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American Journal of Reproductive Immunology, ISSN 1046-7408, 06/2018, Volume 80, Issue S1, pp. 69 - 70
Journal Article
Science Signaling, ISSN 1945-0877, 07/2013, Volume 6, Issue 284, pp. ra59 - ra59
Although tumors tend to be associated with immune cells and inflammation, this immune response often fails to eliminate the cancer and instead promotes cancer... 
Autocrine signalling
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2008, Volume 3, Issue 11, p. e3554
Journal Article
Immunity, ISSN 1074-7613, 03/2011, Volume 34, Issue 3, pp. 288 - 290
As Th17 cell developmental requirements continue to be studied, demonstrate in this issue of Immunity that autocrine TGF- beta cytokine promotes Th17 cell... 
Autocrine signalling
Journal Article
Blood, ISSN 1528-0020, 02/2011, Volume 117, Issue 6, pp. 1938 - 1946
The retrovirus, human T-cell-lymphotrophic virus-1 (HTLV-I) is the etiologic agent of adult T-cell leukemia (ATL) and the neurological disorder... 
Autocrine signalling
Journal Article
Blood, ISSN 1528-0020, 12/2010, Volume 116, Issue 26, pp. 5948 - 5956
Adult T-cell leukemia (ATL), a heterogeneous disease, can be divided into smoldering, chronic, lymphoma, and acute types clinically. In addition to different... 
Autocrine signalling
Journal Article
Cancer Letters, ISSN 0304-3835, 2016, Volume 376, Issue 2, pp. 318 - 327
Journal Article
Oncogene, ISSN 0950-9232, 01/2013, Volume 32, Issue 3, pp. 363 - 374
The endothelium plays a pivotal role in the progression of solid tumors and is considered a highly relevant target for therapy. However, it emerges that... 
chicken embryo chorioallantoic membrane | high-mobility group box 1 | VEGF | autocrine | tumor angiogenesis | SIGNALING PATHWAYS | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | HMGB1 | CELL BIOLOGY | NEURITE OUTGROWTH | ONCOLOGY | ENDOTHELIAL-CELLS | IN-VIVO | GENETICS & HEREDITY | GENE-EXPRESSION | CARDIOVASCULAR-DISEASES | END-PRODUCTS RAGE | DEPENDENT MECHANISM | Autocrine Communication | Colorectal Neoplasms - blood supply | Humans | Gene Expression Regulation, Neoplastic | Neovascularization, Pathologic | Chorioallantoic Membrane - metabolism | Chick Embryo | Receptors, Immunologic - antagonists & inhibitors | Phenotype | Animals | Endothelial Cells - cytology | HMGB1 Protein - metabolism | Colorectal Neoplasms - pathology | Receptor for Advanced Glycation End Products | Colorectal Neoplasms - metabolism | Cell Movement | Care and treatment | Chromosomal proteins | Physiological aspects | Genetic aspects | Neovascularization | Research | Health aspects | Endothelium | Cancer | Angiogenesis | Cancer therapies | Tumors | Platelet-derived growth factor | Migration | photodynamic therapy | Antibodies | Paracrine signalling | Matrix metalloproteinase | Drug resistance | angiogenesis inhibitors | HMGB1 protein | Feedback | Toll-like receptors | Autocrine signalling | Vascular endothelial growth factor | Advanced glycosylation end products | Cytokines | Data processing | Inflammation | Growth factor receptors | TLR4 protein | Gene expression | Embryos | Endothelial cells | Chorioallantoic membrane | Solid tumors | Integrins
Journal Article
Nature cell biology, ISSN 1465-7392, 11/2018, Volume 20, Issue 11, pp. 1328 - 1328
In the version of this Article originally published, in ref. 34 the first author's name was spelled incorrectly. The correct reference is: Rodón, L. et al.... 
Autocrine signalling | Bioenergetics | AMP | Glioblastoma | Tumors | Cancer
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 08/2018, Volume 128, Issue 8, pp. 3583 - 3594
T cells must migrate in order to encounter antigen-presenting cells (APCs) and to execute their varied functions in immune defense and inflammation. ATP... 
BRONCHIOLITIS | MEDICINE, RESEARCH & EXPERIMENTAL | LUNG-TRANSPLANT | ACTIVATION | CHEMOKINES | CHEMOATTRACTANT | INFLAMMATION | IMMUNE SYNAPSE | DYNAMICS | NEUTROPHIL CHEMOTAXIS | RELEASE | Jurkat Cells | CD4-Positive T-Lymphocytes - cytology | Humans | Receptors, Purinergic P2X4 - immunology | Adenosine Triphosphate - genetics | Adenosine Triphosphate - immunology | Inflammation - immunology | Mitochondria - immunology | Autocrine Communication - genetics | CD4-Positive T-Lymphocytes - immunology | Cell Movement - immunology | Animals | Autocrine Communication - immunology | Mitochondria - genetics | Inflammation - genetics | Mice | Mice, Inbred BALB C | Receptors, Purinergic P2X4 - genetics | Mitochondria | Usage | Analysis | Transplantation of organs, tissues, etc | Research | T cells | Adenosine triphosphate | Cell migration | Chemokines | Transplants & implants | Leukocyte migration | Syngeneic grafts | Purine receptors | Lung transplantation | Lymphocytes T | Calcium influx | Inflammatory diseases | Cell adhesion & migration | Cell activation | Lymphocytes | Xenografts | Autocrine signalling | Immune system | Antigens | Lymphatic system | Immunomodulation | Therapeutic applications | Neutrophils | Inflammation | T cell receptors | SDF-1 protein | Calcium (mitochondrial) | CD4 antigen | Graft rejection | Lungs | Antigen-presenting cells
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2013, Volume 110, Issue 18, pp. 7312 - 7317
Hypoxia, or low oxygen tension, is a major regulator of tumor development and aggressiveness. However, how cancer cells adapt to hypoxia and communicate with... 
Angiogenesis | Messenger RNA | Glioma | Cell lines | Hypoxia | Exosomes | Brain hypoxia | Endothelial cells | Tumors | Cancer | Blood vessels | CNS | Biomarker | ANGIOGENESIS | MULTIDISCIPLINARY SCIENCES | blood vessels | MICROVESICLES | MECHANISMS | biomarker | CANCER | AUTOCRINE VEGF | MICROENVIRONMENT | GROWTH | GENE-EXPRESSION | ASSOCIATION | CAVEOLIN-1 | Exosomes - metabolism | Blood Vessels - pathology | Humans | Brain Neoplasms - pathology | Gene Expression Regulation, Neoplastic | Brain Neoplasms - blood supply | Neoplasm Proteins - metabolism | Neovascularization, Pathologic - pathology | Glioma - genetics | Cell Transformation, Neoplastic - genetics | Pericytes - pathology | Glioma - pathology | Female | Autocrine Communication | Paracrine Communication | Endothelial Cells - metabolism | Pericytes - metabolism | Brain Neoplasms - genetics | Signal Transduction - genetics | Transcriptome - genetics | Mice, SCID | Glioma - blood supply | Xenograft Model Antitumor Assays | Animals | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Mice | Tissue Donors | Cell Hypoxia - genetics | Cell Transformation, Neoplastic - pathology | Endothelial Cells - pathology | Proteome - metabolism | Physiological aspects | Metastasis | Research | Gliomas | Health aspects | Cancer cells | Biological Sciences | Clinical Medicine | Basic Medicine | Neurologi | Neurology | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Cell and Molecular Biology | Klinisk medicin | Cancer and Oncology | Cell- och molekylärbiologi | Cancer och onkologi
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2017, Volume 127, Issue 10, pp. 3702 - 3716
Autoreactive CD4 T cells that differentiate into pathogenic Th17 cells can trigger autoimmune diseases. Therefore, investigating the regulatory network that... 
ENCEPHALOMYELITIS | MEDICINE, RESEARCH & EXPERIMENTAL | MYELIN OLIGODENDROCYTE GLYCOPROTEIN | IL-6-DEFICIENT MICE | TGF-BETA | MULTIPLE-SCLEROSIS | EPIGENETIC CONTROL | RHEUMATOID-ARTHRITIS SUSCEPTIBILITY | HELPER T-CELLS | MEDIATED REGULATION | PLASTICITY | Autoimmunity | Myelin-Oligodendrocyte Glycoprotein - immunology | Myelin-Oligodendrocyte Glycoprotein - genetics | Humans | Encephalomyelitis, Autoimmune, Experimental - immunology | Organ Specificity - immunology | Cell Differentiation - genetics | Interleukins - genetics | Organ Specificity - genetics | Autocrine Communication - immunology | Interleukins - immunology | Encephalomyelitis, Autoimmune, Experimental - genetics | Th17 Cells - pathology | Interleukin-6 - genetics | Multiple Sclerosis - genetics | MicroRNAs - immunology | Autocrine Communication - genetics | Mice, Knockout | Cell Differentiation - immunology | Animals | Interleukin-6 - immunology | Multiple Sclerosis - immunology | Th17 Cells - immunology | Mice | MicroRNAs - genetics | Research | Cell differentiation | T cells | Analysis | Multiple sclerosis | Oligodendrocyte-myelin glycoprotein | Encephalomyelitis | Disease | Cytokines | Myelin | Helper cells | Lymphocytes T | Inflammation | T cell receptors | Kinases | Gene expression | Experimental allergic encephalomyelitis | CD4 antigen | Interleukin 6 | T-cell receptor | Lymphocytes | Rheumatoid arthritis | MicroRNAs | Rodents | Stem cells | Interleukin 21 | Autocrine signalling | Autoimmune diseases
Journal Article