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American journal of physiology. Lung cellular and molecular physiology, ISSN 1522-1504, 11/2016, Volume 311, Issue 5, pp. L868 - L880
Journal Article
PloS one, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, pp. e56407 - e56407
Background: Nuclear factor-kappa B (NF-kappa B) is a central transcriptional factor and a pleiotropic regulator of many genes involved in acute lung injury... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Bronchoalveolar Lavage Fluid | Protein Binding - genetics | Cell Count | Pneumonia - pathology | Cell Survival - genetics | Acute Lung Injury - genetics | Male | NF-kappa B - metabolism | Vascular Endothelial Growth Factor A - metabolism | Protective Agents - therapeutic use | Vascular Endothelial Growth Factor A - genetics | Lipopolysaccharides | Alveolar Epithelial Cells - pathology | Protective Agents - pharmacology | Protein Binding - drug effects | Pulmonary Edema - complications | Pulmonary Edema - pathology | Vascular Cell Adhesion Molecule-1 - genetics | Acute Lung Injury - drug therapy | Acute Lung Injury - metabolism | Alveolar Epithelial Cells - ultrastructure | Pneumonia - genetics | Diterpenes - pharmacology | Pulmonary Edema - genetics | Cell Survival - drug effects | Diterpenes - therapeutic use | Cytokines - metabolism | DNA - metabolism | Down-Regulation - drug effects | Acute Lung Injury - pathology | Pulmonary Edema - drug therapy | Gene Expression Regulation - drug effects | Animals | Pneumonia - drug therapy | Transcription Factor RelA - metabolism | Pneumonia - complications | Mice | Mice, Inbred BALB C | Vascular Cell Adhesion Molecule-1 - metabolism | Peroxidase - metabolism | Vascular cell adhesion molecule 1 | Phosphorylation | Epithelial cells | Kinases | Macrophages | Inactivation | Cell adhesion & migration | Interleukin 6 | Ultrastructure | Rodents | Inhibition | Alveoli | Vascular endothelial growth factor | Drug dosages | Deoxyribonucleic acid--DNA | Medicinal plants | Pathogens | Edema | NF-κB protein | Deactivation | Therapeutic applications | Traditional Chinese medicine | Leukocytes (neutrophilic) | Inflammation | Tumor necrosis factor-α | Gene expression | Medicine | Hospitals | Lungs | Respiratory distress syndrome | Rheumatoid arthritis | Hypoxia | In vivo methods and tests | Laboratory animals | In vitro methods and tests | Binding sites | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
Kidney international, ISSN 0085-2538, 05/2012, Volume 81, Issue 10, pp. 942 - 948
Journal Article
Journal of cellular and molecular medicine, ISSN 1582-1838, 11/2017, Volume 21, Issue 11, pp. 2937 - 2949
...‐induced EMT and pulmonary fibrosis by inhibiting the Src pathway. Five days after administrating bleomycin to mimic acute lung injury (ALI... 
epithelial–mesenchymal transition | nintedanib | ventilator‐induced lung injury | Src | pulmonary fibrosis | ventilator-induced lung injury | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Cadherins - metabolism | Humans | Transforming Growth Factor beta1 - metabolism | Actins - metabolism | Acute Lung Injury - genetics | Epithelial-Mesenchymal Transition - drug effects | Pulmonary Fibrosis - genetics | Tidal Volume | Epithelial-Mesenchymal Transition - genetics | Ventilator-Induced Lung Injury - drug therapy | src-Family Kinases - deficiency | Actins - genetics | Collagen Type I - genetics | Respiration, Artificial - adverse effects | Indoles - pharmacology | Acute Lung Injury - drug therapy | Acute Lung Injury - metabolism | Lung - metabolism | Pulmonary Fibrosis - metabolism | Cadherins - genetics | Bleomycin - toxicity | Disease Models, Animal | Lung - pathology | Collagen Type I - metabolism | Ventilator-Induced Lung Injury - genetics | Drug Administration Schedule | Signal Transduction | Administration, Oral | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | src-Family Kinases - antagonists & inhibitors | Gene Expression Regulation | Transforming Growth Factor beta1 - genetics | Mice, Knockout | Animals | Acute Lung Injury - chemically induced | Lung - drug effects | Pulmonary Fibrosis - chemically induced | Mice | Pulmonary Fibrosis - drug therapy | src-Family Kinases - genetics | Ventilator-Induced Lung Injury - metabolism | Ventilator-Induced Lung Injury - etiology | Acute respiratory distress syndrome | Cell death | Collagen | Fibrosis | Stem cells | Permeability | Muscle proteins | Gene expression | Vascular endothelial growth factor | Growth factors | Mesenchyme | Staining | Smooth muscle | Kinases | E-cadherin | Signal transduction | Bleomycin | Actin | Rodents | Inhibition | Alveoli | Protein-tyrosine kinase | Tyrosine | Lung diseases | Malondialdehyde | Signaling | Pulmonary fibrosis | Respiratory distress syndrome | Ventilation | Microvasculature | Apoptosis | Index Medicus | Original
Journal Article
Journal of Thoracic and Cardiovascular Surgery, The, ISSN 0022-5223, 2016, Volume 151, Issue 3, pp. 861 - 869
Journal Article
The Journal of thoracic and cardiovascular surgery, ISSN 0022-5223, 03/2016, Volume 151, Issue 3, pp. 841 - 849
...–conditioned media 30 minutes before injury. To determine the degree of lung injury, we assessed for changes in lung vascular permeability, proinflammatory cytokines and cellular infiltrates in bronchoalveolar lavage, and histopathology... 
Cardiothoracic Surgery | lung ischemia–reperfusion injury | mesenchymal stromal (stem)–conditioned media | mesenchymal stromal (stem) cell therapy | acute lung graft rejection | mesenchymal stromal (stem)-conditioned media | lung ischemia-reperfusion injury | Cardiac & Cardiovascular Systems | Respiratory System | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Surgery | Science & Technology | Immunotherapy - methods | Lung Injury - pathology | Capillary Permeability | Rats, Long-Evans | Male | Mesenchymal Stromal Cells - immunology | Time Factors | Inflammation Mediators - metabolism | Lung Injury - metabolism | Lung Injury - prevention & control | Lung - metabolism | Reperfusion Injury - metabolism | Disease Models, Animal | Lung - pathology | Cytokines - metabolism | Paracrine Communication | Reperfusion Injury - pathology | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Macrophages - metabolism | Animals | Reperfusion Injury - prevention & control | T-Lymphocyte Subsets - metabolism | Reperfusion Injury - immunology | Bronchoalveolar Lavage Fluid - chemistry | Lung - blood supply | Lung Injury - immunology | Lung - immunology | Culture Media, Conditioned - metabolism | Medical colleges | Immunotherapy | Stem cells | Index Medicus | Abridged Index Medicus | Lung ischemia reperfusion injury | MSC conditioned media (MSC-CM) | mesenchymal stromal (stem) cell (MSC) therapy
Journal Article