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Journal of Biological Chemistry, ISSN 0021-9258, 11/2005, Volume 280, Issue 46, pp. 38247 - 38258
Journal Article
Cell stress & chaperones, ISSN 1355-8145, 07/2012, Volume 18, Issue 1, pp. 11 - 23
...), PKR-like endoplasmic reticulum kinase (PERK), inositol-requiring 1 (IRE1), activating transcription factor 6 (ATF6), and ATF4... 
Polymerase chain reaction | Messenger RNA | Plasmids | Genes | HeLa cells | Gene expression regulation | Stress response | Endoplasmic reticulum | Apoptosis | Unfolded protein response | Biochemistry, general | Neurosciences | Gene regulation | ER stress | BiP | UPRE | Biomedicine general | Cell Biology | Biomedicine | Immunology | Cancer Research | ERSE | Life Sciences & Biomedicine | Science & Technology | Transcription Factor CHOP - genetics | Up-Regulation | Humans | Endoplasmic Reticulum - metabolism | Endoplasmic Reticulum Stress - genetics | Gene Regulatory Networks | Activating Transcription Factor 6 - genetics | X-Box Binding Protein 1 | DNA-Binding Proteins - metabolism | Heat-Shock Proteins - genetics | Antineoplastic Agents - pharmacology | DNA Damage - drug effects | Promoter Regions, Genetic | Unfolded Protein Response - genetics | Stress, Physiological - genetics | Heat-Shock Proteins - metabolism | Activating Transcription Factor 4 - genetics | Etoposide - pharmacology | Proteasome Endopeptidase Complex - chemistry | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Regulatory Factor X Transcription Factors | Transcription Factors - metabolism | Activating Transcription Factor 6 - metabolism | Activating Transcription Factor 4 - metabolism | HeLa Cells | Proteasome Endopeptidase Complex - metabolism | Transcription Factor CHOP - metabolism | Doxorubicin - pharmacology | Index Medicus | Immunoglobulins | Transcription | Activating transcription factor 6 | DNA damage | Etoposide | Homeostasis | Regulatory sequences | CCAAT/enhancer-binding protein | Chaperones | Doxorubicin | Promoters | Stress | Membrane proteins | Protein folding | Activating transcription factor 1 | Post-translation | BiP protein | Original Paper
Journal Article
Nature (London), ISSN 1476-4687, 02/2019, Volume 567, Issue 7749, pp. 530 - 534
... activation of nuclear receptor transcription factors NR4A1 (also... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Chromatin - metabolism | CD8-Positive T-Lymphocytes - pathology | Receptors, Steroid - metabolism | Humans | Transcription Factors - deficiency | Male | NF-kappa B - metabolism | Gene Expression Profiling | Adoptive Transfer | Transcription Factor AP-1 - metabolism | DNA-Binding Proteins - deficiency | DNA-Binding Proteins - metabolism | Melanoma, Experimental - immunology | Neoplasms - genetics | CD8-Positive T-Lymphocytes - metabolism | Female | Lymphocytes, Tumor-Infiltrating - metabolism | Receptors, Steroid - deficiency | Antigens, CD19 - immunology | Mice, Inbred C57BL | Melanoma, Experimental - pathology | Survival Rate | Nuclear Receptor Subfamily 4, Group A, Member 1 - deficiency | Nuclear Receptor Subfamily 4, Group A, Member 2 - deficiency | Receptors, Thyroid Hormone - metabolism | Transcription Factors - metabolism | Animals | Melanoma, Experimental - genetics | Lymphocytes, Tumor-Infiltrating - pathology | Neoplasms - immunology | Receptors, Chimeric Antigen - immunology | Cell Line, Tumor | Nuclear Receptor Subfamily 4, Group A, Member 1 - metabolism | Receptors, Thyroid Hormone - deficiency | Mice | Nuclear Receptor Subfamily 4, Group A, Member 2 - metabolism | CD8-Positive T-Lymphocytes - immunology | Neoplasms - pathology | Chromatin - genetics | Lymphocytes, Tumor-Infiltrating - immunology | Transcription factors | Antigen receptors, T cell | Receptors | Causes of | Physiological aspects | Development and progression | Observations | T cells | Tumors | Antigens | Chromatin | DNA binding proteins | Gene expression | Accessibility | PD-1 protein | CD8 antigen | Genomics | Nur77 protein | Effector cells | Genomes | NF-AT protein | Bearing | Consortia | Cell activation | Lymphocytes | Transcription activation | Immunotherapy | DNA methylation | Deoxyribonucleic acid--DNA | Binding | NF-κB protein | Phenotypes | Cell survival | CD19 antigen | Activator protein 1 | Melanoma | Nuclear receptors | Nurr1 protein | Lymphocytes B | Stem cells | Solid tumors | Receptor mechanisms | Cancer | Index Medicus
Journal Article
Journal Article
Nature immunology, ISSN 1529-2916, 03/2010, Volume 11, Issue 5, pp. 411 - 418
.... and its downstream target, the transcription factor XBP1. Previously described ER-stress target genes of XBP1 were not induced by TLR signaling... 
Life Sciences & Biomedicine | Immunology | Science & Technology | RNA, Small Interfering - genetics | Endoribonucleases - genetics | Tularemia - metabolism | Toll-Like Receptor 2 - genetics | Membrane Glycoproteins - metabolism | Lipopeptides - pharmacology | Transcriptional Activation - drug effects | NADPH Oxidases - metabolism | Tularemia - genetics | Protein Splicing - genetics | Transcriptional Activation - immunology | X-Box Binding Protein 1 | Protein Splicing - drug effects | NADPH Oxidases - genetics | Stress, Physiological - drug effects | TNF Receptor-Associated Factor 6 - genetics | Transcription Factor CHOP - biosynthesis | Transcription Factors - immunology | Cytokines - genetics | Protein-Serine-Threonine Kinases - metabolism | DNA-Binding Proteins - immunology | Endoribonucleases - metabolism | Macrophages - pathology | Stress, Physiological - genetics | Myeloid Differentiation Factor 88 - genetics | Toll-Like Receptor 4 - genetics | Signal Transduction - genetics | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Macrophages - metabolism | Signal Transduction - drug effects | Tunicamycin - pharmacology | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Mice | Stress, Physiological - immunology | Endoribonucleases - immunology | Transcription Factor CHOP - genetics | NADPH Oxidases - immunology | DNA-Binding Proteins - metabolism | Signal Transduction - immunology | Macrophages - virology | Mice, Mutant Strains | Tularemia - immunology | Membrane Glycoproteins - immunology | Macrophages - immunology | Cytokines - immunology | Cell Line | Francisella tularensis - immunology | Protein Splicing - immunology | Protein-Serine-Threonine Kinases - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Immunity, Innate | NADPH Oxidase 2 | Regulatory Factor X Transcription Factors | Mice, Inbred C3H | Membrane Glycoproteins - genetics | Transcription Factors - metabolism | Animals | TNF Receptor-Associated Factor 6 - metabolism | Macrophages - drug effects | Protein-Serine-Threonine Kinases - immunology | Francisella tularensis - pathogenicity | Myeloid Differentiation Factor 88 - metabolism | Cytokines - biosynthesis | Cell receptors | Transcription factors | Immune response | Physiological aspects | Genetic aspects | Research | Health aspects | Francisella tularensis | Index Medicus
Journal Article