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Nephron, ISSN 1660-8151, 09/2018, Volume 140, Issue 2, pp. 105 - 110
Background: Acute kidney injury (AKI) is a frequent complication of both cardiac and major non-cardiac surgery. AKI is independently associated with morbidity,... 
Biomarkers | Perioperative | Acute kidney injury | NONCARDIAC SURGERY | CARDIAC-SURGERY | BEDSIDE | UROLOGY & NEPHROLOGY | RISK | RENAL FUNCTIONAL RESERVE | COHORT | FAILURE | INTRAOPERATIVE HYPOTENSION
Journal Article
Critical Care Medicine, ISSN 0090-3493, 07/2019, Volume 47, Issue 7, pp. 993 - 996
Critical care physicians continue to be challenged to recognize an environment that has the potential to result in acute kidney injury, with its associated... 
RECOVERY | acute kidney stress | BYPASS | renal ischemia | CARDIAC-SURGERY | TISSUE INHIBITOR | RISK | acute kidney injury | CELL-CYCLE ARREST | cell cycle arrest biomarkers | CRITICAL CARE MEDICINE
Journal Article
Kidney International, ISSN 0085-2538, 08/2012, Volume 82, Issue 4, pp. 412 - 427
Endothelial progenitor cells are known to reverse acute kidney injury by paracrine mechanisms. We previously found that microvesicles released from these... 
exosome | ischemia–reperfusion | acute kidney injury | ischemia-reperfusion | HORIZONTAL TRANSFER | STEM-CELLS | CONTRIBUTE | REGENERATION | FAILURE | EXPERIMENTAL GLOMERULONEPHRITIS | REPAIR | EPITHELIAL-CELLS | MESSENGER-RNA | UROLOGY & NEPHROLOGY | PLATELET-ACTIVATING-FACTOR | Chemotaxis, Leukocyte | Cell Proliferation | Epithelial Cells - metabolism | Kidney - blood supply | Capillaries - pathology | Kidney - pathology | Rats, Wistar | Endothelial Cells - transplantation | Cell-Derived Microparticles - transplantation | Male | MicroRNAs - metabolism | Acute Kidney Injury - genetics | Stem Cells - metabolism | Cell Hypoxia | Stem Cell Transplantation | Kidney - metabolism | Transfection | RNA Interference | Time Factors | Cell-Derived Microparticles - pathology | Cell-Derived Microparticles - metabolism | Kidney Tubules - pathology | Kidney Tubules - metabolism | Capillaries - metabolism | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | Disease Models, Animal | Ribonuclease III - genetics | Ribonuclease III - metabolism | Reperfusion Injury - pathology | Acute Kidney Injury - pathology | Endothelial Cells - metabolism | Cells, Cultured | Gene Expression Regulation | Rats | Epithelial Cells - pathology | Acute Kidney Injury - prevention & control | Regeneration | Oligonucleotides - metabolism | Animals | Reperfusion Injury - prevention & control | Fibrosis | Stem Cells - pathology | Acute Kidney Injury - metabolism | Endothelial Cells - pathology | Apoptosis | Index Medicus | Cell proliferation | Intravenous administration | Paracrine signalling | mRNA | Leukocytes | Ribonuclease | Endothelial cells | Kidney | Angiogenesis | Ischemia | Stem cells | Hypoxia | miRNA | Capillaries | Injuries
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2014, Volume 63, Issue 1, pp. 62 - 70
Objectives This study sought to evaluate the safety and efficacy of rosuvastatin in preventing contrast-induced acute kidney injury (CI-AKI) in patients with... 
Cardiovascular | Internal Medicine | kidney | diabetes mellitus | contrast medium | statins | angiography | C-REACTIVE PROTEIN | CARDIAC & CARDIOVASCULAR SYSTEMS | CARDIOTHORACIC SURGERY | INDUCED NEPHROPATHY | RISK | ATORVASTATIN | SERUM CREATININE | TRIAL | PERCUTANEOUS CORONARY INTERVENTION | PRETREATMENT | STATIN THERAPY | Hydroxymethylglutaryl-CoA Reductase Inhibitors - administration & dosage | Prospective Studies | Follow-Up Studies | Humans | Middle Aged | Renal Insufficiency, Chronic - diagnostic imaging | Male | Fluorobenzenes - administration & dosage | Renal Insufficiency, Chronic - complications | Diabetes Mellitus, Type 2 - diagnostic imaging | Dose-Response Relationship, Drug | Contrast Media - adverse effects | Time Factors | Angiography - methods | Female | Acute Kidney Injury - chemically induced | Diabetes Mellitus, Type 2 - complications | Pyrimidines - administration & dosage | Rosuvastatin Calcium | Treatment Outcome | Acute Kidney Injury - prevention & control | Angiography - adverse effects | Sulfonamides - therapeutic use | Pyrimidines - therapeutic use | Hydroxymethylglutaryl-CoA Reductase Inhibitors - therapeutic use | Fluorobenzenes - therapeutic use | Sulfonamides - administration & dosage | Prevention | Diabetics | Medical colleges | Care and treatment | Chronic kidney failure | Rosuvastatin | Diabetes | Diabetes therapy | Antilipemic agents | Heart failure | Oxidative stress | Heart attacks | Medical imaging | Statistical analysis | Hydration | Clinical trials | Kidney diseases | Drug therapy | Acute coronary syndromes | Cholesterol | Index Medicus | Abridged Index Medicus
Journal Article
Kidney International, ISSN 0085-2538, 12/2012, Volume 82, Issue 12, pp. 1271 - 1283
Autophagy is induced in renal tubular cells during acute kidney injury; however, whether this is protective or injurious remains controversial. We address this... 
acute kidney injury | autophagy | Atg7 | cisplatin | ischemia–reperfusion | Ischemia-reperfusion | APOPTOSIS | ACTIVATION | ACUTE-RENAL-FAILURE | PATHOLOGICAL ROLE | ischemia-reperfusion | CISPLATIN NEPHROTOXICITY | CELL-DEATH | PATHOPHYSIOLOGY | UROLOGY & NEPHROLOGY | MICE | BODY FORMATION | SELF-DIGESTION | Microtubule-Associated Proteins - genetics | Blood Urea Nitrogen | JNK Mitogen-Activated Protein Kinases - metabolism | Acute Kidney Injury - genetics | Autophagy - drug effects | Reperfusion Injury - blood | Chloroquine - pharmacology | Time Factors | Reperfusion Injury - chemically induced | Acute Kidney Injury - chemically induced | Microtubule-Associated Proteins - deficiency | Reperfusion Injury - genetics | Cytoprotection | Disease Models, Animal | Kidney Tubules, Proximal - pathology | Reperfusion Injury - pathology | Signal Transduction | Acute Kidney Injury - pathology | Acute Kidney Injury - blood | Mice, Inbred C57BL | Cells, Cultured | Tumor Suppressor Protein p53 - metabolism | Biomarkers - blood | Sirolimus - pharmacology | Acute Kidney Injury - prevention & control | Cisplatin | Mice, Knockout | Autophagy-Related Protein 7 | Animals | Reperfusion Injury - prevention & control | Kidney Tubules, Proximal - metabolism | Creatinine - blood | Mice | Enzyme Activation | Apoptosis | Kidney Tubules, Proximal - drug effects | Index Medicus
Journal Article
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