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Journal of Hepatology, ISSN 0168-8278, 2017, Volume 67, Issue 2, pp. 349 - 359
Graphical abstract Summarized effects of ATF3 and in vivo -jetPEI ATF3 siRNA on the oxidative stress-mediated hepatic steatosis and/or T2D. 
Gastroenterology and Hepatology | In vivo silencing | Activation transcription factor 3 | Oxidative stress | Human patients | Non-alcoholic fatty liver disease | Zucker diabetic fatty rat | PROTEIN | REGULATOR | METABOLISM | DIET | INSULIN-RESISTANCE | BETA-CELL DYSFUNCTION | STRESS | GASTROENTEROLOGY & HEPATOLOGY | ATF3 | PROGRESSION | LIFE | Glucose Intolerance - metabolism | Non-alcoholic Fatty Liver Disease - pathology | RNA, Small Interfering - genetics | Up-Regulation | Liver - pathology | Prospective Studies | Oxidative Stress | Humans | Middle Aged | Diabetes Mellitus, Type 2 - metabolism | Non-alcoholic Fatty Liver Disease - complications | Glucose Intolerance - etiology | Adult | Diabetes Mellitus, Type 2 - etiology | Female | Activating Transcription Factor 3 - genetics | Biomarkers - metabolism | Cross-Sectional Studies | Liver - metabolism | Insulin Resistance | Rats | Non-alcoholic Fatty Liver Disease - metabolism | Activating Transcription Factor 3 - metabolism | Activating Transcription Factor 3 - antagonists & inhibitors | Rats, Zucker | Animals | Aged | Cohort Studies | Transcription factors | Liver diseases | Therapeutic applications | Diabetes mellitus | Activating transcription factor 3 | Homeostasis | siRNA | Inflammation | Glucose | Macrophages | Insulin | Steatosis | Glucose tolerance | Intolerance | Mitochondria | Fatty liver | Biopsy | Oxidation | Molecular biology | Diabetes mellitus (non-insulin dependent) | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 02/2017, Volume 292, Issue 5, pp. 1625 - 1636
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 10, pp. e26744 - e26744
Activating transcription factor 3 (ATF3), which is encoded by an adaptive-response gene induced by various stimuli, plays an important role in the... 
PATHWAYS | APOPTOSIS | MYOCYTES | MULTIDISCIPLINARY SCIENCES | ATF3 GENE | ENDOTHELIAL-CELLS | KINASE | STRESS-INDUCIBLE GENE | EXPRESSION | PROTECTS | FAMILY | Cardiomyopathy, Dilated - pathology | Humans | Cardiomegaly - pathology | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | MAP Kinase Signaling System | Mitogen-Activated Protein Kinase Kinases - metabolism | Disease Models, Animal | Cardiomyopathy, Dilated - complications | Mice, Inbred C57BL | Activating Transcription Factor 3 - deficiency | Cardiomegaly - physiopathology | Myocardium - pathology | Activating Transcription Factor 3 - metabolism | Cardiomyopathy, Dilated - metabolism | Cardiomegaly - complications | Myocytes, Cardiac - pathology | Myocardium - enzymology | Animals | Cardiomyopathy, Dilated - physiopathology | Fibrosis | Myocytes, Cardiac - metabolism | Mice | Ventricular Pressure | Hemodynamics | Cardiomegaly - metabolism | Heart | Heart enlargement | Genes | Neonates | Neurosciences | Animal models | Transcription factors | Cytotoxicity | Kinases | Cell growth | Pathways | Ischemia | Rodents | Aorta | Tumorigenesis | Cardiology | Angiotensin II | Heart diseases | Deoxyribonucleic acid--DNA | Cardiovascular system | Activating transcription factor 3 | Extracellular signal-regulated kinase | Heat shock proteins | Cardiomyocytes | JNK protein | Gene expression | Signaling | Banding | Angiotensin | Laboratory animals | Hypertrophy | Apoptosis | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 04/2017, Volume 135, Issue 21, pp. 2041 - 2057
BACKGROUND—Hypertensive ventricular remodeling is a common cause of heart failure. However, the molecular mechanisms regulating ventricular remodeling remain... 
Heart failure | Fibroblast | P38 mitogenactivated protein kinases | Activating transcription factor 3 | FIBROSIS | heart failure | CARDIAC & CARDIOVASCULAR SYSTEMS | CARDIOMYOPATHY | p38 mitogen-activated protein kinases | BETA | HYPERTROPHY | fibroblast | INHIBITION | INFLAMMATION | DISEASE | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | CELL | activating transcription factor 3 | ATF3 | Fibroblasts - enzymology | Hypertrophy, Left Ventricular - enzymology | Ventricular Function, Left | Heart Failure - enzymology | Humans | Heart Failure - physiopathology | Male | MAP Kinase Kinase 3 - genetics | Heart Failure - prevention & control | Ventricular Remodeling | Time Factors | Hypertension - chemically induced | p38 Mitogen-Activated Protein Kinases - metabolism | Acetylation | Angiotensin II | Binding Sites | Heart Failure - etiology | Activating Transcription Factor 3 - genetics | Disease Models, Animal | Promoter Regions, Genetic | Genetic Predisposition to Disease | Signal Transduction | Hypertrophy, Left Ventricular - etiology | Hypertrophy, Left Ventricular - prevention & control | Activating Transcription Factor 3 - deficiency | Cells, Cultured | Myocardium - pathology | Fibroblasts - pathology | Activating Transcription Factor 3 - metabolism | MAP Kinase Kinase 3 - metabolism | Mice, Knockout | Myocardium - enzymology | Phenotype | Animals | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Fibrosis | Protein Kinase Inhibitors - pharmacology | Histones - metabolism | Hypertrophy, Left Ventricular - physiopathology | Transforming Growth Factor beta - metabolism | Histone Deacetylase 1 - metabolism | Index Medicus | Abridged Index Medicus | p38 MAPK | cardiac fibroblast
Journal Article
Journal Article
Journal Article