X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (65605) 65605
Book Chapter (430) 430
Book / eBook (14) 14
Dissertation (7) 7
Conference Proceeding (5) 5
Newspaper Article (3) 3
Publication (3) 3
Book Review (2) 2
Newsletter (2) 2
Government Document (1) 1
Magazine Article (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
science & technology (58774) 58774
life sciences & biomedicine (47063) 47063
humans (36340) 36340
animals (35310) 35310
mice (21084) 21084
cell biology (16524) 16524
gene expression (15440) 15440
biochemistry & molecular biology (15383) 15383
proteins (14516) 14516
transcriptional activation (14178) 14178
signal transduction (13239) 13239
science & technology - other topics (11599) 11599
multidisciplinary sciences (11562) 11562
research article (11516) 11516
immunology (11393) 11393
male (10044) 10044
female (9585) 9585
kinases (8606) 8606
research (8560) 8560
apoptosis (8530) 8530
medicine (8470) 8470
activation (8469) 8469
phosphorylation (8392) 8392
cell line (8356) 8356
cells, cultured (8123) 8123
science (8099) 8099
oncology (7684) 7684
enzyme activation (7361) 7361
cell line, tumor (7077) 7077
transcription factors - metabolism (7050) 7050
genes (6919) 6919
transcription factors (6909) 6909
genetic aspects (6892) 6892
molecular sequence data (6855) 6855
cell activation (6699) 6699
promoter regions, genetic (6558) 6558
inflammation (6340) 6340
biology (6251) 6251
analysis (6212) 6212
rodents (6095) 6095
mutation (5922) 5922
cancer (5869) 5869
base sequence (5852) 5852
cytokines (5716) 5716
protein binding (5696) 5696
mice, inbred c57bl (5672) 5672
physiological aspects (5666) 5666
gene expression regulation (5451) 5451
transcription factors - genetics (5434) 5434
transcription, genetic (5424) 5424
rats (5208) 5208
dna-binding proteins - metabolism (4918) 4918
cell proliferation (4904) 4904
transfection (4897) 4897
binding sites (4740) 4740
biochemistry (4407) 4407
health aspects (4402) 4402
transcription (4315) 4315
mice, knockout (4264) 4264
signaling (4070) 4070
lymphocytes (4057) 4057
amino acid sequence (4053) 4053
genetics & heredity (4015) 4015
lymphocyte activation (3962) 3962
rna, messenger - metabolism (3933) 3933
t cells (3912) 3912
enzyme activation - drug effects (3740) 3740
dna-binding proteins - genetics (3732) 3732
deoxyribonucleic acid--dna (3712) 3712
rna, messenger - genetics (3677) 3677
transcription activation (3642) 3642
immune system (3602) 3602
genetics (3591) 3591
cell cycle (3548) 3548
lymphocytes t (3547) 3547
life sciences (3527) 3527
signal transduction - drug effects (3466) 3466
tumors (3451) 3451
cell differentiation (3321) 3321
physiology (3285) 3285
dna (3241) 3241
genetic transcription (3229) 3229
macrophages (3166) 3166
immune response (3144) 3144
rna (3112) 3112
microbiology (3110) 3110
enzymes (3089) 3089
nf-kappa b - metabolism (3066) 3066
neurosciences (3054) 3054
medicine, research & experimental (2984) 2984
research & experimental medicine (2983) 2983
abridged index medicus (2964) 2964
blotting, western (2894) 2894
molecular biology (2847) 2847
reverse transcriptase polymerase chain reaction (2831) 2831
flow cytometry (2798) 2798
stem cells (2791) 2791
studies (2723) 2723
metabolism (2719) 2719
genomes (2710) 2710
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (66052) 66052
Japanese (219) 219
Chinese (26) 26
Norwegian (24) 24
Dutch (15) 15
French (15) 15
German (13) 13
Portuguese (10) 10
Russian (7) 7
Polish (3) 3
Spanish (3) 3
Croatian (1) 1
Hungarian (1) 1
Italian (1) 1
Korean (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Nature immunology, ISSN 1529-2916, 03/2010, Volume 11, Issue 5, pp. 411 - 418
.... and its downstream target, the transcription factor XBP1. Previously described ER-stress target genes of XBP1 were not induced by TLR signaling... 
Life Sciences & Biomedicine | Immunology | Science & Technology | RNA, Small Interfering - genetics | Endoribonucleases - genetics | Tularemia - metabolism | Toll-Like Receptor 2 - genetics | Membrane Glycoproteins - metabolism | Lipopeptides - pharmacology | Transcriptional Activation - drug effects | NADPH Oxidases - metabolism | Tularemia - genetics | Protein Splicing - genetics | Transcriptional Activation - immunology | X-Box Binding Protein 1 | Protein Splicing - drug effects | NADPH Oxidases - genetics | Stress, Physiological - drug effects | TNF Receptor-Associated Factor 6 - genetics | Transcription Factor CHOP - biosynthesis | Transcription Factors - immunology | Cytokines - genetics | Protein-Serine-Threonine Kinases - metabolism | DNA-Binding Proteins - immunology | Endoribonucleases - metabolism | Macrophages - pathology | Stress, Physiological - genetics | Myeloid Differentiation Factor 88 - genetics | Toll-Like Receptor 4 - genetics | Signal Transduction - genetics | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Macrophages - metabolism | Signal Transduction - drug effects | Tunicamycin - pharmacology | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Mice | Stress, Physiological - immunology | Endoribonucleases - immunology | Transcription Factor CHOP - genetics | NADPH Oxidases - immunology | DNA-Binding Proteins - metabolism | Signal Transduction - immunology | Macrophages - virology | Mice, Mutant Strains | Tularemia - immunology | Membrane Glycoproteins - immunology | Macrophages - immunology | Cytokines - immunology | Cell Line | Francisella tularensis - immunology | Protein Splicing - immunology | Protein-Serine-Threonine Kinases - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Immunity, Innate | NADPH Oxidase 2 | Regulatory Factor X Transcription Factors | Mice, Inbred C3H | Membrane Glycoproteins - genetics | Transcription Factors - metabolism | Animals | TNF Receptor-Associated Factor 6 - metabolism | Macrophages - drug effects | Protein-Serine-Threonine Kinases - immunology | Francisella tularensis - pathogenicity | Myeloid Differentiation Factor 88 - metabolism | Cytokines - biosynthesis | Cell receptors | Transcription factors | Immune response | Physiological aspects | Genetic aspects | Research | Health aspects | Francisella tularensis | Index Medicus
Journal Article
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 12/2017, Volume 47, Issue 6, pp. 1129 - 1141.e5
.... T cell exhaustion during chronic infection was driven by high amounts of T cell receptor (TCR)-induced transcription factors IRF4, BATF, and NFATc1... 
differentiation | memory | transcription | exhaustion | BATF | NFAT | NFAT_AP-1_IRF4 composite element | NAICE | metabolic function | chronic infection | IRF4 | CD8 | TCF1 | Life Sciences & Biomedicine | Immunology | Science & Technology | Lymphocytic choriomeningitis virus - immunology | Hepatocyte Nuclear Factor 1-alpha - immunology | Hepatocyte Nuclear Factor 1-alpha - genetics | Humans | Basic-Leucine Zipper Transcription Factors - immunology | Lymphocyte Depletion | Interferon Regulatory Factors - deficiency | Lymphocytic Choriomeningitis - immunology | NFATC Transcription Factors - immunology | HEK293 Cells | Receptors, Antigen, T-Cell - immunology | Cell Differentiation | Lymphocytic Choriomeningitis - genetics | Lymphocytic Choriomeningitis - virology | Signal Transduction | Lymphocyte Activation | Gene Expression Regulation | Interferon Regulatory Factors - genetics | Basic-Leucine Zipper Transcription Factors - genetics | Mice, Knockout | Interferon Regulatory Factors - immunology | Lymphocytic choriomeningitis virus - growth & development | Animals | CD8-Positive T-Lymphocytes - virology | Immunologic Memory | Mice | Receptors, Antigen, T-Cell - genetics | Programmed Cell Death 1 Receptor - immunology | CD8-Positive T-Lymphocytes - immunology | Programmed Cell Death 1 Receptor - genetics | NFATC Transcription Factors - genetics | Regulators | Transcription factors | PD-1 protein | CD8 antigen | Genes | Memory | Chronic infection | Viruses | Infections | Lymphocytes T | T-cell receptor | Signal transduction | Receptors | Lymphocytes | Tumor necrosis factor-TNF | Hepatocyte nuclear factor 1 | Antigens | Immunological memory | Memory cells | Tumor cells | T cell receptors | Metabolism | Cell differentiation | Cells | Interferon regulatory factor 4 | Exhaustion | Receptor mechanisms | Metabolic disorders | Viral infections | Index Medicus
Journal Article
BMC genomics, ISSN 1471-2164, 09/2019, Volume 20, Issue 1, pp. 718 - 718
Journal Article
Nature (London), ISSN 1476-4687, 02/2019, Volume 567, Issue 7749, pp. 530 - 534
... activation of nuclear receptor transcription factors NR4A1 (also... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Chromatin - metabolism | CD8-Positive T-Lymphocytes - pathology | Receptors, Steroid - metabolism | Humans | Transcription Factors - deficiency | Male | NF-kappa B - metabolism | Gene Expression Profiling | Adoptive Transfer | Transcription Factor AP-1 - metabolism | DNA-Binding Proteins - deficiency | DNA-Binding Proteins - metabolism | Melanoma, Experimental - immunology | Neoplasms - genetics | CD8-Positive T-Lymphocytes - metabolism | Female | Lymphocytes, Tumor-Infiltrating - metabolism | Receptors, Steroid - deficiency | Antigens, CD19 - immunology | Mice, Inbred C57BL | Melanoma, Experimental - pathology | Survival Rate | Nuclear Receptor Subfamily 4, Group A, Member 1 - deficiency | Nuclear Receptor Subfamily 4, Group A, Member 2 - deficiency | Receptors, Thyroid Hormone - metabolism | Transcription Factors - metabolism | Animals | Melanoma, Experimental - genetics | Lymphocytes, Tumor-Infiltrating - pathology | Neoplasms - immunology | Receptors, Chimeric Antigen - immunology | Cell Line, Tumor | Nuclear Receptor Subfamily 4, Group A, Member 1 - metabolism | Receptors, Thyroid Hormone - deficiency | Mice | Nuclear Receptor Subfamily 4, Group A, Member 2 - metabolism | CD8-Positive T-Lymphocytes - immunology | Neoplasms - pathology | Chromatin - genetics | Lymphocytes, Tumor-Infiltrating - immunology | Transcription factors | Antigen receptors, T cell | Receptors | Causes of | Physiological aspects | Development and progression | Observations | T cells | Tumors | Antigens | Chromatin | DNA binding proteins | Gene expression | Accessibility | PD-1 protein | CD8 antigen | Genomics | Nur77 protein | Effector cells | Genomes | NF-AT protein | Bearing | Consortia | Cell activation | Lymphocytes | Transcription activation | Immunotherapy | DNA methylation | Deoxyribonucleic acid--DNA | Binding | NF-κB protein | Phenotypes | Cell survival | CD19 antigen | Activator protein 1 | Melanoma | Nuclear receptors | Nurr1 protein | Lymphocytes B | Stem cells | Solid tumors | Receptor mechanisms | Cancer | Index Medicus
Journal Article
Journal Article
Cell stress & chaperones, ISSN 1355-8145, 07/2012, Volume 18, Issue 1, pp. 11 - 23
...), PKR-like endoplasmic reticulum kinase (PERK), inositol-requiring 1 (IRE1), activating transcription factor 6 (ATF6), and ATF4... 
Polymerase chain reaction | Messenger RNA | Plasmids | Genes | HeLa cells | Gene expression regulation | Stress response | Endoplasmic reticulum | Apoptosis | Unfolded protein response | Biochemistry, general | Neurosciences | Gene regulation | ER stress | BiP | UPRE | Biomedicine general | Cell Biology | Biomedicine | Immunology | Cancer Research | ERSE | Life Sciences & Biomedicine | Science & Technology | Transcription Factor CHOP - genetics | Up-Regulation | Humans | Endoplasmic Reticulum - metabolism | Endoplasmic Reticulum Stress - genetics | Gene Regulatory Networks | Activating Transcription Factor 6 - genetics | X-Box Binding Protein 1 | DNA-Binding Proteins - metabolism | Heat-Shock Proteins - genetics | Antineoplastic Agents - pharmacology | DNA Damage - drug effects | Promoter Regions, Genetic | Unfolded Protein Response - genetics | Stress, Physiological - genetics | Heat-Shock Proteins - metabolism | Activating Transcription Factor 4 - genetics | Etoposide - pharmacology | Proteasome Endopeptidase Complex - chemistry | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Regulatory Factor X Transcription Factors | Transcription Factors - metabolism | Activating Transcription Factor 6 - metabolism | Activating Transcription Factor 4 - metabolism | HeLa Cells | Proteasome Endopeptidase Complex - metabolism | Transcription Factor CHOP - metabolism | Doxorubicin - pharmacology | Index Medicus | Immunoglobulins | Transcription | Activating transcription factor 6 | DNA damage | Etoposide | Homeostasis | Regulatory sequences | CCAAT/enhancer-binding protein | Chaperones | Doxorubicin | Promoters | Stress | Membrane proteins | Protein folding | Activating transcription factor 1 | Post-translation | BiP protein | Original Paper
Journal Article
Annual review of genomics and human genetics, ISSN 1527-8204, 2008, Volume 9, Issue 1, pp. 183 - 196
Journal Article
American journal of respiratory and critical care medicine, ISSN 1535-4970, 10/2008, Volume 178, Issue 8, pp. 838 - 846
.... Measurements and Main Results: Compared with COPD and donor lungs, protein levels of ER stress mediators, such as processed p50 activating transcription factor (ATF... 
C/EBP-homologous protein | X-box binding protein-1 | Type II alveolar epithelial cell | Interstitial lung disease | Lung fibrosis | Respiratory System | Life Sciences & Biomedicine | Critical Care Medicine | General & Internal Medicine | Science & Technology | Immunohistochemistry | Humans | Middle Aged | Oxidative Stress - physiology | Endoplasmic Reticulum - metabolism | Male | Pulmonary Fibrosis - genetics | Pulmonary Disease, Chronic Obstructive - pathology | Activating Transcription Factor 6 - genetics | X-Box Binding Protein 1 | RNA - genetics | Activating Transcription Factor 6 - biosynthesis | Endoplasmic Reticulum - pathology | Pulmonary Alveoli - metabolism | Female | Pulmonary Fibrosis - metabolism | Pulmonary Disease, Chronic Obstructive - metabolism | Pulmonary Disease, Chronic Obstructive - genetics | In Situ Nick-End Labeling | Severity of Illness Index | Gene Expression | Pulmonary Alveoli - pathology | Activating Transcription Factor 4 - genetics | Transcription Factors - biosynthesis | Pulmonary Fibrosis - pathology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Regulatory Factor X Transcription Factors | Blotting, Western | Activating Transcription Factor 4 - biosynthesis | Apoptosis - physiology | DNA-Binding Proteins - biosynthesis | Index Medicus | Abridged Index Medicus | C | interstitial lung disease | EBP-homologous protein | type II alveolar epithelial cell | lung fibrosis | E. Interstitial Lung Disease
Journal Article