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Journal Article
Stem cells (Dayton, Ohio), ISSN 1549-4918, 09/2004, Volume 22, Issue 5, pp. 770 - 778
Murine embryonic stem (mES) cells remain undifferentiated in the presence of leukemia inhibitory factor (LIF... 
Human embryonic stem cells | Self‐renewal | Leukemia inhibitory factor | STAT3 | Self-renewal | Cell & Tissue Engineering | Life Sciences & Biomedicine | Hematology | Oncology | Biotechnology & Applied Microbiology | Science & Technology | Cell Biology | Membrane Glycoproteins - metabolism | Humans | Culture Media, Conditioned - pharmacology | Antigens, CD - genetics | Cell Culture Techniques - methods | Protein Subunits - metabolism | Antigens, CD - metabolism | Interleukin-6 | DNA-Binding Proteins - metabolism | Cell Differentiation - genetics | Cell Nucleus - metabolism | Trans-Activators - genetics | Phosphorylation - drug effects | Receptors, Cytokine - metabolism | Active Transport, Cell Nucleus - genetics | Leukemia Inhibitory Factor | Receptors, Cytokine - genetics | Protein Subunits - genetics | Cell Division - genetics | Cell Line | Cytokine Receptor gp130 | Pluripotent Stem Cells - immunology | Gene Expression Regulation - genetics | Signal Transduction - genetics | DNA-Binding Proteins - genetics | Cell Division - drug effects | Leukemia Inhibitory Factor Receptor alpha Subunit | Membrane Glycoproteins - genetics | Gene Expression Regulation - drug effects | Pluripotent Stem Cells - metabolism | Proteins - genetics | Stem Cell Transplantation - methods | Animals | Proteins - metabolism | Cell Nucleus - genetics | Signal Transduction - drug effects | Cell Differentiation - drug effects | Active Transport, Cell Nucleus - drug effects | Pluripotent Stem Cells - drug effects | Trans-Activators - metabolism | Cell Proliferation - drug effects | Mice | Proteins - pharmacology | STAT3 Transcription Factor | Cell Nucleus - drug effects | Receptors, OSM-LIF | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 4/2003, Volume 100, Issue 7, pp. 3878 - 3882
Journal Article
Radiation research, ISSN 0033-7587, 2013, Volume 179, Issue 1, pp. 53 - 61
We previously established annexin A2 as a radioresponsive protein associated with anchorage independent growth in murine epidermal cells... 
Ionizing radiation | Cell nucleus | Epidermal cells | Genes | Keratinocytes | Skin | Signals | Directional control | Annexins | Radiation dosage | Biology | Life Sciences & Biomedicine - Other Topics | Biophysics | Life Sciences & Biomedicine | Radiology, Nuclear Medicine & Medical Imaging | Science & Technology | Transcription, Genetic - drug effects | Cell Death - radiation effects | Skin - cytology | Humans | Annexin A2 - deficiency | Transcription, Genetic - radiation effects | NF-kappa B - metabolism | Stem Cells - cytology | Transcriptome - radiation effects | Stress, Physiological - radiation effects | Cell Differentiation - genetics | Cell Death - genetics | Cell Nucleus - radiation effects | Stress, Physiological - drug effects | Cell Death - drug effects | Homeostasis - drug effects | Active Transport, Cell Nucleus - genetics | Active Transport, Cell Nucleus - radiation effects | Cell Differentiation - radiation effects | Stress, Physiological - genetics | Gene Silencing | Stem Cells - radiation effects | Radiation Tolerance - genetics | Homeostasis - radiation effects | Transcriptome - drug effects | X-Rays - adverse effects | Radiation Tolerance - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Animals | Annexin A2 - genetics | Cell Differentiation - drug effects | Active Transport, Cell Nucleus - drug effects | Stem Cells - drug effects | Mice | Cell Nucleus - drug effects | Annexin A2 - metabolism | Homeostasis - genetics | Index Medicus | Space life sciences | RADIATION, THERMAL, AND OTHER ENVIRON. POLLUTANT EFFECTS ON LIVING ORGS. AND BIOL. MAT
Journal Article
Journal Article
Cell death and differentiation, ISSN 1476-5403, 09/2016, Volume 23, Issue 12, pp. 2007 - 2018
Journal Article
PloS one, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, pp. e91770 - e91770
.... Hyperactivation of YAP by mutating its five inhibitory phosphorylation sites (YAP-5SA) increased ovarian cancer cell proliferation, resistance to chemotherapeutic drugs, cell migration, and anchorage-independent growth... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Paclitaxel - pharmacology | Prognosis | Apoptosis - drug effects | Humans | Ovarian Neoplasms - pathology | Drug Resistance, Neoplasm | Phosphoproteins - metabolism | DNA-Binding Proteins - metabolism | Cell Nucleus - metabolism | Muscle Proteins - metabolism | Female | Ovarian Neoplasms - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Ovarian Neoplasms - diagnosis | Cisplatin - pharmacology | Carcinogenesis - drug effects | Transcription Factors - metabolism | Cell Movement - drug effects | Animals | Cell Transformation, Neoplastic | Active Transport, Cell Nucleus - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Adaptor Proteins, Signal Transducing - metabolism | Cell Nucleus - drug effects | Chemotherapy | Oncology, Experimental | Research | Gene expression | Ovarian cancer | Cancer | Cell proliferation | Drugs | Phosphorylation | Transcription factors | Ovarian carcinoma | Mesenchyme | Downstream effects | Drug resistance | Kinases | Cancer therapies | Proteins | Liver cancer | Signal transduction | Cell growth | Cell cycle | Life sciences | Tumorigenesis | Survival | Patients | Yes-associated protein | Pathology | Signaling | Medical prognosis | In vivo methods and tests | Laboratory animals | Cell migration | Apoptosis | Tumors | Index Medicus
Journal Article
Cancer letters, ISSN 0304-3835, 02/2015, Volume 357, Issue 2, pp. 520 - 526
.... In the present study, we first observed that blocking of NF-κB function in KRAS mutant A549 cell line with an IκBα mutant (IκBαM... 
Hematology, Oncology and Palliative Medicine | NF-κB | KRAS | Lung cancer | Chemoresistance | p53 | P53 | Life Sciences & Biomedicine | Oncology | Science & Technology | Cell Cycle - genetics | Lung Neoplasms - drug therapy | ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Paclitaxel - pharmacology | Apoptosis - drug effects | Heterocyclic Compounds, 3-Ring - pharmacology | Humans | Lung Neoplasms - metabolism | ras Proteins - metabolism | Apoptosis - genetics | NF-kappa B - metabolism | Tumor Suppressor Protein p53 - genetics | Cell Nucleus - metabolism | I-kappa B Kinase - metabolism | I-kappa B Kinase - antagonists & inhibitors | Tumor Burden - genetics | Antineoplastic Agents - pharmacology | Lung Neoplasms - genetics | Proto-Oncogene Proteins - metabolism | Carcinogenesis - genetics | Tumor Suppressor Protein p53 - metabolism | Proto-Oncogene Proteins - genetics | Cisplatin - pharmacology | Xenograft Model Antitumor Assays - methods | Blotting, Western | Carcinogenesis - drug effects | Drug Resistance, Neoplasm - genetics | Animals | Transcription Factor RelA - metabolism | Tumor Burden - drug effects | Active Transport, Cell Nucleus - drug effects | Cell Line, Tumor | Mice, Inbred BALB C | Pyridines - pharmacology | Mutation | Cell Cycle - drug effects | Cell Nucleus - drug effects | Drug Resistance, Neoplasm - drug effects | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 08/2010, Volume 5, Issue 8, pp. e12293 - e12293
Background: The PI3K-Akt signal pathway plays a key role in tumorigenesis and the development of drug-resistance... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Prognosis | Carcinoma, Ductal - genetics | Carcinoma, Ductal - pathology | Humans | Drug Resistance, Neoplasm | Breast Neoplasms - metabolism | Cell Nucleus - metabolism | Forkhead Transcription Factors - metabolism | Carcinoma, Ductal - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Neoplasm Invasiveness | Lymphatic Metastasis | Forkhead Transcription Factors - genetics | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Active Transport, Cell Nucleus - drug effects | Survival Analysis | Tamoxifen - pharmacology | Carcinoma, Ductal - diagnosis | Cell Line, Tumor | Breast Neoplasms - diagnosis | Cell Proliferation - drug effects | Cell Nucleus - drug effects | Forkhead Box Protein O3 | Doxorubicin - pharmacology | Anthracyclines | Chemotherapy | Cell death | Analysis | Breast cancer | Drug resistance | Tamoxifen | Health aspects | Cancer | Cell proliferation | Cell culture | Deregulation | Transcription factors | Phosphorylation | Downstream effects | Colorectal cancer | Estrogen | Cytotoxicity | AKT protein | Kinases | Cancer therapies | Doxorubicin | Lymph nodes | Metastases | Breast carcinoma | Surgery | Cell cycle | Tumorigenesis | Localization | Relocation | FOXO3 protein | Medical research | Invasiveness | Tumor cell lines | College campuses | Survival | 1-Phosphatidylinositol 3-kinase | Medicine | Studies | Pathology | Signaling | Medical prognosis | Cytoplasm | Apoptosis | Lymph | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, pp. e97957 - e97957
.... The exposure of cells to DHA recruited the adapter protein beta-arrestin1/2 to FFAR4, but not to a related lipid receptor... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Inflammasomes - metabolism | Calcium - metabolism | Humans | NF-kappa B - metabolism | Arrestins - metabolism | Autophagy - drug effects | Cell Nucleus - metabolism | Interleukin-1beta - biosynthesis | Macrophages - immunology | Interleukin-1beta - secretion | Intracellular Space - drug effects | Bone Marrow Cells - cytology | Mice, Inbred C57BL | Macrophages - cytology | Gene Expression Regulation - drug effects | Docosahexaenoic Acids - pharmacology | Macrophages - metabolism | Animals | beta-Arrestins | Active Transport, Cell Nucleus - drug effects | Intracellular Space - metabolism | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Mice | Receptors, G-Protein-Coupled - genetics | HeLa Cells | Cell Nucleus - drug effects | Macrophage Activation - drug effects | Biotechnology | G protein-coupled receptors | Laboratories | Macrophages | Docosahexaenoic acid | Autophagy | Cell activation | Immunology | Rodents | Bone marrow | Life sciences | Inhibition | Adenosine triphosphate | Chronic illnesses | Translocation | NF-κB protein | Adapters | Priming | Inflammation | Exposure | IL-1β | Metabolism | Fatty acids | Nuclear transport | Infectious diseases | Molecular modelling | Adapter proteins | Phagocytosis | Cancer | Apoptosis | Index Medicus | Activation
Journal Article