 - genetics | RNA, Messenger - immunology | Aryl Hydrocarbon Hydroxylases - biosynthesis | Active Transport, Cell Nucleus - physiology | Humans | Aryl Hydrocarbon Hydroxylases - genetics | Protein Biosynthesis - immunology | Gene Knockdown Techniques | Interleukins - genetics | RNA, Messenger - biosynthesis | Cell Nucleus - metabolism | T-Lymphocytes - metabolism | Interleukins - immunology | Protein Biosynthesis - genetics | Cytochrome P-450 CYP1A1 - immunology | Aryl Hydrocarbon Hydroxylases - immunology | RNA, Messenger - genetics | Receptors, Aryl Hydrocarbon - genetics | Up-Regulation - genetics | Cell Nucleus - genetics | T-Lymphocytes - cytology | Up-Regulation - immunology | Cytochrome P-450 CYP1B1 | T-Lymphocytes - immunology | Cytochrome P-450 CYP1A1 - biosynthesis | Lymphocyte Activation - physiology | Receptors, Aryl Hydrocarbon - immunology | Interleukins - secretion | Physiological aspects | Polycyclic aromatic hydrocarbons | RNA | T cells | Dioxin | Immunotherapy | Lymphocytes | T cell receptors | Life Sciences
Journal Article
International Journal of Molecular Medicine, ISSN 1107-3756, 10/2017, Volume 40, Issue 4, pp. 1029 - 1036
A large body of evidence indicates that particulate matter (PM)2.5 is associated with various negative effects on human health. However, the impact and... 
Aryl hydrocarbon receptor signaling | Cytochrome P450 1A1 | Lipid synthesis | Inflammatory cytokines | Particulate matter 2.5 | MEDICINE, RESEARCH & EXPERIMENTAL | cytochrome P450 1A1 | ACNE | inflammatory cytokines | AHR | aryl hydrocarbon signaling | lipid synthesis | POLLUTION | ARYL-HYDROCARBON RECEPTOR | PM2.5 | SKIN | particulate matter 2.5 | CELL-DIFFERENTIATION | EXPRESSION | EXPOSURE | WATER | Cytochrome P-450 CYP1A1 - genetics | Interleukin-8 - genetics | Epithelial Cells - metabolism | Epithelial Cells - drug effects | Humans | Interleukin-1alpha - metabolism | Aryl Hydrocarbon Receptor Nuclear Translocator - metabolism | Sebaceous Glands - cytology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Complex Mixtures - pharmacology | Particulate Matter - pharmacology | Receptors, Aryl Hydrocarbon - metabolism | Aryl Hydrocarbon Receptor Nuclear Translocator - agonists | Interleukin-1alpha - genetics | Interleukin-8 - metabolism | Epithelial Cells - cytology | Interleukin-6 - metabolism | Basic Helix-Loop-Helix Transcription Factors - agonists | Basic Helix-Loop-Helix Transcription Factors - genetics | Interleukin-6 - genetics | Signal Transduction | Sebaceous Glands - drug effects | Gene Expression Regulation | Receptors, Aryl Hydrocarbon - genetics | Cytochrome P-450 CYP1A1 - metabolism | G1 Phase Cell Cycle Checkpoints - genetics | Receptors, Aryl Hydrocarbon - agonists | Lipid Metabolism - drug effects | Aryl Hydrocarbon Receptor Nuclear Translocator - genetics | Cell Proliferation - drug effects | Sebaceous Glands - metabolism | G1 Phase Cell Cycle Checkpoints - drug effects | Cell Line, Transformed
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2013, Volume 110, Issue 27, pp. 11115 - 11120
Smoking is a major risk factor for osteoporosis and fracture, but the mechanism through which smoke causes bone loss remains unclear. Here, we show that the... 
Bone resorption | Enzymes | Cell lines | Stem cells | Bones | Osteoclasts | Cultured cells | Bone marrow cells | Cigarette smoking | Aryl hydrocarbon receptors | Osteoblast | Skeletal remodeling | Toxicology | Bone formation | toxicology | TARGET | RESORPTION | MULTIDISCIPLINARY SCIENCES | osteoblast | AHR | skeletal remodeling | CANCER | IN-VITRO | INHIBITION | METABOLISM | bone formation | CIGARETTE-SMOKE | HEALTH | OSTEOCLAST DIFFERENTIATION | Smoking - adverse effects | Cytochrome P-450 CYP1A1 - genetics | Receptors, Aryl Hydrocarbon - deficiency | Aryl Hydrocarbon Hydroxylases - biosynthesis | Humans | Aryl Hydrocarbon Hydroxylases - genetics | Male | Carcinogens - toxicity | Cytochrome P-450 CYP1A2 - genetics | Cytochrome P-450 CYP1A2 - deficiency | Bone Resorption - metabolism | Female | Cytochrome P-450 CYP1A1 - deficiency | Disease Models, Animal | Bone Resorption - etiology | Benzo(a)pyrene - toxicity | Smoke - adverse effects | Mice, Inbred C57BL | Receptors, Aryl Hydrocarbon - genetics | Smoking - genetics | Receptors, Aryl Hydrocarbon - physiology | Tobacco - toxicity | Mice, Knockout | Enzyme Induction - genetics | Animals | Aryl Hydrocarbon Hydroxylases - deficiency | Cytochrome P-450 CYP1A2 - biosynthesis | Bone Resorption - pathology | Cytochrome P-450 CYP1B1 | Polychlorinated Dibenzodioxins - toxicity | Mice | Cytochrome P-450 CYP1A1 - biosynthesis | Osteoporosis | Carcinogens | Cocarcinogens | Hydrocarbons | Physiological aspects | Health aspects | Smoking | Index Medicus | Biological Sciences
Journal Article
Archives of Toxicology, ISSN 0340-5761, 1/2017, Volume 91, Issue 1, pp. 313 - 324
The mechanisms by which pollutants participate in the development of diverse pathologies are not completely understood. The pollutant... 
Biomedicine, general | Persistant organic pollutant | Biomedicine | Environmental Health | HepaRG | Occupational Medicine/Industrial Medicine | ADH | Dioxin | Pharmacology/Toxicology | RAT | MOUSE | PERSISTENT ORGANIC POLLUTANTS | DRUG-METABOLIZING-ENZYMES | HEPATOCELLULAR-CARCINOMA | MESSENGER-RNA | ARYL-HYDROCARBON RECEPTOR | LIVER | GENE-EXPRESSION | ETHANOL | TOXICOLOGY | Gene Expression Regulation, Enzymologic - drug effects | Receptors, Aryl Hydrocarbon - antagonists & inhibitors | Humans | Alcohol Dehydrogenase - antagonists & inhibitors | Male | Isoenzymes - chemistry | Hepatocytes - metabolism | Aryl Hydrocarbon Receptor Nuclear Translocator - metabolism | Carcinogens, Environmental - toxicity | Hepatocytes - cytology | Basic Helix-Loop-Helix Transcription Factors - metabolism | RNA Interference | Isoenzymes - metabolism | Receptors, Aryl Hydrocarbon - metabolism | Female | Aryl Hydrocarbon Receptor Nuclear Translocator - agonists | Hepatocytes - drug effects | Alcohol Dehydrogenase - chemistry | Alcohol Dehydrogenase - metabolism | Basic Helix-Loop-Helix Transcription Factors - agonists | Basic Helix-Loop-Helix Transcription Factors - genetics | Aryl Hydrocarbon Receptor Nuclear Translocator - antagonists & inhibitors | Isoenzymes - genetics | Mice, Inbred C57BL | Cells, Cultured | Receptors, Aryl Hydrocarbon - genetics | Basic Helix-Loop-Helix Transcription Factors - antagonists & inhibitors | Random Allocation | Methylcholanthrene - toxicity | Receptors, Aryl Hydrocarbon - agonists | Animals | Polychlorinated Biphenyls - toxicity | Signal Transduction - drug effects | Pesticides - toxicity | Alcohol Dehydrogenase - genetics | Cell Line, Tumor | Polychlorinated Dibenzodioxins - toxicity | Ligands | Aryl Hydrocarbon Receptor Nuclear Translocator - genetics | Isoenzymes - antagonists & inhibitors | Herbicides | Liver diseases | Alcoholism | Liver | Physiological aspects | Mice | Alcohol dehydrogenase | Retinoids | Pollutants | Pathology | Alcohol | Metabolism | Hepatology | Cells | Index Medicus
Journal Article
Science, ISSN 0036-8075, 9/2010, Volume 329, Issue 5997, pp. 1345 - 1348
Although practiced clinically for more than 40 years, the use of hematopoietic stem cell (HSC) transplants remains limited by the ability to expand these cells... 
Hep G2 cells | Cytokines | REPORTS | Stem cells | Multipotent stem cells | Bone marrow | Cultured cells | Transplantation | Aryl hydrocarbon receptors | Hematopoietic stem cells | Blood | EX-VIVO EXPANSION | ENGRAFTMENT | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | BIOLOGY | MICE | UMBILICAL-CORD BLOOD | BINDING | TRANSPLANTATION | Antigens, CD34 - analysis | Cell Proliferation | Receptors, Aryl Hydrocarbon - antagonists & inhibitors | Species Specificity | Cell Count | Humans | Aryl Hydrocarbon Hydroxylases - genetics | Antigens, CD - analysis | Multipotent Stem Cells - drug effects | Hematopoiesis | Polychlorinated Dibenzodioxins - pharmacology | Hematopoietic Stem Cells - physiology | Receptors, Aryl Hydrocarbon - metabolism | Multipotent Stem Cells - physiology | Hematopoietic Stem Cells - drug effects | Signal Transduction | Purines - metabolism | Purines - pharmacology | Cells, Cultured | Hematopoietic Stem Cell Transplantation | Hematopoietic Stem Cells - metabolism | Mice, SCID | AC133 Antigen | Aryl Hydrocarbon Hydroxylases - metabolism | Cell Lineage | Animals | Multipotent Stem Cells - cytology | Small Molecule Libraries | Hematopoietic Stem Cells - cytology | Cytochrome P-450 CYP1B1 | Mice, Inbred NOD | Mice | Glycoproteins - analysis | Cytokines - pharmacology | Peptides - analysis | Physiological aspects | Arylation
Journal Article
Toxicological Sciences, ISSN 1096-6080, 09/2011, Volume 123, Issue 1, pp. 26 - 36
Journal Article
Toxicological Sciences, ISSN 1096-6080, 12/2012, Volume 130, Issue 2, pp. 349 - 361
The aryl hydrocarbon receptor (AHR) and AHR nuclear translocator (ARNT) activated complex regulates genes in response to the environmental contaminant... 
Chromatin immunoprecipitation (ChIP) | AHR nuclear translocator (ARNT) | ChIP with next-generation sequencing (ChIP-Seq) | Aryl hydrocarbon receptor (AHR) | HUMAN PROMOTERS | CYP1A2 GENE | HUMAN CELL-LINES | ESTROGEN-RECEPTOR | ARYL-HYDROCARBON RECEPTOR | GENE-EXPRESSION | SIM PROTEINS | TOXICOLOGY | RETINOID-X-RECEPTOR | DIOXIN-RESPONSIVE ENHANCER | TRANSCRIPTIONAL REGULATION | Oligonucleotide Array Sequence Analysis | Humans | Sp1 Transcription Factor - metabolism | Gene Expression Profiling | Aryl Hydrocarbon Receptor Nuclear Translocator - metabolism | Gene Regulatory Networks | Breast Neoplasms - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Chromatin Immunoprecipitation | MCF-7 Cells | Nucleotide Motifs | Receptors, Aryl Hydrocarbon - metabolism | Female | Environmental Pollutants - toxicity | Gene Expression Regulation, Neoplastic - drug effects | Basic Helix-Loop-Helix Transcription Factors - drug effects | Binding Sites | Basic Helix-Loop-Helix Transcription Factors - genetics | Response Elements | Signal Transduction | Receptors, Aryl Hydrocarbon - genetics | Receptors, Aryl Hydrocarbon - drug effects | Sequence Analysis, DNA | Hepatocyte Nuclear Factor 3-alpha - metabolism | Breast Neoplasms - genetics | Polychlorinated Dibenzodioxins - toxicity | Aryl Hydrocarbon Receptor Nuclear Translocator - genetics | High-Throughput Nucleotide Sequencing | Index Medicus
Journal Article