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Gastroenterology, ISSN 0016-5085, 2015, Volume 148, Issue 1, pp. 148 - 157.e7
Background & Aims Chronic stress alters the hypothalamic–pituitary–adrenal axis, increases gut motility, and increases the perception of visceral pain. We... 
Gastroenterology and Hepatology | HPA | Epigenetic Modification | Pain Sensitivity | Visceral Hyperalgesia | METHYLATION | SENSORY FIBERS | WATER AVOIDANCE STRESS | GUT | RECEPTOR | TRPV1 | GASTROENTEROLOGY & HEPATOLOGY | EXPOSURE | Epigenesis, Genetic | E1A-Associated p300 Protein - genetics | Male | E1A-Associated p300 Protein - metabolism | Ganglia, Spinal - physiopathology | Receptors, Glucocorticoid - metabolism | Visceral Pain - diagnosis | RNA Interference | Time Factors | Histones | Transcription, Genetic | Acetylation | Disease Models, Animal | Abdominal Pain - diagnosis | DNA (Cytosine-5-)-Methyltransferase 1 | Visceral Pain - physiopathology | Abdominal Pain - metabolism | Rats, Sprague-Dawley | Visceral Pain - genetics | Receptor, Cannabinoid, CB1 - genetics | Hyperalgesia - genetics | Abdominal Pain - genetics | Pain Threshold | Chronic Disease | Stress, Psychological - complications | Stress, Psychological - physiopathology | Hyperalgesia - psychology | Stress, Psychological - psychology | DNA (Cytosine-5-)-Methyltransferases - metabolism | TRPV Cation Channels - metabolism | DNA Methylation | Stress, Psychological - metabolism | Visceral Pain - psychology | Hyperalgesia - diagnosis | Promoter Regions, Genetic | Hyperalgesia - metabolism | Abdominal Pain - physiopathology | Gene Expression Regulation | Visceral Pain - metabolism | TRPV Cation Channels - genetics | Hyperalgesia - physiopathology | Abdominal Pain - psychology | DNA (Cytosine-5-)-Methyltransferases - genetics | Receptor, Cannabinoid, CB1 - metabolism | Stress, Psychological - genetics | Animals | Receptors, Glucocorticoid - genetics | Pain Measurement | Ganglia, Spinal - metabolism | Stress (Psychology) | Epigenetic inheritance | Genes | epigenetic modification | pain sensitivity | Visceral hyperalgesia
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 2016, Volume 68, Issue 11, pp. 2795 - 2805
ObjectiveMevalonate kinase deficiency (MKD) is a rare metabolic disease characterized by recurrent inflammatory episodes. This study was undertaken to describe... 
MVK | HYPER-IGD | PERIODIC FEVER SYNDROME | ACIDURIA | HYPERIMMUNOGLOBULINEMIA-D | AUTOINFLAMMATORY DISEASES | MUTATIONS | SPECTRUM | RHEUMATOLOGY | Arthralgia - etiology | Uveitis - etiology | Humans | Child, Preschool | Lymphadenopathy - etiology | Male | Arthralgia - physiopathology | Arthritis - genetics | Stomatitis, Aphthous - genetics | Cerebellar Diseases - physiopathology | Amyloidosis - genetics | Skin Diseases - physiopathology | Amyloidosis - etiology | Child | Stomatitis, Aphthous - etiology | Infant, Newborn | Myalgia - physiopathology | Amyloidosis - physiopathology | Genotype | Headache - physiopathology | Vomiting - genetics | Phenotype | Diarrhea - genetics | Abdominal Pain - genetics | Adolescent | Age of Onset | Intellectual Disability - etiology | Uveitis - genetics | Vomiting - physiopathology | Conjunctivitis - etiology | Conjunctivitis - genetics | Arthritis - physiopathology | Headache - etiology | Skin Diseases - genetics | Mevalonate Kinase Deficiency - physiopathology | Myalgia - genetics | Cerebellar Diseases - etiology | Diarrhea - physiopathology | Infant | Mevalonate Kinase Deficiency - genetics | Intellectual Disability - genetics | Pharyngitis - genetics | Arthritis - etiology | Female | Registries | Retrospective Studies | Arthralgia - genetics | Pharyngitis - physiopathology | Abdominal Pain - etiology | Pharyngitis - etiology | Lymphadenopathy - genetics | Abdominal Pain - physiopathology | Myalgia - etiology | Phosphotransferases (Alcohol Group Acceptor) - genetics | Headache - genetics | Intellectual Disability - physiopathology | Skin Diseases - etiology | Cerebellar Diseases - genetics | Conjunctivitis - physiopathology | Mevalonate Kinase Deficiency - complications | Stomatitis, Aphthous - physiopathology | Vomiting - etiology | Diarrhea - etiology | Lymphadenopathy - physiopathology | Uveitis - physiopathology | Kinases | Genotype & phenotype | Index Medicus | Abridged Index Medicus
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 07/2015, Volume 309, Issue 2, pp. G87 - G99
Quiescent phases of inflammatory bowel disease (IBD) are often accompanied by chronic abdominal pain. Although the transient receptor potential vanilloid 1... 
Inflammatory bowel disease | Transient receptor potential vanilloid 1 | Visceral pain | Substance P | Peripheral sensitization | PHYSIOLOGY | GENE-RELATED PEPTIDE | PRIMARY AFFERENT NEURONS | transient receptor potential vanilloid 1 | ULCERATIVE-COLITIS | inflammatory bowel disease | visceral pain | POTENTIAL VANILLOID 1 | INFLAMMATORY-BOWEL-DISEASE | IBS-LIKE SYMPTOMS | peripheral sensitization | CHRONIC-PANCREATITIS | PRIMARY SENSORY NEURONS | QUALITY-OF-LIFE | substance P | SUBSTANCE-P | GASTROENTEROLOGY & HEPATOLOGY | Hyperalgesia - chemically induced | Dextran Sulfate | Visceral Pain - chemically induced | Colitis - genetics | Humans | Colon - innervation | Ganglia, Spinal - physiopathology | TRPV Cation Channels - deficiency | Behavior, Animal | TRPV Cation Channels - metabolism | Transfection | Time Factors | Colitis - chemically induced | HEK293 Cells | Disease Models, Animal | Abdominal Pain - chemically induced | Acute Disease | Hyperalgesia - metabolism | Signal Transduction | Visceral Pain - physiopathology | Mice, Inbred C57BL | Abdominal Pain - physiopathology | Hyperalgesia - prevention & control | Abdominal Pain - metabolism | Visceral Pain - metabolism | TRPV Cation Channels - genetics | Hyperalgesia - physiopathology | Mice, Knockout | Visceral Pain - genetics | Animals | Colitis - physiopathology | Hyperalgesia - genetics | Abdominal Pain - genetics | Colitis - metabolism | Pain Threshold | Substance P - metabolism | Neurons, Afferent - metabolism | Pain Measurement | Ganglia, Spinal - metabolism | Abdominal pain | Care and treatment | Inflammation | Colitis | Dextran | Gastrointestinal diseases
Journal Article
PLoS Medicine, ISSN 1549-1277, 08/2016, Volume 13, Issue 8, p. e1002090
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 03/2018, Volume 314, Issue 3, pp. G448 - G457
Journal Article
Journal of Urology, The, ISSN 0022-5347, 2014, Volume 192, Issue 5, pp. 1564 - 1568
Journal Article