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Journal of Neuroscience, ISSN 0270-6474, 2013, Volume 33, Issue 32, pp. 13204 - 13224
The spatiotemporal organization of neurotransmitter receptors in postsynaptic membranes is a fundamental determinant of synaptic transmission and information... 
QUANTAL TRANSMISSION | NMDA RECEPTORS | IONOTROPIC GLUTAMATE RECEPTORS | EXCITATORY SYNAPSES | ACTIVITY-DEPENDENT REGULATION | MONTE-CARLO-SIMULATION | IMMUNOGOLD LOCALIZATION | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | TERM SYNAPTIC PLASTICITY | POSTSYNAPTIC DENSITY | Disks Large Homolog 4 Protein | Action Potentials - genetics | Calcium Channels - metabolism | Embryo, Mammalian | Male | Neurons - cytology | Protein Transport - physiology | Green Fluorescent Proteins - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Nanostructures | Excitatory Postsynaptic Potentials - drug effects | Synapses - metabolism | Stochastic Processes | Homer Scaffolding Proteins | Female | Membrane Proteins - metabolism | Calcium Channels - genetics | Intracellular Signaling Peptides and Proteins - genetics | Action Potentials - drug effects | Excitatory Postsynaptic Potentials - genetics | Receptors, AMPA - metabolism | Green Fluorescent Proteins - metabolism | Membrane Proteins - genetics | Cells, Cultured | Rats | Synapses - ultrastructure | Hippocampus - cytology | Mutation - genetics | Synaptic Membranes - ultrastructure | Protein Transport - genetics | GABA Antagonists - pharmacology | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Models, Biological | Picrotoxin - pharmacology | Synaptic Membranes - metabolism | Index Medicus
Journal Article
Neuron, ISSN 0896-6273, 09/2013, Volume 79, Issue 6, pp. 1152 - 1168
Genetic variation in neuregulin and its ErbB4 receptor has been linked to schizophrenia, although little is known about how they contribute to the disease... 
PARVALBUMIN-POSITIVE INTERNEURONS | GLUTAMIC-ACID DECARBOXYLASE | NEUREGULIN 3 NRG3 | CORTICAL GABAERGIC INTERNEURONS | GENE-EXPRESSION | FUNCTIONAL CONNECTIVITY | PSYCHIATRIC-DISORDERS | NEUROSCIENCES | PREFRONTAL CORTEX | CEREBRAL-CORTEX | WORKING-MEMORY | Interneurons - physiology | Receptor, Epidermal Growth Factor - genetics | Action Potentials - genetics | Synaptic Transmission - genetics | Parvalbumins - metabolism | Receptor, Epidermal Growth Factor - deficiency | Male | Schizophrenia - complications | Green Fluorescent Proteins - genetics | Schizophrenia - pathology | Statistics as Topic | Schizophrenia - genetics | Social Behavior | Cognition Disorders - etiology | Disease Models, Animal | Animals, Newborn | Maze Learning - physiology | Brain - physiopathology | RNA, Untranslated | Electroporation | Mice, Transgenic | Cognition Disorders - genetics | Transcription Factors - genetics | Mutation - genetics | Nerve Tissue Proteins - genetics | Action Potentials - physiology | Nerve Tissue Proteins - metabolism | Proteins - genetics | Motor Activity - genetics | LIM-Homeodomain Proteins - genetics | Patch-Clamp Techniques | Phenotype | Animals | Proteins - metabolism | Brain - pathology | Glutamate Decarboxylase - metabolism | Mice | Receptors, GABA-A - metabolism | In Vitro Techniques | Receptor, ErbB-4 | Schizophrenia | Disease susceptibility | Genetic aspects | Biological products | Tissue engineering | Brain | Ultrasonic imaging | Rodents | Migration | Colleges & universities | Kinases | University colleges | Index Medicus
Journal Article
Nature Neuroscience, ISSN 1097-6256, 01/2013, Volume 16, Issue 1, pp. 64 - 70
Cerebrocortical injuries such as stroke are a major source of disability. Maladaptive consequences can result from post-injury local reorganization of cortical... 
STROKE | IN-VITRO | PHOTOTHROMBOTIC BRAIN INFARCTION | EPILEPTIFORM ACTIVITY | BIOCYTIN | RATS | EPILEPSY | MODEL | RELAY NEURONS | EPILEPTOGENESIS | NEUROSCIENCES | Membrane Potentials - genetics | Electric Stimulation | Age Factors | Ion Channels - genetics | Brain Injuries - complications | Electroencephalography | Optogenetics | Neural Pathways - physiology | Cerebral Cortex - physiopathology | Thalamus - physiology | Light | Neurons - physiology | Lysine - metabolism | Neurons - drug effects | Spectrum Analysis | Action Potentials - drug effects | Cyclic Nucleotide-Gated Cation Channels - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Disease Models, Animal | Animals, Newborn | Neural Inhibition - genetics | Lysine - analogs & derivatives | Wakefulness - genetics | Cyclic Nucleotide-Gated Cation Channels - genetics | Rats | Biophysics | Rats, Sprague-Dawley | Action Potentials - physiology | Biophysical Phenomena - physiology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - genetics | Microscopy, Confocal | Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels | Patch-Clamp Techniques | Animals | Ion Channels - metabolism | Electric Capacitance | Seizures - etiology | Luminescent Proteins - genetics | Models, Neurological | In Vitro Techniques | Brain Injuries - pathology | Luminescent Proteins - metabolism | Brain | Care and treatment | Physiological aspects | Thalamus | Seizures (Medicine) | Research | Injuries | Index Medicus
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2013, Volume 33, Issue 10, pp. 4456 - 4467
Journal Article
CELL, ISSN 0092-8674, 10/2004, Volume 119, Issue 1, pp. 19 - 31
Ca(v)1.2, the cardiac L-type calcium channel, is important for excitation and contraction of the heart. Its role in other tissues is unclear. Here we present... 
FUNCTIONAL EXPRESSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | HUMAN GENETIC-DISEASE | SUDDEN CARDIAC DEATH | CA2+ CHANNEL | MOLECULAR DETERMINANTS | LONG-QT SYNDROME | LANGE-NIELSEN-SYNDROME | PERVASIVE DEVELOPMENTAL DISORDERS | STATIONARY NIGHT BLINDNESS | POTASSIUM CHANNEL | CELL BIOLOGY | Limb Deformities, Congenital - genetics | Action Potentials - genetics | Autistic Disorder - physiopathology | Calcium - metabolism | Humans | Genetic Diseases, Inborn - genetics | Male | Cell Membrane - genetics | Arrhythmias, Cardiac - physiopathology | Mutation, Missense - genetics | Oocytes | Brain - metabolism | Genetic Diseases, Inborn - complications | Female | Cell Membrane - metabolism | Neurons - metabolism | Child | Abnormalities, Multiple - genetics | Arrhythmias, Cardiac - genetics | Infant, Newborn | Brain Chemistry - genetics | CHO Cells | Autistic Disorder - genetics | Heart - physiopathology | Cricetinae | Brain - physiopathology | Xenopus laevis | Abnormalities, Multiple - physiopathology | Syndrome | Animals | Calcium Channels, L-Type - genetics | Pedigree | Myocytes, Cardiac - metabolism | Limb Deformities, Congenital - complications | Arrhythmias, Cardiac - complications | Genetic Diseases, Inborn - physiopathology | Mice | Calcium Channels, L-Type - metabolism | Autistic Disorder - complications | Calcium Signaling - genetics | Index Medicus
Journal Article
Nature Neuroscience, ISSN 1097-6256, 01/2010, Volume 13, Issue 1, pp. 76 - 83
Journal Article
Journal Article