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PloS one, ISSN 1932-6203, 2018, Volume 13, Issue 10, p. e0206389
Journal Article
Gut, ISSN 0017-5749, 03/2012, Volume 61, Issue 3, pp. 416 - 426
...) and murine models of steatohepatitis and fibrosis. It was recently shown that monocyte/macrophage infiltration into the liver upon injury is critically regulated... 
FIBROSIS | STEATOSIS | INSULIN-RESISTANCE | INFLAMMATION | BONE-MARROW | DISEASE | MONOCYTE SUBSETS | MICE | SPIEGELMER | GASTROENTEROLOGY & HEPATOLOGY | CCR2 | Liver - pathology | Carbon Tetrachloride | Liver Cirrhosis, Experimental - etiology | Bone Marrow Cells - physiology | Male | Chemical and Drug Induced Liver Injury, Chronic - drug therapy | Liver Cirrhosis, Experimental - prevention & control | Non-alcoholic Fatty Liver Disease | Chemokine CCL2 - physiology | Bone Marrow Cells - drug effects | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury, Chronic - complications | Macrophages - physiology | Aptamers, Nucleotide - pharmacology | Drug Evaluation, Preclinical - methods | Acute Disease | Cytokines - metabolism | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Fatty Liver - prevention & control | Aptamers, Nucleotide - therapeutic use | Chemotaxis - drug effects | Chemical and Drug Induced Liver Injury, Chronic - pathology | Disease Progression | Fatty Liver - drug therapy | Animals | Chemical and Drug Induced Liver Injury - complications | Chemical and Drug Induced Liver Injury - drug therapy | Macrophages - drug effects | Mice | Chemokine CCL2 - antagonists & inhibitors | Fatty Liver - etiology | Care and treatment | Liver diseases | Fibrosis | Models | Research | Macrophages | Chemokine receptors | Studies | Cytokines | Polyethylene glycol | Rodents | Liver | Clinical trials | Tumor necrosis factor-TNF | Inflammation | Diabetes | Metabolic disorders | Chemokines | Index Medicus | Abridged Index Medicus
Journal Article
British Journal of Clinical Pharmacology, ISSN 0306-5251, 07/2016, Volume 82, Issue 1, pp. 238 - 248
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2015, Volume 149, Issue 7, pp. 1896 - 1909.e14
Journal Article
The Journal of pathology, ISSN 1096-9896, 2018, Volume 247, Issue 1, pp. 110 - 122
Ibuprofen is a worldwide used non‐steroidal anti‐inflammatory drug which may cause acute liver injury (ALI... 
toxicity | POPULATION | JNK | MEDIATOR | ibuprofen | liver failure | NECROSIS | HEPATITIS | RESPIRATION | drug-induced liver injury (DILI) | HEPATOTOXICITY | OUTCOMES | VANISHING BILE-DUCT | STEVENS-JOHNSON-SYNDROME | drug‐induced liver injury (DILI) | PATHOLOGY | ONCOLOGY | Liver - pathology | Phosphorylation | Liver - enzymology | Humans | Liver Failure, Acute - enzymology | Hepatocytes - pathology | Male | Mitogen-Activated Protein Kinase 9 - genetics | Liver Failure, Acute - genetics | Cell Death | Ibuprofen | Mitogen-Activated Protein Kinase 8 - genetics | Liver Failure, Acute - prevention & control | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury - enzymology | Disease Models, Animal | Chemical and Drug Induced Liver Injury - prevention & control | Signal Transduction | Mitogen-Activated Protein Kinase 9 - metabolism | Mice, Inbred C57BL | Mitogen-Activated Protein Kinase 8 - metabolism | Chemical and Drug Induced Liver Injury - genetics | Mitogen-Activated Protein Kinase 9 - deficiency | Mice, Knockout | Animals | Liver Failure, Acute - pathology | Enzyme Activation | Hepatocytes - enzymology | Enzymes | Liver diseases | Analysis | Liver | Transplantation of organs, tissues, etc | Histochemistry | Animal models | Protein kinase C | Syngeneic grafts | Toxicity | Cytotoxicity | AKT protein | Activation | Transplantation | Histopathology | Allografts | Pathways | Clonal deletion | Rodents | Animal tissues | Biocompatibility | Nonsteroidal anti-inflammatory drugs | Injuries | MAP kinase | Histology | Inflammation | siRNA | Investigations | Hepatocytes | Transaminases | Mice | Intoxication | Cytoplasm | Liver transplantation | Biochemical markers | Index Medicus
Journal Article
世界胃肠病学杂志:英文版(电子版), ISSN 1007-9327, 2015, Volume 21, Issue 18, pp. 5473 - 5481
AIM: To assess the effects of dihydromyricetin(DHM) as a hepatoprotective candidate in reducing hepatic injury and accelerating hepatocyte proliferation after carbon tetrachloride(CCl4) treatment.METHODS... 
Dihydromyricetin | Tumor | factor-α | regeneration | necrosis | Liver | Liver regeneration | Tumor necrosis factor-α | PATHWAYS | APOPTOSIS | ACTIVATION