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Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2009, Volume 119, Issue 10, pp. 2898 - 2913
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2012, Volume 122, Issue 7, pp. 2661 - 2671
Journal Article
Journal Article
Journal Article
Nature, ISSN 0028-0836, 03/2010, Volume 464, Issue 7285, pp. 104 - 107
Injury causes a systemic inflammatory response syndrome (SIRS) that is clinically much like sepsis. Microbial pathogen-associated molecular patterns (PAMPs)... 
SHOCK | SIRS | OPERATED CALCIUM INFLUX | HUMAN NEUTROPHILS | PEPTIDES | MODELS | MOBILIZATION | CHEMOTAXIS | MULTIDISCIPLINARY SCIENCES | RECEPTORS | TRAUMA | Liver - pathology | Phosphorylation | Humans | Male | Mitochondria - immunology | Liver - injuries | N-Formylmethionine Leucyl-Phenylalanine - metabolism | Liver - immunology | Systemic Inflammatory Response Syndrome - blood | Muscle, Skeletal - immunology | Sepsis - metabolism | Receptors, Formyl Peptide - metabolism | Systemic Inflammatory Response Syndrome - immunology | Neutrophils - metabolism | Calcium Signaling | Femur - injuries | Sepsis - immunology | CpG Islands - immunology | Neutrophils - enzymology | Wounds and Injuries - pathology | Fractures, Bone - immunology | Wounds and Injuries - blood | Cells, Cultured | Neutrophils - immunology | Wounds and Injuries - complications | Rats | Wounds and Injuries - immunology | Acute Lung Injury - pathology | DNA, Mitochondrial - blood | Rats, Sprague-Dawley | DNA, Mitochondrial - immunology | Immunity, Innate - immunology | Systemic Inflammatory Response Syndrome - pathology | Animals | Mitochondria - secretion | Acute Lung Injury - immunology | Fractures, Bone - pathology | Systemic Inflammatory Response Syndrome - complications | Muscle, Skeletal - pathology | Toll-Like Receptor 9 - metabolism | Sepsis - microbiology | Mitogen-Activated Protein Kinases - metabolism | N-Formylmethionine Leucyl-Phenylalanine - immunology | Infection | Peptides | Physiological aspects | Mitochondrial DNA | Inflammation | Research | Risk factors | Fractures | Bacterial proteins | Rheumatoid arthritis | Trauma | Deoxyribonucleic acid--DNA | Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 08/2012, Volume 18, Issue 8, pp. 1217 - 1223
Sepsis, a systemic inflammatory response to infection, commonly progresses to acute lung injury (ALI), an inflammatory lung disease with high morbidity. We... 
MEDICINE, RESEARCH & EXPERIMENTAL | VIVO | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | RESPIRATORY-DISTRESS-SYNDROME | CELL BIOLOGY | IN-VITRO | INFLAMMATION | HEPARAN-SULFATE | MICE | DEPENDENT MECHANISM | CELL | UNFRACTIONATED HEPARIN | Intestinal Perforation - complications | Humans | Heparitin Sulfate - antagonists & inhibitors | Male | Adoptive Transfer | Heparitin Sulfate - metabolism | Pulmonary Alveoli - enzymology | Glucuronidase - deficiency | Glucuronidase - analysis | Receptors, Tumor Necrosis Factor, Type I - deficiency | Glucuronidase - physiology | Respiratory Insufficiency - enzymology | Ventilator-Induced Lung Injury - enzymology | Acute Lung Injury - prevention & control | Endotoxemia - physiopathology | Intercellular Adhesion Molecule-1 - biosynthesis | Endothelium - enzymology | Disease Models, Animal | Acute Lung Injury - physiopathology | Lipopolysaccharides - toxicity | Pulmonary Alveoli - pathology | Mice, Inbred C57BL | Neutrophils - physiology | Ventilator-Induced Lung Injury - pathology | Lung - physiopathology | Mice, Knockout | Gene Expression Regulation - drug effects | Respiratory Insufficiency - pathology | Glycocalyx - physiology | Animals | Cell Adhesion - physiology | Endotoxemia - complications | Receptors, Tumor Necrosis Factor, Type I - physiology | Intercellular Adhesion Molecule-1 - genetics | Tumor Necrosis Factor-alpha - physiology | Mice | Enzyme Activation | Acute Lung Injury - etiology | Intestinal Perforation - microbiology | Endothelium - physiology | Care and treatment | Glycocalyx | Cellular control mechanisms | Neutrophils | Cell adhesion | Physiological aspects | Sepsis | Development and progression | Research | Properties | Respiratory physiology | Endothelium | Leucocytes | Glycoproteins | Lung diseases | Cell adhesion & migration | Immune system | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, pp. e56407 - e56407
Background: Nuclear factor-kappa B (NF-kappa B) is a central transcriptional factor and a pleiotropic regulator of many genes involved in acute lung injury.... 
INDUCED MITOGENIC FACTOR | IN-VITRO | ACTIVATION | INHIBITION | PI-3K/AKT-NF-KAPPA-B SIGNALING PATHWAY | MULTIDISCIPLINARY SCIENCES | ALVEOLAR FLUID CLEARANCE | EPITHELIAL SODIUM-CHANNEL | PANICULATA | EXPRESSION | RESPIRATORY-DISTRESS-SYNDROME | Bronchoalveolar Lavage Fluid | Protein Binding - genetics | Cell Count | Pneumonia - pathology | Cell Survival - genetics | Acute Lung Injury - genetics | Male | NF-kappa B - metabolism | Vascular Endothelial Growth Factor A - metabolism | Protective Agents - therapeutic use | Vascular Endothelial Growth Factor A - genetics | Lipopolysaccharides | Alveolar Epithelial Cells - pathology | Protective Agents - pharmacology | Protein Binding - drug effects | Pulmonary Edema - complications | Pulmonary Edema - pathology | Vascular Cell Adhesion Molecule-1 - genetics | Acute Lung Injury - drug therapy | Acute Lung Injury - metabolism | Alveolar Epithelial Cells - ultrastructure | Pneumonia - genetics | Diterpenes - pharmacology | Pulmonary Edema - genetics | Cell Survival - drug effects | Diterpenes - therapeutic use | Cytokines - metabolism | DNA - metabolism | Down-Regulation - drug effects | Acute Lung Injury - pathology | Pulmonary Edema - drug therapy | Gene Expression Regulation - drug effects | Animals | Pneumonia - drug therapy | Transcription Factor RelA - metabolism | Pneumonia - complications | Mice | Mice, Inbred BALB C | Vascular Cell Adhesion Molecule-1 - metabolism | Peroxidase - metabolism | Vascular cell adhesion molecule 1 | Phosphorylation | Epithelial cells | Lung | Kinases | Macrophages | Inactivation | Cell adhesion & migration | Interleukin 6 | Ultrastructure | Rodents | Inhibition | Alveoli | Vascular endothelial growth factor | Drug dosages | Deoxyribonucleic acid--DNA | Medicinal plants | Pathogens | Edema | NF-κB protein | Deactivation | Therapeutic applications | Traditional Chinese medicine | Leukocytes (neutrophilic) | Inflammation | Tumor necrosis factor-α | Gene expression | Medicine | Hospitals | Lungs | Respiratory distress syndrome | Rheumatoid arthritis | Hypoxia | In vivo methods and tests | Laboratory animals | In vitro methods and tests | Binding sites | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article