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Journal Article
Journal Article
PloS one, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, pp. e56407 - e56407
Background: Nuclear factor-kappa B (NF-kappa B) is a central transcriptional factor and a pleiotropic regulator of many genes involved in acute lung injury... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Bronchoalveolar Lavage Fluid | Protein Binding - genetics | Cell Count | Pneumonia - pathology | Cell Survival - genetics | Acute Lung Injury - genetics | Male | NF-kappa B - metabolism | Vascular Endothelial Growth Factor A - metabolism | Protective Agents - therapeutic use | Vascular Endothelial Growth Factor A - genetics | Lipopolysaccharides | Alveolar Epithelial Cells - pathology | Protective Agents - pharmacology | Protein Binding - drug effects | Pulmonary Edema - complications | Pulmonary Edema - pathology | Vascular Cell Adhesion Molecule-1 - genetics | Acute Lung Injury - drug therapy | Acute Lung Injury - metabolism | Alveolar Epithelial Cells - ultrastructure | Pneumonia - genetics | Diterpenes - pharmacology | Pulmonary Edema - genetics | Cell Survival - drug effects | Diterpenes - therapeutic use | Cytokines - metabolism | DNA - metabolism | Down-Regulation - drug effects | Acute Lung Injury - pathology | Pulmonary Edema - drug therapy | Gene Expression Regulation - drug effects | Animals | Pneumonia - drug therapy | Transcription Factor RelA - metabolism | Pneumonia - complications | Mice | Mice, Inbred BALB C | Vascular Cell Adhesion Molecule-1 - metabolism | Peroxidase - metabolism | Vascular cell adhesion molecule 1 | Phosphorylation | Epithelial cells | Kinases | Macrophages | Inactivation | Cell adhesion & migration | Interleukin 6 | Ultrastructure | Rodents | Inhibition | Alveoli | Vascular endothelial growth factor | Drug dosages | Deoxyribonucleic acid--DNA | Medicinal plants | Pathogens | Edema | NF-κB protein | Deactivation | Therapeutic applications | Traditional Chinese medicine | Leukocytes (neutrophilic) | Inflammation | Tumor necrosis factor-α | Gene expression | Medicine | Hospitals | Lungs | Respiratory distress syndrome | Rheumatoid arthritis | Hypoxia | In vivo methods and tests | Laboratory animals | In vitro methods and tests | Binding sites | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
American journal of physiology. Lung cellular and molecular physiology, ISSN 1522-1504, 11/2016, Volume 311, Issue 5, pp. L868 - L880
Acute lung injury (ALI) and acute respiratory distress syndrome (ARDS) are diseases with high mortality... 
Acute lung injury | Suppressor of cytokine signaling- 3 | STAT3 inhibitor | JAK/STAT pathway | Respiratory System | Physiology | Life Sciences & Biomedicine | Science & Technology | Inflammation - pathology | Leukocytes, Mononuclear - metabolism | Humans | Pneumonia - pathology | Lipopolysaccharides | Respiratory Distress Syndrome, Adult - blood | Acute Lung Injury - prevention & control | Gene Deletion | Inflammation Mediators - metabolism | Integrases - metabolism | Myeloid Cells - drug effects | Acute Lung Injury - metabolism | Bronchoalveolar Lavage Fluid - cytology | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Pneumonia - genetics | Cell Separation | Mice, Inbred C57BL | Sulfonamides - pharmacology | Acute Lung Injury - pathology | Gene Expression Regulation - drug effects | Macrophages - metabolism | Animals | Acute Lung Injury - chemically induced | Inflammation - genetics | Myeloid Cells - metabolism | Macrophages - drug effects | Anthraquinones - pharmacology | Chemokines - metabolism | Suppressor of Cytokine Signaling 3 Protein - metabolism | Acute respiratory distress syndrome | Prevention | Causes of | Health aspects | Plasma | Molecules | Phosphorylation | Respiratory distress syndrome | Mortality | Genes | Cells | Index Medicus | Call for Papers | STAT pathway | suppressor of cytokine signaling-3, STAT3 inhibitor | JAK | acute lung injury
Journal Article