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Developmental Cell, ISSN 1534-5807, 03/2012, Volume 22, Issue 3, pp. 489 - 500
Journal Article
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 12/2014, Volume 96, Issue 6, pp. 1023 - 1035
miR‐199a‐5p inhibits monocyte/macrophage differentiation via down‐regulating ACVR1B, further reducing phosphorylation of Smad2/3, resulting in decreased... 
cell proliferation and cell cycle | phosphorylation of Smad2/3 | PU.1 | TGF‐β signaling pathway | Phosphorylation of Smad2/3 | TGF-β signaling pathway | Cell proliferation and cell cycle | TRANSCRIPTION FACTORS | TGF-BETA | PROTEIN | CANCER CELLS | MONOCYTIC DIFFERENTIATION | DOWN-REGULATION | CELL-PROLIFERATION | ACUTE MYELOID-LEUKEMIA | IMMUNOLOGY | CELL BIOLOGY | GROWTH-FACTOR-BETA | TGF- signaling pathway | HEMATOLOGY | phosphorylation of Smad2 | Activin Receptors, Type I - antagonists & inhibitors | Phosphorylation | Monocytes - cytology | Humans | Neoplasm Proteins - metabolism | Trans-Activators - physiology | CCAAT-Enhancer-Binding Protein-alpha - biosynthesis | Transfection | RNA Interference | CCAAT-Enhancer-Binding Protein-alpha - physiology | Colony-Forming Units Assay | Activin Receptors, Type I - physiology | Smad2 Protein - physiology | Genes, Reporter | Transduction, Genetic | RNA, Small Interfering - pharmacology | Retinoblastoma Protein - metabolism | Cells, Cultured | Gene Expression Regulation, Developmental - drug effects | Hematopoiesis - genetics | Fetal Blood - cytology | Macrophages - cytology | Activin Receptors, Type I - genetics | Hematopoietic Stem Cells - cytology | Proto-Oncogene Proteins - physiology | CCAAT-Enhancer-Binding Protein-alpha - genetics | Cell Line, Tumor | HL-60 Cells | Protein Processing, Post-Translational | MicroRNAs - physiology | Index Medicus
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 04/2015, Volume 30, Issue 4, pp. 733 - 741
Journal Article
Journal Article
Developmental Dynamics, ISSN 1058-8388, 01/2008, Volume 237, Issue 1, pp. 259 - 269
textabstractAlthough transforming growth factor-beta (TGF-β) signaling negatively regulates branching morphogenesis in early lung development, few studies to... 
Human | Mouse | Rat | Transforming growth factor | Development | TGF-β | Alveolarization | Septation | development | rat | BRONCHIAL EPITHELIAL-CELLS | ANATOMY & MORPHOLOGY | BRANCHING MORPHOGENESIS | BRONCHOPULMONARY-DYSPLASIA | EMBRYONIC LUNG | DEVELOPMENTAL BIOLOGY | INDUCTION | II-RECEPTOR | alveolarization | mouse | septation | TGF-beta | TRANSFORMING-GROWTH-FACTOR | DIFFERENTIATION | transforming growth factor | human | EXPRESSION | MOUSE LUNG | Immunohistochemistry | Receptors, Transforming Growth Factor beta - genetics | Humans | Immunoblotting | Intracellular Signaling Peptides and Proteins - metabolism | Smad4 Protein - genetics | Smad2 Protein - genetics | Activin Receptors, Type I - physiology | Smad6 Protein - genetics | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Smad7 Protein - metabolism | Smad2 Protein - metabolism | Proteoglycans - metabolism | Smad6 Protein - physiology | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | Proteoglycans - physiology | Activin Receptors, Type II - genetics | Lung - embryology | Mice | Smad6 Protein - metabolism | Intracellular Signaling Peptides and Proteins - physiology | Smad3 Protein - physiology | Proteoglycans - genetics | Activin Receptors, Type II - metabolism | Smad3 Protein - metabolism | Receptors, Transforming Growth Factor beta - physiology | Endoglin | Smad3 Protein - genetics | Smad7 Protein - physiology | Gene Expression Regulation, Developmental | Lung - metabolism | Smad7 Protein - genetics | Smad2 Protein - physiology | Protein-Serine-Threonine Kinases - physiology | Mice, Inbred C57BL | Smad4 Protein - physiology | Protein-Serine-Threonine Kinases - genetics | Transforming Growth Factor beta - physiology | Activin Receptors, Type I - metabolism | Activin Receptors, Type I - genetics | Smad4 Protein - metabolism | Animals | Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - physiology | Activin Receptors, Type II - physiology | Transforming Growth Factor beta - metabolism | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2013, Volume 123, Issue 11, pp. 4714 - 4730
Journal Article
Circulation, ISSN 0009-7322, 02/2005, Volume 111, Issue 4, pp. 435 - 441
BACKGROUND: Pulmonary arterial hypertension (PAH) is a potentially fatal vasculopathy that can develop at any age. Adult-onset disease has previously been... 
Signal transduction | Receptors, growth factor | Activin receptors, type I | Pulmonary heart disease | Cell adhesion molecules | pulmonary heart disease | activin receptors, type I | cell adhesion molecules | CARDIAC & CARDIOVASCULAR SYSTEMS | HEREDITARY HEMORRHAGIC TELANGIECTASIA | MECHANISM | signal transduction | ENDOGLIN | OUTFLOW TRACT | BMPR-II | HAPLOINSUFFICIENCY | receptors, growth factor | GENE | DISEASE | GERMLINE MUTATIONS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | BRANCHPOINT SEQUENCE | Sequence Deletion | Receptors, Transforming Growth Factor beta - genetics | Humans | Child, Preschool | Infant | Male | Receptors, Cell Surface | Telangiectasia, Hereditary Hemorrhagic - genetics | Mutation, Missense | Receptors, Transforming Growth Factor beta - physiology | Endoglin | Heart Defects, Congenital - genetics | RNA Splicing | DNA Mutational Analysis | Germ-Line Mutation | Female | Vascular Cell Adhesion Molecule-1 - genetics | Activin Receptors, Type I - physiology | Child | Infant, Newborn | Telangiectasia, Hereditary Hemorrhagic - complications | Genetic Predisposition to Disease | Protein-Serine-Threonine Kinases - physiology | Protein-Serine-Threonine Kinases - genetics | Transforming Growth Factor beta - physiology | Exons - genetics | Genotype | Hypertension, Pulmonary - genetics | Vascular Cell Adhesion Molecule-1 - physiology | Codon, Nonsense | Activin Receptors, Type I - genetics | Bone Morphogenetic Protein Receptors, Type II | Activin Receptors, Type II | Signal Transduction - physiology | Amino Acid Motifs - genetics | Hypertension, Pulmonary - etiology | Amino Acid Substitution | Antigens, CD | Index Medicus | Abridged Index Medicus
Journal Article
Developmental Biology, ISSN 0012-1606, 2006, Volume 296, Issue 2, pp. 298 - 314
Transforming growth factor beta (TGF-β) proteins play important roles in morphogenesis of many craniofacial tissues; however, detailed biological mechanisms of... 
Cleft palate | Mandible | Palatal fusion | Cranial neural crest | Alk5 | Cleft face | Craniofacial malformation | craniofacial malformation | cleft palate | DEVELOPMENTAL BIOLOGY | ALK5 | CAUSES CLEFT-PALATE | cranial neural crest | cleft face | NEURAL CREST CELLS | TOOTH DEVELOPMENT | WNT1-CRE-MEDIATED DELETION | mandible | GENE-EXPRESSION | MYOSTATIN-DEFICIENT MICE | CRANIOFACIAL DEVELOPMENT | TRANSFORMING GROWTH FACTOR-BETA-3 | palatal fusion | MESENCHYMAL INTERACTIONS | 1ST BRANCHIAL ARCH | Receptors, Transforming Growth Factor beta - genetics | Face - abnormalities | Humans | Protein-Serine-Threonine Kinases | Receptors, Transforming Growth Factor beta - physiology | Ectoderm - physiology | Epithelium - embryology | Activin Receptors, Type I - physiology | Neural Crest - cytology | Rats | Epithelium - abnormalities | Mice, Transgenic | Signal Transduction - genetics | Activin Receptors, Type I - genetics | Palate - embryology | Protein Isoforms - physiology | Mice, Knockout | Neural Crest - physiology | Animals | Palate - abnormalities | Mesoderm - physiology | Signal Transduction - physiology | Face - embryology | Mice | Transforming Growth Factor beta - metabolism | Neural Crest - abnormalities | Protein Isoforms - genetics | Index Medicus | Medical and Health Sciences | Hematology | Medicin och hälsovetenskap | craniofacial | Klinisk medicin | Clinical Medicine | Hematologi | malformation
Journal Article
Biology of Reproduction, ISSN 0006-3363, 06/2006, Volume 74, Issue 6, pp. 1051 - 1059
The bovine dominant follicle (DF) model was used to identify molecular mechanisms potentially involved in initial growth of DF during the low FSH milieu of... 
Estradiol receptor | Follicle-stimulating hormone receptor | Follicular development | Gene regulation | Apoptosis | GONADOTROPIN RECEPTORS | BOVINE ESTROUS-CYCLE | YEAST LONGEVITY | LUTEAL-PHASE | estradiol receptor | follicle-stimulating hormone receptor | apoptosis | follicular development | REPRODUCTIVE BIOLOGY | MESSENGER-RIBONUCLEIC-ACID | gene regulation | OVARIAN-FOLLICLES | GENE-EXPRESSION | CD30 LIGAND | RAT GRANULOSA-CELLS | ANTRAL FOLLICLES | Activin Receptors, Type I - analysis | Luteinizing Hormone - analysis | Estradiol - genetics | Receptors, FSH - genetics | Theca Cells - cytology | Apoptosis Regulatory Proteins - analysis | Estrogen Receptor beta - analysis | RNA, Messenger - analysis | Granulosa Cells - chemistry | Follicle Stimulating Hormone - physiology | Ovarian Follicle - chemistry | Estrogen Receptor beta - genetics | Receptors, LH - genetics | Cattle | Receptors, FSH - physiology | Ovarian Follicle - physiology | Apoptosis Regulatory Proteins - genetics | Female | Activin Receptors, Type I - physiology | Follicle Stimulating Hormone - genetics | Receptors, LH - physiology | Estradiol - physiology | Receptors, FSH - analysis | Estradiol - analysis | Progesterone - analysis | Estrogen Receptor beta - physiology | Luteinizing Hormone - genetics | Cell Survival | Gene Expression Regulation - genetics | Ovarian Follicle - growth & development | RNA, Messenger - genetics | Gene Expression Regulation - physiology | Activin Receptors, Type I - genetics | Follicular Fluid - chemistry | Receptors, LH - analysis | Follicle Stimulating Hormone - analysis | Animals | Luteinizing Hormone - physiology | Apoptosis Regulatory Proteins - physiology | Progesterone - physiology | Signal Transduction - physiology | Progesterone - genetics | Granulosa Cells - cytology | Theca Cells - chemistry | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 02/2003, Volume 278, Issue 6, pp. 3751 - 3761
Transforming growth factor-beta (TGF-beta) elicits cellular effects by activating specific Smad proteins that control the transcription of target genes.... 
SIGNAL-TRANSDUCTION | TGF-BETA | PROTEIN | EPITHELIAL-CELLS | CANCER CELLS | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | N-TERMINAL KINASE | GENE-EXPRESSION | C-JUN | JNK PATHWAY | Fibronectins - biosynthesis | Receptors, Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - chemistry | Humans | Protein-Serine-Threonine Kinases | Molecular Sequence Data | Receptors, Transforming Growth Factor beta - physiology | Adaptor Proteins, Vesicular Transport | DNA-Binding Proteins - metabolism | Base Sequence | X-Linked Inhibitor of Apoptosis Protein | Activin Receptors, Type I - physiology | Genes, Tumor Suppressor | Mink | Amino Acid Sequence | Mutagenesis, Site-Directed | Activin Receptors, Type I - metabolism | DNA Primers | Activin Receptors, Type I - genetics | Cell Division - physiology | Plasminogen Activator Inhibitor 1 - biosynthesis | Adaptor Proteins, Signal Transducing | Animals | Proteins - metabolism | Receptors, Transforming Growth Factor beta - metabolism | Smad Proteins | Protein Binding | Trans-Activators - metabolism | Enzyme Activation | Activin Receptors, Type I - chemistry | Tumor Suppressor Proteins | Mitogen-Activated Protein Kinases - metabolism | Index Medicus | Proteins/metabolism | Receptors; Transforming Growth Factor beta/chemistry/genetics/metabolism/physiology | Trans-Activators/metabolism | Medical and Health Sciences | Medicin och hälsovetenskap | Adaptor Proteins; Vesicular Transport | DNA-Binding Proteins/metabolism | Fibronectins/biosynthesis | Research Support; Non-U.S. Gov't | Natural Sciences | Plasminogen Activator Inhibitor 1/biosynthesis | Cell Division/physiology | Mutagenesis; Site-Directed | Naturvetenskap | Mitogen-Activated Protein Kinases/metabolism | Activin Receptors; Type I/chemistry/genetics/metabolism/physiology
Journal Article