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Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2017, Volume 37, Issue 4, pp. 657 - 663
Journal Article
Journal Article
Cell, ISSN 0092-8674, 2003, Volume 112, Issue 3, pp. 303 - 315
Metamorphosis of the Drosophila brain involves pruning of many larval-specific dendrites and axons followed by outgrowth of adult-specific processes. From a... 
MUSHROOM BODIES | REPRESSIBLE CELL MARKER | NERVOUS-SYSTEM | CAENORHABDITIS-ELEGANS | ECDYSONE RECEPTOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | II RECEPTOR | LARVAL DEVELOPMENT | C-ELEGANS | METAMORPHOSIS | MOSAIC ANALYSIS | CELL BIOLOGY | Receptors, Steroid - metabolism | Mushroom Bodies - growth & development | Activin Receptors, Type II - deficiency | Male | Neurons - cytology | Brain - growth & development | DNA-Binding Proteins - deficiency | Mutation, Missense - genetics | Brain - metabolism | Proteins | Cell Differentiation - genetics | Neuronal Plasticity - genetics | Protein Isoforms - metabolism | Larva - growth & development | Trans-Activators - genetics | Mushroom Bodies - cytology | Female | Neurons - metabolism | Mushroom Bodies - metabolism | Brain - cytology | Larva - metabolism | Neural Pathways - growth & development | Activin Receptors, Type I - deficiency | Drosophila - cytology | Signal Transduction - genetics | DNA-Binding Proteins - genetics | Activin Receptors, Type I - genetics | Larva - cytology | Animals | Trans-Activators - deficiency | Receptors, Steroid - genetics | Genes, Lethal - genetics | Receptors, Cell Surface - deficiency | Activin Receptors, Type II - genetics | Drosophila Proteins - deficiency | Neural Pathways - cytology | Neural Pathways - metabolism | Drosophila - growth & development | Drosophila - metabolism | Drosophila Proteins - genetics | Transforming Growth Factor beta - metabolism | Protein Isoforms - genetics | Receptors, Cell Surface - genetics | Smad2 Protein | Neuroplasticity | Physiological aspects | Hormone receptors | Cellular signal transduction | Transforming growth factors | Analysis
Journal Article
Molecular Therapy, ISSN 1525-0016, 08/2017, Volume 25, Issue 8, pp. 1974 - 1987
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2018, Volume 38, Issue 8, pp. 1821 - 1836
OBJECTIVE—Diabetic macular edema is a major cause of visual impairment. It is caused by blood-retinal barrier breakdown that leads to vascular... 
Capillary permeability | Mice | Activin receptors | Occludin | ANTI-VEGF | HEREDITARY HEMORRHAGIC TELANGIECTASIA | OCULAR ANGIOGENESIS | activin receptors | mice | occludin | BLOOD-RETINAL BARRIER | capillary permeability | VE-CADHERIN | PERIPHERAL VASCULAR DISEASE | DIABETIC MACULAR EDEMA | TIGHT JUNCTION PROTEINS | HEMATOLOGY | MOLECULAR-BASIS | ENDOTHELIAL GROWTH-FACTOR | 1 ALK1 | Activin Receptors, Type II - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Streptozocin | Growth Differentiation Factors - biosynthesis | Humans | Diabetes Mellitus, Experimental - genetics | Capillary Permeability - drug effects | Male | Vascular Endothelial Growth Factor A - metabolism | Diabetes Mellitus, Experimental - therapy | Hyperglycemia - genetics | Hyperglycemia - chemically induced | Diabetic Retinopathy - chemically induced | Growth Differentiation Factors - pharmacology | Growth Differentiation Factors - genetics | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Macular Edema - genetics | Macular Edema - prevention & control | Cell Line | Macular Edema - chemically induced | Human Umbilical Vein Endothelial Cells - drug effects | Endothelial Cells - metabolism | Mice, Inbred C57BL | Activin Receptors, Type I - deficiency | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Activin Receptors, Type I - metabolism | Macular Edema - metabolism | Activin Receptors, Type I - genetics | Diabetic Retinopathy - metabolism | Diabetic Retinopathy - genetics | Haploinsufficiency | Mice, Knockout | Hyperglycemia - metabolism | Diabetic Retinopathy - prevention & control | Animals | Signal Transduction - drug effects | Activin Receptors, Type II - genetics | Blood Glucose - metabolism | Hyperglycemia - therapy | Smad Proteins - metabolism | Endothelial Cells - drug effects | Genetic Therapy - methods
Journal Article
Journal Article
Developmental Biology, ISSN 0012-1606, 2003, Volume 261, Issue 1, pp. 251 - 267
Guidance and patterning of axons are orchestrated by cell-surface receptors and ligands that provide directional cues. Interactions between the Robo receptor... 
Activin receptor-like kinase 1 (Alk1) | Angiogenesis | Neuronal/vascular guidance | Slit | HHT | Roundabout (Robo) | Vascular patterning | Vascular development | Endothelial migration | Vascular sprouting | NEURONAL MIGRATION | vascular sprouting | RETINAL AXONS | ZEBRAFISH | angiogenesis | endothelial migration | BLOOD-VESSEL FORMATION | activin receptor-like kinase 1 (Alk1) | REPULSIVE AXON GUIDANCE | DEVELOPMENTAL BIOLOGY | SLIT PROTEINS | MIDLINE | ROUNDABOUT RECEPTOR | slit | roundabout (Robo) | vascular development | neuronall/vascular guidance | CELL-MIGRATION | GROWTH-FACTORS | vascular patterning | Receptors, Immunologic - chemistry | Receptors, Immunologic - physiology | Humans | Molecular Sequence Data | Phylogeny | RNA, Messenger - metabolism | In Situ Hybridization | Gene Expression Regulation, Developmental | Activin Receptors, Type I - physiology | Endothelium, Vascular - embryology | Amino Acid Sequence | Cell Line | Signal Transduction | Intercellular Signaling Peptides and Proteins | RNA, Messenger - genetics | Activin Receptors, Type I - deficiency | Zebrafish | Chromosome Mapping | Activin Receptors, Type I - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Sequence Homology, Amino Acid | Animals | Activin Receptors, Type II | Ligands | Mice | Receptors, Immunologic - genetics | Cell Movement | Axons | Motility | Developmental biology | Research | Cell migration | Cells | Endothelium
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 04/2016, Volume 310, Issue 8, pp. H984 - H994
Bone morphogenetic protein (BMP) signaling contributes to the development of cardiac hypertrophy. However, the identity of the BMP type I receptor involved in... 
Cardiac hypertrophy | BMP signaling | BMP type I receptor | ALK2 | Calcineurin | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | CARDIOMYOPATHY | KINASE | calcineurin | GROWTH | PERIPHERAL VASCULAR DISEASE | MICE | CHANNELS | ATROGIN-1 | cardiac hypertrophy | Bone Morphogenetic Protein 2 - pharmacology | Phosphorylation | Cardiomegaly - pathology | Dose-Response Relationship, Drug | Myocytes, Cardiac - enzymology | Phenylephrine - pharmacology | Transfection | RNA Interference | Time Factors | Bone Morphogenetic Protein Receptors, Type I - deficiency | Angiotensin II | Disease Models, Animal | Pyrazoles - pharmacology | Signal Transduction | Mice, Inbred C57BL | NFATC Transcription Factors - metabolism | Activin Receptors, Type I - deficiency | Cells, Cultured | Bone Morphogenetic Protein Receptors, Type I - genetics | Activin Receptors, Type I - metabolism | Smad Proteins - genetics | Cardiomegaly - enzymology | Pyrimidines - pharmacology | Bone Morphogenetic Protein Receptors, Type I - metabolism | Inhibitor of Differentiation Protein 1 - metabolism | Activin Receptors, Type I - genetics | Rats, Sprague-Dawley | Bone Morphogenetic Protein 4 - pharmacology | Mice, Knockout | Myocytes, Cardiac - pathology | Animals | Cardiomegaly - prevention & control | Myocytes, Cardiac - drug effects | Fibrosis | Cardiomegaly - chemically induced | Inhibitor of Differentiation Protein 1 - genetics | Cardiomegaly - genetics | Smad Proteins - metabolism | Physiological aspects | Bone morphogenetic proteins | Heart enlargement | Health aspects | Angiotensin | Integrative Cardiovascular Physiology and Pathophysiology
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 10/2014, Volume 34, Issue 10, pp. 2232 - 2236
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 05/2018, Volume 38, Issue 5, pp. 1216 - 1229
OBJECTIVE—ALK1 (activin-receptor like kinase 1) is an endothelial cell-restricted receptor with high affinity for BMP (bone morphogenetic protein) 9 TGF-β... 
endothelium | ARTERIOVENOUS-MALFORMATIONS | HEREDITARY HEMORRHAGIC TELANGIECTASIA | PROTEIN | telangiectasia | ANGIOGENESIS | ENDOTHELIAL-CELL MIGRATION | mice | hereditary hemorrhagic | rare diseases | RECEPTOR-LIKE KINASE-1 | GENE | retina | MOLECULAR REGULATION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | humans | EXPRESSION | TYPE-2 | Growth Differentiation Factor 2 - pharmacology | Telangiectasia, Hereditary Hemorrhagic - pathology | Phosphatidylinositol 3-Kinase - antagonists & inhibitors | Humans | Hyperplasia | Neovascularization, Pathologic | Telangiectasia, Hereditary Hemorrhagic - genetics | Retinal Telangiectasis - genetics | Case-Control Studies | Gene Deletion | Phosphatidylinositol 3-Kinase - metabolism | Vascular Endothelial Growth Factor A - pharmacology | Genetic Predisposition to Disease | Signal Transduction | Mice, Inbred C57BL | Activin Receptors, Type I - deficiency | Cells, Cultured | Angiogenesis Inhibitors - pharmacology | Retinal Telangiectasis - enzymology | Telangiectasia, Hereditary Hemorrhagic - drug therapy | Telangiectasia, Hereditary Hemorrhagic - enzymology | Activin Receptors, Type I - genetics | Retinal Telangiectasis - drug therapy | Mice, Knockout | Phenotype | Animals | Human Umbilical Vein Endothelial Cells - enzymology | Activin Receptors, Type II - genetics | Retinal Telangiectasis - pathology | Human Umbilical Vein Endothelial Cells - pathology | Protein Kinase Inhibitors - pharmacology | Enzyme Activation | Mutation | Endothelial Cells - pathology | Endothelial Cells - enzymology | Endothelial Cells - drug effects | Proteïnes quinases | Rats as laboratory animals | Retina | Hemorrhage | Hemorràgia | Endothelium | Vascular endothelial growth factors | Vascular diseases | Factor de creixement de l'endoteli vascular | Malalties vasculars | Endoteli | Rates (Animals de laboratori) | Protein kinases
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2016, Volume 105, pp. 1 - 11
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 02/2013, Volume 33, Issue 2, pp. 305 - 310
OBJECTIVE—Vessels in brain arteriovenous malformations are prone to rupture. The underlying pathogenesis is not clear. Hereditary hemorrhagic telangiectasia... 
platelet-derived growth factor receptor-β | iron deposition | brain arteriovenous malformation | pericyte | activin receptor-like kinase 1 | HEREDITARY HEMORRHAGIC TELANGIECTASIA | BARRIER | INJURY | CEREBRAL ARTERIOVENOUS-MALFORMATIONS | INTRACRANIAL HEMORRHAGE | CAPILLARIES | platelet-derived growth factor receptor-beta | IN-VIVO | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | PERICYTES | Dependovirus - genetics | Microglia - metabolism | Telangiectasia, Hereditary Hemorrhagic - pathology | Blood Vessels - pathology | Actins - metabolism | Neovascularization, Pathologic | Telangiectasia, Hereditary Hemorrhagic - genetics | Vascular Endothelial Growth Factor A - metabolism | Pericytes - enzymology | Vascular Endothelial Growth Factor A - genetics | Brain - blood supply | Pericytes - pathology | Microglia - pathology | Blood Vessels - enzymology | Disease Models, Animal | Receptor, Platelet-Derived Growth Factor beta - metabolism | Gene Transfer Techniques | Macrophages - pathology | Proto-Oncogene Proteins c-sis - metabolism | Mice, Inbred C57BL | Activin Receptors, Type I - deficiency | Telangiectasia, Hereditary Hemorrhagic - enzymology | Activin Receptors, Type I - genetics | Iron - metabolism | Mice, Knockout | Macrophages - metabolism | Animals | Mice | Fibrin - metabolism | Genetic Vectors | PDGFR-β
Journal Article
Angiogenesis, ISSN 0969-6970, 10/2016, Volume 19, Issue 4, pp. 451 - 461
Journal Article