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1. Full Text Nicotine Promotes Initiation and Progression of KRAS-Induced Pancreatic Cancer via Gata6- Dependent Dedifferentiation of Acinar Cells in Mice
by Hermann, Patrick C and Sancho, Patricia and Cañamero, Marta and Martinelli, Paola and Madriles, Francesc and Michl, Patrick and Gress, Thomas and de Pascual, Ricardo and Gandia, Luis and Guerra, Carmen and Barbacid, Mariano and Wagner, Martin and Vieira, Catarina R and Aicher, Alexandra and Real, Francisco X and Sainz, Bruno and Heeschen, Christopher
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2014, Volume 147, Issue 5, pp. 1119 - 1133.e4
Gastroenterology and Hepatology | Metastasis | Pancreas | Progenitor Cells | Mouse Model | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Pancreatic Neoplasms - metabolism | Neoplastic Stem Cells - drug effects | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Carcinoma, Pancreatic Ductal - metabolism | Epithelial-Mesenchymal Transition - drug effects | Mice, 129 Strain | Carcinoma, Pancreatic Ductal - genetics | Neoplastic Stem Cells - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | Neoplastic Stem Cells - pathology | Pancreatic Neoplasms - chemically induced | Liver Neoplasms - secondary | Nicotinic Agonists - toxicity | Proto-Oncogene Proteins c-akt - metabolism | Neoplastic Cells, Circulating - drug effects | GATA6 Transcription Factor - deficiency | Liver Neoplasms - genetics | Pancreatic Neoplasms - pathology | Pancreas - pathology | alpha7 Nicotinic Acetylcholine Receptor - genetics | Pancreas - metabolism | Mice, Knockout | Acinar Cells - pathology | Signal Transduction - drug effects | Mice, Nude | Liver Neoplasms - metabolism | Cell Line, Tumor | alpha7 Nicotinic Acetylcholine Receptor - metabolism | Mutation | Carcinoma, Pancreatic Ductal - secondary | Carcinoma, Pancreatic Ductal - chemically induced | Neoplastic Cells, Circulating - pathology | Neoplastic Cells, Circulating - metabolism | Cell Transformation, Neoplastic - chemically induced | Extracellular Signal-Regulated MAP Kinases - metabolism | Carcinoma, Pancreatic Ductal - prevention & control | Tumor Suppressor Protein p53 - genetics | Transfection | Proto-Oncogene Proteins p21(ras) - deficiency | Gene Expression Regulation, Neoplastic - drug effects | Pancreatic Neoplasms - prevention & control | Tumor Cells, Cultured | Proto-Oncogene Proteins p21(ras) - metabolism | Acinar Cells - metabolism | Cell Dedifferentiation - drug effects | Metformin - pharmacology | Mice, Inbred C57BL | Tumor Suppressor Protein p53 - metabolism | Pancreas - drug effects | Pancreatic Neoplasms - genetics | Nicotine - toxicity | Cell Transformation, Neoplastic - metabolism | Proto-Oncogene Proteins c-myc - metabolism | GATA6 Transcription Factor - metabolism | Animals | Acinar Cells - drug effects | GATA6 Transcription Factor - genetics | Cell Transformation, Neoplastic - pathology | Pancreatic cancer | Stem cells | Nicotine | Index Medicus | Abridged Index Medicus
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2. Full Text How to make a functional β-cell
by Pagliuca, Felicia W and Melton, Douglas A
Development (Cambridge), ISSN 0950-1991, 06/2013, Volume 140, Issue 12, pp. 2472 - 2483
Cell | Mammalian metabolism | Diabetes mellitus | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Diabetes Mellitus - pathology | Acinar Cells - metabolism | Glucagon-Secreting Cells - cytology | Cell Proliferation | Pluripotent Stem Cells - cytology | Humans | Insulin Resistance | Cellular Reprogramming | Endoderm - metabolism | Pluripotent Stem Cells - metabolism | Insulin-Secreting Cells - metabolism | Animals | Endoderm - cytology | Diabetes Mellitus - therapy | Glucagon-Secreting Cells - metabolism | Glucose - metabolism | Cell Transdifferentiation | Cell Differentiation | Insulin-Secreting Cells - cytology | Insulin-Secreting Cells - transplantation | Acinar Cells - cytology | Index Medicus | β cell | Primers
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3. Full Text Induction of Expandable Tissue-Specific Stem/Progenitor Cells through Transient Expression of YAP/TAZ
by Panciera, Tito and Azzolin, Luca and Fujimura, Atsushi and Di Biagio, Daniele and Frasson, Chiara and Bresolin, Silvia and Soligo, Sandra and Basso, Giuseppe and Bicciato, Silvio and Rosato, Antonio and Cordenonsi, Michelangelo and Piccolo, Stefano
Cell stem cell, ISSN 1934-5909, 12/2016, Volume 19, Issue 6, pp. 725 - 737
Cell & Tissue Engineering | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Acinar Cells - metabolism | Reproducibility of Results | Cell Proliferation | Mice, Inbred C57BL | Organoids - cytology | Neurons - cytology | Neural Stem Cells - cytology | Organ Specificity | Phosphoproteins - metabolism | Stem Cells - metabolism | Mammary Glands, Animal - cytology | Transcription Factors - metabolism | Cell Lineage | Regeneration | Animals | Pancreas, Exocrine - cytology | Female | Cell Differentiation | Adaptor Proteins, Signal Transducing - metabolism | Neural Stem Cells - metabolism | Cell Cycle Proteins | Acinar Cells - cytology | Index Medicus
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4. Full Text Enhancer hijacking activates oncogenic transcription factor NR4A3 in acinic cell carcinomas of the salivary glands
by Haller, Florian and Bieg, Matthias and Will, Rainer and Körner, Cindy and Weichenhan, Dieter and Bott, Alexander and Ishaque, Naveed and Lutsik, Pavlo and Moskalev, Evgeny A and Mueller, Sarina K and Bähr, Marion and Woerner, Angelika and Kaiser, Birgit and Scherl, Claudia and Haderlein, Marlen and Kleinheinz, Kortine and Fietkau, Rainer and Iro, Heinrich and Eils, Roland and Hartmann, Arndt and Plass, Christoph and Wiemann, Stefan and Agaimy, Abbas
Nature communications, ISSN 2041-1723, 12/2019, Volume 10, Issue 1, pp. 368 - 368
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Chromatin - metabolism | Translocation, Genetic | Multigene Family | Salivary Glands - pathology | Salivary Proteins and Peptides - genetics | Cell Proliferation | Receptors, Steroid - metabolism | Salivary Gland Neoplasms - genetics | Epigenesis, Genetic | Humans | Carcinoma, Acinar Cell - pathology | Gene Expression Regulation, Neoplastic | Male | Genetic Loci | DNA-Binding Proteins - metabolism | Carcinoma, Acinar Cell - metabolism | Receptors, Thyroid Hormone - genetics | Salivary Gland Neoplasms - metabolism | Female | Chromatin - chemistry | Acinar Cells - metabolism | Carcinoma, Acinar Cell - genetics | DNA-Binding Proteins - genetics | Salivary Proteins and Peptides - metabolism | Chromosomes, Human, Pair 4 - metabolism | Chromosomes, Human, Pair 9 - metabolism | Receptors, Thyroid Hormone - metabolism | Chromosomes, Human, Pair 4 - chemistry | Acinar Cells - pathology | Animals | Enhancer Elements, Genetic | Receptors, Steroid - genetics | Chromosomes, Human, Pair 9 - chemistry | Mice | Primary Cell Culture | Salivary Gland Neoplasms - pathology | Salivary Glands - metabolism | Cohort Studies | Binding | Cell proliferation | Chromatin | Transcription factors | Carcinoma | Salivary glands | Pathogenesis | Salivary gland | Chromosome 4 | Gene expression | Phosphoproteins | Clusters | Saliva | Index Medicus
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5. Full Text Most Nonparotid “Acinic Cell Carcinomas” Represent Mammary Analog Secretory Carcinomas
by Bishop, Justin A and Yonescu, Raluca and Batista, Denise and Eisele, David W and Westra, William H
The American journal of surgical pathology, ISSN 0147-5185, 07/2013, Volume 37, Issue 7, pp. 1053 - 1057
S100 | Minor salivary gland carcinoma | Acinic cell carcinoma | ETV6-NTRK3 translocation | Mammaglobin | Mammary analog secretory carcinoma | Pathology | Life Sciences & Biomedicine | Surgery | Science & Technology | Translocation, Genetic | Humans | Middle Aged | Carcinoma, Acinar Cell - pathology | Male | Mouth Neoplasms - metabolism | Breast Neoplasms - metabolism | Submandibular Gland Neoplasms - metabolism | Young Adult | Carcinoma, Acinar Cell - metabolism | Salivary Gland Neoplasms - metabolism | Aged, 80 and over | Biomarkers, Tumor - metabolism | Adult | Female | Retrospective Studies | Acinar Cells - metabolism | Submandibular Gland Neoplasms - pathology | S100 Proteins - metabolism | Acinar Cells - pathology | Carcinoma, Acinar Cell - surgery | Breast Neoplasms - pathology | Mammaglobin A - metabolism | Mouth Neoplasms - pathology | Aged | Salivary Gland Neoplasms - pathology | Salivary Gland Neoplasms - surgery | Index Medicus
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6. Full Text Aging dependent effect of nuclear tau
by Gil, Laura and Federico, Concetta and Pinedo, Fernando and Bruno, Francesca and Rebolledo, Ana B and Montoya, Juan J and Olazabal, Isabel M and Ferrer, Isidre and Saccone, Salvatore
Brain research, ISSN 0006-8993, 12/2017, Volume 1677, pp. 129 - 137
Breast acinar cells | AT100 | Alzheimer disease | Neurons | Epithelial cells | Aging | Nuclear tau | Confocal microscopy | Aging marker | Colorectal mucosa | Cancer | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Pyramidal Cells - metabolism | Intestinal Mucosa - metabolism | Phosphorylation | Epithelial Cells - metabolism | Humans | Middle Aged | Carcinoma, Acinar Cell - pathology | tau Proteins - metabolism | Breast - metabolism | Alzheimer Disease - pathology | Breast Neoplasms - metabolism | Young Adult | Carcinoma, Acinar Cell - metabolism | Cell Nucleus - metabolism | Heterochromatin - metabolism | Adult | Breast - pathology | Severity of Illness Index | Cellular Senescence - physiology | Epithelial Cells - pathology | Hippocampus - pathology | Aging - pathology | Hippocampus - metabolism | Breast Neoplasms - pathology | Alzheimer Disease - metabolism | Fluorescent Antibody Technique | Pyramidal Cells - pathology | Aging - metabolism | Intestinal Mucosa - pathology | Alzheimer's disease | Fluorescein | Colorectal cancer | Index Medicus
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7. Full Text PTPN14 is required for the density-dependent control of YAP1
by Wang, Wenqi and Huang, Jun and Wang, Xin and Yuan, Jingsong and Li, Xu and Feng, Lin and Park, Jae-Il and Chen, Junjie
Genes & development, ISSN 0890-9369, 09/2012, Volume 26, Issue 17, pp. 1959 - 1971
YAP1 | CRL2 | Hippo pathway | PTPN14 | Tumor suppressor | Cell density | Genetics & Heredity | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Cell Biology | Protein Structure, Tertiary | Acinar Cells - metabolism | Cell Line | Phosphorylation | Cell Count | Humans | Ubiquitin-Protein Ligases - metabolism | Cytoplasm - metabolism | Phosphoproteins - genetics | Phosphoproteins - metabolism | Amino Acid Motifs | Gene Knockdown Techniques | Protein Transport | Acinar Cells - pathology | Cell Nucleus - metabolism | Protein Tyrosine Phosphatases, Non-Receptor - genetics | Protein Tyrosine Phosphatases, Non-Receptor - metabolism | Adaptor Proteins, Signal Transducing - genetics | HEK293 Cells | Cell Line, Tumor | Receptors, Cytokine - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Cell Transformation, Neoplastic - pathology | Acinar Cells - cytology | Tumor suppressor genes | Usage | Gene targeting | Research | Translocation (Genetics) | Index Medicus | cell density | hippo pathway | CRL2LRR1 | tumor suppressor | Research Paper
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8. Full Text Oncogenic KRAS Reduces Expression of FGF21 in Acinar Cells to Promote Pancreatic Tumorigenesis in Mice on a High-Fat Diet
by Luo, Yongde and Yang, Yaying and Liu, Muyun and Wang, Dan and Wang, Feng and Bi, Yawei and Ji, Juntao and Li, Suyun and Liu, Yan and Chen, Rong and Huang, Haojie and Wang, Xiaojie and Swidnicka-Siergiejko, Agnieszka K and Janowitz, Tobias and Beyaz, Semir and Wang, Guoqiang and Xu, Sulan and Bialkowska, Agnieszka B and Luo, Catherine K and Pin, Christoph L and Liang, Guang and Lu, Xiongbin and Wu, Maoxin and Shroyer, Kenneth R and Wolff, Robert A and Plunkett, William and Ji, Baoan and Li, Zhaoshen and Li, Ellen and Li, Xiaokun and Yang, Vincent W and Logsdon, Craig D and Abbruzzese, James L and Lu, Weiqin
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 11/2019, Volume 157, Issue 5, pp. 1413 - 1428.e11
Gene Regulation | KLB | Signaling | FGFR1 | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Pancreatic Intraductal Neoplasms - pathology | Pancreatic Intraductal Neoplasms - genetics | Pancreatic Neoplasms - metabolism | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Carcinoma, Pancreatic Ductal - metabolism | Gene Expression Regulation, Neoplastic | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Fibroblast Growth Factors - genetics | Carcinoma, Pancreatic Ductal - prevention & control | PPAR gamma - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Tumor Suppressor Protein p53 - genetics | Carcinoma, Pancreatic Ductal - genetics | Fibroblast Growth Factors - metabolism | Pancreatitis - genetics | Pancreatic Cyst - pathology | Cell Transformation, Neoplastic - genetics | Diet, High-Fat | Membrane Proteins - metabolism | Pancreatic Neoplasms - prevention & control | Pancreatic Intraductal Neoplasms - prevention & control | PPAR gamma - genetics | Pancreatic Intraductal Neoplasms - metabolism | Proto-Oncogene Proteins p21(ras) - metabolism | Acinar Cells - metabolism | Signal Transduction | Membrane Proteins - genetics | Down-Regulation | Pancreatic Neoplasms - pathology | Tumor Suppressor Protein p53 - metabolism | Mice, Transgenic | Pancreatic Neoplasms - genetics | Transcription Factors - genetics | Carcinoma, Pancreatic Ductal - pathology | Cell Transformation, Neoplastic - metabolism | Pancreatic Cyst - genetics | Transcription Factors - metabolism | Acinar Cells - pathology | Animals | Pancreatitis - pathology | Mutation | Pancreatitis - metabolism | Cell Transformation, Neoplastic - pathology | Pancreatic Cyst - metabolism | Analysis | Fibroblast growth factors | Tumors | Index Medicus | Abridged Index Medicus | signaling | gene regulation
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9. Full Text YAP1 and TAZ Control Pancreatic Cancer Initiation in Mice by Direct Up-Regulation of JAK–STAT3 Signaling
by Gruber, Ralph and Panayiotou, Richard and Nye, Emma and Spencer-Dene, Bradley and Stamp, Gordon and Behrens, Axel
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2016, Volume 151, Issue 3, pp. 526 - 539
Gastroenterology and Hepatology | Inflammation | PanINs | Pancreatic Cancer Progression | Mouse Model | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Acinar Cells - metabolism | Up-Regulation | Pancreatic Neoplasms - etiology | Pancreatic Neoplasms - pathology | Risk Factors | Carcinogenesis - genetics | Pancreas - pathology | Pancreatic Neoplasms - genetics | Janus Kinases - metabolism | Signal Transduction - genetics | Pancreatitis - complications | Pancreatitis - chemically induced | Phosphoproteins - metabolism | Transcription Factors - metabolism | Animals | Pancreatitis - pathology | Mice | Mutation | Adaptor Proteins, Signal Transducing - metabolism | Cell Cycle Proteins | STAT3 Transcription Factor - metabolism | Proto-Oncogene Proteins p21(ras) - metabolism | Animal genetics | Medical colleges | Pancreatic cancer | Pancreatitis | Index Medicus | Abridged Index Medicus | Original Research | HBSS, Hank’s balanced salt solution | ADM, acinar-to-ductal metaplasia | PDAC, pancreatic ductal adenocarcinoma | TGFα, transforming growth factor–α | PBS, phosphate-buffered saline | PanIN, pancreatic intraepithelial neoplasia | IL, interleukin | ES, embryonic stem cell | CMV, cytomegalovirus
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