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The Plant Cell, ISSN 1040-4651, 5/2007, Volume 19, Issue 5, pp. 1603 - 1616
Journal Article
Gastroenterology, ISSN 0016-5085, 2008, Volume 134, Issue 5, pp. 1424 - 1435
Background & Aims: The isolation and culture of primary enteric neurons is a difficult process and yields a small number of neurons. We developed fetal and... 
Gastroenterology and Hepatology | SURVIVAL | NERVOUS-SYSTEM | SEROTONIN TRANSPORTER | SMOOTH-MUSCLE | GDNF | PATHWAY | NEUROTROPHIC FACTOR | TRANSGENIC MOUSE | RECEPTORS | GASTROENTEROLOGY & HEPATOLOGY | MICE LACKING | Immunohistochemistry | Neuroglia - transplantation | ELAV-Like Protein 4 | Proto-Oncogene Proteins c-ret - biosynthesis | Serotonin - genetics | Membrane Glycoproteins - biosynthesis | Colon - innervation | Synaptophysin - genetics | S100 Proteins - genetics | Muscle, Smooth - physiology | Neurons - metabolism | S100 Proteins - biosynthesis | Serotonin - biosynthesis | Peripherins | Muscle, Smooth - innervation | S100 Calcium Binding Protein beta Subunit | Enteric Nervous System - metabolism | Glial Cell Line-Derived Neurotrophic Factor - biosynthesis | Glial Cell Line-Derived Neurotrophic Factor - genetics | Mice, Transgenic | Xenopus Proteins | Reverse Transcriptase Polymerase Chain Reaction | Ubiquitin Thiolesterase - genetics | Blotting, Western | Nerve Growth Factors - genetics | Synaptophysin - biosynthesis | Mice | ELAV Proteins - genetics | Nestin | Colon - embryology | Gastrointestinal Motility - physiology | Neurons - cytology | Actins - genetics | RNA - genetics | tau Proteins - biosynthesis | Neuroglia - cytology | tau Proteins - genetics | Gene Expression Regulation, Developmental | Intermediate Filament Proteins - genetics | Nerve Tissue Proteins - biosynthesis | Female | Isometric Contraction - physiology | Cell Line | Actins - biosynthesis | ELAV Proteins - biosynthesis | Mice, Inbred C57BL | Enteric Nervous System - embryology | Intermediate Filament Proteins - biosynthesis | Nerve Growth Factors - biosynthesis | Nerve Tissue Proteins - genetics | Colon - surgery | Membrane Glycoproteins - genetics | Pregnancy | Animals | Neuroglia - metabolism | Ubiquitin Thiolesterase - biosynthesis | Proto-Oncogene Proteins c-ret - genetics | Polymerase chain reaction | Physiological aspects | Biological response modifiers | Neurons | Index Medicus | Abridged Index Medicus | differentiation | Enteric neurons | proliferation | nNOS | Immorto | Piebald
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 12, pp. e116142 - e116142
Rationale: Replication deficient adenoviruses (Ad) vectors are common tools in gene therapy. Since Ad vectors are known to activate innate and adaptive... 
PATHOGENESIS | MATRIX | ACTIVATION | HIPPEL-LINDAU-PROTEIN | MULTIDISCIPLINARY SCIENCES | IN-VIVO | DISEASE | ANIMAL-MODELS | MEDIATED GENE-TRANSFER | IDIOPATHIC PULMONARY-FIBROSIS | EXPRESSION | Male | Interleukin-13 - biosynthesis | Lung Injury - virology | Proliferating Cell Nuclear Antigen - biosynthesis | Pulmonary Fibrosis - virology | Interleukin-16 - biosynthesis | Bronchoalveolar Lavage Fluid - cytology | Interleukin-1beta - biosynthesis | Adenoviridae - immunology | Integrins - biosynthesis | Matrix Metalloproteinase 2 - biosynthesis | Transforming Growth Factor beta1 - biosynthesis | Disease Models, Animal | Matrix Metalloproteinase 9 - biosynthesis | Actins - biosynthesis | Mice, Inbred C57BL | Inflammation - virology | Pulmonary Fibrosis - immunology | Inflammation - immunology | Bleomycin - administration & dosage | Collagen Type I - biosynthesis | Animals | Interleukin-1alpha - biosynthesis | Mice | Bronchoalveolar Lavage Fluid - chemistry | Wnt Proteins - biosynthesis | Lung Injury - immunology | RNA | Collagen | Adenoviruses | Fibrosis | Bone morphogenetic proteins | Transforming growth factors | Gene therapy | Health aspects | Integrins | Pediatrics | Collagen (type I) | Disease | Pathogenesis | Lung | Immunity | Proteins | Bleomycin | Proliferating cell nuclear antigen | Physiology | Chronic obstructive pulmonary disease | Drug dosages | Deoxyribonucleic acid--DNA | Cytokines | Bronchus | Interleukin 13 | Tumor necrosis factor-α | Gene expression | Interleukin 16 | DNA viruses | Vectors (Biology) | Pulmonary fibrosis | Interleukin 10 | Wnt protein | Laboratories | Staining | Critical care | Inflammatory response | Viruses | mRNA | Infections | Kinases | Tissues | Bronchoalveolar lavage | Rodents | Alveoli | Trachea | Injuries | Expression vectors | Lung diseases | Inflammation | Adaptive immunity | Gelatinase B | Gelatinase A | Signaling | Lungs | Molecular biology | Immunofluorescence | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Cell Cycle, ISSN 1538-4101, 06/2010, Volume 9, Issue 12, pp. 2423 - 2433
Loss of stromal caveolin 1 (Cav-1) is a novel biomarker for cancer-associated fibroblasts that predicts poor clinical outcome in breast cancer and DCIS... 
Proteins | Binding | Landes | Calcium | Biology | Bioscience | Cell | Cycle | Organogenesis | Cancer | Caveolin-1 | Lysosomal degradation | Chloroquine | Myofibroblast | Tumor stroma | TGFbeta signaling | Cancer associated fibroblasts | Autophagy | ACTIVATION | autophagy | TGF-BETA | lysosomal degradation | myofibroblast | SYSTEMIC-SCLEROSIS | cancer associated fibroblasts | CELL BIOLOGY | chloroquine | caveolin-1 | GROWTH | GENE-EXPRESSION | STROMAL FIBROBLASTS | VIMENTIN EXPRESSION | tumor stroma | TENASCIN | MMP-9 | PROGRESSION | Prognosis | Extracellular Matrix Proteins - biosynthesis | Coculture Techniques | Humans | Carcinoma, Intraductal, Noninfiltrating - metabolism | Transforming Growth Factor beta - biosynthesis | Autophagy - drug effects | Actins - genetics | Breast Neoplasms - metabolism | Chloroquine - pharmacology | Smad2 Protein - biosynthesis | Vimentin - genetics | Female | Microfilament Proteins - genetics | Fibroblasts - metabolism | Actins - biosynthesis | Calcium-Binding Proteins - biosynthesis | Vimentin - biosynthesis | Extracellular Matrix Proteins - genetics | Caveolin 1 - genetics | Caveolin 1 - metabolism | Microfilament Proteins - biosynthesis | Phenotype | Cell Line, Tumor | Biomarkers, Tumor - genetics | Transforming Growth Factor beta - metabolism | Biomarkers, Tumor - biosynthesis | Calcium-Binding Proteins - genetics | Index Medicus
Journal Article
Biochemical Pharmacology, ISSN 0006-2952, 08/2015, Volume 96, Issue 3, pp. 247 - 255
The 5-lipoxygenase product 5-oxo-6,8,11,14-eicosatetraenoic acid (5-oxo-ETE) is the most powerful human eosinophil chemoattractant among lipid mediators and... 
Eicosanoids | Inflammation | 5-Lipoxygenase | OXE receptor | Neutrophils | Eosinophils | 5-OXO-6,8,11,14-EICOSATETRAENOIC ACID | OXIDATIVE STRESS | RADICAL PRODUCTION | RECEPTOR | POTENT CHEMOATTRACTANT | HUMAN EOSINOPHILS | LIQUID-CHROMATOGRAPHY | 5-OXO-EICOSATETRAENOATE | PHARMACOLOGY & PHARMACY | LEUKOTRIENE | 5-OXO-EICOSANOIDS | Asthma - metabolism | Eosinophils - metabolism | Receptors, Eicosanoid - antagonists & inhibitors | Eosinophils - drug effects | Arachidonate 5-Lipoxygenase - genetics | Humans | Actins - metabolism | Male | Arachidonic Acids - biosynthesis | Actins - genetics | Cynodon - chemistry | Allergens - immunology | Asthma - immunology | Female | Bronchoalveolar Lavage Fluid - cytology | Eosinophils - pathology | Chemotaxis - immunology | Cynodon - immunology | Benzeneacetamides - pharmacology | Neutrophils - metabolism | Disease Models, Animal | Neutrophils - pathology | Cats | Gene Expression | Arachidonate 5-Lipoxygenase - metabolism | Benzothiazoles - pharmacology | Neutrophils - drug effects | Polymerization | Chemotaxis - drug effects | Arachidonic Acids - pharmacology | Asthma - chemically induced | Asthma - genetics | Leukotriene B4 - pharmacology | Animals | Receptors, Eicosanoid - metabolism | Prostaglandin D2 - pharmacology | Receptors, Eicosanoid - genetics | Primary Cell Culture | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2010, Volume 107, Issue 29, pp. 12828 - 12833
While many functions of the p53 tumor suppressor affect mitochondrial processes, the role of altered mitochondrial physiology in a modulation of p53 response... 
Up regulation | Pyrimidines | HCT116 cells | Epithelial cells | Cell lines | HeLa cells | Actins | Biosynthesis | Respiration | Apoptosis | p53 tumor suppressor | NQO1 and NQO2 | Dihydroorotate dehydrogenase | Mitochondrial electron transport chain | dihydroorotate dehydrogenase | mitochondrial electron transport chain | ACTIVATION | PROTEASOMAL DEGRADATION | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | PROTHYMOSIN-ALPHA | apoptosis | HYPOXIA | ARREST | INHIBITION | TUMOR-SUPPRESSOR | S-PHASE | STRESS | Mitochondria - enzymology | Electron Transport Complex III - metabolism | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | Pyrimidines - biosynthesis | Membrane Potential, Mitochondrial - drug effects | Oxidoreductases Acting on CH-CH Group Donors - antagonists & inhibitors | Oxidoreductases Acting on CH-CH Group Donors - deficiency | Protein Processing, Post-Translational - drug effects | Cell Respiration - drug effects | Methacrylates - pharmacology | Potassium Cyanide - pharmacology | Quinone Reductases - metabolism | Tumor Suppressor Protein p53 - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Isoxazoles - pharmacology | Up-Regulation - drug effects | Electron Transport Complex III - antagonists & inhibitors | Signal Transduction - drug effects | Cell Line, Tumor | NAD(P)H Dehydrogenase (Quinone) - metabolism | Thiazoles - pharmacology | Proteasome Endopeptidase Complex - metabolism | Observations | Health aspects | Signal transduction | Tissue | Enzymes | DNA damage | Electron transfer | Organic chemicals | Tumors | Index Medicus | Phosphorylation | Carcinoma | Serine | Data processing | Nuclei | p53 protein | Electron transport chain | Mitochondria | Cytochrome bc1 | NAD(P)H dehydrogenase (quinone) | Hypoxia | Tumor suppressor genes | Intoxication | pyrimidines | Electron transport | p14ARF protein | Biological Sciences
Journal Article
Pediatric Blood & Cancer, ISSN 1545-5009, 08/2015, Volume 62, Issue 8, pp. 1414 - 1420
BackgroundInfantile hemangioma (IH) is a benign vascular neoplasm resulting from the abnormal proliferation of endothelial cells and pericytes in infants.... 
eNOS | infantile hemangioma | VEGF | propranolol | Infantile hemangioma | Propranolol | ENOS | ANGIOGENESIS | NITRIC-OXIDE SYNTHASE | PANCREATIC-CANCER | ACTIN DYNAMICS | PROGENITOR-CELL | ONCOLOGY | SIGNALING PATHWAY | IN-VIVO | INFANTILE HEMANGIOMAS | PEDIATRICS | HEMATOLOGY | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | Norepinephrine - pharmacology | Vascular Endothelial Growth Factor A - biosynthesis | Phosphorylation | Humans | Nitric Oxide Synthase Type III - biosynthesis | Propranolol - therapeutic use | Infant | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Proliferating Cell Nuclear Antigen - biosynthesis | Neoplasm Invasiveness - pathology | Angiogenesis Inhibitors - therapeutic use | Nitric Oxide Synthase Type III - metabolism | Chromones - pharmacology | Hemangioma - drug therapy | Phosphatidylinositol 3-Kinases - biosynthesis | Proto-Oncogene Proteins c-akt - metabolism | Infant, Newborn | Adrenergic beta-Antagonists - therapeutic use | NG-Nitroarginine Methyl Ester - pharmacology | Nitric Oxide - biosynthesis | Cyclin A2 - biosynthesis | Cyclin-Dependent Kinase Inhibitor p21 - biosynthesis | Endothelial Cells - metabolism | Pericytes - metabolism | Cells, Cultured | Morpholines - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Cinnamates - pharmacology | Nitric Oxide Synthase Type III - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - biosynthesis | Vascular Endothelial Growth Factor Receptor-1 - pharmacology | Cell Cycle Checkpoints - drug effects | Neovascularization, Pathologic - drug therapy | Cell Proliferation - drug effects | Cyclin D2 - biosynthesis | Proteins | Propranolol hydrochloride | Hemangioma | Vascular endothelial growth factor | Cell proliferation | Regression | AKT protein | Infants | Endothelial cells | 1-Phosphatidylinositol 3-kinase | Cyclin D2 | Vascular endothelial growth factor receptors | Antiangiogenics | Cell cycle | Cofilin | Pericytes | Norepinephrine | Inhibition | Pretreatment | Cancer | Index Medicus
Journal Article