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Journal of Clinical Investigation, ISSN 0021-9738, 10/2009, Volume 119, Issue 10, pp. 2898 - 2913
Journal Article
Journal Article
Nature, ISSN 0028-0836, 03/2010, Volume 464, Issue 7285, pp. 104 - 107
Injury causes a systemic inflammatory response syndrome (SIRS) that is clinically much like sepsis. Microbial pathogen-associated molecular patterns (PAMPs)... 
SHOCK | SIRS | OPERATED CALCIUM INFLUX | HUMAN NEUTROPHILS | PEPTIDES | MODELS | MOBILIZATION | CHEMOTAXIS | MULTIDISCIPLINARY SCIENCES | RECEPTORS | TRAUMA | Liver - pathology | Phosphorylation | Humans | Male | Mitochondria - immunology | Liver - injuries | N-Formylmethionine Leucyl-Phenylalanine - metabolism | Liver - immunology | Systemic Inflammatory Response Syndrome - blood | Muscle, Skeletal - immunology | Sepsis - metabolism | Receptors, Formyl Peptide - metabolism | Systemic Inflammatory Response Syndrome - immunology | Neutrophils - metabolism | Calcium Signaling | Femur - injuries | Sepsis - immunology | CpG Islands - immunology | Neutrophils - enzymology | Wounds and Injuries - pathology | Fractures, Bone - immunology | Wounds and Injuries - blood | Cells, Cultured | Neutrophils - immunology | Wounds and Injuries - complications | Rats | Wounds and Injuries - immunology | Acute Lung Injury - pathology | DNA, Mitochondrial - blood | Rats, Sprague-Dawley | DNA, Mitochondrial - immunology | Immunity, Innate - immunology | Systemic Inflammatory Response Syndrome - pathology | Animals | Mitochondria - secretion | Acute Lung Injury - immunology | Fractures, Bone - pathology | Systemic Inflammatory Response Syndrome - complications | Muscle, Skeletal - pathology | Toll-Like Receptor 9 - metabolism | Sepsis - microbiology | Mitogen-Activated Protein Kinases - metabolism | N-Formylmethionine Leucyl-Phenylalanine - immunology | Infection | Peptides | Physiological aspects | Mitochondrial DNA | Inflammation | Research | Risk factors | Fractures | Bacterial proteins | Rheumatoid arthritis | Trauma | Deoxyribonucleic acid--DNA | Index Medicus
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2012, Volume 122, Issue 7, pp. 2661 - 2671
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2012, Volume 122, Issue 6, pp. 1973 - 1990
EC activation and dysfunction have been linked to a variety of vascular inflammatory disease states. The function of microRNAs (miRNAs) in vascular EC... 
CELL-ADHESION MOLECULE-1 | TRANSCRIPTION FACTORS | MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | DEPENDENT INDUCTION | LEUKOCYTE RECRUITMENT | HUMAN-ENDOTHELIAL-CELLS | ACUTE MYELOID-LEUKEMIA | PROGNOSTIC-SIGNIFICANCE | EXPRESSION PROFILES | SIGNAL-TRANSDUCTION PATHWAY | alpha Karyopherins - metabolism | Endotoxemia - therapy | Leukocytes - pathology | Vasculitis - metabolism | Acute Lung Injury - therapy | Endotoxemia - genetics | Humans | NF-kappa B - immunology | Critical Illness | Acute Lung Injury - genetics | MicroRNAs - metabolism | NF-kappa B - metabolism | Endotoxemia - pathology | Vasculitis - immunology | Leukocytes - immunology | Vasculitis - genetics | alpha Karyopherins - immunology | E-Selectin - genetics | Endotoxemia - immunology | Vascular Cell Adhesion Molecule-1 - genetics | Acute Lung Injury - metabolism | Vascular Cell Adhesion Molecule-1 - immunology | Vasculitis - therapy | Lipopolysaccharides - toxicity | Endothelial Cells - metabolism | E-Selectin - metabolism | E-Selectin - immunology | MicroRNAs - immunology | Acute Lung Injury - pathology | Endothelial Cells - immunology | Animals | NF-kappa B - genetics | alpha Karyopherins - genetics | Acute Lung Injury - immunology | Mice | MicroRNAs - genetics | Endothelial Cells - pathology | Endotoxemia - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Leukocytes - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
International Immunopharmacology, ISSN 1567-5769, 05/2014, Volume 20, Issue 1, pp. 24 - 32
Journal Article
Immunity, ISSN 1074-7613, 04/2015, Volume 42, Issue 4, pp. 640 - 653
Acute lung injury (ALI) remains a serious health issue with little improvement in our understanding of the pathophysiology and therapeutic approaches. We... 
BACTERIAL LIPOPOLYSACCHARIDE | STERILE INFLAMMATION | SIGNALING PATHWAY | P2X RECEPTORS | ALPHA PRODUCTION | ENDOTHELIAL-CELLS | MOUSE MODEL | IN-VIVO | ACUTE LUNG INJURY | IMMUNOLOGY | INFLAMMATORY RESPONSE | Interleukin-1alpha - immunology | Macrophages, Alveolar - pathology | Calcium - metabolism | Humans | Capillary Permeability - immunology | Neutrophil Infiltration | Necrosis - pathology | Receptors, Purinergic P2X7 - genetics | Necrosis - immunology | Cadherins - immunology | Adenosine Triphosphate - metabolism | HEK293 Cells | Interleukin-1alpha - genetics | Cadherins - genetics | Receptors, Purinergic P2X7 - immunology | Macrophages, Alveolar - immunology | Neutrophils - pathology | Lung - pathology | Protein Precursors - genetics | Signal Transduction | Neutrophils - drug effects | Gene Expression Regulation | Neutrophils - immunology | Lipopolysaccharide Receptors - immunology | Mice, Transgenic | Protein Precursors - immunology | Acute Lung Injury - pathology | Necrosis - chemically induced | Endothelial Cells - immunology | Animals | Acute Lung Injury - chemically induced | Macrophages, Alveolar - drug effects | Lung - drug effects | Lipopolysaccharides - pharmacology | Lipopolysaccharide Receptors - genetics | Acute Lung Injury - immunology | Mice | Endothelial Cells - pathology | Intubation, Intratracheal | Lung - immunology | Endothelial Cells - drug effects | Index Medicus | endothelial cells | alveolar macrophage | Ca2+ influx | MyD88 | IL-1α | neutrophil | P2X7R | lung injury | necrosis | ARDS | ALI | sepsis | lung inflammation
Journal Article
Journal Article