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Artificial Cells, Nanomedicine, and Biotechnology, ISSN 2169-1401, 12/2019, Volume 47, Issue 1, pp. 2343 - 2351
Recent papers suggest that oncogenic Ras participate in regulating tumour cells proliferation and metastasis. This work linked Ras with H1.4 modification in... 
non-small-cell lung carcinoma (NSCLC) | histone modification | histone H1.4 | Ras mutation | PKA | MATERIALS SCIENCE, BIOMATERIALS | ACETYLATION | ENGINEERING, BIOMEDICAL | PROLIFERATION | CANCER | HISTONE H3 | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | TARGETS
Journal Article
Artificial Cells, Nanomedicine, and Biotechnology, ISSN 2169-1401, 12/2019, Volume 47, Issue 1, pp. 2882 - 2890
Background: Histone H1.5 has been considered as a novel cancer marker as its expression is associated with various human cancers. The objective of this study... 
Glioma | GSK3 | AKT signaling | Ras mutation | H1.5 phosphorylation | CELLS | MATERIALS SCIENCE, BIOMATERIALS | ACETYLATION | ENGINEERING, BIOMEDICAL | PROLIFERATION | CANCER | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GROWTH | TARGETS | MUTATIONS
Journal Article
Artificial Cells, Nanomedicine, and Biotechnology, ISSN 2169-1401, 12/2019, Volume 47, Issue 1, pp. 1207 - 1215
Histone H4 acetylation at lysine 12 (H4K12ac) has been reported to be associated with the poor prognosis of pancreatic cancer. The study intends to study... 
MDM2 | Ras-ERK1/2 signalling | Osteosarcoma | H4K12ac | HDAC6 | HAT1 | TARGET | MATERIALS SCIENCE, BIOMATERIALS | METASTASIS | CELL MOTILITY | ENGINEERING, BIOMEDICAL | HISTONE ACETYLATION | CANCER | GENE | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | RESISTANCE | MUTATIONS | EXPRESSION
Journal Article
Artificial Cells, Nanomedicine, and Biotechnology, ISSN 2169-1401, 12/2019, Volume 47, Issue 1, pp. 2618 - 2623
Cellular senescence is strongly tied to vascular disease. The current study aims to examine ways that endothelial cellular senescence can be prevented and the... 
Salicin | TNF-α | endothelial dysfunction | SASP | HUVECs | cell senescence | MATERIALS SCIENCE, BIOMATERIALS | EFFICACY | TNF-alpha | ACETYLATION | ENGINEERING, BIOMEDICAL | OLDER-ADULTS | ADHERENCE | EXERCISE | P53 | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | WILLOW BARK EXTRACT | EXPRESSION
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 12/2019, Volume 10, Issue 1, pp. 1897 - 1897
The cellular decision regarding whether to undergo proliferation or death is made at the restriction (R)-point, which is disrupted in nearly all tumors. The... 
UBIQUITINATION | AML1-ETO | ACETYLATION | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | PROTEINS | QUANTITATIVE-ANALYSIS | POLYCOMB | CANCER | KINASES | FAMILY | Chromatin - metabolism | RNA, Small Interfering - genetics | Myeloid-Lymphoid Leukemia Protein - metabolism | TOR Serine-Threonine Kinases - metabolism | Core Binding Factor Alpha 3 Subunit - antagonists & inhibitors | Humans | Polycomb-Group Proteins - metabolism | TOR Serine-Threonine Kinases - genetics | Cell Cycle Checkpoints - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Cyclin-Dependent Kinase 4 - antagonists & inhibitors | Chromatin - chemistry | MAP Kinase Kinase 1 - antagonists & inhibitors | Butadienes - pharmacology | Signal Transduction | Epithelial Cells - pathology | MAP Kinase Kinase 1 - metabolism | Imidazoles - pharmacology | Cyclin-Dependent Kinase 4 - metabolism | Piperazines - pharmacology | Cell Cycle Checkpoints - drug effects | Polycomb-Group Proteins - genetics | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cell Line, Tumor | MAP Kinase Kinase 4 - genetics | RNA, Small Interfering - metabolism | ras Proteins - genetics | Core Binding Factor Alpha 3 Subunit - metabolism | Epithelial Cells - metabolism | Nitriles - pharmacology | Cyclin-Dependent Kinase 4 - genetics | Epithelial Cells - drug effects | ras Proteins - metabolism | Drosophila melanogaster - genetics | Chromatin Assembly and Disassembly - drug effects | Drosophila melanogaster - metabolism | MAP Kinase Kinase 1 - genetics | TOR Serine-Threonine Kinases - antagonists & inhibitors | MAP Kinase Kinase 4 - metabolism | HEK293 Cells | MAP Kinase Kinase 4 - antagonists & inhibitors | Chromatin - drug effects | Histone-Lysine N-Methyltransferase - genetics | Drosophila melanogaster - cytology | Gene Expression Regulation | p38 Mitogen-Activated Protein Kinases - genetics | Sirolimus - pharmacology | Animals | Histone-Lysine N-Methyltransferase - metabolism | Myeloid-Lymphoid Leukemia Protein - genetics | Core Binding Factor Alpha 3 Subunit - genetics | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Index Medicus
Journal Article
SCIENTIFIC REPORTS, ISSN 2045-2322, 04/2019, Volume 9, Issue 1, pp. 6304 - 9
Here we demonstrate a mode of reciprocal regulation between GFI1 and p53 that controls the induction of apoptosis in T cells. We show that GFI1 prevents... 
TARGET | DNA-BINDING | GENE | PATHWAY | MULTIDISCIPLINARY SCIENCES | C-TERMINAL DOMAIN | EXPRESSION | Demethylation | Transcription | Lysine | p53 Protein | DNA damage | Post-translation | Lymphocytes T | C-Terminus | Acetylation | Methylation | Apoptosis
Journal Article
by Zhang, F and Li, LS and Ma, J and Gong, H
SCIENTIFIC REPORTS, ISSN 2045-2322, 02/2019, Volume 9, Issue 1, pp. 1 - 8
The MoS2-catalyzed transamidation reaction with high yields using N,N-dimethylformamide and other amides as carbonyl sources is developed here. The protocol is... 
HIGHLY EFFICIENT PROCEDURE | N,N-DIMETHYLFORMAMIDE | METHANOL | ACYLATION | MULTIDISCIPLINARY SCIENCES | N-FORMYLATION | CARBOXAMIDES | CATALYST | SECONDARY-AMINES | SOLVENT | CARBOXYLIC-ACIDS | Amides | Heterocyclic amines | Amines | Molybdenum disulfide | Dimethylformamide | Acetylation
Journal Article
JOURNAL OF EXPERIMENTAL & CLINICAL CANCER RESEARCH, ISSN 1756-9966, 03/2019, Volume 38, Issue 1, pp. 108 - 108
Journal Article
by Ye, T and Wei, LW and Shi, J and Jiang, K and Xu, HZ and Hu, LL and Kong, LK and Zhang, Y and Meng, SS and Piao, HZ
BMC CANCER, ISSN 1471-2407, 07/2019, Volume 19, Issue 1, pp. 706 - 706
Background: Glioblastoma (GBM) is an extremely deadly form of brain cancer with limited treatment options and thus novel therapeutic modalities are necessary.... 
SURVIVAL | ACETYLATION | BEVACIZUMAB | STAT3 | RECURRENT GLIOBLASTOMA | COMBINATION | VORINOSTAT | Endoplasmic reticulum stress | RADIATION-THERAPY | TEMOZOLOMIDE | PHASE-II TRIAL | NF-kappa B | Glioma | ONCOLOGY | Sirt1 | Care and treatment | Genetic aspects | Research | Glioblastoma multiforme | Endoplasmic reticulum | Patient outcomes | Proteins | Gliomas | Growth | Vorinostat | Stress (Physiology) | Health aspects | Acetates | Apoptosis | NF-κB
Journal Article
Nature Communications, ISSN 2041-1723, 12/2019, Volume 10, Issue 1, pp. 625 - 12
The Elongator complex catalyzes posttranscriptional tRNA modifications by attaching carboxy-methyl (cm(5)) moieties to uridine bases located in the wobble... 
COMPLEX | COGNATE | CODE | PROTEIN | MECHANISM | MULTIDISCIPLINARY SCIENCES | ARCHITECTURE | HISTONE ACETYLATION | BINDING | GENOME | REVEALS | Post-transcription | tRNA Ala | tRNA | Methionine | Substrates | Lysine | Substrate specificity | N-Terminus | Uridine | Acetyltransferase | Catalysis | Elongation | Crystal structure | Index Medicus
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2019, Volume 9, Issue 1, pp. 1626 - 11
Recent concepts in evolutionary biology suggest that epigenetic mechanisms can translate environmental selection pressures into heritable changes in phenotype.... 
COEVOLUTION | DNA METHYLATION | CLONING | MULTIDISCIPLINARY SCIENCES | LARVAE | INSECTS | EXPRESSION | PROTEASE INHIBITORS | HEMOLYMPH | Adaptations | Larvae | Phenotypes | Proteinase | MiRNA | Parasite resistance | Innate immunity | Proteinase inhibitors | Parasites | Coevolution | Fungi | DNA methylation | Epigenetics | Heritability | Acetylation | Methylation
Journal Article