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Journal of Surgical Research, ISSN 0022-4804, 2013, Volume 185, Issue 2, pp. 740 - 747
Journal Article
Scientific Reports, ISSN 2045-2322, 06/2014, Volume 4, Issue 1, pp. 5204 - 5204
Journal Article
The Journal of Pathology, ISSN 0022-3417, 06/2016, Volume 239, Issue 2, pp. 152 - 161
Leakage of fluid from blood vessels, leading to oedema, is a key feature of many diseases including hyperoxic acute lung injury ( HALI ), which can occur when... 
hyperoxic acute lung injury | VEGFR | pulmonary oedema | hyperoxia | VEGF | vascular leak | VEGFR-3 | VEGFR-2 | VEGF-D | LYMPHATIC ENDOTHELIUM | METASTASIS | MOUSE | TYROSINE KINASES | VASCULAR-PERMEABILITY | TUMOR ANGIOGENESIS | FACTOR RECEPTOR-3 | OXYGEN-TOXICITY | PATHOLOGY | CELL-DEATH | ONCOLOGY | ENDOTHELIAL GROWTH-FACTOR | Bronchoalveolar Lavage Fluid | Humans | Male | Pulmonary Edema - etiology | Oxygen - metabolism | Vascular Endothelial Growth Factor D - administration & dosage | Hyperoxia - complications | Hyperoxia - metabolism | Pulmonary Edema - complications | Female | Pulmonary Edema - pathology | Acute Lung Injury - metabolism | Lung - metabolism | Vascular Endothelial Growth Factor D - genetics | Pulmonary Edema - metabolism | Lung - pathology | Signal Transduction | Mice, Inbred C57BL | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Mice, SCID | Acute Lung Injury - pathology | Mice, Knockout | Hyperoxia - pathology | Xenograft Model Antitumor Assays | Vascular Endothelial Growth Factor D - metabolism | Animals | Cell Line, Tumor | Mice, Inbred NOD | Mice | Vascular Endothelial Growth Factor Receptor-3 - genetics | Acute Lung Injury - complications | Acute respiratory distress syndrome | Dropsy | Edema | Vascular endothelial growth factor | Index Medicus | VEGF‐D | VEGFR‐3 | VEGFR‐2 | Original Papers | Original Paper
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 10/2014, Volume 15, Issue 11, pp. 19355 - 19368
Acute lung injury (ALI) and its severe manifestation of acute respiratory distress syndrome (ARDS) are well-known illnesses. Uncontrolled and self-amplified... 
Emodin | E-selectin | MCP-1 | Nuclear factor-κB (NF-κB) | Acute respiratory distress syndrome (ARDS) | Acute lung injury (ALI) | Lipopolysaccharide (LPS) | lipopolysaccharide (LPS) | BIOCHEMISTRY & MOLECULAR BIOLOGY | CHEMISTRY, MULTIDISCIPLINARY | acute respiratory distress syndrome (ARDS) | emodin | acute lung injury (ALI) | MURINE MODEL | BERLIN DEFINITION | GENE | CHRONIC INFLAMMATORY DISEASES | INDUCED PULMONARY-FIBROSIS | PATHWAY | nuclear factor-kappa B (NF-kappa B) | EXPRESSION | ALI/ARDS | ARDS | Pneumonia - pathology | Acute Lung Injury - genetics | Male | NF-kappa B - metabolism | Lipopolysaccharides - adverse effects | E-Selectin - genetics | Inflammation Mediators - metabolism | Pulmonary Edema - pathology | Acute Lung Injury - drug therapy | Acute Lung Injury - metabolism | Bronchoalveolar Lavage Fluid - cytology | Chemokine CCL2 - metabolism | Pulmonary Edema - metabolism | Gene Expression | Emodin - pharmacology | Emodin - administration & dosage | Chemokine CCL2 - genetics | E-Selectin - metabolism | Enzyme Activation - drug effects | Acute Lung Injury - pathology | Pulmonary Edema - drug therapy | Protein Kinase Inhibitors - administration & dosage | Animals | Pneumonia - drug therapy | Acute Lung Injury - chemically induced | Signal Transduction - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Bronchoalveolar Lavage Fluid - chemistry | Pneumonia - metabolism | Peroxidase - metabolism | Index Medicus | nuclear factor-κB (NF-κB)
Journal Article
Chest, ISSN 0012-3692, 06/2009, Volume 135, Issue 6, pp. 1440 - 1447
Acute lung injury (ALI) and ARDS are common clinical syndromes that are underdiagnosed. Clara cell secretory protein (CC16) is an antiinflammatory protein... 
Clara cell protein | CC16 | biomarker pulmonary edema | acute lung injury | ARDS | Acute lung injury | Biomarker pulmonary edema | EARLY-DIAGNOSIS | UTEROGLOBIN | BIOMARKER | SECRETORY PROTEIN | RESPIRATORY-DISTRESS-SYNDROME | BRONCHOALVEOLAR LAVAGE | BRONCHIOLITIS OBLITERANS | SURFACTANT PROTEIN | BLOOD BARRIER | RESPIRATORY SYSTEM | SERUM-LEVELS | Critical Care - methods | Prognosis | Acute Lung Injury - therapy | Humans | Middle Aged | Critical Illness | Male | Reference Values | Respiratory Distress Syndrome, Adult - metabolism | Uteroglobin - metabolism | Case-Control Studies | Pulmonary Edema - therapy | Sensitivity and Specificity | Pulmonary Edema - diagnosis | Statistics, Nonparametric | Adult | Female | Acute Lung Injury - metabolism | Bronchoalveolar Lavage Fluid - cytology | Pulmonary Edema - metabolism | Respiratory Distress Syndrome, Adult - therapy | Biomarkers - metabolism | Intensive Care Units | Enzyme-Linked Immunosorbent Assay | Risk Assessment | Biomarkers - analysis | Probability | Respiration, Artificial | Respiratory Distress Syndrome, Adult - diagnosis | Acute Lung Injury - diagnosis | Aged | Respiratory Mucosa - metabolism | Proteins | Usage | Lung diseases | Physiological aspects | Diagnosis | Research | Biological markers | Pulmonary edema | Index Medicus | Abridged Index Medicus | Original Research
Journal Article
FASEB Journal, ISSN 0892-6638, 06/2013, Volume 27, Issue 6, pp. 2207 - 2219
Journal Article
Immunology, ISSN 0019-2805, 10/2014, Volume 143, Issue 2, pp. 241 - 257
Acute lung injury ( ALI ) from a variety of clinical disorders, characterized by diffuse inflammation, is a cause of acute respiratory failure that develops in... 
lipopolysaccharide | wogonin | peroxisome proliferator‐activated receptor | acute lung injury | nuclear factor | Lipopolysaccharide | Acute lung injury | Peroxisome proliferator-activated receptor γ | Wogonin | Nuclear factor-κB | DOWN-REGULATION | nuclear factor-kappa B | IMMUNOLOGY | SCUTELLARIA-RADIX | RESPIRATORY-DISTRESS-SYNDROME | AIRWAY DISEASE | GENE-EXPRESSION | PPAR-GAMMA | ALVEOLAR MACROPHAGES | BLOOD MONOCYTES | MOUSE MACROPHAGES | peroxisome proliferator-activated receptor gamma | SKIN INFLAMMATION | PPAR gamma - drug effects | Pneumonia - prevention & control | Acute Lung Injury - genetics | NF-kappa B - metabolism | PPAR gamma - metabolism | Lipopolysaccharides | Transfection | RNA Interference | Time Factors | Acute Lung Injury - prevention & control | Pneumonia - chemically induced | Inflammation Mediators - metabolism | Pneumonia - immunology | Female | Acute Lung Injury - metabolism | Lung - metabolism | Macrophages - immunology | Disease Models, Animal | PPAR gamma - genetics | Pneumonia - genetics | Cell Line | Cytokines - metabolism | Anti-Inflammatory Agents - pharmacology | Flavanones - pharmacology | Mice, Inbred C57BL | Gene Expression Regulation | Macrophages - metabolism | Animals | Acute Lung Injury - chemically induced | Signal Transduction - drug effects | Lung - drug effects | Acute Lung Injury - immunology | Macrophages - drug effects | Mice | Pneumonia - metabolism | Lung - immunology | Acute respiratory distress syndrome | Mitogens | Inflammation | Cytokines | Rodents | Immune system | Index Medicus | nuclear factor-κB | Original | peroxisome proliferator-activated receptor γ
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2017, Volume 12, Issue 6, pp. e0179822 - e0179822
Ischemia-reperfusion (IR)-induced acute lung injury (ALI) is implicated in several clinical conditions including lung transplantation, cardiopulmonary bypass... 
PATHWAYS | THERAPEUTIC APPLICATIONS | CYTOKINES | MULTIDISCIPLINARY SCIENCES | RATS | ACETAZOLAMIDE | ACTIVATORS | HYPERCAPNIA | TRANSPLANTATION | Capillary Permeability - physiology | Microvessels - metabolism | Reperfusion Injury - drug therapy | Capillary Permeability - drug effects | Microvessels - pathology | Male | Pulmonary Edema - etiology | Pulmonary Edema - pathology | Acute Lung Injury - drug therapy | Acute Lung Injury - metabolism | Hypertension, Pulmonary - drug therapy | Lung - metabolism | Reperfusion Injury - complications | Drug Evaluation, Preclinical | Neutrophils - metabolism | Pulmonary Edema - metabolism | Reperfusion Injury - metabolism | Disease Models, Animal | Neutrophils - pathology | Lung - pathology | Cytokines - metabolism | Reperfusion Injury - pathology | Tissue Culture Techniques | Neutrophils - drug effects | Microvessels - drug effects | Hypertension, Pulmonary - metabolism | Carbonic Anhydrase Inhibitors - pharmacology | Acute Lung Injury - pathology | Pulmonary Edema - drug therapy | Rats, Sprague-Dawley | Sodium-Potassium-Exchanging ATPase - metabolism | Animals | Lung - drug effects | Lung - blood supply | Acute Lung Injury - etiology | Hypertension, Pulmonary - etiology | Hypertension, Pulmonary - pathology | Complications and side effects | Ischemia | Lung diseases | Acetazolamide | Dosage and administration | Research | Drug therapy | Risk factors | Syngeneic grafts | Body weight | Lung | Carbon dioxide | Lung transplantation | Pleural effusion | Transplantation | Interleukin 6 | Reperfusion | Restoration | Surgery | Xenografts | Interleukin 1 | Attenuation | Physiology | Injuries | Hypertension | Edema | Pneumothorax | Cytokines | Internal medicine | Pulmonary arteries | Mortality | Leukocytes (neutrophilic) | Inflammation | Permeability | Na+/K+-exchanging ATPase | Interleukin 17 | Ostomy | Hospitals | Inhibitors | Carbonic anhydrase | Lungs | Tumor necrosis factor | Laboratory animals | Transport | Index Medicus
Journal Article