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American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 07/2014, Volume 307, Issue 2, pp. L158 - L172
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2009, Volume 106, Issue 38, pp. 16357 - 16362
Recent studies have suggested that bone marrow-derived multipotent mesenchymal stem cells (MSCs) may have therapeutic applications in multiple clinical... 
Pulmonary alveoli | Lungs | Epithelial cells | Medical treatment | Endotoxins | Small interfering RNA | Pulmonary edema | Average fixed cost | Mesenchymal stem cells | Lung injury | Acute respiratory distress syndrome | Keratinocyte growth factor | Alveolar fluid clearance | pulmonary edema | keratinocyte growth factor | MULTIDISCIPLINARY SCIENCES | ALPHA | SODIUM-CHANNEL | INTRATRACHEAL INSTILLATION | REPAIR | ENGRAFTMENT | alveolar fluid clearance | KERATINOCYTE | EPITHELIAL FLUID TRANSPORT | EDEMA | DECREASES EXPRESSION | acute respiratory distress syndrome | STROMAL CELLS | Fibroblast Growth Factor 7 - pharmacology | Lung Injury - surgery | Humans | Middle Aged | Culture Media, Conditioned - pharmacology | Male | Transplantation, Homologous | Mesenchymal Stromal Cells - cytology | Pulmonary Alveoli - metabolism | Pulmonary Alveoli - drug effects | Pulmonary Edema - prevention & control | Adult | Female | Pulmonary Edema - pathology | Leukocyte Count | Lung - metabolism | Pulmonary Edema - metabolism | Neutrophils - pathology | Acute Disease | Lung - pathology | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Sodium Channel Blockers - pharmacology | Recombinant Proteins - pharmacology | Escherichia coli - chemistry | Fibroblast Growth Factor 7 - genetics | Mesenchymal Stem Cell Transplantation - methods | Body Fluids - metabolism | Perfusion | Amiloride - pharmacology | In Vitro Techniques | Lung - surgery | Lung Injury - chemically induced | Care and treatment | Escherichia coli infections | Research | Lung diseases | Stem cells | Bone marrow | E coli | Ribonucleic acid--RNA | Index Medicus | Biological Sciences
Journal Article
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 07/2013, Volume 305, Issue 2, pp. C182 - C189
Inspiration of a high concentration of oxygen, a therapy for acute lung injury (ALI), could unexpectedly lead to reactive oxygen species (ROS) production and... 
Reactive oxygen species | Inflammation | Hyperoxia | Injury | Lung | ACTIVATION | PHYSIOLOGY | hyperoxia | INTERLEUKIN-1-BETA | INDUCTION | RESPIRATORY-DISTRESS-SYNDROME | CELL BIOLOGY | lung | EPITHELIAL-CELLS | inflammation | reactive oxygen species | ENDOTHELIAL-CELLS | GENE-EXPRESSION | MACROPHAGE INFLAMMATORY PROTEIN-2 | II PNEUMOCYTES | injury | TNF-ALPHA | Tumor Necrosis Factor-alpha - metabolism | Interleukin-6 - analysis | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Oxygen - therapeutic use | Tumor Necrosis Factor-alpha - genetics | Bronchoalveolar Lavage Fluid - immunology | Acute Lung Injury - genetics | Oxygen - adverse effects | Inflammasomes - adverse effects | Acute Lung Injury - prevention & control | DNA Fragmentation | Hyperoxia - complications | Hyperoxia - metabolism | Chemokine CXCL2 - genetics | Lung - metabolism | Chemokine CXCL2 - metabolism | Genetic Predisposition to Disease | Interleukin-1beta - analysis | Lung - pathology | Gene Expression Regulation - physiology | Mice, Knockout | Gene Expression Regulation - drug effects | Carrier Proteins - genetics | Oxygen - administration & dosage | Animals | Carrier Proteins - metabolism | Acute Lung Injury - chemically induced | Mice | Acute respiratory distress syndrome | Physiological aspects | Hypoxia | Health aspects | Proteins | Tissue | Oxygen | Lung diseases | Apoptosis | Index Medicus
Journal Article
Journal Article
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 01/2018, Volume 233, Issue 1, pp. 688 - 698
In conclusion, we have shown in present study that LPS challenge soared the autophagy level, inhibiting autophagy aggravated LPS‐induced lung microvascular... 
autophagy | lipopolysaccharide | acute lung injury | microvascular barrier | APOPTOSIS | ACTIVATION | PHYSIOLOGY | PERMEABILITY | MODEL | RESPIRATORY-DISTRESS-SYNDROME | PROTECTS | CELL BIOLOGY | LIVER | Capillary Permeability | Autophagy-Related Protein 7 - metabolism | Humans | Acute Lung Injury - genetics | Autophagy-Related Protein 5 - genetics | Autophagy | Lipopolysaccharides | Endothelial Cells - ultrastructure | Transfection | RNA Interference | Lung - ultrastructure | Blood-Air Barrier - ultrastructure | Pulmonary Edema - pathology | Acute Lung Injury - metabolism | Lung - metabolism | Zonula Occludens-1 Protein - metabolism | Pulmonary Edema - metabolism | Disease Models, Animal | Endothelial Cells - metabolism | Mice, Inbred C57BL | Cells, Cultured | Acute Lung Injury - pathology | Autophagy-Related Protein 7 - genetics | Pulmonary Edema - chemically induced | Stress Fibers - metabolism | Animals | Blood-Air Barrier - metabolism | Acute Lung Injury - chemically induced | Autophagy-Related Protein 5 - metabolism | Lung - blood supply | Acute respiratory distress syndrome | Permeability | Mitogens | Endothelium | Lung | Interleukin | Hemorrhage | Metastases | Bronchoalveolar lavage | Rodents | Interleukin 1 | Tumor necrosis factor-TNF | Inhibition | Alveoli | Injuries | Edema | Cytokines | Bronchus | Lactate dehydrogenase | Pharmacology | L-Lactate dehydrogenase | Zonula occludens-1 protein | Endothelial cells | Tumor necrosis factor | Infiltration | In vivo methods and tests | Microvasculature | Lactic acid | Viability | Phagocytosis | Integrity | Index Medicus
Journal Article