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Autophagy, ISSN 1554-8627, 06/2012, Volume 8, Issue 6, pp. 867 - 869
The term autophagic cell death (ACD) initially referred to cell death with greatly enhanced autophagy, but is increasingly used to imply a death-mediating role... 
apoptosis | autophagic cell death | autophagy | cell death | necrosis | Proteins | Binding | Landes | Calcium | Biology | Bioscience | Cell | Cycle | Organogenesis | Cancer | Autophagic cell death | Cell death | Autophagy | Apoptosis | Necrosis | GROWTH | NEURON DEATH | DEGRADATION | DROSOPHILA-MELANOGASTER OOGENESIS | CELL BIOLOGY | Humans | Animals | Index Medicus | Editor's Corner
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 11/2007, Volume 11, Issue 6, pp. 1214 - 1224
Journal Article
Toxicon, ISSN 0041-0101, 05/2018, Volume 146, pp. 114 - 119
Fumonisins are a type of mycotoxin produced by Fusarium spp., mainly F. proliferatum and F. vertieilliodes. Fumonisins represent a potential hazard to the... 
Autophagic cell death | Fumonisin B | Apoptosis | Hemocytes | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2016, Volume 11, Issue 7, pp. e0158761 - e0158761
Background Myelodysplastic syndrome (MDS) is a group of heterogeneous hematopoietic stem cell malignancies with a high risk of transformation into acute... 
APOPTOSIS | UBIQUITIN-PROTEASOME SYSTEM | PROTEIN | CANCER CELLS | POOR-PROGNOSIS | MULTIDISCIPLINARY SCIENCES | DOUBLE-EDGED-SWORD | RESISTANCE | RISK MYELODYSPLASTIC SYNDROME | PROTECTIVE AUTOPHAGY | SELECTIVE AUTOPHAGY | Cell Survival - drug effects | RNA, Small Interfering - genetics | Myelodysplastic Syndromes - metabolism | Humans | Ubiquitin-Conjugating Enzymes - genetics | Bortezomib - pharmacology | Blotting, Western | Ubiquitin-Conjugating Enzymes - metabolism | Fluorescent Antibody Technique | Cell Line, Tumor | Myelodysplastic Syndromes - genetics | Cell Death - drug effects | Tumor Cells, Cultured | Autophagy-Related Proteins - genetics | Autophagy-Related Proteins - metabolism | Real-Time Polymerase Chain Reaction | Complications and side effects | Bortezomib | Physiological aspects | Dosage and administration | Genetic aspects | Research | Gene expression | Drug therapy | Myelodysplastic syndromes | Cell proliferation | Biotechnology | Transformation | Leukemia | Multiple myeloma | Staining | AKT protein | Kinases | Autophagy | Cancer therapies | Myelodysplastic syndrome | Western blotting | Proteins | Signal transduction | Cell cycle | Bone marrow | Cell survival | Hematology | Risk groups | Myeloid leukemia | Mortality | Caspase | Tumor cell lines | Electron microscopy | Patients | Survival | Hemopoiesis | Signaling | Sensitivity | Hospitals | DNA microarrays | Cell death | Medical prognosis | Cell lines | Stem cells | Sensitivity enhancement | Transformations | Annexin V | Cholecystokinin | Acute myeloid leukemia | Viability | Phagocytosis | Apoptosis | Index Medicus
Journal Article
Cell Death and Disease, ISSN 2041-4889, 07/2013, Volume 4, Issue 7, pp. e720 - e720
Journal Article
Neuroscience, ISSN 0306-4522, 02/2018, Volume 371, pp. 60 - 74
Although the receptor-interacting protein 1 kinase (RIP1K)-regulated necroptosis can be evoked by cerebral ischemia, the effects of RIP1K in mediating neuronal... 
neuron | ISCHEMIC stroke | autophagic-lysosomal pathway | astrocyte | neuroprotection | RIP1K | NECROPTOSIS | RAT | FOCAL CEREBRAL-ISCHEMIA | PROGRAMMED NECROSIS | INJURY | NEUROSCIENCES | INHIBITION | SIGNALING PATHWAY | RECEPTOR-INTERACTING PROTEIN | 3-METHYLADENINE | Neurons - pathology | Cell Hypoxia - physiology | Astrocytes - pathology | Brain Ischemia - metabolism | Male | Autophagy - physiology | GTPase-Activating Proteins - metabolism | Autophagy - drug effects | Brain - metabolism | Stroke - pathology | Lysosomes - metabolism | Neuroprotective Agents - pharmacology | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Lysosomes - pathology | Neurons - metabolism | Neurons - drug effects | Protein-Serine-Threonine Kinases - metabolism | Lysosomes - drug effects | Astrocytes - drug effects | Cell Hypoxia - drug effects | GTPase-Activating Proteins - antagonists & inhibitors | Cells, Cultured | Stroke - drug therapy | Random Allocation | Rats, Sprague-Dawley | Mice, Inbred ICR | Brain - drug effects | Glucose - deficiency | Stroke - metabolism | Animals | Signal Transduction - drug effects | Brain Ischemia - drug therapy | Brain Ischemia - pathology | Brain - pathology | Protein Kinase Inhibitors - pharmacology | Astrocytes - metabolism | Stroke (Disease) | Ischemia | Neurons | Cell death | Analysis | Cathepsins | Biochemistry | Permeability | Index Medicus
Journal Article
Autophagy, ISSN 1554-8627, 08/2017, Volume 13, Issue 8, pp. 1404 - 1419
Journal Article